Thorax, Abdomen, and Pelvis Flashcards
thoracotomy
surgical creation of an opening through thoracic wall to enter a pleural cavity, periosteum maintained for regeneration. H shape cuts through perichondrium of one or more costal cartilages shelling out segments of costal cartilage gaining entrance. incision such that the periosteum of the rib is maintained for regeneration.æ
sternal biopsy
used for bone marrow needle biopsy due to breadth and subcutaneous position. needle pierces thin cortical bone and enters vascular trabecular (spongy) bone. used to obtain specimens of bone marrow for transplantation and detection of metastatic cancer.æ
sternotomy
gaining access of thoracic cavity for surgical procedures on heart and great vessels sternum is divided in median plain and retracted and reunited and held together with wire sutures shut to maintain shape afterwards. Posterolateral aspects of 5th-7th intercostal spaces ae important sites for posterior thoractomy incisions. Have pt lie contralaterally, abducting limb forearm beside head accessing 4th intercostal. use h shaped incision to incisde superficial aspect of periosteum ensheathing rib, stripping periosteum from rib removing wide segment of rib to gain better acess if need to remove lung. median sternotomy as major exposure for open-heart surgery.
thoracic outlet syndrome
Superior thoracic aperture, outlet, emphasizing important nerves and arteries pass thorugh aperture into lower neck and upper limb. w/ various type of thoracic outlet syndrome, costoclavicular syndrome- pallor and coldness of skin of upper limb and diminished radial pulse resulting from compression of subclavian artery between clavicle and 1st rib. compression of brachial plexus and/or subclavian vessels due to abnormal cervical rib, altered first rib or scalenes attachment, etc. present with loss of feeling in their arm causing entrapment point because of first rib. Scalene, interscalene triangle, brachial plexus in nerve artery vein. Brachial plexus, subclavian artery and vein can be caught by clavicle, abnormal first rib or abnormal 7 can develop thoracic outlet syndrome. Has many squeeze points leading to many pressure blocking points.
rib fracture
not usually (1, 2, 11, or 12 because protected) caused by crush injuries, penetrating chest wounds. intense pain because of expansion and contraction of rib cage during respiration requires palliation by anesthesizing intercostal nerve block. will typically fracture either at the point of impact or around the angle of the rib. ribs are angled and when breaks and fracture point pushes internally.
rib dislocation
ex. slipping rib syndrome, dislocation of sternocostal joint are displacement of costal cartilage from sternum causing severe pain during deep breathing. produces lump-like deformity at dislocation site common in body contact sports, complications are pressure on or damage to nearby nerves, vessels, and muscles. rib seperation is dislocation fo costochondral junction between rib and costal cartilage 3-10th rib seperation can tear perichondrium and periosteum moving superiorly overriding above. involvement of costal cartilage from sternum, with rib separation involving the rib and the costal cartilage; both will be painful. If not being held in place commonly snaps
diaphragm paralysis
able to see by paradoxical movmeent. paralysis and eventual atrophy of half diaphragm because of injury to its motor supply from phrenic nerve from ipsalateral, unless have accessory phrenic nerve does not affect other half because each half has seperate nerve supple. supposed to descend on inspiration but is pushed superiorly by abdominal viscera and compressed by active side. paralyzed dome descends during expiration pushed down by positive pressure in lungs. can be short term by injecting anesthetic agent around nerve where it is on anterior surface of anterior scalene muscle. paradoxical motion: Chest wall expands, diaphragm pulls down central tendon contracts so when inspiring breath in abdomen goes out and diaphragm domes down opposite on expiration. Breathing in lowering pressure wall is brought in and as exhale .Diaphragm central tendon muscle on end phrenic nerve for each diaphragm- both hemidiaphragms go up if paralyze one wont contract and flatten down and opposite one does stomach contents come up into area when chest wall flails out.
breast changes during pregnancy
include branching of lactiferous ducts, in breast tissues during menstrual cycle and prengnacy. Mammary glands prepared for secretion by midpregnancy, do not produce milk until shortly before baby is born. colostrum, creamy white to yellowish premilk fluid, secrete from nipples during last trimester of prengnacy and during initial episodes of nursing. colostrum is rich in protein, immune agents, and growth factor for infant’s intestines. multiparous women (given birth more than two time) breast is large and pendulous. postmenopausal- small from lack of fat and atrophy of glandular tissue.
fibrocystic breast change
covering large group of benign condition in 80% of women, related to cyclic changes in maturation and involution of glandular tissue. fibroadenoma=– most common breast mass peak between 20-25 years benign neoplasms of glandular epithelium accompanied by significant increase in periductal connective tissue present firm, painless, mobile, solitary palpabel masses may grow rapidly during adolescence but should be checked later
breast cancer
carcinoma of breast are malignant tumors common in postmenopausal women, adenocarcinomas arising from epithelial cells of lactiferous ducts in mammary gland lobules-most common. if enter lymph vessel pass through 2-3 groups of lymph nodes before entering venous system spreading via lymphatics. can interfere w/ drainage caused by lymphedema may deviate nipple causing thickened, leather-like appearance of skin. prominent or puffy skin between dimples pores give orange-ppeel appearance (peau d’orange) A peau dÍorange appearance from skin edema following lymphatic involvement, vs. retraction of skin from involvement of suspensory (Cooper’s) ligaments, may be signs of advanced breast cancer. larger dimples (fingertip +) from cancerous invasion of glandular tissue and fibrosis (fibrous degeneration), causing shorteneing or places traction on suspensory ligaments. subareolar cancer cause inversion of nipple by pulling on or shortning suspensory ligaments. carries cancer to axillary to cervical and parasternal (infraclavicular and supraclavicular) leaving to supraclavicular lymph nodes, opposite breast or abdomen. posterior intercostal veins drain into azygos/hemiazygos system along body of vertebrae and internal venous plexus can spread to brain. can continguity to pectoral fascia, pectoralis major, interpectoral nodes causing breast elevation when muscle contracts and is advanced sign. ex. in men metastatisized to bone, pleura, lung, lver, and skin subareolar mass or secretion from nipple infiltrating pectoral fascia, major, and apical lymph nodes. superolateral quadrant most involved in breast cancer. Carcinoma of the breast typically arises as an adenocarcinoma of the lacteriferous duct epithelium. Auxiliary process tail of Spence Breast cancer- ductal (3-4 things adipose, suspensions ligaments (big retinaculicutus elaborated ones holding adipose and glandular tissue together, ducts, mammary lobules). Skin dimpling- cancer is grabbing onto of suspensions ligaments, nipple retraction 20 separate ducts feeding into it suggest cancer there. Large mass, contours, peak dÍorgange (small tenting) infiltration of local lymphatic sweating out and having them exaggerated.
mammography
used to detect breast masses. appearing as a large jagged density, thickened over tumor use as guide when removing breast tumors, cysts, and abscesses.æ
breast surgery
incisions placed in inferior breast quadrants when possible because less vascular than superior ones. transition between thoracic walla nd breast most abrupt inferiorly producing line/crease/deep skin fold, inferior cutaneous crease. incisions along crease are least evident hidden by overlap of breast. incisions made near areola directed radially to either side of nipple.æ
modified masectomy
whole beast is removed w/ lymph nodes, axillary fat, investing fascia over chest wall mucsles. preserve pectoralis, serratus anterior, and latissimus dorsi and long thoracic and thoracodorsal nerves. Given concerns of metastasis from breast cancer, the lymphatics of breast need to be considered. Central axillary nodes are the most frequently palpable midway between the anterior and posterior axillary folds. Overall, paralleling the venous drainage, the breast mostly drains to axillary nodes, but with the medial part of the breast draining to parasternal (internal thoracic) lymph nodes and the lower quadrants into abdominal nodes.
Radical mastectomy
masectomy (breast excision)- breast is removed down to retromamary space (simple). more extensive involves removal of breast, pectoral muscles, fat, fascia, and as many lymph nodes as possible in axilla and pectoral region.
lumpectomy or quadrantectomy-æ
breast conserving surgery tumor and surrounding tissues removes followed by radiation.
intercostal nerve block
uses local anestheis of an intercostal space by injecting local anestethtic agent around intercostal nerves and collateral branches. particualr area of skin receives innervation from two adjacent nerves, overlapping of contiguous dermatomes occurs. complete lsos of sensation does not occur unless two or more intercostal nerves in adjacent intercostal spaces are anesthetized. image reminds us that intercostal nerves are the ñvulnerableî part of the intercostal neurovascular bundle, and so could be infiltrated just deep to the rib. Given that dermatomal patterns are likely overlapping, one should block sequential nerves in order to assure loss of sensation for a particular region.
pulmonary collapse
aka Atalectasis: sufficient amount of air enters pleural cavity, surface tension adhering visceral to parietal pleura (lung to wall) is broken causing lung to collapse because of inherent elastciity (elastic recoil) when collapses (atelectasis) pleural cavity normall potential space becomes full of air. one can collapse w/out the other. can be caused following surgery ex. air in but not out. Infection or mass blocking out lungs causing it to shrink (atelctasis spirometry to expand lung and not let patients get pneumonia prone.
pneumothorax
entry of air into pleural cavity resulting from pentrating wound of parietal pleura or rupture of lung from trauma ex. bullet, knife, broken rib tearing pareital pleura, leakage from lung through opening in visceral pleura resulting in partial collapse of lung. hydrothorax- accumulation of significant amount of fluid in pleural cavity from pleural effusion Hearts failing fluid building up hydrostatically pushing into recess, infection or inflammatory infusions (transfudate and exudate) cancer making capillaries leaky. ex. Tension pneumothorax can be deadly, as a one-way valve effect of a wound can lead to air trapping, deviation of the trachea, shift of the mediastinum, and compression of the functioning lung. hyperresonant over the air-filled side, with some decreased resonance on the compressed lung side. Air in pleural cavity, chest go out lung go in, pleural cavity pressure low if make it atmospheric from inside (spontaneous pneuomothorax from a bulla), whole from outside (have space, open gagging wound lung collapsed if flap will let air in but not out building up pressure fast trachea deviating because of not hard structures leading to increased distress. Lungs like to expand, but also have recoil. Rings like to go out. Barrel chest, lungs are more stretchy than normal so as trapping air and not able to collapse as much chest wall is going out because of air trapped and lung not as recoiled.
hemothorax
caused by chest wound where blood enters pleural cavity from injury to major intercostal vessel blood does not clot well because of smooth pleural surface and defibrinating action of respiratory movements. dullness to percussion over the fluid
chylothorax
lymph from a torn (or lacerated) thoracic duct entering pleural cavity. chyle is pale white or yellowish lymph fluid in thoracic duct contianing fat absorbed by intestines. major lymphatic passage behind lung and esophagus thoracic duct might get leakage of lymph into the costal diaphragmatic recess. This may stem pleural leakage, e.g., from excessive hydrostatic pressure, as with cardiac disease, or with inflammation and leakage, as from infection or cancer. empyema- typically from bacterial pneumonia, e.g., from exudates of neutrophils forming pus (via leaking tissues from the inflammation).
thoracentesis (3-6)
with excessive pleural air or fluid contents, chest tube placement meeded and is when a hypodermic needle is inserted through intercostal space into pleural cavity to obtain sample of pleural fluid or remove blood or pus. avoiding damage to intercostal nerve and cessel inserted superior to rib, high enough to avoid colalteral branches. For pneumothorax: a small tube at the 2nd or 3rd intercostal space, more midclavicular line, given that the lung will have collapsed a bit with the air, with subsequent loss of negative intrapleural pressure. This position should also minimize targeting of the subclavian vessels and brachial plexus components, but will require going through pectoralis major. Many clinicians will put the chest tube in the 4th/5th intercostal space for pneumothorax, in addition to pleural effusion. The 5th intercostal space is low enough to capture contents, but high enough to minimize diaphragm damage. Midaxillary around 5th interspace donÍt put near sternum because of heart, donÍt want to go lower diaphragm goes up not hit diaphragm to draw out fluid and reexpand fluid. Air goes up fluid goes down.
auscultation and variations
ausculatation of lungs (assessing air flow thorugh tracheobronchial tree into lung w/ stethoscope). percussion of lungs (tapping chest over lungs with finger) including root of neck to detect sounds in apices of lungs helps establish whether underlying tissues are air-fileld (resonant), fluid-filled (dull), or solid (flat). base of lung- inferior part of posterior costal surface of inferior lobe listening at inferoposteriro aspect of thoracic wall at level of T10. note the need to have multiple listening points so as to include different lobes of the lungs and the pleural recesses inferiorly. ex. Hollow tone- normal lung, hyperressonant- in pneumothorax . Fusion, consolidate- flatter tone
bronchi and foreign body aspiration
right bronchus is wider and shorter running more vertically than left bronchus, aspirated foreign bodies are more liekly to enter and lodge in it or a branch. Foreign body aspiration: changes in auscultation could also occur with aspiration. The right main stem bronchus is somewhat larger and more vertical than the left main stem bronchus, so that aspirated materials will tend to end up in aspects of the right lung
pleural sensitivity and innervation
Pleurisy: inflammation of the pleural surfaces can generate a pleural rub. Visceral pleura insensitive to pain because innervated by autonomic (motor and visceral afferent) reaching visceral w/ bronchial vessel receiving no nerves of sensation. parietal is sensitve to pain (costal pleura) richly supplied by branches of somatic intercostal and phrenic nerves. irritation of parietal pleura produces local pain and reffered pain to areas sharing innervation by same segments of spinal cord.
pleural injury
irriation of costal and peripheral parts of diaphragmatic pleura results in local pain and reffered pain along intercsotal nerves to thoracic and abdominal walls. irritation of mediastinal and central diaphragmatic areas of parietal pleura result in pain refferred to root of neck and over shouldr (C3-5)
pleuritis
normal: moist, smooth pleurae make no sound detectable by ausculation (listening to breath sounds). pleuritis- inflammation of pleurae makes lung surfaces rough w/ resutling friction may be heart w/ stethoscope creating sharp, stabbing pain, on exertion like climbing stairs when rate and depth of respirations increased slightly.æ
heart failure
Cor pulmonale- trouble getting blood through the lungs. Right side of heart much lower pressure than right. If pressure goes up or having trouble blood getting in right side is going to go into failure continuing working have right sided failure secondary to a lung disease. Right heart failure- backs up into legs, secondary to lung disease . Left heart failure- backup into lung harder to oxygenate, higher capillary pressure . Clubbing- sift tissue development associated w/ lung disease . Obstructive conditions such as COPD and asthma, where it is difficult to exhale,
pulmonary fibrosis
where the lung will have difficulty expanding with inhalation. idiopathic pulmonary fibrosis (IPF) chronic restrictive lung disease. chronic restrictive lung diseases include reduced compliance that cause chronic inflammation, fibrosis, need for more pressure to inflate stiffened lungs. IPF specific form of firbosing interstitial pneumonia mainly affects people over age of 50 cigarette smoking major risk factor. For example, the increased recoil of the fibrotic lung will counteract the recoil of the chest wall, and lead to a new, shrunken equilibrium point between the inward recoil of the lung and the outward recoil of the chest wall. Digital clubbing is a distinctive sign, but its formation is still controversial, presumably based on growth factors triggered by hypoxia. trouble getting lung in ex. Pulmonary fibrosis, hard lung use of accessory muscles and intercostal spaces. Work of respiration lose weight working, diaphragm high and chest is sinking in because lungs not strong. Lung is usually dense or honeyconed
pulmonary embolism
The right and left pulmonary arteries dividing into lobar and segmental branches and increasingly small vessels from there are at risk of pulmonary thromboembolism, especially stemming from deep venous thrombosis of the lower extremities. Increased risk of thromboembolism via the Virchow triad of stasis, endothelial injury, and hypercoagulability. Features contributing to the triad are often asked for in the history with a patient with a suspected pulmonary embolism. Embolism- deep femoral veins straight shot up inferior vena cava, right atrium into pulmonary trunk narrowing vessels are pulmonary arteries travels out to edge infarct not he margin or large saddle blocking pulmonary trunk along the way. Virchow- father of pathology. VirchowÍs triad risk factors for clotting stasis (bedridden), hypercoagulable (medications with potentially activating coagulation cascade or platelet adhesion), blood lining (endothelial damage activation) . obstruction of pulmonary artery by blood clot (embolus) common cause of morbidity (sickness) and mortality (death). embolus in pulmonary artery forms when blood clot, fat globule or air bubble travels in blood to lungs from leg vein. embolus passes through right side of heart to lung through pulmonary artey. embolus may block pulmonary artery or branch. immediate result is partial or complete obstruction of blood flow to lungs. obstruction results in sector of lung ventilated but not perfused w/ blood. large embolus occludes a pulmonary artery, person suffers acute respiratory distress because of major decrease in oxygenation of blood owing to blockage of blood flow through lung. medium-sized embolus may block artery supplying bronchopulmonary segment, producing pulmonary infarct, area of dead lung tissue. Lung naturally filters venous clots larger than circulating blood cells accommodating small clots by fibrinolytic (clot buster) mechanisms. VirchowÍs triad: venous stasis (bed rest), trauma (fracture, tissue injury), coagulation disorders (inherited or acquired). Caused y postoperative and postpartum immobility and some hormone medications increasing risk of blood clots most are silent and small. Larger obstruct medium sizes lead to infarct or obstruction of large vessel. W/out infarct present as tachypnea, anxiety, dyspnea, syncope, and vagus substernal pressure. Saddle embolus- completely occluding right pulmonary and partially obstructing trunk and left aa.
bronchoscopy
endoscope for inspecting interiro of tracheobronchial treaa for diagnsotic purposes can observe ridge, carina- between orifices of main bronchi. cartilaginous projection of last tracheal ring. if tracheobronchial lymph nodes in angle between main bronchi are enlarged because cancer cells have metastasized from bronchogenic carcinoma ex. carina distorted widened posterioly and immobile. Leading cause of cancer-related deaths most caused by cigarette smoking from alveolar lining cells of lungparenchyma or epithelium o trachebronchial tree. types: squamous (bronchiogenic)-impinge on adjacent structures involving sympathetic trunk and brachial plexus- pancoast syndrome compromises it to head leadin to horner syndrome, adenocarcinoma (intrapulmonary bronchi),
lung cancer
Lung cancer as arising from the epithelium of the airways (bronchogenic carcinoma) may involve diagnosis with bronchoscopy. Note the carina as a distinctive ridge at the tracheal bifurcation into bronchi. Distortion of the carina can occur with expanded nodes, cancerous infiltration, etc. bronchogenic carcinoma- common type of lung cancer aririsng from epithelium of bronchial tree, usually metastaizes widley because of arrangemnt of lymphatics tumer cells enter systemic circualtion by invading wall of sinusoid or veunle in lung and transported thorugh pulmonary veins, left heart, and aorta to rest of body (cranium and brain) lung cancer- caused by cigarette smoking.
pericarditis
inflammation of pericardium source for pericardial effusion enerating cardiac tamponade. usually causes chest pain makes surfaces rough and resulting friction rub sound like rustle of silk when listening. normal- layers of serous pericardium make no detectable sound during auscultation.æ
pericardial effusion
inflammatory diseases produce pericardial effusion (passage of fluid from pericardial capillaries into pericardial cavity) heart becomes compressed
cardiac tamponade
with the Beck triad of hypotension, muffled heart sounds, and jugular venous distension apparent with acute tamponade. when heart becomes compressed, unable to expand and fill fully and ineffectual. potentially lethal as fibrous pericardium touch and inelastic. heart volume compromised by fluid outside heart inside pericardial cavity.æcardiomegaly- w/ slow increase in size of heart, pericardium gradullay enlarges allowing enlargement of heart to occur w/out compression.æhemopericardium- stab wounds piercing heart occur w/out compression but can cause blood to enter pericardial cavity can also produce this or may reult from perforaation of weakened area of heart muscle after heart attack. as blood accumulates, heart is compressed and circulation fails.æpericardiocentesis- drainage of serous fluid from pericardial cavity usually necessary to relieve cardiac tamponade, removing excess fluid using wide-bore needle may be inserted through left subcostal angle 5-6th intercostal space near sternum. Caused by ruptured aortic aneurysm, ruptured myocardial infarct, penetrating injury compromises bating hear and decreases venous return and cardiac output removed by tap. vascular bundle on either side is phrenic nerve. Visceral and parietal side on inside of pericardial sac. Fibrous component of parietal so over time if slowly fill it slow long distentional stretch. Acute blood in there fibrous component stretch and had tamponade. Blood and fluid lubricate it against sac. Heart sounds are distant because more between you and valves, compression not able to fill as much going back up neck veins and not as much pressure in BeckÍs triad. Aim close to sternal body but not too close avoiding thoracic aorta. Pericarditis- decreased cardiac output creaking sound
atrial septal defect
(ASDs), if they remain small, have less shunting issues with the lower pressure difference between the right and left atria. Around 10% of adults have a patent foramen ovale, which may remain asymptomatic, but also could be an opening that allows for paradoxical emboli to enter the systemic circulation if right atrial pressure is ñboostedî to greater than left atrial pressure. congenital anomalies of interatrial septum, related to closure of oval foramen (atrial septal defects). probe-sized patency (defect) appears in superior part of oval fossa. usually no clinical significance, but large ASDs allow oxygenated blood from lungs to be shunted from left atrium through defect into right atrium, causing enlargement of right atrium and ventricle and dilation of pulmonary trunk. ostium secundum defects from incomplete closure of foramen ovale, larger defects require a patch sutured. smaller use percutaneous transcatheter approach using septal occluder deployed and secured. threading catheter through IVC catheter positioned to pass directly into atrial defect and deployed.
ventricular septal defect
(VSDs) are the most common, as the membranous part of the interventricular septum is the last to fuse and so can remain patent between the two ventricles. The higher pressure generated by the left ventricle will force additional blood into the right ventricle, or a left to right shunt. perimembranous- where muscular septum and membranous septum of endocardial cushion should fuse) membranous part of IV septum develops separately from muscular part and has complex embryological origin place for ventricular septal defects causing left to right shunt of blood through defect. large shunt increases pulmonary blood flow, causing pulmonary disease (hypertension) and may cause cardiac failure.
tetralogy of Fallot
results from maldevelopment of spiral septum dividing truncus arteriousus into pulmonary trunk and aorta. involving pulmonary stenosis or narrowing of right ventricular outflow tract, overriding transposed aorta, right ventricuar hypertrophy, ventricular septal defect (VSD) surgical repair done on cardiopulmonary bypass to close VSD and provide unobstructed flow into pulmonary trunk. Stenotic pulmonary outflow tract widened by inserting patch into wall (pericardial) increasing volume of subpulmonic stenosis and/or pulmonary artery stenosis. Linear structure whereby atrium feeds into ventricle feeds into aortic and pulmonary trunk. Ventricle becomes name becoming ventral creating twist left atrium on back side. Separate right and left circulations have septa built in between atria and ventricle. Partition several round of septa premium, Secundum creating foramen ovale from umbilical cord to left atrium. Interventricular septum muscular part sticking up with membranous part Causing tetralogy of allot. Partition blows arteriouss finish membranous portion of septum but does not happen in this condition. Tetralogy of Fallot is defined by four issues of: Pulmonary stenosis, Ventricular septal defect, Overriding aorta, Right ventricular hypertrophy. The tetralogy is caused by abnormal neural crest cell migration and unequal partition of the aortic-pulmonic septum in the developing heart (causing the first three features, and the right ventricular hypertrophy compensatory from the pulmonic stenosis). Affected children may have ñtet spells,î where subsequent squatting tends to decrease a right_ left shunt and increase pulmonary flow following the increase in peripheral systemic resistance. spiral divide between aortic arch and pulmonary trunk around same size. Going down forming membranous part of interventricular septum running into muscular part. In this condition it doesnÍt occur. Neural crest cell- pharyngeal arches and doing 75/25 instead of 50/50 more on aorta side (overriding aorta) a lot bigger than the pulmonary side, pulmonary stenosis, not migrating and forming full interventricular septum leaving a defect. Pulmonitic artery so stenotic right side hypertrophy to work harder to get it to lungs. Not as much blood flow through pulmonic artery get right to left shunt because of the hypertrophy over time causing deoxygenated blood through systemic circulation, congenital heart defect usually this.æ
patent ductus arteriosus
not part of the tetralogy, but for a child with TOF, a PDA can alleviate some of the cyanosis, as blood will be driven from the higher pressure aorta into the lower pressure pulmonary arteries and hence through the pulmonary circulation back to the left atrium, thereby bypassing the stenotic pulmonary valve region.
valvular heart disease
valves of heart disturb pumping efficiency of heart. Produces either stenosis (narrowing) or insufficiency. result in increased worload of the heart. restruction of high-pressure blood flow (stenosis); and passage of blood through narrow opening into larger vessel or chamber (stenosis and regurgitation) produce turbulence setting up eddies (small whirlpools) producing vibrations that are audible as murmurs. superficial vibratory sensation-thrills may be felt on skin over area of turbulence.can be replaced surgically w/ valvuloplasty w/ artificial valve prostheses from synthetic materials or xenografted valves (pigs).
stenosis
failure of valve to open fully, slowing blood flow from chamber. can have a great effect on hemodynamics, and so create distinctive murmurpatterns, e.g., the diastolic murmur of aortic regurgitation. Aortic stenosis will be commonly seen in the elderly, due to in increased pressure. ex. mitral stenosis leads to left atrial dialtion caused by RHD
external genitalia and hypaxialmuscles that anchor and move them (and the anus). insufficiency
valvular insuffiency or regurgitation, is failure of valve to close completely owing to nodule formation or scarring and contraction of cusps so that the edges do not mee tor align. This allows variable amount of blood (depending upon severity) to flow back into chamber ejected from. Calcified valve sound reflecting pressure of ventricle with ventricle, Hypertrophy to get pressure crescendo—decrescendo. Cardiac output decreased causing left in carotid artery causing syncope. Aortic regurgitation: caused by congentially malformed leaflets, RHD< IE< anklyosing spondylitis, Marfan’s syndrome, aortic root dilation. Mitral regurgitation: caused by abnormalities of valve leaflets, rupture of paipllary muscle or chordae tendinae, papillary muscle fibrosis, IE, left ventricular enlargement.
mitral valve prolapse
insufficient or incompetent valve in which one or btoh leaflets are enlarged, redunandt or floppy and extending back into left atrium during systole. blood regurgitates into left atrium when ventricle contracts producing mumur. which classically presents with a late systolic click as the floppy valve leaflets prolapse into the left atrium and thereby suddenly tense up the chordae tendinae. AV valves leaking, causing click then murmur, leaflet has … part of leaflet floppy depending upon tension upon it causing regurgitation. When have increased pressure Click shortens and comes later left Ventricular not as taut click earlier w/ longer murmur.
aortic valve stenosis
most frequent abnormaly resulting in left ventricular hypertrophy. majority of cases of aortic stenosis result from degenerative calcification. Caused by rhemuatic heat disease (RHD), congenital bicuspid valve
pulmonary valve stenosis
(narrowing), valve cusps are fused, forming dome w/ narrow central opening. infundibular pulmonary stenosis, conus arteriosus is underdeveloped, producing restriction of right ventricular outflow. degree of hypertrophy of right ventricle is variable
coronary artery disease:
from atherosclerosis result in reduced blood supply to vital myocardial tissue. common sites for occlusion are LAD, right main, and left circumflex coronary arteries. Atherosclerosis can be triggered by trubulent blood flow at arterial branches, which triggers sheer stress on endothelium causing damage and inflammatory response. Macrophages may take up available oxidized lipids and become foam cells. Additional triggering of cytokines can stimulate smooth muscle and connective tissue proliferation that contribute to plaque development. Plaque rupture can trigger thrombosis. happens more on left because of higher workload than right
infarction of each main artery and its affect on heart and what leads it could be found in
caused by sudden occlusion of major artery by an embolus, region of myocardium supplied by occluded vessel becomes infarcted (rendered virtually bloodless) and undergoes necrosis (pathological tissue death). patterns reflect the supply areas of the different coronary arteries, e.g., LAD generating an anterior infarct involving much of the interventricular septum. Anterior infarct affects anterior LAD (left ventricular surface anterior 2/3rds of interventricular septum could lead to bundle branch block leads 2/3. Right coronary- affects posterior wall of ventricle posterior 1/3 of IVS (if right-dominant coronary circulation). Left circumflex- affects alteral wall of left ventricle can also affect posterior wall if left dominant coronary circulation in leads 5+6
angioplasty
surgeons use percutaneous transluminal coronary angioplasty, pass catheter w/ small inflatable balloon attached to tip into obstructed coronary artery. When it reaches obstruction, balloon is inflated, flattening atherosclerotic plaque against vessel’s wall, vessel stretched to increase size of lumen, improving blood flow. Thrombokinase injected through catheter dissolving blood clot after dialtion of vessel intravascular stent introduced to maintain dilation.
coronary artery bypass graft
(CABG), also called “the cabbage procedure,” offers a surgical approach for revascularization. Veins or arteries from elsewhere in the patient’s body are grafted to the coronary arteries to improve the blood supply. In a saphenous vein graft a portion of the great saphenous vein is harvested from the patient’s lower limb. Alternatives include internal thoracic artery and radial artery grafts.
referred pain
Heart insensitive to touch, cutting, cold, and heat. ischemia and accumulation fo metabolic products stimualte pain endings in myocardium. viscerable afferent pain fibers run centrally in middle and inferior cervical branches in thoracic cardiac branches of sympathetic trunk axon to their spinal nerve source (T1(Ulnar side of arm)-T4 (nipple) and the corresponding dorsal root ganglia that then are interpreted as pain in those dermatomes. With cell bodies in the same spinal ganglion and central processes that enter the spinal cord through the same posterior root. Cardiac referred pain is a phenomenon whereby noxious stimuli originating in the heart are perceived by the person as pain arising from a superficial part of the body— the skin on the medial aspect of the left upper limb, for example. s. visceral pain afferents from the heart enter the upper thoracic spinal cord along with somatic afferents, both converging in the spinal cord’s posterior horn. Angina pectoris (“strangling of the chest”) is usually described as pressure, discomfort, or a feeling of choking or breathlessness in the left chest or substernal region that radiates to the left shoulder and arm, as well as the neck, jaw and teeth, abdomen, and back. The higher brain center’s interpretation of this visceral pain may initially be confused with somatic sensations from the same spinal cord levels. Somatic pain is “mapped” on the brain’s sensory cortex, but a similar symptomatic mapping of visceral sensations does not occur. This may explain why pain from visceral structures is often mistakenly perceived as somatic pain
conducting system damage
Damage to the conducting system, often resulting from ischemia caused by CAD, produces distur- bances of cardiac muscle contraction. Because the anterior IV branch (LAD branch) supplies the AV bundle in most people and because branches of the RCA supply both the SA and the AV nodes, parts of the conducting system of the heart are likely to be affected by their occlusion. Damage to the AV node or bundle results in a heart block because the atrial excitation does not reach the ventricles. As a result, the ventricles begin to contract independently at their own rate (25–30 times per minute), which is slower than the lowest nor- mal rate of 40–45 times per minute. Damage to one of the bundle branches results in a bundle branch block, in which excita- tion passes along the unaffected branch and causes a normally timed systole of that ventricle only. The impulse then spreads to the other ventricle, producing a late asynchronous contraction.
anterior mediastinum masses
include: thymoma (thymus tumor associated w/ myasthenia gravis), thyroid mass (cause enlarged gland to extend inferiorly and displace trachea starts in tongue created in development as thyroid migrates down thyroglossal cyst w/ passageway may not have been sealed throughout developing causing mass), teratoma (remnants of germ cells, hair, teeth, parts of organs from totipotent cells), lymphoma (lymph nodes Hodgkin’s and primary mediastinal B cell tumor w associated w/ mainstream bronchus or trachea of throacic artery and vein). symptoms include: retrosternal pain, cough, dyspnes, SVC syndrome, choking sensation.
middle mediastinum masses
similar to anterior include surrounding heart from lymph nodes, aortic aneurysm (aneurysm that is atherosclerotic in origin, may rupture in any part of mediastinum), vascular dilation (enlarged pulmonary trunk or cardiomegaly), and cysts (bronchogenic at tracheal bifurcation, and pericardial cysts).
posterior mediastinal masses
associated w/ pain, neurologic symptoms or swallowing difficulty caused by neurogenic tumors (peripheral nerves or sheath cells) or esophageal lesions (diverticular and tumors)
azygos drainage
The azygos system, as it drains the body wall, can be an alternative venous pathway if either of the venaecavaeare obstructed. Hemiazygoshave accessory as well. an acces- sory azygos vein parallels the main azygos vein on the right side. Other people have no hemi-azygos system of veins. A clinically important variation, although uncommon, is when the azygos system receives all the blood from the IVC, except that from the liver. In these people, the azygos system drains nearly all the blood inferior to the diaphragm, except from the digestive tract. When obstruction of the SVC occurs superior to the entrance of the azygos vein, blood can drain inferiorly into the veins of the abdominal wall and return to the right atrium through the IVC and azygos system of veins.
abdominopelvic venous drainage routes
three collateral routes, formed by valveless veins of trunk are available for venous blood to return to heart when IVC is obstructed or ligated; inferior epigastric vein(tributaries of the external iliac veins of the inferior caval system, anastomose in the rectus sheath with the superior epigastric veins, which drain in sequence through the internal thoracic veins of the supe- rior caval system.)superficial epigastric(or superficial circumflex iliac veins, normally tributaries of the great saphenous vein of the inferior caval system, anastomose in the subcutaneous tis- sues of the anterolateral body wall with one of the tribu- taries of the axillary vein, commonly the lateral thoracic vein. When the IVC is obstructed, this subcutaneous col- lateral pathway—called the thoraco-epigastric vein—becomes particularly conspicuous). epidural venous plexus inside the vertebral column communicates with the lumbar veins of the inferior caval system and the tributaries of the azygos system of veins, which is part of the superior caval system
thoracic aneurysm
distal part of the ascending aorta receives a strong thrust of blood when the left ventricle con- tracts. Because its wall is not yet reinforced by fibrous pericardium (the fibrous pericardium blends with the aortic adventitia at the beginning of the arch), an aneurysm (localized dilation) may develop. An aortic aneurysm is evident on a chest film (radiograph of the thorax) or a magnetic resonance an- giogram as an enlarged area of the ascending aorta silhouette. Individuals with an aneurysm usually complain of chest pain that radiates to the back. The aneurysm may exert pressure on the trachea, esophagus, and recurrent laryngeal nerve, causing difficulty in breathing and swallowing. While abdominal aortic aneurysms (AAAs) are more common and due to atherosclerosis, the location of thoracic aneurysms can impact on different symptoms, e.g., impact on the trachea, left recurrent laryngeal nerve. The risk of dissection or rupture can be followed by the size of the aneurysm. Thoracic aneurysm- atherosclerosis and degenerative conditions of aorta ex. Intima (weakens media), media (swelling, Marfan syndrome connective tissue fibrolytic in elastic organs, tertiary syphilis (long ongoing inflammatory response found w/ heave. Left recurrent laryngeal might be dysphasia or hoarseness vague symptomology)), adventitia.
recurrent laryngeal nerve injury
The recurrent laryngeal nerves supply all the intrin- sic muscles of the larynx, except one. Consequently, any investigative procedure or disease process in the superior mediastinum may involve these nerves and affect the voice. Because the left recurrent laryngeal nerve hooks around the arch of the aorta and ascends between the trachea and the esophagus, it may be involved when there is a bron- chial or esophageal carcinoma, enlargement of mediastinal lymph nodes, or an aneurysm of the arch of the aorta. In the latter condition, the nerve may be stretched by the dilated arch of the aorta.
coarctation of aorta
coarctation of the aorta, the arch of the aorta or descending aorta has an abnormal narrowing (stenosis) that diminishes the caliber of the aortic lumen, producing an obstruction to blood flow to the inferior part of the body. The most common site for a coarctation is near the ligamentum arteriosum. When the coarctation is inferior to this site (postductal coarctation), a good collateral circulation usually develops between the prox- imal and distal parts of the aorta through the intercostal and internal thoracic arteries. The increased blood flow in some arteries may lead to enlargement of those vessels. can lead to a visibly increased collateral circulation within the intercostal and internal thoracic arteries through their anastomoses, and thereby may lead to rib notching. In coarctation, with the proximal aorta compromised, blood flow goes through the subclavian arteries to the internal thoracic arteries to the anterior intercostal arteries to the posterior intercostal arteries and so into the thoracic aorta distal to the obstruction. Squeezed aorta to visceral aorta gets blood out and down through workaround subclavian feed into internal thoracic into anterior intercostal posterior intercostal come off aorta and they anasthemose. Reverse flow back through posterior intercostal into aorta working around becoming tortuous pulsating eroding costal groove creating rib notching. Strong in arms but not in the feet. The marked CXR highlights the coarctation itself (blue) and the rib notching that occurs with the large amount of additional flow through the intercostal vessels as a result of this altered pathway
thoracic surface anatomy landmark lines
see sternum, clavicle, breasts, lung, and heart
Several bony landmarks and imaginary vertical lines facilitate anatomical descriptions, identification of thoracic areas, and location of lesions such as a bullet wound:
• Anterior median (midsternal) line indicates the inter- section of the median plane with the anterior thoracic wall.
• Midclavicular lines pass through the midpoints of the clavicles, parallel to the anterior median line.
• Anterior axillary line runs vertically along the anterior axillary fold, which is formed by the border of the pectoralis major as it spans from the thorax to the humerus (arm bone)
• Midaxillary line runs from the apex (deepest part) of the axilla, parallel to the anterior axillary line.
• Posterior axillary line, also parallel to the anterior axillary line, is drawn vertically along the posterior axillary fold formed by the latissimus dorsi and teres major muscles as they span from the back to the humerus.
• Posterior median (midvertebral) line is a vertical line at the intersection of the median plane with the vertebral column. Scapular lines are parallel to the posterior median line and cross the inferior angles of the scapulae.
thoracic imaging
B. Describe the layers of the abdominal wall
epidermis> dermis> subcutaneous tissue (superficial fascia)> fatty layer of subcutaneous tissue (camper fascia)>deep membranous layer of subcutaneous tissue (scapa fascia)right over muscles deep to fascia on top of muscles> abdominal muscles (external, internal, transversus abominus) w/ investing deep fascia in between that is superficial, intermediate, and deep> transversalis fascia (endoabdominal fascia), extraperioneal fascia w/ fat, parietal peritoneum (innvervation of these layer 1-8 via spinal nerves leading to sharp localizing pain if involved: visceral peritoneum (everything in refferred pain pattern> peritoneal cavity. Paracentesis, e.g., to drain abdominal ascites fluid, will penetrate into here> visceral peritoneum. Involvement of this layer that covers the visceral organs will typically generate referred pain patterns. when doing surgeries no transceting muscles (irreversible eath of muscle fibers), splits between except rectus abdominis transected because muscle fibers short and nerves located and preserves. suture membranous layer of subcutaneous tissue as seperate layer because of strength, space between membranous layer and deep fascia
B2. describe layers of inguinal canal
The gonads develop superiorly and developmentally migrate to their inferior positions, as directed by an embryonic gubernaculum. The male inguinal canal contains the spermatic cord as the testis reaches its final location in the scrotum, attached by a short scrotal ligament (gubernaculum testis) within the cord. Note that there is a bit of peritoneal cavity “captured” as the tunica vaginalis. Process vaginalis- gubernaculum shorten sealing sheath behind testis descended (tunica vaginalis) and gubernaculum had testicular ligament at inferior pole of testis. Ovaries pull down and into labia majors more of round ligament of uterus hanging off of ovary and connected to uterus ovarian and into canal of round ligament. The male process of testicular migration can lead to alterations, e.g., cryptorchidism, if the testis does not complete its migration. Deep inguinal ring in transversalis fascia, superficial inguinal ring through aponeurosis external oblique, sealed processed vaginalis The female inguinal canal contains the round ligament of the uterus as its remnant of the gubernaculum. Internal spermatic fascia- transversus fascia Cremation fascia from internal oblique pull things up Aponeurosis off of external oblique external spermatic fascia
Within the context of basic embryology, describe the relationships of thoracic organs in adult
Thoracic Organs
Linear structure whereby atrium feeds into ventricle feeds into aortic and pulmonary trunk. Ventricle becomes name becoming ventral creating twist left atrium on back side. Separate right and left circulations have septa built in between atria and ventricle. Partition several round of septa premium, Secundum creating foramen ovale from umbilical cord to left atrium. Interventricular septum muscular part sticking up with membranous part Causing tetralogy of allot. Partition blows arteriouss finish membranous portion of septum but does not happen in this condition.
C. Within the context of basic embryology, describe the relationships of abdominal organs in the adult
Holding it onto the back wall. the gut, supported by a dorsal mesentery, forms as a long tube that elongates and rotates. Holding it onto the back wall. Three major vessels coming off aorta (celiac, superior mesenteric, and inferior mesenteric) developmentally create foregut w/ stomach and such. Midgut- small intestine and half of large intestine. Hindgut- last part of large intestine. In the developing proximal gut, there is an additional gastrohepatic ligament- ventral mesentery in which the liver forms close to the stomach (expanding shifts and switch places), with its adult remnant known as the lesser omentum. Can see folds of midgut coming out then rolling and coming back in. Adult segment falciparum ligament- sic cal shape between liver and stomach. spleen ligaemounts holes to stomach along way. Dorsal mesentery of stomach along greater curvature decides to expand and fold in creating double fold adhering to top of small intestine aka greater omentum The liver develops as an endodermal outgrowth of the duodenum in the ventral mesentery, dividing it into the falciform ligament between liver and umbilicus, and lesser omentum between liver and stomach. The greater omentum will be better visualized on the next slide. The stomach, duodenum and the other viscera (liver, pancreas, spleen) that develop in the mesogastrium are not part of the primary loop, so they are supplied by branches off of the celiac trunk. While all this is going on, the rest of the gut is elongating, forming a primary intestinal loop or region that is supplied by the superior mesenteric artery. From the left colic flexure distally, this developing digestive tract is supplied by the inferior mesenteric artery. Foregut developed into celiac artery, midgut into superior mesenteric artery, hindgut into inferior mesenteric artery
C. Within the context of basic embryology, describe the relationships of pelvic organs in the adult
During development, the testes in effect push through the layers of the abdominal wall, so that each layer contributes to the spermatic fascia that cover the spermatic cord and inguinal canal. From superficial to deep: From the external oblique aponeurosis: external spermatic fascia and superficial inguinal ring. From the internal oblique: cremasteric fascia and loops of cremaster muscle. From the transversalis fascia: internal spermatic fascia and deep inguinal ring. there is fusion of much of the dorsal mesentery of the abdominal gut to the parietal peritoneum of the dorsal wall, so that many structures originally in the peritoneal cavity, e.g., pancreas, duodenum, ascending and descending colon, are considered retroperitoneal. Other organs such as the kidneys develop deep to the peritoneal cavity and so are retroperitoneal in that manner. supported by a dorsal mesentery, forms as a long tube that elongates and rotates
D. Combining an understanding of visceral regions and quadrants, and visceral pain pathways, explain common abdominal pain presentations.
Heart insensitive to touch, cutting, cold, and heat. ischemia and accumulation fo metabolic products stimualte pain endings in myocardium. viscerable afferent pain fibers run centrally in middle and inferior cervical branches in thoracic cardiac branches of sympathetic trunk axon to their spinal nerve source (T1(Ulnar side of arm)-T4 (nipple) and the corresponding dorsal root ganglia that then are interpreted as pain in those dermatomes. With cell bodies in the same spinal ganglion and central processes that enter the spinal cord through the same posterior root. Cardiac referred pain is a phenomenon whereby noxious stimuli originating in the heart are perceived by the person as pain arising from a superficial part of the body— the skin on the medial aspect of the left upper limb, for example. s. visceral pain afferents from the heart enter the upper thoracic spinal cord along with somatic afferents, both converging in the spinal cord’s posterior horn. Angina pectoris (“strangling of the chest”) is usually described as pressure, discomfort, or a feeling of choking or breathlessness in the left chest or substernal region that radiates to the left shoulder and arm, as well as the neck, jaw and teeth, abdomen, and back. The higher brain center’s interpretation of this visceral pain may initially be confused with somatic sensations from the same spinal cord levels. Somatic pain is “mapped” on the brain’s sensory cortex, but a similar symptomatic mapping of visceral sensations does not occur. This may explain why pain from visceral structures is often mistakenly perceived as somatic pain
abdominal incisions
When closing abdominal skin incisions, surgeons suture the membranous layer of subcutaneous tissue as a separate layer because of its strength. Between the membranous layer and the deep fascia cov- ering the rectus abdominis and external oblique muscles is a potential space where fluid may accumulate (e.g., urine from a ruptured urethra). Although no barriers (other than gravity) prevent fluid from spreading superiorly from this space, it cannot spread inferiorly into the thigh because the membranous layer of subcutaneous tissue attaches to the pubic bone and fuses with the deep fascia of the thigh (fascia lata) along a line inferior and parallel to the inguinal ligament. Operation- nonlaproscopic down to lines alba (aponeurosis forming midline connection of muscles)– no blood vessels or nerves only connective tissue have to recline it up and connect it can cause herniation. Note that the abdominal wall muscles and their aponeuroses dominate the pattern of the anterior abdominal wall. The linea alba is a favored midline for incisions, with proper alignment of its connective tissue is needed for closure to heal. Location of incision chosen allows adequate exposure, cosmetic effect, type or operation, bones surrounding, avoiding n.,a.,v. aiming for favorable healing. instead of transecting msucles, causing irreversible necrosis (death) of muscle fibers will split between fibers. The rectus abdominis is an excep- tion and can be transected because its muscle fibers are short and its nerves entering the lateral part of the rectus sheath can be located and preserved. Cutting a motor nerve paralyzes the muscle fibers supplied by it, thereby weakening the anterolat- eral abdominal wall. However, because of overlapping areas of innervation between nerves in the abdominal wall, one or two small branches of nerves may be cut without a noticeable loss of motor supply to the muscles or loss of sensation to the skin. Abdominal wall hernias often are called ventral hernias to distinguish them from inguinal hernias. However, all are technically abdominal wall hernias. Other than inguinal hernias, most common types of abdominal hernias include:
• Umbilical hernia: usually seen up to age 3 years and after 40.
• Linea alba hernia: often seen in the epigastric region and more common in males; rarely contains
visceral structures (e.g., bowel).
• Linea semilunaris (spigelian) hernia: usually occurs in midlife and develops slowly.
• Incisional hernia: occurs at the site of a previous laparotomy scar.
incisional hernias
concern with the potential entrapment of a loop of bowel. Umbilical hernias represent a congenital weakness in that region, given the embryological herniation of the developing intestinal tract. If the muscular and aponeurotic layers of the abdomen do not heal properly, a hernia may occur through the defect. An incisional hernia is a protrusion of omentum (fold of peritoneum) or an organ through a surgical incision or scar. Innermost part of subcutaneous tissue not as fatty more thicker more easily caught caught. Transversalis fascia on inside of transverse muscle, fat…, parietal peritoneum. Weakness in abdominal wall unpleasant lump, incarceration or trapping of internal organs. Umbilical- physiologici hernia of umbilical
endoscopic surgery
Many abdominopelvic surgical procedures are now performed using an endoscope, in which tiny per- forations into the abdominal wall allow the entry of remotely operated instruments, replacing the larger conven- tional incisions. Thus, the potential for nerve injury, incisional hernia, or contamination through the open wound and the time required for healing are minimized.
abdominal protuberance
Inspection of the abdomen may indicateprotrusion (the “six Fs”Food , Fluid (duller) , Fat , Feces (firmer left colon) , Flatus (gas hypertympanic) , Fetus ). Eversion of the umbilicus may be a sign of increased intra-abdominal pressure, usually resulting from ascites (a possible indication for paracentesis)(abnormal accumulation of serous fluid in the peritoneal cavity) or a large mass (e.g., a tumor, a fetus, or an enlarged organ such as the liver). Excess fat accumulation owing to overnourishment most commonly involves the subcutaneous fatty layer; however, there may also be excessive depositions of extraperitoneal fat.