theresa maurer Flashcards

1
Q

habitual abortion

A

spontaneous abortion that has terminated the course of 3 or more consecutive pregnancies

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2
Q

first trimester bleeding and pain

A

ectopic pregnancy should always be suspected

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3
Q

Threatened abortion
s/s
Rx
diagnosis

A

bleeding in first half pregnancy
fresh or old brown blood
may or may not having cramping and low backache
Rx: pelvic rest, bed rest, no sex, notify midwife if there is fever, a gush, increased bleed or increase low back pain.
Rx: heavy bleeding/febrile, no bleeding but ctx and abnormal ultrasound-immediate physician eval
slight bleeding and no other abnormal findings, reiterate Rx regular RTC
gentle spec exam, screen vaginitis cervicitis
gentle bimanual exam, uterine size, effacement, dilation, membrane status

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4
Q

febrile

A

signs of fever

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5
Q

inevitable abortion
s/s

eval

treatment

follow up

A

combo of bleeding ctx, ROM, dilation
assess gestational age, amount bleeding, ab pain, emotional status, previous or stat hematocrit, dilation, vital signs
choices: if bleeding is not excessive, pain not excessive and emotional status good, hematocrit 30%
1. go to physician for induced abortion D&C
2. go home await spontaneous abortion
take temp every 4 hours
call midwife if a pad is soaked in less than 1 hr
clots larger than 2.5 cm
fever of 100 +
when aborted call midwife
followup: counsel, support, sex 2-4weeks, genetic counseling.

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6
Q

incomplete abortion

A

placenta is not expelled with fetus
can cause bleeding or infection
D&C

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7
Q
missed abortion
s/s
diagnosis
plan
follow up
A

vag spotting bleeding, low ab back pain
fundal height ceases to increase, uterus decreases
regressed mammary changes
loses weight
persistent amenorrhea
no fetal heart tones
diagnosis: ultrasound
severe coagulation problems may occur from carrying non viable fetus
order coagulation serums, prothrombin,fibronogen, platelets, partial prothrombin, endocrinologic workup, uterine abnormalities screen
followup: counsel, support, sex 2-4weeks, genetic counseling.

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8
Q

incompetent cervix
s/s
predisposed

A

rare in primigravidas
evidence in 2nd trimester
painless dilation, ROM, expulsion of fetus, vag discharge free from infection s/s (itching, odor etc)
previous fetal loss, cervical surgery

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9
Q

cerclage

reason for

follow up

A

Classic shirodkar-permanent suture stays until birth, csection delivery
Modified Shirodkar- purse string suture each pregnancy removed at delivery
Mcdonalds- purse string suture each pregnancy removed at delivery

can be done if cervix is 4 cm
vaginal exam every 2 weeks
counseling
client to report any s/s of bleeding, ROM, foul odor, fever.

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10
Q

suspicious history of incompetent cervix- assesment

A

History- s/s previous loss, infections in relation, discharge etc.
gestational age, genetic abnormalities, previous suction abortios, cervical traumas
Pelvic ex- consis, length, dilation (internal, ext) membranes, position station present part

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11
Q

history of cervical trauma without fetal loss

plan

A

vaginal exam every two weeks starting at 2nd trimester until fetal viability or

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12
Q

history of fetal loss but no current signs of cervical incompetence

A

vaginal exams every 1-2 weeks starting in 2nd trimester until viability or dilation

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13
Q

cerclage is removed in these cases

A

infection, suture not intact, or laceration cervix
after ROM, impending labor or 38 weeks, labor start
all unless it is a Classic Shirodkar suture

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14
Q

hydadtiform mole

complete/partial

A

chorionic villi are clear vessels with pedicles hanging like grapes, mass of cyst clear vesicles
complete- all vesicles
partial- vesicles with non viable fetus and sac
some are benign with potential to be malignant & proceed choriocarcinoma

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15
Q

benign

A

not harmful

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16
Q

malignant

A

infectious volatile

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17
Q

hydadtiform mole
likelyhood
s/s

A

10 times higher in women over 45 years
persistent nausea and vomiting, uterine bleeding evident at 12 weeks, brown blood, intermittent, anemia, large for dates baby, shortness of breath, tender ovaries, not fht, parts or palpation.
PIH, preeclampsia, eclampsia, before 24 weeks!
very high Hcg levels

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18
Q

ectopic pregnancy
predisposed
types
diagnosis

A

pelvic infections, IUD, precious ectopic & tubal preg

types: cervical, tubal, ovarian, abdominal
order quantitative Hcg & ultrasound

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19
Q

cervical ectopic pregnancy

s/s

A

rare,not lasting after 20 wks
painless bleeding at implantation
cervical mass, distention, thinning of cervical wal, dilation of ex os, enlarged fundus

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20
Q

tubal ectopic pregnancy
s/s

differential diagnosis

labs

A

most common 95% or more cases
classic case of tubal cases knowing or unknowing suspected pregnancy but spotting has substituted menses.
s/s sharp stabbing pain, tearing, low ab pain
hypotension, shock, tenderness, painful exam, tender boggy mass to one side of uterus

pain in the neck or shoulder* especially inhaling
displaced uterus
low Hcg or neg pregnancy tests

differential: salpingitis, threatened/inc abortion, twisted ovarian cyst, ruptured corpus luteum follicular cyst

CBC with differential and quantitative beta Hcg
ultrasound

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21
Q

abdominal ectopic pregnancy

s/s

A

s/s sharp stabbing pain, tearing, low ab pain
hypotension, shock, tenderness, painful exam, tender boggy mass to one side of uterus, displaced uterus

pain in the neck or shoulder* especially inhaling
gastrointestinal upset, transverse lie, painful fetal movement, very audible heart tones, small parts outside of uterus, cervical displace, dilation no effacement.

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22
Q

hyperemesis gravidarum
s/s
plan

A

s/s: excessive nausea and vomiting, extends past 1st trimester, poor appetite and intake, weight loss, dehydration,electrolyte imbalance, extreme response to psycho social events, acidosis (starvation), alkalosis (no hydrochloric acid), hypokolemia(low potassium)
labs: BUN and electrolytes
urine dipstick for acetone and glucose
blood gases, serum pH,
spilling glucose and acetone- immediate eval, glucose to assess for diabetes
dehydration IV fluids
continues after IV fluids admit to hospital

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23
Q
Tuberculosis mycobacterium
s/s
history
labs
treatment
A

initial lesion develops on lung, exudate inflammed, necrosis of lung tissue-airborne droplets
s/s- fever, night sweats, weight loss, persistent colds and cough, chronic cough with yellow green mucous
pleurisy(chest pain, inflammation) rales

previously infected, exposure to, history, environmental, poverty, drug use, socioeconomic

Mantoux test, PPD test, not if active infection!
chest xray and sputum test
BCG vaccine can cause positive test- xrays everty 2 years. scar on arm from vaxx

treatment: INH (isoniazid chemotherapy) for women under age 35 during pregnancy with positive PPD neg xray and post PPD and post xray. If not in pregnancy then after pregnancy, treatment continued if pregnancy occurs mid treatment.
reportable disease.

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24
Q

A Sickle Cell screen should be offered to which group of clients?

A

african descent

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25
Q
cultures prone to certain disease
african
mediterranean
french canadian
jewish
A

Sickle cell anemia is found primarily in 1/375 birth of African-Americans.
Thalasemia is predominantly found in those of Mediterranean descent;
TaySachs disease is found in those of French Canadian descent
Ashkenazi Jewish descent.

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26
Q

hepatitis
most common in US
rare forms

A
inflammation of the liver via an infection
hep A and hep B
hep D is secondary to B -mediterranean
D & E rare- Asia, south america
reportable diseases
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27
Q
Hep A (infectious hepatitis)
route of transmission

s/s

A

most common worldwide- impoverished pop, poor hygienic
fecal oral route is transmission - contaminated food and water
s/s- flu, fatigue, malaise, weak, nausea, low fever, upper right pain, rapid onset, 10-15 days acute to resolve 2 months
no risks to newborn,no transmission
vaccine for at risk or close contact ISG (immune serum globin)
routine screen

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28
Q
Hep B (serum hepatitis)
route of transmission
s/s
transmission
treatment
labs
A

transmission through blood, blood products, needles , saliva, vag secretions, semen.
1-6 months incubation
may lead to chronic carrier, active hep, liver disease, cirrhosis.
s/s rash fever, fatigue, weak, nausea, vomit, low fever, upper right pain, tender enlarged liver, headache

vertical transmission to the baby is common, any route of delivery, postnatal close contact
infects all body fluids but breast milk!!!
breastfeeding contraindicated if cracked sore nipples

90% change of transmission, 90% become carriers, transmitted to offspring, 25% die of cirrhosis, liver carcinoma

babies receive hep b vax at birth within 1 hour 90-95% protection, hep b vax is given to mothers who havent been vaxxed prior

IgM- active antibody IgG- antigen
routine screen

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29
Q

Hepatitis C

A

40-50% of hep cases in US
low incidence of vertical

transmission to baby

blood test positive for hepc antibody

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30
Q

rubella (German measles)

risks to newborn
s/s
labs
treatment

A

1st trimester infection- 20% chance of congenital malformations
1st month- 50% malformations
glaucoma, cardiac defects, deafness, brain and deafness, SA, eyes, heart, brain CNS
s/s low fever, drowsy, sore throat, rash, spreads rapid, swollen glands, lasts 3-5 days

routine screen- rubella titres
1:10 or above immunity
below 1:10- lack of immunity offer vaxx POSTPARTUM
1:64 or higher ACTIVE infection- consult -series of titers consult
live vaxx not in pregnancy!
1st trimester- option to abort
avoid pregnancy 3 months after vax

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31
Q

cytomegalovirus
s/s
transmission

A

common viral infection-various strains
most asymptomatic:
fever sore throat, swollen glands, fatigue
transmission is effective in fetal period (10-40 weeks)
not seen until birth
risks: microcephaly, hydrocephaly, small eyes, seizures, blindness, encephalitis. IUGR, oligiohydraminos
no vaxx and no treatment
test babies saliva, urine, blood high concentrations

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32
Q

toxoplasmosis
s/s
transmission
testing

A

parasitic protozoa, infected meats, raw or undercooked, cat feces, soil. raw milk

s/s fatigue, malaise, muscle pain, swollen lymph
death, preterm, CNS, anecephalus, hyrdocephalus, eye and brain defects. hydrops
the earlier the infection the more severe disease. 1st and 2nd trimester most transmission.
3rd transmission always congenital anomalies

preconception, skin test 48-72 hrs
ELISA lab, high titers recent infection, rising titers current infection, low titers immunity
IgG antibodies from previous infection protects baby
consult with virologist, routine screen
Spiraymicin
cordocentesis can check baby for infection

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33
Q

varicella (herpes zoster=shingles)
s/s
risks
treatment

A

highly contagious viral infection form of HSV
25-40% of babies have congenital varicella syndrome
infection in 1st 20 weeks of pregnancy greatest risk

incubation 10-21 days
contagious 2 days before lesions and 7-10 days after lesions are crusted
fever chills, malaise, lesions head neck to trunk, pneumonia, chest pain, cough, fever

risks: cataracts, chorioretinitis, limb hypoplasia, dydronepherosis, micorcephaly,
maternal pneumonia 40% maternal death

varicella contracted at the very end of pregnancy , no antibodies, 5% babies die
treatment VZIG- exposed first 20 weeks and 6 days before delivery 2 days after delivery

routine testing serological for antibodies, vax prior to pregnancy no conception atleast 3 months!

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34
Q

why are pregnant women more susceptible to UTI

A

normal hydronephrosis may cause urine stasis

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35
Q

preventing UTI

A

drink 8 glasses of water, empty bladder, cranberry, good hygeine

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36
Q

presence of bacteria significant in clean catch

A

50,000 bacteria of same species per mililitre

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37
Q

100,000 of nonpatho, or 100,000 mixed

A

contaminated specimen

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38
Q

bacteria reported in urinalysis

A

coliforms, e coli, enterococci, klebsiella.

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39
Q

s/s of UTI
risks
treatment

A

urinary frequency, dysuria, pain, suprapubic pain
preterm labor and birth, low birth weight, pyelonephritis, inflamed kidneys
amoxicillin, ampicillin repeat culture 2 weeks, if doesn’t work 2nd round of diff antibiotics, suppressive therapy if doesn’t work

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40
Q

recurring UTI factors

A

diabetes, sickle cell trait, history of UTI

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41
Q

cystitis
s/s
labs

A

inflammation of the bladder, bacterial infection
s/s urgency, frequency, dysuria, low ab pain, hematuria
culture- nitrates, high WBC, high bact, rbc in urine, blood in urine
treatment same

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42
Q
acute pyelonephritis
cause
s/s
risks
treatment
A

inflammation of both kidneys, bacterial
2% freq in pregnant women

compression of ureters by uterus
dilation and decreased tone of ureters from progesterone
urine stasis- decreased bladder tone, urine stasis
s/s fever, chills, hematuria,nause vomit, hx of UTI, frequency CVA tenderness (right side kidney), low abpain
risks- septic shock, respiratory distress, anemai, pretern labor/delivery
treatment: refer to physician IV therapy, electrolytes, IV antibiotics.suppression therapy

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43
Q

why is hemoglobin lowered in pregnancy

A

hemodilution results in blood expansion and increase in plasma but not rbc’s, ration of rbc’s to plasma is disproportionate.

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44
Q

why does a women need increased iron in pregnancy

A

because of the increase in rbc’s

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45
Q

factors that effect hemoglobin

A

race, sex, elevation, smoking meds,
Africans have a hemoglobin 1 gdl higher
women who smoke or live higher altitudes have a higher low

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46
Q

anemia levels and definition

A

decrease in # of rbc or decrease in the concentration
less than 12 in nonpregnant
less than 10 in pregnant

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47
Q

s/s of anemia

A

fatigue, dizzy, malaise, headache, sore tongue, skin pallor, hx of heavy menses, close pregnancies, pale nails, hx anemia, nause vomit no appetite, PICA

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48
Q

initial lab test of rbc size

A

microcytic-decreased rbc size, iron deficient, thallasemia, lead disease
normocytic- blood loss, sicke cell, G6PD, med side effects
macrocytic-vitamin B 12 deficient, folic acid deficient

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49
Q

high iron foods

A

green leafy veggies, liver, egg yolk, raisins, fortified

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50
Q

treatment anemia

true anemia= hypochromic*

A

start on iron, folic acid and vita C supplements

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51
Q

hemoglobin below 9 after supplementing

A

hx, reeval, do labs, consult

CBC, reticulocyte count, serum iron, serum ferritin, TIBS (binding), platelet count, hemog electrophoresis

52
Q

MCV levels

A

high Mean Corpuscular Value = folate, b12 deficient

low MCV= iron deficiency

53
Q

hemoglobin electrophoresis

A
AA = normal
AS= sickle cell trait carrier
SS= sickle cell disease (genetic disorder) africans
54
Q

women with sickle cell trait have higher incidence of these conditions

A

asymptomatic bacteriuria, pyelonephritis close monitoring is needed

55
Q

G6PD

A

x linked genetic disease affects enzyme G6PD, associated with rbc’s
prevalent in Mediterranean, African americans

56
Q

hemolysis

A

destruction of rbc

57
Q

treatment of G6PD clients

A

obtain status if not sure
avoid fava beans, sulfa drugs, oxidant drugs, surgeries, diagnose any and treat infections prompt such as UTI to reduce hemolysis risk
notify physician immediately in case of csection

58
Q

normal pregnancy increase cardiac output by %?

when does increase occur?

A

40%

occurs early pregancy peaks at 20 weeks to 24 weeks

59
Q

cardiac output increases, when, %?

A

during pregnancy and during birth

50% during contractions, highest immediate postpartum

60
Q

heart disease/congestive failure

s/s

A

persistent rales in lungs, decreased physical activity, dysnpea, cyanosis, edema lower extremities
pregnancy can progress disease
rheumatic fever, heart failure, heart murmurs, dysrythmia, cardia enlargement, clubbed hands

61
Q

treatment and s/s of heart disease

A

collaborative care, iron folic acid, close monitor 20-24 weeks, counseling

62
Q

normal blood pressure in pregnancy rise and falls

how to take

A

drop in second trimester, rise in third trimester
sitting up with arm at heart level at rest, no crossed legs
left side supine,

63
Q

hypotension pressure

A

90/60 mm Hg indicate low blood pressure, or hypotension

64
Q

hypertension

A

140/90 mm Hg in pregnancy indicate high blood pressure, or hypertension

65
Q

asthma

A

1 in 4 pregnant women
risks: perinatal mortality, hyperemisis gravidarum, preterm, chronic hypertension, preeclampsia, hemorrhage, low birth weight

66
Q

OTC drugs that induce asthma attacks

A

aspirin and ibuprofen aleve,

67
Q

medications contraindicated in pregnant women with asthma

A

demoral, morphine and hembate

68
Q

type 1 diabetes

A

true insulin dependent

occurs before age 40

69
Q

type 2

A

occurs after age 40

obesity can be controlled with diet, exercise, oral hypoglycemic agents

70
Q

gestational diabetes

s/s
risks

A

abnormal carbohydrate metabolism
glucose monitor, diet exercise
diabetes mellitus cannot be diagnosed until postpartum
routine screen all women at 28 weeks
s/s polyuria, polydipsia (excessive thirst), weight loss, poor healing, polyhydraminos

shoulder dystocia, macrosomia, hypoglycemia, respiratory distress

71
Q

screen for women with history of diabetes, previous large baby, recurring candida, glycosuria

A

initial screen, 28 weeks, 34-36 weeks

72
Q

secondary risk factors to diabetes

A

preeclampsia, polyhydraminos, large for dates babies

screen at first signs and again at 34-36 weeks

73
Q

most efficacious diabetic screening

A

fasting blood sugar, 1 - 2hr postglucose challenge

74
Q

glucose challenge test

A

dont eat since midnight before test. drink entire glucose(100 g glucose)
mixture and draw blood at 1 or 2 hr mark

75
Q

abnormal testing values for glucose challenge and fasting plasma, GTT

A

fasting plasma of > 105mg per 100 ml
1 -hr 50g glucose challenge of >135 mg
2-hr 75g glucose challenge of > 120 mg

76
Q

does a woman with known or obivous diabetic get a GTT

A

no

77
Q

3-hr 100g GTT diagnostic for diabetes if

A

fasting 90mg
1 hr 165mg
2 hr 145 mg
3 hr 125 mg

78
Q

normal fast blood sugar, abnormal 1-2 hr, and abnormal GTT is gestational diabetic T or F

A

true

79
Q

values for plasma are 15% higher then whole blood values in glucose testing T or F

A

true

80
Q

any post and neg combo of fasting and 1 /2hr do GTT

T or F?

A

tue

81
Q

dietary guidelines for diabetes

A

30 calories per kg, 50% carb, 20 protein, 30 fat

82
Q

mulitple gestation

s/s

A

large for dates, rapid growth in 2nd trimester, palpation of multiple small parts, more then one fetal heart tone

83
Q

antepartum management of multips

A

early signs of preterm labor, s/s preeclampsia
monitor fetal growth, nutrition calories and protein added for each baby, weekly appointments for weight gain, fetal growth and preeclampsia, weekly from 34 weeks for cervical changes

84
Q

rh - mom and rh- dad =

A

negative baby no worries

85
Q

rh - mom

labs

A

screened initial ABO blood type if Rh- then:
28 weeks & 36 weeks for titers below 1:16
rhogam is offered prophylactic at 28 weeks is last about 12 weeks.
a positive titer from rhogam should be no higher then 1:4
If mom is sensitized with postive Coombs check titers at 26 to 28 weeks, 32-36 weeks, and 38 weeks

indirect Coombs test if antibodies are present a titer is obtained and a

86
Q

reasons for giving Rhogam to Rh- neg mother

A

uterine bleeding, trauma, hemorrhage, invasive procedures, SA, miscarriage, ectopic pregnancy, transfusion, postpartum in 72 hrs, prophylactic at 28 weeks
Rh- is the most sensitizing antibody!
NO rhogam if antibodies present and mother sensitized

87
Q

HDFN

A

hemolytic disease of the fetus newborn

88
Q

% of mothers sensitized in first pregnancy

A

13% of rh- mothers will be sensitized in first pregnancy

89
Q

risk of having a subsequently affected baby if not receiving Rhogam in previous postpartum?

A

90-100% risk of affecting next baby

90
Q

kernicterus

A

brain damage from extreme jaundice, sensitization, bilirubin fills the brain

91
Q

hydrops fetalis

A

extreme hemolysis form of Rh incompatibility, shifted edema

92
Q

how do antibodies attack baby in Rh- incompatibility

A

IgG antibodies cross the placenta and attack the baby

93
Q

Rhogam class C category

A

thimerosal= toxic mercury based
50-300ug
given deep Im- side of muscle
atlease 72 hours before antibodies can be produced

94
Q

labs for baby of rh- mother

A

blood typing from cord
bilirubin test
hemoglobin and cbc

95
Q

hydraminos
predisposed factors
risks
plan/labs

A

factors : multip, diabetes, erythroblastosis, fetal malform
risks:
cord prolapse, hemorrhage!
fetal malpresentation
abruptio placenta
uterine dysfunction
s/s: uterine enlarged, tense uterine wall, hard to get fht, uterine fluid thrill, dyspnea, vulvar edema, pressur pains, nausea vomit, frequent change in baby lie
plan: screen diabetes, screen ABO/Rh disease
ultrasound
consult physician

96
Q
oligohydraminos
predisposed
s/s 
risks
plan
A

congenital anomalies, IUGR, PROM, postmature syndrome
s/s- molding contour of fetus, not ballotable, lagging fundal height.
risks: variable decels, less fluid for cord cushion, poor tolerance for labor, cord compression,
plan: hydration, bed rest, nutrition, ultrasound, AFI check, NST’s, kick counts, fetal monitor

97
Q

Amniotic fluid production in pregnancy

A

increases in preg until it reaches about 1000 ml by third trimester, gradually decreases around 34 weeks to about 800 ml

98
Q

signs of fetal demise

risks

A

ceased: fht, uterine growth, movement, retrogressed breast changes, fetal skull collapse with exam,
Spaldings sign- excessive overlap of bones
exaggerated curvature of spine
Birth 2/3 weeks of demise
risks: deseminated intravascular coagulation DIC, test prothrombin, platelet and fibrogen. drop in platelets fibro consult, incrase in prothrombin consult induce labor.
encouraged to view, touch and hold infant
dont try to diagnose death, most have no cause even after autopsy

99
Q

hypertensive disorders of pregnancy

A
preeclampsia
eclampsia
chronic hypertensive
PIH
proteinuria, edema
100
Q

hypertension

A

140/90 or higher! or 30 rise in top, 15 rise on bottom from base

101
Q

MAP

A

= D x 2 + S
_________
3
2 readings 6 hours apart

102
Q

proteinuria

A

more than .3 g in 24 hr spec/ 1-2+ on stick

2 or more occasions when 6 hours apart

103
Q

edema

A

sudden weight gain, unquestionable in hands and feet

104
Q

hypotension

predisposed factors

A

hydadtiform mole trophoblastic disease, multips, chronic hypertensive vascular disease (20%), chronic renal disease, diabetes, fetal hydrops, age 35+, previous hx of preeclampsia

105
Q

preeclampsia
s/s
labs
Rx

A

headaches, blurred vision, upper edema, proteinuria increased weight and BP
labs: hemoglobin & hematocrit, platelet, liver function, kidney profile(BUN), urine protein, coagulation profile

leaning towards: bed rest, 2 hr in am and pm on left sideprotein, high protein high calorie
before 36 weeks ultrasound for IUGR, introuterineplacental insufficiency
NST, fetal kick count, liver & kidney function tests

106
Q

progressive severe eclampsia

A

hyperflexia, headaches, visual disturbances, epigastric pain, oliguria, elevating BP, 160/100 severe, increasing proteinuria 3+ 4+

107
Q

HELLP

A

may develop from preeclampsia,

hemolysis, elevated liver enzymes, low platelets,

108
Q

eclampsia

A

when preeclampsia progresses to convulsions
most common prior to delivery can happy 10 days postpartum
magnesium sulfate to prevent future seizure

109
Q

placenta previa

predisposed

A

more common in women: multiparity, 35+, multip, large placenta ( erythroblastosis), previous c section, smoking hx of IUD

110
Q

signs of placenta previa

A

PAINLESS bleeding sudden onset
ultrasound repeat for vag bleeding
5-10% also have placenta accreta

111
Q

complete placenta previa

A

hospitalization with labs:
Rh, indirect Coombs, platelets, DIC. NST, fetal kick
risk of hemorrhage, fetal distress, maternal distress

112
Q

marginal and partial previa delivery

A

can be vaginally , presenting part is like tampon

113
Q

placenta accreta

A

2 or more cesections

114
Q

abruptio placenta

predisposing factors

A

hypertension, preeclampsia, folic acid deficient, ab trauma, short cord, malnutrition, sudden uterine decrease, ROM in polyhydraminos, difficult external version, cocaine usage

115
Q

abruptio placenta

s/s

A

painful LOCALIZED tenderness, hypertonic boardlike uterus, decreased fetal movements, fetal distress, uterine enlargement, maternal shock. back pain, colicky uterus,

116
Q

abruption placenta management

A

period of observation and obtain findings
management is delivery, vaginal if no distress.

emergent csection and immediate transfer
start IV, trendelburg position, vital signs, heart tones, oxygen, warm blankets

117
Q

trendelburg postion

A

supine with feet at 15-30 degree elevation

118
Q

size dates discrepancy definition

A

woman is sure of her dates and LMP, EDD is determined an uterus is smaller or larger for dates
ultrasound and complete intake

119
Q

common reasons for size date descrp

A

3rd trimester- large baby
inaccurate dates
1st 2nd trimester- anomalies, disease, infection. small

120
Q

differential diagnosis of size dates discrepancy

A

multip, IUGR, diabetes, thyroid (smaller), malnutrition, polyhydraminos, oligohydraminos, fetal lie, anomalies, station, hypertension preeclampsia, psychosocial, infections, drugs. placenta previa( larger longitudinal, smaller for tranverse, myomata (larger)

121
Q

myomata

A

fibroids

122
Q

IUG, SGA

A

intrauterine growth restriction
small for gestational age
most SGA is because of IUGR
SGA can also be malnutrion, or genetically predisposed smaller babies

123
Q

symetrical

A

compromised growth of body length head and weight

124
Q

assymetrical

A

compromised growth of body length and weight but head is normal

125
Q

IUGR risk factors

A

preeclampsia
renal disease, poor nutrition, poor weight gain,infection,amomalies, multip, previous IUGR, drug use, pre-pregancy less then 90 lbs, anemia, diabetes, alcohol, hypoglycemia,

126
Q

postmaturity syndrome

A

post dates plus oligiohydraminos, mec stained fluid, Newborn with:
lost of subcutaneous fat, long fingernails, peeling wrinkled skin, alert facies, no lanugo, no vernix