Theoretical Perspectives of Trauma Flashcards
The primary theoretical limitation of early conditioning models and fear reduction of trauma reaction approaches:
The inability of these theories to provide adequate explanations of all the core features of PTSD and the role of responses and emotions other than fear (Brewin & Holmes, 2003; Foa & Kozak, 1986; McGarvey & Collins, 2001).
Contemporary theories also enjoy the advancement of technology that has led to…….. and use the role of neuropsychology to expand…….
To an increase in knowledge in the field of neuropsychology.
Expand our understanding of the normal and abnormal processes involved in trauma exposure
When and who first identified the dual nature of PT symptoms as both physiological and psychological:
Responses to trauma are associated with (3 F’s)
Each of these responses have 3 elements to them.
As early as the 1940s, Kardiner (1941) and other theorists.
Fight, Flight or Freeze
Elements of: physiological/arousal, cognitive and affective.
Physiological Response to Trauma
- purpose of the physiological response
- 2 systems responsible for mediating the process
(and how they act - by releasing what chemicals in brain)
Purpose involves a number of systems that prepare the body to respond (fight, flight or freeze) to the threat and subsequently return the body to homeostasis.
The autonomic nervous system and the hypothalamic-pituitary-adrenal axis. These systems act primarily via the “release of endogenous stress responsive neurohormones including cortisol, epinephrine and norepinephrine, vasopressin, oxytocins and endogenous opioids” (van der Kolk, 1994, p. 255)
Normal physiological response to a stressor in the environment:
- Incoming stimuli are processed in ….. however the …. is responsible for….
- This activity directs activation of …… leading to secretion of ……. (3 hormones) into bloodstream
- This then results in secretion of …… via adrenal medulla into ………. + what does this do to prepare the body
- How does the body react to the last step (characteristics of flight or fight) and how they help the body’s senses
- What happens in the brain (hormone is trasnformed) when step 4 is occuring.. and what it allows the individual to focus on
- What is important for activation of the flight/fight response and for returning the body to homeostasis and how does this occur in the brain
- What are also released into the brain to act as an analgesic - and how does this help a person in a fight or flight situation
- Incoming stimuli are initially processed in the cerebral cortex. However, the amygdala is responsible for the determination of threat and the activation of a series of appropriate neuro-chemical systems
- This activity directs activation of the hypothalamus, leading to the secretion of corticotropin-releasing hormone (CRH) and arginine vasopressin, signalling the anterior pituitary gland to secrete adrenocorticotropic hormone (ACTH) into the bloodstream.
- This then results in the secretion of glucocorticoids (primarily cortisol), epinephrine and norepinephrine (NE) via the adrenal medulla and adrenal cortex. This cascade of neuro-chemical activity activates the body, allowing it to deal with the current threat, known as the flight or fight reaction
- Thus, the body reacts via increased heart rate and respiration, dilation of the pupils, and release of sugar into the blood. All of these functions serve to heighten the body’s senses and access strength and energy in order to deal with the crisis
- Simultaneously, in the brain, adrenalin is transformed into norepinephrine, allowing the individual’s attentional resources to focus narrowly on the threat at hand.
- Glucocorticoids are responsible for the activation of the flight/fight response and for returning the body to homeostasis. This occurs via the delivery of information from the glucocorticoid receptors to the hippocampus, which in turn signals the hypothalamus to stop releasing glucocorticoids
- Endorphins are also released into the brain and act as an analgesic, thus improving the likelihood of survival by ensuring that the individual will not be preoccupied by physical pain
The encoding of the trauma memory and the response is adaptive for future survival; the retrieval of such information allows for…
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Kolb (1987) proposed that encoding trauma information efficiently is compromised when ….
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Kolb suggested that excessive peritraumatic stimulation of the central nervous system might result in….
Increased speed and efficiency of the body in activating emergency systems, should a similar trauma event present again.
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Excessive traumatic stimulation overwhelms the brain’s capacity to process the information, resulting in subsequent PTSD symptoms.
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Posttraumatic neuronal changes
NEUROBIOLOGICAL MODEL OF PTSD
The resultant changes signify a permanent sensory imprint of ____________________ , which imposes an imbalanced degree of __________. This, in turn, results in behavioural reactions of _____ or ____ that are focused on defending against future trauma via the activation or deactivation of biological systems in a bid to secure __________.
The resultant changes signify a permanent sensory imprint of maladaptive processing of the experience, which imposes an imbalanced degree of emotional importance. This, in turn, results in behavioural reactions of activation or restraint that are focused on defending against future trauma via the activation or deactivation of biological systems in a bid to secure homeostasis
NEUROBIOLOGICAL MODEL OF PTSD
According to Sherin and Nemeroff, the lack of baseline cortisol which is consistently reported at the time of trauma may facilitate the ________ of the central CRH-NE cascade. This is thought to result in amplified and prolonged __________.
Inadequate _______ effects of the gamma-aminobutyric acid, neuropeptide Y and serotonin neurotransmitters may further ________ this amplification of _________
It is argued that the PTSD feature of the ________of learned fear to unrelated trauma contexts, as well as ________ in discerning threatening and non-threatening stimuli, are associated with an ________ functioning hippocampus
According to Sherin and Nemeroff, the lack of baseline cortisol which is consistently reported at the time of trauma may facilitate the over-activation of the central CRH-NE cascade. This is thought to result in amplified and prolonged stress responses
Inadequate regulatory effects of the gamma-aminobutyric acid, neuropeptide Y and serotonin neurotransmitters may further exaggerate this amplification of stress responsiveness
It is argued that the PTSD feature of the generalisation of learned fear to unrelated trauma contexts, as well as deficits in discerning threatening and non-threatening stimuli, are associated with an abnormally functioning hippocampus
NEUROBIOLOGICAL MODEL OF PTSD
Common in individuals meeting the ____ diagnostic criteria is abnormal _______ function, as well as overactive _______ response, in combination, these deficits are thought to be in part responsible for paranoia, hyper-vigilance, behavioural activation, exaggerated ______ responses and the further acquisition of ______ associations
Disrupted _____ cortical functions may, in turn, facilitate ______ symptomology due to the inadequate suppression of stress responses and fear associations, as well as extinction
Common in individuals meeting the PTSD diagnostic criteria is abnormal hippocampus function, as well as overactive amygdala response, in combination, these deficits are thought to be in part responsible for paranoia, hyper-vigilance, behavioural activation, exaggerated stress responses and the further acquisition of fear associations
Disrupted prefrontal cortical functions may, in turn, facilitate PTSD symptomology due to the inadequate suppression of stress responses and fear associations, as well as extinction
NEUROBIOLOGICAL MODEL OF PTSD
M____ distrubance is one of the core characteristics of PTSD.
And what does this result in? (Characteristics)
What is the Declarative Memory System
Impairment of the Declarative Memory System is attributed to…. (and a feature of)
Memory disturbance is one of the core clinical characteristics of PTSD. Typically associated with PTSD are intrusive memories, which are generally accompanied by high levels of arousal and behavioural responses, and fragmented or impoverished memory, which is associated with poor encoding and retrieval
General impairment of the declarative memory system (also known as explicit memory) = consists of facts and events that can be explicitly stored and consciously recalled or “declared”.
Impairment of the Declarative Memory System is also a feature of PTSD (van der Kolk & Fisler, 1995), and is attributed to an abnormally functioning hippocampus.
Integrated Model of Foa and Colleagues:
Foa and Kozak proposed what theory? (FNT)
Trauma info biased by extreme peritraumatic emotions would result in ________________________ of the event.
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Meaning of process: Extinction
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Abnormal processes of learning and extinction in people with PTSD may be a consequence of (hormone/chemical)
Accordingly, trauma information that is biased by extreme peritraumatic emotions would arguably result in a fragmented and disorganised memory of the event.
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Extinction = the gradual weakening of a conditioned response that results in the behaviour decreasing or disappearing
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Abnormal processes of learning and extinction observed in individuals with PTSD may be a consequence of altered norepinephrine and stress hormone activity
Integrated Model of Foa and Colleagues:
Norepinephrine enhances the encoding of ____memories.
Glucocorticoids block the retrieval of _______memories.
The combination of elevated noradrenergic activity and relative hypocortisolism may strengthen the ______ of _______ memories, thereby explaining the reduced inhibition of ______ retrieval; both of which are thought to trigger the re-experience phenomenon in PTSD.
Norepinephrine enhances the encoding of fear memories.
Glucocorticoids block the retrieval of emotional memories.
The combination of elevated noradrenergic activity and relative hypocortisolism (low cortisol level) may strengthen the encoding of traumatic memories, thereby explaining the reduced inhibition of memory retrieval; both of which are thought to trigger the re-experience phenomenon in PTSD
Foa and Rothbaum (1998)
As inflexibility of _____ and ______ increases, so too does the individual’s vulnerability to developing ____.
For those who have poor self and worldviews, this vulnerability acts to confirm their _________.
Oppositely: those with positive self and worldviews, the trauma _____ and ______ their previously held beliefs.
Therefore hindering successful _________________ of the trauma information..
Foa and Rothbaum (1998) proposed that, as inflexibility of self and worldviews increases, so too does the individual’s vulnerability to developing PTSD.
For those with poor self and worldviews, this vulnerability occurs because the trauma acts to confirm previously held beliefs.
In contrast, for those with rigid inflexible positive self and worldviews, the trauma contradicts and challenges previously held beliefs. This arguably hinders the successful information processing of the trauma information
In Foa and colleagues expanded and contemporary model:
Greated emphasis placed on ______ appriasals of ____ and _____ during and after trauma.
This includes post trauma symptoms and responses that intensify perceptions of ______.
Model proposes that pre, peri and post traumatic beliefs may interact to strengthen current beliefs of ______ and _____ which are believed to underlie chronic ____.
In Foa and colleagues expanded and contemporary model, greater emphasis is placed on the role of negative appraisals of responses and behaviours during the trauma and after the trauma.
Including post-trauma symptoms and the responses of others that intensify perceptions of incompetence.
For that reason, the model proposes that pre-trauma, peritraumatic and posttraumatic beliefs may interact to strengthen current beliefs of incompetence and danger, which are believed to underlie chronic PTSD
Foa and Kozak (1986) proposed exposure therapy as an effective treatment for PTSD.
This identified benefits of _____
Countering belief that symptoms are _____
+ 4 other things they looked into and changed.
Other benefits include:
- reuduction in global ____ _____
- cohesive memory _____in integration of ____ memory.
- promotion of personal _______
Identifying the benefits of the habituation of fear, countering the belief that symptoms are permanent, prevention of negative reinforcement of the trauma memory, incorporation of safety information, and disconfirmation of negative trauma information allowing for the modification of negative trauma memories.
Other benefits have been found to include a reduction in global danger beliefs, promotion of cohesive memory aiding in the integration of the trauma memory, and promotion of a sense of personal resiliency