The Child with an Electrical Injury or Drowning Flashcards

1
Q

What determines the risk of cardiac arrest in electrical injury?

A

RISK OF CARDIAC ARREST

ELECTRICAL INJURY

  • Size of the current
    • usually occurs in the home - low currents and voltage, few need hospitalisation
    • high mortality + severe tissue damage if from high power / voltage external sources e.g.
      • railway
      • high tension cables
      • lightning strike - massive direct current of short duration depolarises the myocardium and causes immediate asystole
    • AC can cause cardiac arrest at lower voltage than DC (alternating and direct current)
    • water decreases resistance of the skin and increases the amount of current that flows through the body
  • Duration of exposure
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2
Q

Describe how increasing current changes the pathophysiology of electrical injury and the differing effects seen.

A

PATHOPHYSIOLOGY

ELECTRICAL INJURY

INCREASING CURRENT

  • > 10 mA: Tetanic contractions of muscles, child may not be able to let go of electrical source
  • 50 mA: tetanic contraction diaphragm + IC muscles –> resp arrest until current disconnected –> hypoxia –> cardiac arrest
  • > 100 mA - 50 A: primary cardiac arrest
  • 50 A - several 100 A: prolonged resp + cardiac arrest + severe burns
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3
Q

How does electrical resistance affect the pathophysiology of electrical injury?

A

ELECTRICAL INJURY

PATHOPHYSIOLOGY

RESISTANCE

  • current follows the path of least resistance through tissues
  • tissue fluid < blood < muscle < nerve < fat < skin < bone
  • water decreases skin resistance allowing more current to flow through the body
  • electrocution generates heat which causes tissue damage
  • most damage - nerves, blood vessels, skin
  • damaged tissues swell
  • swelling of muscle can cause crush or compartment syndrome - FASCIOTOMY
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4
Q

How does high voltage affect the pathophysiology of electrical injury?

A

PATHOPHYSIOLOGY

HIGH VOLTAGE

ELECTRICAL INJURY

  • e.g. lighting, railways, high-tension cables
  • high voltage can cause
    • high current and severe tissue damage OR
    • FLASH BURNS/ ARCING –> severe superficial burns, no damage to deeper structures
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5
Q

Describe the management of electrical injury.

A

ELECTRICAL INJURY

MANAGEMENT

  • FIRST PRIORITY
    • DISCONNECT ELECTRICAL CURRENT !
    • NB high voltage sources can discharge through several cm’s of air
  • PRIMARY SURVEY AND RESUS
    • A
      • Immobilise C-spine as appropriate
      • Open and secure airway (esp if facial injuries)
    • E
      • determine entry and exit point of current + => possible internal injuries
  • SECONDARY SURVEY AND EMERGENCY Mx
    • associated injuries are common
      • falls
      • thrown from the source
      • burns from the current or burning clothes
        • oedema
        • fluid loss
      • fractures, luxations, muscle tearing (from tetanic contractions)
      • myoglobinuria
        • after sig. muscle damage
        • aim UO > 2 ml/ kg/ hr
        • mannitol (diuretics)
        • fluids
        • sodium bicarbonate to alkalinise the urine - helps excretion
  • STABILISATION AND TRANSFER TO DEFINITIVE CARE
    • consider transfer to burns centre
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6
Q

Define Drowning.

A

DROWNING:

A PROCESS RESULTING IN…

PRIMARY RESP IMPAIRMENT…

FROM SUMERSION/ IMMERSION IN A LIQUID MEDIUM.

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7
Q

Describe the pathophysiology of drowning.

A

PATHOPHYSIOLOGY

DROWNING

  • submersion
  • diving reflex
    • –> bradycardia
    • –> apnoea
  • hypoxia
  • acidosis
    • –> tachycardia
    • –> rise in BP
  • breakpoint (20 secs - 5 mins later)
  • breathing
  • fluid inhaled
  • touches the glottis
  • laryngospasm
  • subsides
  • fluid aspirated into lungs
    • –> alveolitis
    • –> pulm. oedema
  • severe hypoxia
  • LOC
  • +/- bradycardia / dysrrhythmias (VF rare)
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8
Q

What are the main problems arising from drowning that require treatment?

A

DROWNING

THERAPY FOR: B H x 3 I

  • Bradycardia/ dysrrhythmias
  • Hypoxia (main cause of death)
  • Hypothermia
    • Submersion injuries
    • Infants and children more at risk due to large body surface: weight ratio
    • protective against neurological sequelae of hypoxia + ischaemia
    • BUT can cause
      • dysrrhythmias (life threatening)
      • coagulation disorders
      • susceptibility to infections
  • Hypovolaemia
  • Injury esp. spinal
    • common in older children
    • often overlooked
    • C-spine injury
      • always suspect if mechanism of drowning unclear
      • rare - 0.5% overall, more rare if < 5 yo
      • RTA, diving accidents
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9
Q

How does the type of water involved in drowning affect the clinical course?

A

DROWNING

TYPE OF WATER

  • Saltwater or freshwater
    • does not predict clinical course
    • same Mx
  • Severely contaminated water
    • infections (unusual org’s)
  • Contaminated with petroleum products e.g. soap, shampoo
    • ARDS
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10
Q

Describe the primary survey and resuscitation of drowning.

A

DROWNING

PRIMARY SURVEY AND RESUS

<> ABCDEFG

  • FIRST PRIORITY
    • remove victim from water w/o risk to the rescuer
    • rescue in a vertical position - venous pooling + CV collapse
    • do NOT delay the rescue for horizontal rescue/ C-spine immobilisation in water
  • EARLY BLS
    • standard algorithm even in hypothermia
    • Rescue breaths in the water - mouth over nose
    • pulses can be difficult to feel - start compressions if doubt
    • if using AED - DRY THE SKIN FIRST!
  • ALS - slight modifications in HYPOTHERMIA
    • ​ (See E)
  • A
    • Immobilise C-spine
    • high risk of aspiration - swallowed water
    • RSI + ETT
    • –> OGT/ NGT
  • B
    • I+V
    • aim sats 94 - 98%
    • O2 + PEEP
    • BEWARE
      • observe for at least 8 hrs
      • 4-6 hrs later resp deterioration (delay)
  • E - HYPOTHERMIA
    • core temp (rectal/ oesophageal)
    • NB therapeutic hypothermia 32- 34 degrees for at least 24 hrs in children who remain comatose improves neurological outcome
    • Prevent further cooling (initially) - adversely affects resus attempts
      • arrythmias
      • VF may be refractory below 30 degrees
    • Changes to ALS in HYPOTHERMIA
      • < 30 DEGREES
        • limit defibrillation to 3 shocks
        • do NOT give antiarrhythmic/ inotropic drugs
        • IF unsuccessful quickly warm above 30 degrees then re-attempt defibrillation
      • 30 - 25 degrees
        • 2 x dose interval for resus drugs
      • Continue
        • until T at least 32 degrees OR
        • cannot be raised despite active measures
      • Intubate if necessary - benefit > small risk of pptating malignant arrythmias
    • REWARMING
      • SLOW - 0.25 - 0.5 degrees/ hour
      • reduces haemodynamic instability due to vasodilatation (most hypothermic children are hypovolaemic, fast rewarming - vasodilation - hypotension)
      • continuous monitoring of BP
      • treat hypotension with fluids
      • avoid overfilling/ pulm oedema
    • Rewarming strategies
      • External (> 30 degrees)
        • remove cold, wet clothing
        • blankets + heating blankets
        • warm air system
        • infrared radiant lamp
      • Core (< 30 degrees)
        • Warm
          • IV fluids (39 degrees)
          • Ventilator gases (42 degrees)
        • Lavage
          • gastric/ bladder (normal saline at 42 degrees)
          • peritoneal (potassium-free dialysate, 42 degrees, 20 ml/kg, 15 min cycles)
          • pleural
          • pericardial
        • Endovascular warming
        • Extracorporeal blood rewarming
          • ECMO / Bypass
          • best in circulatory arrest
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11
Q

Describe the secondary survey of drowning.

A

DROWNING

SECONDARY SURVEY

  • HEAD TO TOE
  • Looking for injuries that occured before immersion incl. spinal
  • Consider ingestion of alcohol/ drugs esp. older children
  • Ix
    • Bloods
      • Baseline incl. U+E
      • Clotting
      • GAS + GLUCOSE
    • MICRO
      • BCx
      • Aspirate culture
    • Radiology
      • CXR
      • C-spine imaging if appropriate
    • ECG
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12
Q

Describe the emergency management and stabilisation of drowning.

A

DROWNING

EMERGENCY Mx & STABILISATION

  • D
    • brain is most vulnerable to hypoxia
    • cerebral impairment before cardiac after submersion
    • raised ICP
      • post hypoxic injury
      • aggressive Rx does not improve prognosis
    • keep normoglycaemic - important for neuro outcome
  • E
    • prophylactic ABx not helpful
    • give if water severely contaminated - Pseudomonas aeruginosa & Aspergillus
    • fever in first 24 hrs not usually due to infection - develops later
    • IVAB e.g. cefotaxime + repeat sputum/ BCx
  • Indications for admission to ITU
    • respiratory insufficiency
    • haemodynamic instability
    • hypothermia
  • NO - barbiturates, calcium channel blockers, surfactants, steroids, free radical scavengers
  • Search for the precipitating cause of drowning - esp channelopathy e.g. long QT syndrome
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13
Q

What are the prognostic indicators in drowning?

A

DROWNING

PROGNOSTIC INDICATORS

THE RIVER IS GONNA BE COLD

  • Temperature - CORE - low
    • hypoxic brain damage is reduced when the brain cools before the heart stops
    • Improved prognosis - protects vital organs
      • pre-existing hypothermia
      • rapid cooling after submersion
    • Increased survival
      • < 33 on arrival
      • < 10 water
    • more pronounced in small children (larger surface area: WT ratio)
  • Respiratory effort - time to
    • Good prognosis - w/1 3 mins of starting BLS
      • Little - no chance of survival - no resp effort after 40 mins of full CPR - unless resp depressed by
        • hypothermia
        • medication
        • alcohol
  • Immersion time
    • v. small chance of intact neurological recovery/ survival - > 10 mins submersion
  • Gas - poor prognosis if:
    • pH < 7.1 despite Rx
    • pO2 < 8 despite Rx
  • BLS - time to
    • poor prognosis - delay of > 10 mins
    • starting BLS at the scene greatly improves survival
  • Coma - persistent
    • GCS < 5 - BAD

NB - duration of resuscitation efforts is not prognostic

DISCONTINUATION OF RESUS:

  • in hospital - only after consideration of prognostic indicators
  • out of hospital - DO NOT discontinue resuscitation out of hospital UNLESS CLEAR EVIDENCE OF FUTILITY e.g.
    • major trauma
    • rigor mortis
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14
Q

What is the outcome of drowning?

A

OUTCOME OF DROWNING

  • BLS at the scene
    • YES - 70% survival
    • NO - 40% survival, even with maximal Tx
  • Survival after in hospital CPR
    • 70% - complete recovery
    • 25% - mild neuro deficit
    • 5% - severely disable, persistent vegetative state
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