The Child with an Electrical Injury or Drowning Flashcards
What determines the risk of cardiac arrest in electrical injury?
RISK OF CARDIAC ARREST
ELECTRICAL INJURY
-
Size of the current
- usually occurs in the home - low currents and voltage, few need hospitalisation
- high mortality + severe tissue damage if from high power / voltage external sources e.g.
- railway
- high tension cables
- lightning strike - massive direct current of short duration depolarises the myocardium and causes immediate asystole
- AC can cause cardiac arrest at lower voltage than DC (alternating and direct current)
- water decreases resistance of the skin and increases the amount of current that flows through the body
- Duration of exposure
Describe how increasing current changes the pathophysiology of electrical injury and the differing effects seen.
PATHOPHYSIOLOGY
ELECTRICAL INJURY
INCREASING CURRENT
- > 10 mA: Tetanic contractions of muscles, child may not be able to let go of electrical source
- 50 mA: tetanic contraction diaphragm + IC muscles –> resp arrest until current disconnected –> hypoxia –> cardiac arrest
- > 100 mA - 50 A: primary cardiac arrest
- 50 A - several 100 A: prolonged resp + cardiac arrest + severe burns
How does electrical resistance affect the pathophysiology of electrical injury?
ELECTRICAL INJURY
PATHOPHYSIOLOGY
RESISTANCE
- current follows the path of least resistance through tissues
- tissue fluid < blood < muscle < nerve < fat < skin < bone
- water decreases skin resistance allowing more current to flow through the body
- electrocution generates heat which causes tissue damage
- most damage - nerves, blood vessels, skin
- damaged tissues swell
- swelling of muscle can cause crush or compartment syndrome - FASCIOTOMY
How does high voltage affect the pathophysiology of electrical injury?
PATHOPHYSIOLOGY
HIGH VOLTAGE
ELECTRICAL INJURY
- e.g. lighting, railways, high-tension cables
- high voltage can cause
- high current and severe tissue damage OR
- FLASH BURNS/ ARCING –> severe superficial burns, no damage to deeper structures
Describe the management of electrical injury.
ELECTRICAL INJURY
MANAGEMENT
- FIRST PRIORITY
- DISCONNECT ELECTRICAL CURRENT !
- NB high voltage sources can discharge through several cm’s of air
- PRIMARY SURVEY AND RESUS
- A
- Immobilise C-spine as appropriate
- Open and secure airway (esp if facial injuries)
- E
- determine entry and exit point of current + => possible internal injuries
- A
- SECONDARY SURVEY AND EMERGENCY Mx
- associated injuries are common
- falls
- thrown from the source
- burns from the current or burning clothes
- oedema
- fluid loss
- fractures, luxations, muscle tearing (from tetanic contractions)
- myoglobinuria
- after sig. muscle damage
- aim UO > 2 ml/ kg/ hr
- mannitol (diuretics)
- fluids
- sodium bicarbonate to alkalinise the urine - helps excretion
- associated injuries are common
- STABILISATION AND TRANSFER TO DEFINITIVE CARE
- consider transfer to burns centre
Define Drowning.
DROWNING:
A PROCESS RESULTING IN…
PRIMARY RESP IMPAIRMENT…
FROM SUMERSION/ IMMERSION IN A LIQUID MEDIUM.
Describe the pathophysiology of drowning.
PATHOPHYSIOLOGY
DROWNING
- submersion
- diving reflex
- –> bradycardia
- –> apnoea
- hypoxia
- acidosis
- –> tachycardia
- –> rise in BP
- breakpoint (20 secs - 5 mins later)
- breathing
- fluid inhaled
- touches the glottis
- laryngospasm
- subsides
- fluid aspirated into lungs
- –> alveolitis
- –> pulm. oedema
- severe hypoxia
- LOC
- +/- bradycardia / dysrrhythmias (VF rare)
What are the main problems arising from drowning that require treatment?
DROWNING
THERAPY FOR: B H x 3 I
- Bradycardia/ dysrrhythmias
- Hypoxia (main cause of death)
- Hypothermia
- Submersion injuries
- Infants and children more at risk due to large body surface: weight ratio
- protective against neurological sequelae of hypoxia + ischaemia
- BUT can cause
- dysrrhythmias (life threatening)
- coagulation disorders
- susceptibility to infections
- Hypovolaemia
- Injury esp. spinal
- common in older children
- often overlooked
- C-spine injury
- always suspect if mechanism of drowning unclear
- rare - 0.5% overall, more rare if < 5 yo
- RTA, diving accidents
How does the type of water involved in drowning affect the clinical course?
DROWNING
TYPE OF WATER
- Saltwater or freshwater
- does not predict clinical course
- same Mx
- Severely contaminated water
- infections (unusual org’s)
- Contaminated with petroleum products e.g. soap, shampoo
- ARDS
Describe the primary survey and resuscitation of drowning.
DROWNING
PRIMARY SURVEY AND RESUS
<> ABCDEFG
- FIRST PRIORITY
- remove victim from water w/o risk to the rescuer
- rescue in a vertical position - venous pooling + CV collapse
- do NOT delay the rescue for horizontal rescue/ C-spine immobilisation in water
- EARLY BLS
- standard algorithm even in hypothermia
- Rescue breaths in the water - mouth over nose
- pulses can be difficult to feel - start compressions if doubt
- if using AED - DRY THE SKIN FIRST!
- ALS - slight modifications in HYPOTHERMIA
- (See E)
- A
- Immobilise C-spine
- high risk of aspiration - swallowed water
- RSI + ETT
- –> OGT/ NGT
- B
- I+V
- aim sats 94 - 98%
- O2 + PEEP
- BEWARE
- observe for at least 8 hrs
- 4-6 hrs later resp deterioration (delay)
- E - HYPOTHERMIA
- core temp (rectal/ oesophageal)
- NB therapeutic hypothermia 32- 34 degrees for at least 24 hrs in children who remain comatose improves neurological outcome
- Prevent further cooling (initially) - adversely affects resus attempts
- arrythmias
- VF may be refractory below 30 degrees
- Changes to ALS in HYPOTHERMIA
- < 30 DEGREES
- limit defibrillation to 3 shocks
- do NOT give antiarrhythmic/ inotropic drugs
- IF unsuccessful quickly warm above 30 degrees then re-attempt defibrillation
- 30 - 25 degrees
- 2 x dose interval for resus drugs
- Continue
- until T at least 32 degrees OR
- cannot be raised despite active measures
- Intubate if necessary - benefit > small risk of pptating malignant arrythmias
- < 30 DEGREES
- REWARMING
- SLOW - 0.25 - 0.5 degrees/ hour
- reduces haemodynamic instability due to vasodilatation (most hypothermic children are hypovolaemic, fast rewarming - vasodilation - hypotension)
- continuous monitoring of BP
- treat hypotension with fluids
- avoid overfilling/ pulm oedema
- Rewarming strategies
- External (> 30 degrees)
- remove cold, wet clothing
- blankets + heating blankets
- warm air system
- infrared radiant lamp
- Core (< 30 degrees)
- Warm
- IV fluids (39 degrees)
- Ventilator gases (42 degrees)
- Lavage
- gastric/ bladder (normal saline at 42 degrees)
- peritoneal (potassium-free dialysate, 42 degrees, 20 ml/kg, 15 min cycles)
- pleural
- pericardial
- Endovascular warming
- Extracorporeal blood rewarming
- ECMO / Bypass
- best in circulatory arrest
- Warm
- External (> 30 degrees)
Describe the secondary survey of drowning.
DROWNING
SECONDARY SURVEY
- HEAD TO TOE
- Looking for injuries that occured before immersion incl. spinal
- Consider ingestion of alcohol/ drugs esp. older children
- Ix
- Bloods
- Baseline incl. U+E
- Clotting
- GAS + GLUCOSE
- MICRO
- BCx
- Aspirate culture
- Radiology
- CXR
- C-spine imaging if appropriate
- ECG
- Bloods
Describe the emergency management and stabilisation of drowning.
DROWNING
EMERGENCY Mx & STABILISATION
- D
- brain is most vulnerable to hypoxia
- cerebral impairment before cardiac after submersion
- raised ICP
- post hypoxic injury
- aggressive Rx does not improve prognosis
- keep normoglycaemic - important for neuro outcome
- E
- prophylactic ABx not helpful
- give if water severely contaminated - Pseudomonas aeruginosa & Aspergillus
- fever in first 24 hrs not usually due to infection - develops later
- IVAB e.g. cefotaxime + repeat sputum/ BCx
- Indications for admission to ITU
- respiratory insufficiency
- haemodynamic instability
- hypothermia
- NO - barbiturates, calcium channel blockers, surfactants, steroids, free radical scavengers
- Search for the precipitating cause of drowning - esp channelopathy e.g. long QT syndrome
What are the prognostic indicators in drowning?
DROWNING
PROGNOSTIC INDICATORS
THE RIVER IS GONNA BE COLD
- Temperature - CORE - low
- hypoxic brain damage is reduced when the brain cools before the heart stops
- Improved prognosis - protects vital organs
- pre-existing hypothermia
- rapid cooling after submersion
- Increased survival
- < 33 on arrival
- < 10 water
- more pronounced in small children (larger surface area: WT ratio)
- Respiratory effort - time to
- Good prognosis - w/1 3 mins of starting BLS
- Little - no chance of survival - no resp effort after 40 mins of full CPR - unless resp depressed by
- hypothermia
- medication
- alcohol
- Little - no chance of survival - no resp effort after 40 mins of full CPR - unless resp depressed by
- Good prognosis - w/1 3 mins of starting BLS
- Immersion time
- v. small chance of intact neurological recovery/ survival - > 10 mins submersion
- Gas - poor prognosis if:
- pH < 7.1 despite Rx
- pO2 < 8 despite Rx
- BLS - time to
- poor prognosis - delay of > 10 mins
- starting BLS at the scene greatly improves survival
- Coma - persistent
- GCS < 5 - BAD
NB - duration of resuscitation efforts is not prognostic
DISCONTINUATION OF RESUS:
- in hospital - only after consideration of prognostic indicators
- out of hospital - DO NOT discontinue resuscitation out of hospital UNLESS CLEAR EVIDENCE OF FUTILITY e.g.
- major trauma
- rigor mortis
What is the outcome of drowning?
OUTCOME OF DROWNING
- BLS at the scene
- YES - 70% survival
- NO - 40% survival, even with maximal Tx
- Survival after in hospital CPR
- 70% - complete recovery
- 25% - mild neuro deficit
- 5% - severely disable, persistent vegetative state