The Basics of an Eye Exam Flashcards
Direct Pupillary Examination
Constriction of ipsilateral eye
Consensual Pupillary Examination
Constriction of contralateral eye
Synchia
The pupil can become adhered to the front of the lens or to the endothelium
This can cause a secondary glaucoma and create an irregular pupil
Leukocoria
White Pupil - red reflex with camera flash
R/O : Cataract or Retinoblastoma
Epiphora
Watery eye… weepy
Insufficient drainage
Lid position / blocked drainage/ age / trauma / scar
Ocular motility
6 cardinal fields (H shaped pattern)
6 Cardinal fields (H shaped)
Inspection for:
1. Normal conjugate, or parallel movements of eyes (deviation)
2. Abnormal movement (nystagmus…rhythmic fine ascillation)
3. Lid lag
Convergence………sustained to within 5-8 cm
(watch for eye to Trope)
arcus senilis
white ring around eye, normal in elderly, think cholesterol issues in young population
Anesthetics uses
Anesthetize cornea within 15 sec, last 10 mins
Remove corneal foreign bodies
Perform tonometry
Examine damaged corneal surface
cholinergic-blocking ( parasympatholytic) Pupil Dilation
Dilate by paralyzing iris sphincter muscle
Cycloplegia by paralyzing ciliary body muscles
Tropicamide Max pupil dilatation 30 min Effect diminishes 4-5 hrs Side effects: Rare Nausea / vomiting Pallor Vasomotor collapse
Adrenergic-stimulating (sympathomimetic) Pupil Dilation
Phenylephrine 2.5% 10%
Dilates in 30 mins, no effect on accommodation
Pupil remains reactive to light
Combine with Tropicamide for maximal dilatation
Infants combine Cyclopentolate 0.2% & Phenylephrine 1%
Side effects:
acute hypertension or MI (with 10%)
Optic neuritis
idiopathic or associated with multiple sclerosis young adults decreased visual acuity and color vision pain with ocular movement bulbar (disc swelling) retro bulbar (you both see nothing)
Central Retinal Artery Occlusion (CRAO)
True ophthalmic emergency!
Sudden painless and often severe visual loss
Permanent damage to the ganglion cells caused by prolonged interruption of retinal arterial blood flow
Characteristic “ cherry-red spot ”
No optic disc swelling unless there is ophthalmic or carotid artery occlusion
Months later, pale disc due to death of ganglion cells and their axons
Branch Retinal Artery Occlusion (BRAO)
Sector of the retina is opacified and vision is partially lost
Most often due to embolus
Treat as CRAO
Retinal Emboli
Often called a Hollenhorst Plaque
Urgent Situations
Penetrating injuries of the globe
Conjunctival or corneal foreign bodies
Hyphema (blood in front of eye)
Lid laceration (suture if not deep and neither the lid margin nor the canaliculi are involved)
Traumatic optic neuropathy
Radiant energy burns (snow blindness or welder’s burn)
Corneal abrasion
Sudden loss of vison
“Curtains” blocking all or some of Vision