Test Three Flashcards

1
Q

What classifies a superficial burn (first degree)?

A

epidermal tissue only affected, erythema, blanchable, mild swelling, no vesicles or blister initially (sunburn), pink to red, mild edema, pain, no eschar

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2
Q

What classifies as a deep/partial burn (second degree)?

A

involves epidermis and deep dermis layers

  • blisters
  • hospitalization if over 25% of body
  • pink to red, mild to moderate edema, painful, no eschar
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3
Q

What classifies a full thickness burn (3rd and 4th degree)?

A
  • destruction of all skin layers
  • Dry, waxy white, leathery, or hard skin, no pain
  • requires immediate hospitalization
  • black, brown, yellow, edema, eschar
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4
Q

Why should you check creatinine in burn pts?

A

Because they are dehydrated, WBCs and inflammation are hard on the kidneys
-decreased renal blood flow (shock), UO decreased first 48 hours, check BUN, creatinine, and Na

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5
Q

What happens to the pts metabolism from a burn injury?

A

hypermetabolism (high catabolism), increased oxygen use and high calorie needs
-increased secretion of catecholamines, ADH, aldosterone, and cortisol due to stress response

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6
Q

What happens to glucose levels in burn injuries?

A

increase as a result of the stress response and altered uptake across injured tissues

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7
Q

Why is ROM important with burn pts?

A

to avoid contractures

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8
Q

What makes septic shock different from other types of shock?

A
lactic acid (b/c metabolism is high so cells metabolize anerobically bc O2 is used up)
-also temperature
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9
Q

Why do vessels get leaky in shock?

A

-widespread vasodilation
leaky vessels leak electrolytes into interstitial fluids and you can’t check those values (electrolyte abnormalities and edema)

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10
Q

What are the problems/complications with thoracic level spinal cord injuries? (6)

A

loss of movement of the chest, trunk, bowel, bladder and legs (depending on the level of injury), leg paralysis, autonomic dysreflexia due to distended bladder or fecal impaction

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11
Q

What is autonomic dysreflexia?

A

occurs with lesions or injuries above T6, caused by visceral distention from a distended bladder or impacted rectum

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12
Q

What are the symptoms of autonomic dysreflexia?

A

sudden HA, severe hypertension and bradycardia, flushing above the level of injury, pale extremities below level of injury, nausea, nasal stuffiness, dilated pupils, blurred vision, sweating, goose bumps, restlessness

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13
Q

What are the 4 stages in MS?

A

Relapsing-remitting
Primary progressive
Secondary progressive
Progressive-relapsing

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14
Q

Which MS stage is this?

occurs in most cases of MS, symptoms develop and resolve in a few weeks to months and the pt returns to baseline

A

Relapsing-remitting

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15
Q

Which MS stage is this?
involves a steady and gradual neurologic deterioration without remission of symptoms. Progressive disability with no acute attacks

A

Primary progressive

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16
Q

Which MS stage is this?

begins with relapsing-remitting course that later becomes steadily progressive.

A

Secondary progressive

17
Q

Which MS stage is this?
frequent relapses with partial recovery but not a return to baseline (rare). Progressive, cumulative symptoms and deterioration occur over several years

A

Progressive-relapsing

18
Q

Early onset MS signs

A

muscle weakness, spasticity, fatigue stiffness (legs), flexor spasms, hyper DTR, positive babinski, unsteady gait, intension tremor, clumsy, poor coordination, tinnitis, vertigo, hearing loss, slurred speech, blurred vision, diplopsia, nystagmus, ataxia, dysphagia, decreased perception to sensory

19
Q

Treatment for Gullian-Barre

A

treatment of symptoms (pain management), monitor respiratory status, respiratory treatments, monitor cardiac status

  • Plasmapheresis: to remove offending antibodies
  • IV immunoglobulin infusions: to suppress immune response
  • Supportive care: ventilator support
20
Q

How do you diagnose Guillian-Barre?

A

there is no single test but physical exam (cranial nerves mainly VII and reflexes), lumbar puncture, blood test, tensilon test, electromyopgraphy study, MRI/CT, and ABGs

21
Q

What causes “oozing”?

A

Severe hypovolemic shock occurs as a result of an inability of the blood to clot because the platelets and clotting factors were consumed earlier. Capillary leak continues and increases the risk for hemmorhage

22
Q

Myasthenia Gravis – what part of the body is affected?

A

-Enlarged thymus gland

Respiratory muscles. When pt. can’t take a deep breath/positioning, suction

23
Q

What would you look for when a patient comes in and a stroke is suspected?

A

hemiplegia, unilateral neglect, impaired speech, change in LOC, abnormal cranial nerves V, VII, IX, X, and XII, nuchal rigidity, ataxia, paralysis

24
Q

What is plavix?

Adverse effects?

A

an antiplatelet drug (decreases risk for stroke/MI)

-GI bleed, rash, neutropenia, petechiae,