Test 3 Flashcards
nursing interventions for fluid volume excess
Monitor I&O, daly weight, edema, crackles and serum electrolyte levels, LOC
nursing interventions imbalanced nutrition- less than required
monitor I&O, daily weight, BUN, anorexia, nausea, committing, dietary restrictions, oral mucosa, serum electrolytes, serum protein and albumin levels
nursing interventions risk for ineffective renal
monitor I&O, daily weight, BUN and creatinine, serum electrolytes, uremia
nursing interventions risk for decreased cardiac output
monitor HR and ECG changes, peripheral pulses, serum electrolyte levels, Arterial blood gas analysis, BP, LOC
activity intolerance nursing interventions
monitor for fatigue, serum electrolytes, H& H, BUN, creatinine
normal BUN and creatinine
BUN 8-20
CR 0.6-1.2
if these are going down- pt is improving
hyperkalemia EKG
Tall, peaked T wave, prolonged QRS
bradycardia, conduction blocks, v fib, muscle weakness
hypokalemia EKG
prominent U wave, ST depression
PVCs, Vtach, muscle weakness
priority intervention hypok
respirations- muscle weakness increases the WOB
suspect HF- diagnostics?
Echo, cardiac cath, x ray
echo is gold standard for DX
hyperphosphatemia
AKI can reduce excretion of phosphorous causing too much- give aluminum hydroxide to reduce phosphate.
should transfer to a burn center
partial thickness >10%, burn on face, hands, feet, genital/perineum or major joints, third degree, electrical, chemical or inhalation, complicated by comorbidity or special needs, children in hospital ill equipped for burn
Baxters formula
to estimate fluid replacement post burn 4 X kg X %burned. 1/2 within 8 hrs of burn 1/2 over next 16 hours Goal is min of 0.5 mL/kg/hr UO
NMS like S&M
Get hot, stiff, and sweaty, BP, HR, RR all increase and you start to drool
in heart failure BNP is?
greater than 100
Administering potassium chloride
IV or PO. Never IVP, highly vesicant. monitor IV site hourly for phlebitis or infiltration. infiltration=necrosis. never give faster than 10mEq/hr, always dilute and mix well.
Priority assessment for left sided HF
Respiratory! Fluid is backing up into the lungs and are fatigued
Heart Failure treatment all kinds
treat underlying cause, circulatory assist decides, daily weights, sodium and fluid restriction, monitor I&O, monitor electrolytes, drug therapy (diuretics, ACE Inhib, vasodilators, Beta blockers, positive inatropes, morphine, possibly antidysrythmias and or anticoagulants)
ADHF treatment
High fowler’s position, oxygen by NC or mask, biped, continuous ECG, pulse ox, and hemodynamic monitoring, conserve energy, maybe intubation mechanical ventilation and or cardio version, ultrafiltration (UF-removes sodium and water from circulation)
Hypokalemia- ekg, priority assessment
prominent U wave, ST depression
PVCs, VTach risk
priority assess respiratory- muscle weakness
Hyperkalemia ekg
Tall peaked T wave, prolonged QRS
conduction blocks, fib
bradycardia, numb/tingling around mouth
diagnostics for AKI
BUN, creatinine, UO, urine osmolality, urine sodium, and specific gravity. serum electrolytes, renal ultrasounds, renal scan, CT scan
Prerenal AKI
lack of adequate perfusion to kidneys could be from hypotension, dehydration, bradycardia, hypovolemic shock. the kidney is okay and just need to give fluids. if is prolonged, can lead to damage- intrarenal AKI
intrarenal AKI (intrinsic?)
poor perfusion, toxins, uncontrolled HTN or DM has damaged the kidneys. amitriptyline is nephrotoxic, NSAIDS, antibiotics… most common cause acute tubular necrosis. monitor LOC, respiratory (crackles?), weight and give aluminum hydroxide fro bind with phosphorus
post renal aki
urine is blocked and can’t leave kidneys, enlarged prostate, kidney stones, tumor, edematous stomach
clinical manifestations of aki
weakness and fatigue from disturbed Na K exchange, diminished deep tendon reflexes, bradycardia (muscle weakness)
treatment aki
insulin, calcium gluconate to reduce risk of v-fib while potassium high, kayexelate if pt can handle it
hypokalemia sxs
weakness and fatigue, muscle cramps, palpitations, arrhythmias, deliriums, magnesium, and potassium will be low together, dysrhythmias,
causes of high creatinine
could be from medications- cimetidine, trimethoprim
and not kidney damage
increases with more than 5% of nephrons damaged
IV fluids and Na and K
IV fluids will force Na and K to switch places
sodium is extracellular and K is intracellular