Test 3 Flashcards

0
Q

Secondary wound healing process

A

Open wound, granulation fills in wound bed, epithelial cells finish the job.

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1
Q

Primary wound healing process

A

Surgical, allows epithelial cells to migrate and repair tissue. Superficial.

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2
Q

Tertiary wound healing process

A

Open a while before it closes. This occurs to get rid of infection. Allow to heal from secondary first.

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3
Q

Phases of healing of wound

A

Hemostasis.
Inflammatory.
Proliferation.
Maturation.

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4
Q

Hemostasis phase of healing

A

Platelet helps clot blood. Vasoconstriction.

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5
Q

Inflammatory phase of wound

A

Vasodilation. Swelling, increased pain around tissue, increased WBC to fight infection.

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6
Q

Maceration of a wound

A

Water logged edges

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7
Q

Tunneling of wound

A

Passageway under intact skin with an end point

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8
Q

Undermining of a wound

A

Extends under skin or superficial tissue

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9
Q

Stage 1 pressure ulcer/wound

A

Skin in tact but doesn’t Blanche

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10
Q

Stage 2 pressure ulcer/wound

A

Partial thickness. It opens here, blister

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11
Q

Stage 3 pressure ulcer/wound

A

Full thickness skin loss, goes into dermis

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12
Q

Stage 4 pressure ulcer/wound

A

Full thickness, tissue loss. May see bone or muscle.

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13
Q

Stage 5 pressure ulcer/wound

A

Unstageable. Can’t determine depth.

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14
Q

Stage 6 pressure ulcer/wound

A

Deep bruising. The skin is intact so u can’t see how far it goes

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15
Q

4 phases of wound healing

A

Hemostasis
Inflammatory
Proliferation
Remodeling

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16
Q

Hemostasis phase of wound healing

A

Platelets clot blood due to vasoconstriction

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17
Q

Inflammatory phase if wound healing

A

Swelling, increased pain around tissue, elevated WBC to fight infection. Vasodilation

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18
Q

Proliferation phase of wound healing

A

Surface wound-epithelial

Deeper-granulation then epithelial

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19
Q

3 processes of wound healing

A

Primary
Secondary
Tertiary

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20
Q

Primary process of wound healing

A

Surgical, allow epithelial cells to repair superficial tissue

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21
Q

Secondary process of wound healing

A

Open wound, granulation tissue fills in, then epithelial

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22
Q

Tertiary process of wound healing

A

Wound is open a while before it closes to get rid of infection

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23
Q

Lab tests for nutrition

A

Hbg
Prealbumin
Esr
Crp

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24
Q

Esr lab shows what for nutrition

A

Response to treatment

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25
Q

Crp lab shows what for nutrition

A

Inflammation, levels should decrease if levels are improving

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26
Q

Mg values

A

1.3-2.3

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27
Q

Ca levels

A

8-10

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28
Q

K+ levels

A

3-5

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29
Q

NA+ levels

A

135-145

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30
Q

pH levels

A

7.35-7.45

31
Q

Causes of metabolic acidosis

A

Dka
Severe diarrhea
Renal failure
Shock

32
Q

Causes of metabolic alkalosis

A

Severe vomiting
Excessive gi auctioning
Diuretic
Excessive NaHCO3

33
Q

Causes of respiratory acidosis

A

Problems with exhaling:

Copd, anesthesia, pneumonia, atelextasis, lung injury

34
Q

Causes of respiratory alkalosis

A

Hyperventilation:

Anxiety. Fear

35
Q

Cardiovascular problems with immobility

A

Decreased cardiac capacity.
Ortho static hypotension
Thrombus formation

36
Q

Respiratory symptoms for immobility

A

Decreased lung expansion, gas exchange

Pooling of secretions

37
Q

Musculoskeletal symptoms of immobility

A

Decreased muscle mass
Atrophy
Contractures

38
Q

Integumentary symptoms for immobility

A

Tissue hypoexmia

Pressure ulcers

39
Q

Urinary symptoms for immobility

A

Infection

Urinary stasis

40
Q

Negative nitrogen balance symptoms of immobility

A

Wt loss

Decreased muscle mass

41
Q

Gi symptoms of immobility

A

Constipation

Decreased peristalsis

42
Q

Psychologic symptoms of immobility

A

Mood disturbances

43
Q

Metabolic interventions for immobility

A

Increase protein, calories

Supplement with B and C vit’s

44
Q

Respiratory interventions for immobility

A

Expand lungs

Mobilize secretions

45
Q

Cardiovascular interventions for immobility

A

Prevent thrombus formation
Increase fluids
Change positions
Decrease cardiac workload

46
Q

Musculoskeletal interventions for immobility

A

Exercise

Passive/active rom

47
Q

Integumentary interventions of immobility

A

Change positions
Skin care
Trochanter roll

48
Q

Fat soluble vitamins

A

A
D
E
K

49
Q

Water soluble vitamins

A

B

C complex

50
Q

Reasons for npo

A

Rest gi tract
Prevent aspiration
Treat n/v
Let gi function normally after sx

51
Q

Interventions for pt with dysphasia

A

Rest prior to eating

HOB 90 degrees

52
Q

Normal oral intake

A

1100-1400 ml

53
Q

Normal output

A

2200-2700 ml

54
Q

How much i/o is needed to maintain f/e

A

2600 ml

55
Q

Obligatory urine output and amount

A

What u need to rid body of toxins

400-600 ml

56
Q

Output exceeds intake/absorption in these conditions

A

Hypernatremia

Hypokalemia

57
Q

Output is less than intake ith these conditions

A

Hyponatremia

Hypercalcemia

58
Q

Colloid pressure

A

Push fluid into compartments

59
Q

Hydrostatic pressure

A

Push fluid out of compartment

60
Q

Osmolality causes what mechanism to react

A

Thirst

61
Q

Increased Hypernatremia cause cells to

A

Shrivel

62
Q

Hyponatremia cause cells to

A

Cells swell

63
Q

Which hormone goes with which electrolyte
Aldosterone
Aldosterone/natriuretic peptides
ADH

A

Increased K+ excretion
Regulate Na+ excretion
H2O excretion

64
Q

Hyponatremia

How to fix

A

Na+ with NS/LR
Lower diuretic dose
Fluid restriction 800 ml-hr

65
Q

Hypernatremia fix

A

Nutrition.

Increase water decrease salt

66
Q

Hypokalemia fix

A

K+ replacement

Increase meats veggies fruits

67
Q

Hyperkalemia fix

A

KayeXalate to increase K+ excretion

68
Q

Hyponatremia symptoms
If hypovolemia
Hypervolemia

A

Mental changes
Combative
Hypovolemia=tachy
Hyper=Brady

69
Q

Hypovolemia

A

Dehydrated

70
Q

Hypervolemia

A

Fluid overload

71
Q

Hypernatremia symptoms

A

Mental changes

72
Q

Hypocalcemia fix

A

Calcium replacement with V-D

D5W fluids

73
Q

Hypercalcemia fix

A

NS with LASIK

74
Q

Hypokalemia symptoms

A

Prolonged QT waves
Dysthymias
At risk for paralytic ileus

75
Q

Hyperkalemia symptoms

A

Peaked T waves