Test Flashcards
CLINICAL HISTORY:
32yo Female
Dyspnoea
FINDINGS:
Bilateral reticulonodular shadowing with diffuse cystic changes.
Lung volumes are preserved.
No pleural abnormalities demonstrated.
Heart size is normal.
Hila and mediastinal contours are normal.
IMPRESSION:
Lymphangioleiomyomatosis
- BACKGROUND INFORMATION:*
- Almost exclusively present in females*
- May present with spontaneous pneumothorax*
- Can be associated with chylothorax*
- May be independent or part of a greater tuberous sclerosis complex*
- Can be associated with abdominal angiomyolipomas.*
DIFFERENTIAL DIAGNOSIS:
Langerhans Cell Histiocytosis (Usually has a smoking history and present in an older age group)
Overlapping emphysematous and granulomatous disease
FOLLOW UP:
Correlation with smoking history
HRCT
Respiratory referral
Bronchial biopsy / lavage
Serum ACE to r/o sarcoidosis
Genetic testing for TSC
U/S abdomen for AMLs
CLINICAL HISTORY:
75yo Male
Right shoulder pain and paraesthesia
FINDINGS:
A large mass is visualised at the right lung apex.
The left lung is clear.
No pleural abnormality is demonstrated.
Skeletal review is unremarkable, no evidence of rib destruction associated with the mass.
Heart size is normal.
IMPRESSION:
Right apical mass in keeping with Pancoast tumour.
BACKGROUND INFORMATION:
Primary bronchogenic malignancy which presents in the lung apex and invades the surrounding soft tissues. This may result in Pancoast syndrome (malignant invasion of the brachial plexus) in roughly 25% of patients.
DIFFERENTIAL DIAGNOSIS:
- Pulmonary metastasis
- Primary pleural malignancy i.e. mesothelioma
- Chest wall tumour i.e. Ewing’s Sarcoma or PNET
- Pleuro-parenchymal fibrosis i.e. from previous Tuberculosis
FOLLOW UP:
- Respiratory referral
- CT CAP for staging
- Consideration of MRI if neurological symptoms
- Consideration of biopsy
CLINICAL HISTORY:
62yo Male
Pyrexia and cough, ?HAP
FINDINGS:
Extensive airspace shadowing in the right upper lobe with inferior bulging of the transverse fissure. There is evidence of internal cavitation. The left lung is clear.
Small right sided pleural effusion.
Heart size is normal. Normal hilar appearances, no evidence of lymphadenopathy. Mediastinal contours are normal.
IMPRESSION:
Cavitating right upper lobe pneumonia, presence of bulging transverse fissure could suggest Klebsiella as a causative organism.
BACKGROUND INFORMATION:
Klebsiella pneumonia occurs from infection with the klebsiella pneumoniae organism. It is a common gram-negative respiratory infection. If cavitation occurs it happens early in the disease process and progresses quickly. Cavitation is much more common than in pneumococcal infections.
DIFFERENTIAL DIAGNOSIS:
- Mycobacterial infection i.e. Primary Tuberculosis (cavitation comparatively uncommon and usually presents with ipsilateral hilar lymphadenopathy)
- Other necrotising lobar pneumonias
FOLLOW UP:
- Sputum culture to determine organism
- Respiratory referral +/- CT
CLINICAL HISTORY:
New cough, ?NG tube position
FINDINGS:
Multiple dense radiopacities demonstrated in both lung bases suggesting aspiration of barium sulphate, I note that a barium swallow was performed yesterday.
Lungs are otherwise clear, no pleural abnormalities.
NG tube is correctly sited, safe for use.
Heart size is normal, hila and mediastinal contours are normal.
IMPRESSION:
Aspiration of barium sulphate
BACKGROUND:
Some patients may aspirate on barium sulphate during GI radiological investigations. If a patient is pre-determined as an aspiration risk then a non-ionic iodinated contrast study is usually performed.
DIFFERENTIAL DIAGNOSIS:
None
FOLLOW UP:
If new aspiration (as in this scenario) patient would benefit from chest physiotherapy assessment
CLINICAL HISTORY:
Worsening SOB
Weight loss
FINDINGS:
Prominent pleural shadowing throughout the right hemithorax. The left lung and pleura are clear.
Heart size is normal, hila and mediastinal contours are normal.
IMPRESSION:
Appearances are concerning for mesothelioma.
BACKGROUND:
Uncommon primary malignacy which is pleural in the vast majority of cases. It has a strong association with occupational asbestos exposure (predominantly crocidolite).
DIFFERENTIAL DIAGNOSIS:
- Loculated pleural effusion i.e empyema
- Pleural metastases
- Pleural plaques
FOLLOW UP:
- Respiratory referral, assuming appearances are not already known
- Pleural phase CT CAP
CLINICAL HISTORY:
76yo Male
Longstanding cough
FINDINGS:
Bilateral reticular interstitial shadowing with a lower zone predominance. Large calcified pleural plaque on the left side.
Heart size is normal, hilar and mediastinal contours are normal.
IMPRESSION:
Appearances are in suggestive of asbestosis
BACKGROUND:
Asbestosis represents the formation of interstitial lung disease after exposure to asbestos. Presents with a predominantly male distribution due to historic occupational exposure.
DIFFERENTIAL DIAGNOSIS:
- Other interstitial lung diseases (calcified pleural plaques are fairly specific to asbestos exposure).
FOLLOW-UP:
- None if appearances unchanged
- HRCT and respiratory referral if findings not previously characterised
CLINICAL HISTORY:
23yo Female
SOBOE
FINDINGS:
Slight right sided volume loss with a tapering vertical curvilinear opacity in the right lung base, running parallel to the heart border.
Chest appearances are otherwise normal.
IMPRESSION:
Appearances are in keeping with scimitar syndrome.
BACKGROUND:
Scimitar syndrome represents a combination of pulmonary hypoplasia and partial anomalous pulmonary venous return. In the vast majority of cases it is right sided. It may be associated with congenital cardiac abnormalities.
DIFFERENTIAL DIAGNOSIS:
- Pulmonary sequestration
- Right middle lobe atelectasis
- Unilateral absence of pulmonary artery
FOLLOW UP:
Cardiology referral
Consideration of further imaging with ECHO or CT
CLINICAL HISTORY:
Significant dyspepsia
FINDINGS:
The oesophagus is grossly dilated with an air-fluid level visualised in the upper mediastinum. Gastric band is seen in the left upper quadrant.
Heart size is normal, normal hilar appearances.
Lungs are clear, no pleural abnormalities.
IMPRESSION:
Megaoesophagus, presumably related to the visualised gastric band.
BACKGROUND:
Diffuse dilation of the oesophagus which may be related to dysmotility or an obstructive cause.
DIFFERENTIAL DIAGNOSIS:
- Ivor Lewis procedure
FOLLOW UP:
- Upper GI opinion / referral
- Consideration of fluoroscopic assessment
CLINICAL HISTORY:
36yo Male
Longstanding cough, increasing breathlessness.
No history of smoking
FINDINGS:
Lungs are hyperinflated with visible bullae formation in both lower zones suggesting severe emphysematous change. No pleural abnormalities demonstrated.
Heart size is normal. Normal appearances of the right hilum, the left hilum is bulky but unchanged from previous imaging 10 years ago. Mediastinal contours are normal.
IMPRESSION:
Severe emphysematous disease. Considering patient age consideration should be made to alpha-1-antitrypsin deficiency.
BACKGROUND:
Reduced or absent levels of A1AT results in unchecked activity of neutrophil elastase. This causes the elastase in respiratory structures to be degraded, this is typically more severe in the lower zones due to gravitational blood flow. Pathogenesis may also result in cirrhosis.
DIFFERENTIAL DIAGNOSIS:
- COPD - Consideration should be made to patient age range. True COPD should not present severely in young patients. Additionally COPD usually presents with an upper lobe predominance.
- Ritalin (Methylphenidate) lung - Clinical correlation with history of drug use needed.
FOLLOW UP:
- Respiratory referral if new findings
- Consider HRCT
- Consider U/S abdomen if co-existent cirrhosis suspected
- Blood test for A1AT
CLINICAL HISTORY:
30yo Female
Lethargy
FINDINGS:
Anterior mediastinal mass, distension of both paratracheal lines is suggestive of massive lymphadenopathy.
Heart size is normal, the hila are difficult to assess but appear normal.
Lungs are clear, no pleural abnormalities.
IMPRESSION:
Anterior mediastinal mass suggestive of massive lymphadenopathy.
Consideration should be made to lymphoma but further imaging and laboratory testing is required.
(Hodgkin Lymphoma confirmed on biopsy)
BACKGROUND:
Lymphoma is a primary lymphatic malignancy which can be divided into Hodgkin and non-Hodgkin types. Mediastinal manifestations are common. Mediastinal involvement on initial presentation is more common in the Hodgkin type.
DIFFERENTIAL DIAGNOSIS:
Mediastinal lymphadenopathy:
- Very wide differential, but sarcoidosis is commonly considered one of the major differential diagnoses. Sarcoidosis almost always symmetrically involves the hila.
Anterior mediastinal mass:
- Thymoma
- Thyroid malignancy
- Teratoma (or other germ-cell tumours)
FOLLOW UP:
- Blood tests: FBC etc
- Serum ACE for sarcoidosis
- CT CAP
CLINICAL HISTORY:
35yo Male
Chest pain and Dyspnoea
Active IVDU
FINDINGS:
Lungs are slightly underinflated.
Multiple cavitating lesions of varying size distributed throughout both lungs with no apparent zonal predominance.
Small/moderate right sided pleural effusion.
The heart appears borderline enlarged but there is no evidence of heart failure. Both hilar contours appear abnormal but this may be due to superimposed lesions. Mediastinal contours are otherwise normal.
Skeletal review is unremarkable.
IMPRESSION:
Given the clinical context appearances are in keeping with multiple septic pulmonary emboli.
BACKGROUND INFORMATION:
- Embolisation of infectious material which is deposited in the lungs via the pulmonary arterial vasculature*
- Can be caused by intravenous drug use, as well as endocarditis, dental disease and infected lines/tubes*
- The variation in nodule size and rapid change in appearances can be explained through repeated episodes of embolic shower*
- Pleural effusions are common and may progress to empyema*
- Patients would usually present clinically with sepsis*
DIFFERENTIAL DIAGNOSIS:
- Cavitating pulmonary metastases (Unlikely in a patient of this age)
- Necrobiotic pulmonary nodules (Clinical history should suggest history of inflammatory bowel disease or rheumatoid arthritis
- Granulomatosis with polyangiitis (Onset is typically later in life (50 years) non-septic presentation)
FOLLOW-UP:
- CT Pulmonary Angiogram for further delineation
CLINICAL HISTORY:
95Yo Female
Pyrexia
FINDINGS:
Consolidation in the left mid/lower zone with effacement of the heart border.
Rounded opacification in the left upper zone containing multiple lucent spheres. Associated deformity of the left upper ribs and left sided volume loss.
Calcific changes over the left mid and lower zone which are presumably pleural in nature.
Granulomata in the right perihilar region in keeping with previous granulomatous disease.
IMPRESSION:
Appearances are in keeping with left sided pulmonary infection, which is probably lingular.
Background left upper zone changes are pathognomonic for plombage, right sided granulomata could suggest previous tuberculous infection.
BACKGROUND:
Plombage was a historical treatment for upper lobe cavitating tuberculosis, prior to the widespread introduction of anti-tuberculous medicines. The inserted material collapsed the adjacent lung, theoretically promoting healing.
DIFFERENTIAL DIAGNOSIS:
- None
FOLLOW UP:
- This patient would need a follow-up CXR in 6-8 weeks to check for resolution of infection. Plombage itself does not need any follow-up unless a complication is suspected
CLINICAL HISTORY:
18Yo Female
Cough with haemoptysis
FINDINGS:
Large well defined mass superimposed over the right hemidiaphragm, with evidence of cavitation.
Several smaller nodules are seen elsewhere in the right lower zone and left mid zone. No pleural abnormality is demonstrated.
Heart size is normal. Hila and mediastinal contours are normal.
IMPRESSION:
Multiple lung nodules, the largest of which shows evidence of cavitation.
Appearances could represent cavitating infection or granulomatosis with polyangiitis. Given the patient’s age malignancy seems unlikely.
BACKGROUND:
- This case shows granulomatosis with polyangiitis which is a type of necrotising granulomatous vasculitis. GPA has systemic manifestations but the pulmonary symptoms include cough and haemoptysis. In approximately 90% of cases the patient has a positive c-ANCA.*
- CXR appearances include cavitating nodules of various sizes, though cavitation in smaller nodules will be difficult to appreciate. Additionally there may be areas of consolidation which could represent areas of pulmonary haemorrhage.*
DIFFERENTIAL DIAGNOSIS:
- Cavitating pulmonary malignancy
- Cavitating infection - patient should present with septic symptoms
FOLLOW-UP:
- CT with contrast for further delineation, depending on the presence of other systemic symptoms or the likelihood of malignancy, the CT may be limited to the chest or cover the whole torso.
- Laboratory testing for c-ANCA may help to point toward GPA.
- Biopsy may be needed to prove presence of granulomatous vasculitis.
CLINICAL HISTORY:
New ITU admission, please confirm ET tube position
FINDINGS:
AP portable projection.
Endotracheal tube in situ with the tip suitably located.
Left sided central line in situ with the tip visualised in the region of the superior vena cava.
Nasogastric tube in situ, the tube is seen to lie outside the borders of the trachea and crosses the carina in a safe zone. the tip is not visualised but lies in the left upper quadrant. Safe for use.
Bilateral consolidation with a symmetrical perihilar and lower zone predominance.
Both hemidiaphragms are effaced, this is likely due to consolidation but it is difficult to rule-out co-existing pleural effusions.
Heart size cannot be accurately assessed but appears grossly normal. Hila are difficult to assess due to overlying consolidation, mediastinal contours appear normal.
IMPRESSION:
All implanted lines and tubes are safe for use.
Bilateral symmetrical consolidation with a perihilar and lower zone predominance.
Appearances are in keeping with ARDS but may also represent diffuse pulmonary haemorrhage, pulmonary oedema or infection. Clinical correlation is advised.
BACKGROUND:
Acute respiratory distress syndrome represents an acute lung injury causing alveolar damage. Alveoli become filled with fluid which decreases oxygenation.
DIFFERENTIAL DIAGNOSIS:
- Pneumonia
- Pulmonary haemorrhage
- Cardiogenic pulmonary oedema - On serial radiographs pulmonary oedema will resolve with diuretic therapy.
FOLLOW-UP:
- CT chest can be used for further delineation if needed
CLINICAL HISTORY:
46Yo Female
Left sided chest pain
FINDINGS:
Innumerable tiny nodules demonstrated bilaterally and evenly throughout the lungs. High density suggests calcification.
No acute pulmonary pathology is demonstrated. No pleural abnormalities demonstrated.
Heart size is normal. Hila and mediastinal contours are normal.
Skeletal review is unremarkable.
IMPRESSION:
Appearances are consistent with previous varicella pneumonia.
No cause for patient’s symptoms identified.
BACKGROUND:
Varicella pneumonia is a viral lung infection caused by the chickenpox virus (varicella-zoster). Initial infection will present with multiple nodules, these may resolve but in some cases (as here) calcify. Most cases of pneumonia are in immunocompromised adults.
DIFFERENTIAL DIAGNOSIS:
Correlation with previous post-infection imaging should show unchanged appearances.
DDX of dense miliary opacities:
- Repeated episodes of pulmonary haemorrhage - patient should have clinical history of this.
- Silicosis - Upper lobe and posterior distribution, other appearances supporting pneumoconiosis.
- Pulmonary alveolar microlithiasis - calcification is more confluent. Middle and lower zonal distribution.
FOLLOW-UP:
- If clinical presentation and previous imaging support varicella pneumonia then no f/u is needed.