(Test #1) Intro to OCMM Lecture Flashcards

1
Q

William G. Sutherland D.O.

A
  • For What Dr Still did for the body, Sutherland did for the cranium
    a) Range of motion
    b) Vectors of motion
    c) Physiologic dynamics of cranial bones and intracranial structures
  • Observed a disarticulated skull and its beveled surfaces in the sutures and wanted to know why they had the design he noted
    a) Thought it reminded him of the gills of a fish (mobility for respiratory mechanism)
  • Described a slow oscillating force in the cranial structures that would cause movement in the cranial bones
  • Described the CNS, CSF, and the dural membranes a functional unit

***** PRIMARY RESPIRATORY MECHANISM

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2
Q

William G. Sutherland D.O. Cont 1

A
  • 1928 Sepp described fluctuations in the CV and CSF
  • Did original research on himself and observations of his patients – 1930’s
    a) 30 years of study before talking about his findings
  • Harold MAGOUN, D.O further expanded the concept in 1966
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3
Q

William G. Sutherland D.O. Cont 2

A
  • Introduced his ideas to the profession
    a) 1943 at Eastern Osteopathic Association Convention
    b) JAOA published April 1944
  • Prior to his teachings the head was considered as not having the ability to have somatic dysfunction
    a) Although Dr Still noted the CSF was important to the function of the CNS
  • Established the Sutherland Cranial Teaching Foundation in 1953
  • Research is ongoing and increasing in this area
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4
Q

Primary Respiratory Mechanism

A
  • Primary
    a) Main internal tissue process of metabolism
  • Respiratory
    b) Exchange of gases
  • Mechanism
    c) Movement of tissue and fluid for a purpose
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5
Q

Basic Tenets of the Classic Cranial Model

A

A. Inherent MOTILITY of the Central Nervous System

B. PLASTICITY and ELASTICITY of the intracranial and intraspinal membranes

C. FLUCTUATION of the cerebrospinal fluid

D. Articular MOBILITY and involuntary motion of
the CRANIAL BONES

E. Articular MOBILITY and involuntary motion of
the SACRUM BETWEEN THE ILIA

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6
Q

PRM

A
  • MOSKALENKO Y. et al
  • The AAO Journal , Vol. 13 No. 2 Summer 2003, page 21-33.
    a) Physiological premises: CRI and PRM
  • Complex article covering modern physiologic interpretation of Dr. Sutherlands thoughts using modern knowledge of cranial CV and CSF flows
  • He traces the history of research in this arena, and discusses the application of current research to the cranial concept.
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7
Q

PRM

Southerland vs Moden Interpretation

Structures

A
1) Sutherland:
A) STRUCTURES OF THE PRM:
- Brain/Spinal cord
- CSF
- Intracranial Membranes
- Intraspinal membranes
- Articular mechanism of cranial bones

2) Modern Interpretation
- Elements form the biophysical structures
- Determine INTERACTION BETWEEN THE VOLUME AND PRESSURE OF THE LIQUID MEDIA of the cranium – blood and CSF

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8
Q

PRM

Southerland vs Moden Interpretation

Dynamic Relations

A
1) Southerland:
A) DYNAMIC RELATIONS OF THE PRM
- Bone mobility is related to and controlled by RECIPROCAL TENSION MEMBRANE
a) Cranial & Spinal dura
b) Falx Cerebri and cerebelli
c) Tentorium cerebelli

2) Modern Interpretation:
- Change in distance of fixed points are due to CSF fluctuations
- Reciprocal motions are determined by the modulatory role of the membranes

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9
Q

PRM

Southerland vs Moden Interpretation

Functioning PRM

A

1) Southerland:
A) FUNCTIONING OF THE PRM
- The brain is the MOTOR for the PRM

2) Modern Interpretation
- SLOW PERIODIC FLUCTUATIONS OF BLOOD VOLUME AND CSF PRESSURE!!!
a) Support brain metabolic supply and water balance of brain tissue

  • These are responsible for the motion of brain tissue and skull bones
  • The fluctuations are functionally connected to chemical and physical homeostatic mechanisms of the brain tissue
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10
Q

Cranial Rhythmic Impulse (CRI)

A
  • Palpable BIPHASIC rhythmic pattern of motion within the cranium.
    a) FLEXION of midline bones with EXTERNAL rotation of paired bones
    b) EXTENSION of midline bones with INTERNAL rotation of paired bones
  • Present in living individuals
  • Normal rate is 10-14 times a minute
  • **Range 6-14*
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11
Q

CRI

A
  • Must be relaxed in order to palpate and contact of the hands is very light
  • Not a visible motion - palpable
  • Separate of the respiratory and circulatory physiologic mechanism
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12
Q

CRI Characteristics

A
  • Rate
  • Rhythm
  • Amplitude
  • Strength
  • Direction
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13
Q

Basic Motion of SBS

A

1) FLEXION:
- SPHENOID will rotate about a transverse axis so that the alae (wings) will move anterior/inferior and the motion at the SBS will be superior or cephalic

  • OCCIPUT will rotate about a transverse axis so that the motion at the SBS will be superior or cephalad and the “bowl” of the occiput will move posterior/inferior
    2) Extension is OPPOSITE Directions
    3) Flexion – head gets shorter in the AP diameter and WIDER in the TRANSVERSE DIAMETER
    4) Extension is OPPOSITE
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14
Q

Hersey and Adams Studies (1993)

A
  • Showing compliance in CAT MODEL
  • SKULL BONES MOVED WITH FORCES FROM FROM THE OUTSIDE AND INSIDE!!!!
  • 30-70 microns of lateral movement at the sagittal suture
  • 250 microns of rotational movement at the parietal bones
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15
Q

Frymann Study (1971)

A
  • MEASURED THE MOTION OF THE HUMAN SKULL
  • Pick mounted on the parietal bones
  • Found rhythm of bones which varied with subjects
  • In most cases, rhythm not synchronized with breathing
  • Problems with pick-offs and artifacts
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16
Q

Zanakis and Colleagues (1995)

A

RECORDING DEVICE:

  • Used infrared device
  • Measured to 10 microns
  • COMPUTER CANCELED OUT COMMON MOVEMENT BETWEEN MARKERS

1) Recorded most data from PARIETALS with reference at BREGMA!!!

2) Data:
- Average movements independent of breathing or heart rate about 7 per minute
- Sometimes fairly symmetrical
- Sometimes very asymmetrical
- Amplitudes of from 0 to 400 microns of motion; usually 100-200 microns

3) Palliators could accurately detect moments shown by system
4) Amplitude usually increased with palpation

17
Q

Kenneth Nelson, D.O.

Inter examiner Reliability

A
  • JAOA June 2006
  • Inter-examiner reliability is impossible to establish due to:
    a) Irregularity of palpation records, perceived still points, and frequency modulation of CRI of 20%
  • Noted the INCONSISTENCY between PALPATION AND INSTRUMENT MEASUREMENTS is explained by the Observation that Clinicians correlated FLEXION with one TH Oscillation and EXTENSION with One Oscillation
    a) Therefore the palpated CRI to recorded ratio is 1:2
18
Q

Moskalenko (1999 and 2001)

A
  • In healthy people FOUND CONTINUOUS CHANGES IN CRANIAL DIMENSIONS (Frontal and Saggital sections) WITH BIPHASIC CHARACTERISTICS using 2-channel bioimpedence imaging (Mean amplitude-.38 mm)
    a) ALSO DESCRIBED CRANIAL VOLUME CHANGES (12-15 ml of intracranial pressure)

b) Found a rate of 6-14 cycles/min

  • DESCRIBES INTERACTION BETWEEN INTRACRANIAL HEMODYNAMICS AND CSF CIRCULATION!!!!!
    a) Rate of 6-12 cycles/min!!!

b) Intracranial origin of bioimpedence related to blood supply and oxygen consumption of cerebral tissue

19
Q

Kenneth Nelson, D.O. 2001 AND 2006

A
  • TRAUBE- HERING- MEYER OSCILLATIONS correlated highly with CRI!!!!!!
  • Used laser doppler flowmetry
  • Rate of 5-10 cycles/min
  • Felt the CRI occurs simultaneously but may represent a different phenomenon
20
Q

Thomas Crow D.O. 2008

A
  • MRI imagery of healthy human subjects

- P

21
Q

Principles of Treatment

A
  • Find greatest restricted pattern of dysfunction
    a) Soma
    b) Cranium
  • Direct vrs Indirect technique
    a) DIRECT: Force into barrier

B) INDIRECT:

i) Balanced Membranous Tension ——-> STILL POINT
ii) Encourage amplitude of dynamic motion

22
Q

Examination

A
  • CRI
    a) Rate
    b) Rhythm
    c) Amplitude
    d) Strength
    e) Direction
  • Motion testing for
    a) Paired and midline bone strain patterns
23
Q

Treatment

A
  • V-spread
    a) Occipital mastoid suture
  • Rocking the Temporals
  • Balance the petrous portion of the temporal bone
  • CV4
  • Condylar decompression
  • Apply techniques of choice to soma dysfunctions