Test 1 Flashcards
Nursing interventions for absence of voiding for significant period of time postoperatively
- Normal positioning of the client—sitting for women and standing for men. Providing reassurance to the client regarding the ability to void
- The use of techniques such as running water, drinking water, or pouring warm water over the perineum may also be of assistance.
- Ambulation, preferably to the bathroom, and the use of a bedside com- mode are additional helpful measures to promote voiding.
- Reinforce
- replace blood loss
- If large volumes of blood have been transfused then haemorrhage may be exacerbated by consumption coagulopathy. It may also be due to preoperative anticoagulants or unrecognised bleeding diathesis.
- Perform clotting screen and platelet count, ensure good intravenous (IV) access. If there is very significant bleeding and it is safe to do so, consider inserting a central venous pressure (CVP) catheter. Give protamine if heparin has been used. Order cross-matched blood. If the clotting screen is abnormal, give fresh frozen plasma (FFP) or platelet concentrates. Consider surgical re-exploration at all times.
- Late postoperative haemorrhage occurs several days after surgery and is usually due to infection damaging vessels at the operation site. Treat the infection and consider exploratory surgery.
Interventions (in proper order) if postoperative bleeding is assessed at the incision site
The blockage of pulmonary arteries by thrombus, fat or air emboli, or neoplastic tissue
PULMONARY EMBOLISM
Definition
- The severity of the clinical manifestations of PE depends on the size of the emboli and the size and number of blood vessels occluded.
- The most common manifestations of PE are anxiety and the sudden onset of unexplained dyspnea, tachypnea, or tachycardia.
- Other manifestations are cough, pleuritic chest pain, hemoptysis, crackles, fever, accentuation of the pulmonic heart sound, and sudden change in mental status as a result of hypoxemia.
- Massive emboli may produce sudden collapse of the client with shock, pallor, severe dyspnea, and crushing chest pain. However, some clients with massive PE do not have pain. The pulse is rapid and weak, the BP is low, and an ECG indicates right ventricular strain.
- Death occurs in more than 60% of clients with massive emboli
- Medium-sized emboli often cause pleuritic chest pain, dyspnea, slight fever, and a productive cough with blood-streaked sputum.
- A physical examination may reveal tachycardia and a pleural friction rub.
- Small emboli frequently are undetected or produce vague, transient symptoms.
- The exception to this is the client with underlying
- cardiopulmonary disease, in whom even small or mediumsized emboli may result in severe cardiopulmonary compromise.
PULMONARY EMBOLISM:
CLINICAL MANIFESTATIONS
- The client should be kept on bedrest in a semi-Fowler’s position to facilitate breathing.
- An IV line should be maintained for medications and fluid therapy. The nurse should know the side effects of medications and observe for them.
- Oxygen therapy should be administered as ordered.
- Careful monitoring of vital signs, ECG, ABGs, and lung sounds is critical to assess the client’s status.
PULMONARY EMBOLISM:
NURSING INTERVENTIONS - ACUTE CARE
A disorder involving a thrombus in a deep vein, most commonly the iliac and femoral veins.
DVT
(called Virchow’s triad)
(1) venous stasis
(2) damage to the endothelium (inner lining of the vein)
(3) hypercoagulability of the blood.
The client at risk for the development of venous thrombosis usually has predisposing conditions to these three disorders
DVT ETIOLOGY
May have no symptoms or have unilateral leg edema, extremity pain, warm skin, and erythema.
If the calf is involved, tenderness may be present on
palpation.
A positive Homans’ sign (pain on forced dorsiflexion
of the foot when the leg is raised) is a classic but very unreliable sign
If the superior vena cava is involved, there may be symptoms in the upper extremities, the neck, the back, and the face.
DVT CLINICAL MANIFESTATIONS
- Nursing care is directed toward the prevention of embolus formation and the reduction of inflammation.
- While the client is receiving nticoagulation therapy, the nurse should closely observe for any indication of bleeding, including epistaxis and bleeding gingivae.
- Urine should be assessed for gross or microscopic hematuria. A smoky appearance to the urine is sometimes noted if blood is present. A Specimen should be checked daily for hematuria.
- Particular attention should be paid to the protection of skin areas that may be traumatized. Surgical incisions should be closely observed for evidence of bleeding.
- Stools should be tested to determine the presence of occult blood from the gastrointestinal tract.
- Mental status changes, especially in the older client, should be assessed as a possible indication of cerebral bleeding. IM injections should not be given
DVT NURSING INTERVENTIONS
Decrease in oxygen resulting in failure to nourish tissues at the capillary level
IMPAIRED TISSUE PERFUSION
WHAT DOES IMPAIRED TISSUE PERFUSION CAUSE?
SHOCK
A syndrome characterized by decreased tissue perfusion and impaired cellular metabolism. This results in an imbalance between the supply of and the demand for oxygen and nutrients. The exchange of oxygen and nutrients at the cellular level is essential to life. When a cell experiences a state of hypoperfusion, the demand for oxygen and nutrients exceeds the supply.
SHOCK
WHICH STAGE OF SHOCK?
aerobic switches to anaerobic, build up of lactic acid, metabolic acidoseous
STAGES OF SHOCK - Intital stage
WHICH STAGE OF SHOCK ?
neurologic changes, maintaining homeostasis, barrow receptors, vessels general vasoconstriction, trying to be maintained, trying to maintain the function of vital organs, kidneys renetangio rensen cycle, decreases urinary output, increases venus return to the heart, attempting to increase blood pressure, GI tract compromised, impaired motility, skin is cool and clamy.
STAGE OF SHOCK - COMPENSATORY STAGE
WHICH STAGE OF SHOCK?
compensating measures in general are failing, proteins leaking through to the interstital space, causes interstitial edema, anascara, blood flow to pulmunary capillaries, leaving vascular space, pulmonary edema, looking for crackles, fluid moves into aveoli, respiratory increase, cardiac input falls further, circulation decreases, cap refil low, temp cold, pulses weak, b/p low, low oxygen, dysrythmea, excemia, MI, complete deteriation of the cardiac system,GI tract mucousa barrier is decreasing, protects lining of bowels, causes major bleeding, increases with shock, ability to absorb nutrietns decreases, immune function compromised
STAGES OF SHOCK - Progressive Stage
• vomiting
• hemohharge
• diahhrea
• diabetes
• excessive urine result of high blood sugar acts like salt
* External loss of whole blood (e.g., hemorrhage from trauma, surgery,
* GI bleeding)
* Loss of other body fluids (e.g., vomiting, diarrhea, excessive diuresis, diabetes insipidus, diabetes mellitus - high blood sugar causes oligoria)
Hypovolemic shock symptoms
Absolute Hypovolemia
* Pooling of blood or fluids (e.g., bowel obstruction)
* Fluid shifts (e.g., burn injuries, ascites)
* Internal bleeding (e.g., fracture of long bones, ruptured spleen, hemothorax, severe pancreatitis)
* Massive vasodilation (e.g., sepsis)
Hypovolemic shock symptoms
Relative Hypovolemia (3rd spacing)
- compensating measures in general are failing
- proteins leaking through to the interstitial space
- causes interstitial edema
- anascara
- blood flow to pulmonary capillaries
- leaving vascular space
- pulmonary edema
- looking for crackles
- fluid moves into alveoli
- respiratory increase
- cardiac input falls further
- circulation decreases
- cap refill low
- temp cold
- pulses weak
- b/p low
- low oxygen
- dysrythmea
- excemia
- MI
- complete deteriation of the cardiac system
- GI tract mucousa barrier is decreasing, protects lining of bowels, causes major bleeding
- increases with shock
- ability to absorb nutrients decreases
- immune function compromised
Common laboratory readings during progressive stage OF SHOCK
• Maintain patent airway
• Optimize oxygenation
with supplemental O2
• Intubation, mechanical
ventilation, if necessary
• Aggressive fluid
resuscitation with
colloids
• Antihistamines (e.g.,
diphenhydramine)
• Epinephrine (subQ, IV,
nebulized)
• Bronchodilators:
nebulized (e.g.,
albuterol)
• Corticosteroids (if
hypotension persists)
• Identify and remove
offending cause
• Prevention via avoidance
of known allergens
• Premedication with
history of prior sensitivity
(e.g., contrast media)
Interventions for anaphylactic shock
Common fluids used in fluid resuscitation
COLLOIDS & ISOTONIC CRYSTALLOIDS