test 1 Flashcards

1
Q

IV fluids – types and why they would be utilized

A

-Isotonic- lactated ringers, 0.9%
-Hypotonic- 0.45% saline- more fluid- too much sodium - hypernatremia
-Hypertonic- 3% saline, more particles- hyponatremia (common symptom is confusion)

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2
Q

sodium

A

135-145

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3
Q

hyponatremia

A

Loss of sodium, water gains that dilute ECF
*Diuretics, impaired aldosterone production
*Vomiting, Diarrhea, GI suctioning
*GI tubes irrigated with water instead of saline
*Manifestations: Headache, lethargy, confusion, convulsions, nausea / vomiting, coma
*Loop diuretics to promote isotonic diuresis
*Give 3% saline
*Restrict fluid

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4
Q

hypernatremia

A

*Sodium is gained in excess of water, or when water is lost in excess of sodium
*Manifestations: thirst, lethargy, weakness, agitation, irritability; can progress to seizures, coma, and death
oReduce sodium in diet
o0.45% saline

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5
Q

potassium

A

3.5-5.0

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6
Q

hypokalemia

A

*Causes inadequate potassium intake; excessive renal, intestinal, or skin losses; redistribution between the ICF and ECF
*Manifestations: weakness, lethargy, hyporeflexia, ST depression on EKG, PVCs, nausea and vomiting, constipation
*Diuretic causes low potassium
*Foods high in potassium= spinach, strawberries, orange, cantaloupe, potatoes
*NEVER GIVE POTASSIUM IV PUSH

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7
Q

hyperkalemia

A

*Causes: impaired renal excretion of potassium from untreated renal failure, adrenal insufficiency, acidosis (causes K to leave the cell)
*Manifestations: cramps, weakness, widened QRS complex, elevated T wave
o Kaexaylate- medication to get rid of excess potassium
oDextrose 50 + Regular insulin

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8
Q

calcium

A

8.4-10

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9
Q

hypocalcemia

A

oCauses: hypoparathyroidism, diarrhea, malnutrition, renal failure, chronic alcohol abuse
oManifestations: anxiety/ confusion, Tetany, Fractures/ irritability, seizures, Chvostek and Troussea signs positive
oLengthening QT interval -> risk for ventricular dysrhythmias
oTreat:
Give Calcium Gluconate IV (never IM or SQ- sloughing of tissue)
*Slowly= cardiotoxic and can cause BP to decrease
*Antidote: Magnesium sulfate IV

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10
Q

Hypercalcemia

A

oCauses: hyperparathyroidism and malignancies
oManifestations: dehydration, renal stones, confusion, severe thirst, polyuria, elevated BP
oParathyroid gland has an issue
oLasix to promote elimination of excess calcium
oCalcitonin to promote uptake of calcium into bones
oSodium phosphate/ potassium phosphate (calcium binds to phosphate, decreasing serum levels
oIsotonic fluids (promote fluid excretion- sodium and calcium excretion accompany one another)

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11
Q

magnesium

A

1.5-2.5

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12
Q

hypomagnesemia

A

oCauses= chronic alcoholism, protein-calorie malnutrition, diabetic ketoacidosis, kidney disease
oManifestations: (CNS depression) tremors, muscle twitching, weakness, excitability, tetany, seizures, tachycardia, PVCs
oCardiac dysrhythmias/ sudden death= hypokalemia + hypomagnesemia= digitalis toxicity

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13
Q

hypermagnesemia

A

oCauses: less common, but can occur with renal failure, especially if magnesium is administered
oManifestations: (CNS depression) Nausea and vomiting, weakness, drowsy, decreased DTRs, bradycardia, hypotension, coma, resp. failure

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14
Q

phosphate

A

2.5- 4.4

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15
Q

hypophosphatemia

A

Can indicate total body deficit or a shift of phosphate into the intracellular space
*Decreased GI absorption of excess renal excretion
*Manifestations: anorexia, weakness, muscle pain, Rhabdomyolysis

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16
Q

hyperphosphatemia

A

Causes: acute or chronic renal failure, rapid administration of phosphate- containing solutions, extensive trauma, heat stroke, or disruption of mechanisms that regulate calcium levels
Manifestations: possibly tetany with low calcium, muscle cramps, pain, muscle spasms

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17
Q

metabolic acidosis

A

*Caused by excess acid or loss of bicarbonate
oTissue hypoxia, diabetic ketoacidosis, acute or chronic renal failure
omanifestations: headache, weakness, fatigue, anorexia, nausea, vomiting, flushed skin, stupor, possible coma, dysrhythmias, cardiac arrest, deep and rapid respirations

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18
Q

metabolic alkalosis

A

*Caused by loss of acid or excess bicarbonate
oSecondary to hospitalization, hypokalemia
*Manifestations: muscle spasms, numbness, tingling, tetany, confusion, dizziness, depressed respirations, and possible respiratory failure
*Treatment: naturally body hypoventilates, retaining CO2; naturally kidneys increase renal excretion of bicarb; sodium chloride plus potassium chloride causes renal excretion of bicarb.

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19
Q

respiratory acidosis

A

*CO2 retention caused by hypoventilation
*Manifestations acute: headache, irritability, decreased level of consciousness, warm skin, blurred vision, cardiac arrest
*Manifestations chronic: weakness, dull headache, impaired memory, personality changes, sleep disturbances, daytime sleepiness
*Treatment: correct underlying cause; naturally kidneys retain bicarb which increases the pH; oxygen, BIPAP, ventilation; bicarb infusion if acidosis is severe

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20
Q

respiratory alkalosis

A

*Causes: anxiety with hyperventilation; high fever or hypoxia; gram- negative bacteria; thyrotoxicosis
*Manifestations: lightheadedness, tremors, tinnitus, panic feeling, difficulty concentrating, sensation of chest tightness, seizures and circumoral and distal extremity paresthesia
*Treatment: resolve underlying cause, severe- breathe in own CO2, sedate the patient

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21
Q

oxygenation methods

A

o Nasal Cannula- 6 L
 Low flow
o Face Mask
 Simple
 Partial rebreather
 Nonrebreather- O2 flow meter all the way up
o High flow
 Venture mask, aerosol mask, trach collar, high flow nasal cannula
o Noninvasive Positive Pressure Ventilation
 CPAP- same level going in and out
 BIPAP- bilevel positive air pressure, respiratory acidosis

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22
Q

oxygenation therapeutic methods

A

oIncentive Spirometry- expand lungs
oChest physiotherapy- break up mucous in lungs
oNebulizer treatment
oMechanical ventilation

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23
Q

fluid volume deficit

A

Decrease in intravascular, interstitial, and/or interstitial fluid in the body
Causes= vomiting, diarrhea, GI suctioning, hot environment, hemorrhage
Manifestations: rapid weight loss, decreased skin turgor and urine output, tachycardia, hypotension
Lab values: sodium, potassium, H/H, serum osmolality/ urine osmolality/ urine specific gravity- rises is kidney conserving water, BUN/ Creatinine
Management
*Treat underlying cause, oral fluids, isotonic solution, Reassess- pulmonary, renal, VS, Neuro

24
Q

fluid volume excess

A

Weight gain, swelling, cramping, shortness of breath, high blood pressure, heart failure
Management: daily weight, sodium/water intake, diuretics or dialysis,

25
functions of the respiratory system
 Performs ventilation and respiration  Acid-base balance  Speech  Sense of smell  Fluid balance
26
anatomy of resp system
Air travels through the upper respiratory system to the lower respiratory system Upper respiratory system- nose, mouth, sinuses, pharynx, and larynx Lower respiratory system- trachea and lungs
27
physiology of resp system
inspiration, expiration, respieration
28
dead space/ shunting
oNo gas exchange at that site oAnatomical- no oxygenated blood going back to heart oAlveolar- large clot in pulmonary artery, low perfusion
29
ventilation/perfusion
ensures continuous delivery of oxygen and removal of carbon dioxide from the body
30
assessment of resp system
oHistory of present illness oFactors that exacerbate or improve symptoms oHealth history- current meds, allergies, past medical history, previous surgeries, family history, occupation/ residence, smoking/ social history, recent travel oPhysical assessment- inspect, palpate, percuss, auscultate
31
normal lung sounds
Bronchial – loud, high-pitched sounds, hollow, longer during expiration than inspiration Bronchiovesicular- medium pitch and intensity of sounds (tubular), heard anteriorly over primary bronchus from 1st to 2nd intercostal space, 1:1 ratio Vesicular- soft, low pitched gentle sounds, heard over all areas of lungs except major bronchi; 3:1 ratio
32
abnormal lung sounds
Crackles- short discrete, bubbling sound; pneumonia, bronchitis, CHF Wheezes- continuous, musical sounds; bronchitis, emphysema, asthma Friction rub- loud , dry creaking sounds; pleural inflammation
33
arterial blood gases
determine oxygenation status
34
pulse oximetry
utilizes wavelengths of light to measure saturation of hemoglobin with oxygen
35
capnography
continuous, monitors pco2 in airway with written tracing
36
capnometry
one time, measures co2 exhaled without continuous tracing
37
sputum analysis
don’t need consent, check for microorganisms and/ or abnormal cell growth
38
chest x-rays
don’t need consent, may identify lungs, heart, and pleural space problems
39
pulmonary function test
do not need consent, evaluate lung volumes to determine function
40
bronchoscopy
need consent, NPO, allows for direct visualization of the respiratory tract down to the level of the secondary bronchi
41
thoracentesis
need consent, NPO, needle is inserted into the pleural space to remove a specimen or excess fluid/ air
42
lung biopsy
- need consent, NPO, small piece of lung tissue removed and analyzed under a microscope
43
influenza
Highly contagious infection rapidly spread; 3 types = A, B, C Pathophys=aerosolization of small droplets from infected individual’s sneezing and coughing. Direct contact with fomites inhaled and deposited on upper respiratory tract epithelial cells Manifestations= more severe than a cold and come on quickly Management- sampling of respiratory secretions for viral culture (10 days to provide confirmation), rapid influenza diagnostic tests, treatment directed toward prevention by annual vaccination
44
pneumonia
Epidemiology- occurs at any time and at any age, 1/3 occurs in persons over 65 years of age, hospital acquired pneumonia develops after 48 hours of hospital admission Pathophys- inflammation of lung parenchyma from bacterial, viral, or fungal infection Mid to severe clinical manifestations: fever, chills, fatigue, tachypnea/ dyspnea, tachycardia, cough (productive or nonproductive), pleuritic chest pain, myalgia/ arthralgia lab tests- ABG, CBC, sputum culture, CMP, chest xray interventions: incentive spirometer, postural drainage education- hand hygiene, take antibiotic fully
45
rhinitis
Classification: allergic, non-allergic, acute, chronic Allergic rhinitis treatment- allergy testing, avoidance of allergen, antihistamines (Benadryl, Zyrtec), Decongestants (Mucinex, rubatussin) Non-allergic rhinitis treatment- nasal spray (corticosteroid), decongestants, possibly antibiotics Nursing interventions: vital signs, nasal drainage, administer prescribed medication, educate on use of medications, avoid allergies, preventing spread of infection
46
rhinosinusitis
Sinus infection Involves inflammation of nasal and paranasal cavities Classified as acute, subacute, and chronic Symptoms: nasal drainage (colored or cloudy), nasal obstruction, facial pain or pressure, weakness, coughing Treatment is based on the cause of infection *Bacterial- antibiotics, analgesics, antipyretics, decongestants *Viral- analgesics, antipyretics, decongestants Nursing interventions: vital signs (mainly oxygenation), medications, examining patient for drainage, percuss sinuses, teaching moments: proper med administration, hand hygiene, report worsening symptoms
47
obstructive sleep apnea
Condition where the upper airway becomes obstructed during sleep At risk patients include obesity, atrial fibrillation, heart failure, type II diabetics -Symptoms include excessive daytime sleepiness, snoring, observed periods of apnea Treatment/ management- combined treatment (CPAP, weight management, oral appliances), those intolerant of CPAP or oral appliances may require surgical procedures Nursing interventions: vital signs, risk assessment (stop bang), CPAP management, teaching moments: importance of taking prescribed medications, weight management
48
Laryngitis
Inflammation of the mucous membranes of the larynx Causes: infection, GERD, environmental pollutants, asthma inhalers, intubation Symptoms: hoarseness, cough, fatigue, postnasal drip  Stridor- extreme airway obstruction (high pitched wheeze) Management/ treatment- rest and humidification, avoidance to smoking or environmental pollutants, bacterial infection: may require antibiotics, hoarseness continues after 5 day- malignancy may be a source Interventions: vital signs, airway assessment, teaching moments- humidification, taking prescribed medications, voice rest, increase fluid intake
49
laryngeal cancer
Cancerous tissue that resides on the larynx Symptoms: voice changes, persistent sore throat, pain with swallowing, difficulty swallowing and eating, feeling of lump or mass in the neck Cuffed vs uncuffed trachea Treatment/management *Dependence of severity: radiation, chemotherapy, surgery (laryngectomy) *Tracheostomy oCordectomy oPartial laryngectomy oTotal laryngectomy Not able to speak, need tracheostomy At risk for aspiration Interventions: vital signs, airway management, administer prescribed medications, communication strategies, teaching moments: involve family, practice oral hygiene
50
laryngeal trauma
Trauma that directly involves the larynx Symptoms: dyspnea, dysphagia, hemoptysis Check airway, Diagnosis- inspection with swelling, discoloration Management/ treatment- intubation, maintain airway, surgery *head of bed 45 degrees or greater *NPO Interventions: vital signs, head elevation, humidification, teaching moments: symptoms of obstructed airway, limit talking (voice rest)
51
Asthma
Chronic lung disease that affects the bronchial airways Symptoms: wheezing, dyspnea, coughing, increased RR, increased HR, anxiety Inability to speak in full sentences Status Asthmaticus-asthma attack that wont stop Treat with anti-inflammatories and inhaled corticosteroids (bronchodilators) Management/ treatment *Pulmonary function tests oSpirometry- measures airflow and lung volumes oForced expiratory volume- measures the amount of air forced out the lungs after one full inspiration (measured over 1 second) oPeak expiratory flow- measures the maximum flow expired during forced expiration oPulse oximeter *Medications oAnti-inflammatories, corticosteroids, bronchodilators oNebulizer *Control of environmental factors and triggers *Interventions oVital signs, respiratory assessment, tripod position, administer oxygen if needed, purse-lip breathing, teaching moments: importance of medication management, proper inhaler administration
52
COPD
Third leading cause of death Primary cause is smoking Copd combination of emphysema and chronic bronchitis Symptoms: increased work of breathing, shortness of breath, barrel chested wheezing Management/ treatment *Pulmonary function tests *Four goals for COPD oAssess and monitor the disease oReduce modifiable risk factors oManage stable COPD oManage exacerbations *Medications oBronchodilators and anticholinergics Nursing interventions: vital signs, assessing respiratory effort and rate, position in semi fowlers, oxygen needed if below 88% *Teaching moments: smoking cessation, medication management, inhaler administration Chronic Bronchitis Emphysema
53
cystic fibrosis
A genetic chronic disease that affects the lungs and digestive, affects glands Survival rate is mid 30s Symptoms: coughing, wheezing, inflammation in nasal passages, repeated lung infections, severe constipation Management/ treatment *Clearing airway secretions (postural drainage/ chest physiotherapy) *Anti-inflammatory *Antibiotics *CFTR modulators *Pancreatic enzymes *Lung transplant Interventions: vital signs, oxygenation, administration of medications, teaching moments: airway clearance techniques, importance of taking medications, nutritional supplements
54
lung cancer
Uncontrolled growth of abnormal cells in the lungs *Non-small cell *Small cell Symptoms: vary based on advancement of disease : chronic cough, bloody sputum, SOB, wheezing, chest pain, weight loss Management/treatment *Surgical treatment – no metastasis oLobectomy- removing entire lobe of lung oPneumonectomy- removal of entire lung oWedge resection- removal of small section of lobe *Chemotherapy *Radiation *Palliative care Interventions *Vital signs, oxygenation, oxygen therapy, pain control (help them breathe), possible chest tube management, teaching moments- smoking cessation, use of medications
55
Tuberculosis
oEpidemiology- potentially life-threatening respiratory infection, 1/3 of world’s population oPathophys- transmitted by aerosolized droplets inhaled from the coughing or sneezing of an infected individual, can remain suspended in air for several hours oMeds- 6 mos to 1 year oClassifications Latent tb infection Primary tb infection Primary progressive tb infection Drug resistant tb oManagement Diagnosis by lab testing, skin test, and chest x-ray Mantoux tuberculin skin test Treatment goal to cure patient and minimize exposure