Term Test 1 Flashcards

1
Q

What constitutes abnormal?

A

Psychological dysfunction within a person that causes distress or dysfunction and a response that is not typically culturally accepted (I.e think of 4 Ds)

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2
Q

Prevalence

A

How common a mental health disorder or symptom is in a population

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3
Q

Why are we interested in prevalence when discussing mental illnesses?

A

Since many mental health disorders are episodic, may emerge at different times during a lifespan

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4
Q

Time limited

A

Illness only present for a period of time then never recurs again

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5
Q

Lifetime prevalence of schizophrenia

A

1%

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6
Q

What are the 3 things that mental health research typically focuses on

A

Diagnosis
Causation
Treatment/outcome

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7
Q

What are some criticisms of the DSM-5

A

Being culturally biased towards American culture

Not providing reliable and valid diagnoses bc some physicians will diagnose same patient differently

Medicalizing potential normal behaviours (I.e tantrums in children)

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8
Q

How do genes affect the body?

A

DNA provide genetic code to mRNA through transcription process that enables protein synthesis, which then serves a function in the body.

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9
Q

What does it mean for mental illnesses to be polygenetic?

A

Many genes are involved in mental disorders, it’s not just one gene with a mutation it’s a bunch of genes with mutations that add up to produce a disorder

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10
Q

Genome wide association studies

A

They look for patterns of single nucleotide point mutations across the genome to see if people with a disorder have the same point mutations

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11
Q

Explain the diathesis stress model

A

A person can have a genetic predisposition to develop a disorder but depending on life stressors will influence the likelihood they develop it. Someone with low genetic predisposition might be able to handle more life stressors and not develop a disorder as opposed to someone with high genetic predisposition

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12
Q

Interacting factors in mental illness development

A

Someone with genetic predisposition might be more likely to face stressors in their life, both genetics and environment interact with each other

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13
Q

Grey matter

A

Mental functions performed by grey matter, neuron rich, actually pink in living brains. Grey matter regions interact with each other by signalling through axons

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14
Q

White matter

A

Religion of the brain containing a lot of axons, look white due to high myelin content

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15
Q

Why might mental illness occur at a neuron level?

A

Might occur when there is a problem on an axonal level where neurons are night communicating properly with other neurons

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16
Q

Central executive

A

Network of brain regions responsible for higher order cognitive thinking

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17
Q

Salience

A

Network of brain regions that become active when paying attention

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18
Q

Default mode

A

Network of brain regions that are most active when the brain is at rest

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19
Q

Glutamate and it’s role in CNS

A

Excitatory, turns on neurons

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20
Q

GABA

A

Generally inhibitory, inhibits neuron from firing action potential

21
Q

Serotonin

A

Present in circuits that influence behaviour, especially information processing

22
Q

Norepinephrine

A

Mobile brain and body for action aka fight or flight

23
Q

Dopamine

A

Exploratory pleasure seeking behaviour

24
Q

Two factor conditioning model

A

Model combines classical and instrumental conditioning and can explain the persistence of fears where a person develops a fear response to a previous neutral stimuli (classical condition) and then develop behaviour to avoid triggering fear through instrumental conditioning

25
Cognitive disortions
Unhelpful thinking styles that result in negative emotions and maladaptive behaviours in mental illness
26
Anxiety
A physical state characterized by physical tension and apprehension about future events
27
fear
A normal emotion that activates in potentially life threatening situations to produce the fight or flight response
28
Panic
Abnormal form of fear
29
What is the fight or flight response produced by?
HPA axis to prepare body for threatening external stimuli
30
Explain the process of the HPA axis
Hypothalamus releases CRF to pituitary gland which secrete ACTH which stimulate the adrenal cortex which then releases cortisol (and a bunch of other hormones)
31
Relationship between anxiety disorders and fight or flight
Normal fight or flight may become dysregulated such that it turns on in response to non threatening stimuli
32
2 types of panic attacks
Expected: person is afraid of specific stimulus and they come in contact with that stimulus and have a panic attack Unexpected: random panic attack not in response to anything
33
GAD
In GAD people are anxious almost all the time in any situation, have uncontrollable worry about many things, occurring for more than 6 months and they spend a lot of time trying to avoid feared situations
34
Course of GAD
Usually begins in late teens early 20s | Considered chronic but can be symptom free for a long time
35
What circuit is relevant in anxiety disorders
Behavioural inhibition system which lies in the limbic system and brings information from either down from the areas of cortex or up from the brain stem and passes them to the cortex which causes us to freeze and evaluate if the situation is dangerous
36
Neurotransmitters causation for GAD
Norepinephrine GABA serotonin
37
Causation child psychology of GAD
If you provide appropriately for you children they learn that they have a good sense of control of their own life, however if children are neglected they may learn that they do not have control of events, or overprotective parents may also a child’s ability to develop a sense of control by not letting them experience any adversity
38
Cognitive causation GAD
Cognitive problems are: Intolerance of uncertainty Positive beliefs about worry Poor problem orientation Cognitive avoidance
39
Drugs to treat GAD
Benzodiazepines: enhance the effect of GABA, immediate short term relief, prescribed for short term period, side effects might include drowsiness, dependence and impaired cognitive function Antidepressants: tend to act as reuptake inhibitors, slower acting, anywhere from 2-6 weeks. 2 used to treat gad are SSRI and SNRI
40
Besides pharmaceuticals to treat GAD, what psych techniques can be used
CBT, recognizing cognitive distortions and developing coping mechanisms to control their anxiety Mindfulness and meditation
41
Panic disorder
Where a person experiences an unexpected panic attack and then develops anxiety around having another panic attack
42
Agoraphobia
Fear of having a panic attack in a place where it would not be convenient to have one
43
Why would someone with agoraphobia or panic disorder avoid physical exercise?
Because some people with panic disorder might have a high trend awareness of bodily cues and so physical stimuli associated with working out may trick the person into thinking they’re having a panic attack
44
Panic disorder course
Usually begins in late adolescence early adulthood Chronic but symptoms tend to differ over time
45
Gender effect in panic disorder
Females 2x as likely to get it but em there is an age effect
46
Treatments for panic disorder and agoraphobia
Benzodiazepines SSRIS SNRIs CBT where a patient is taught relaxation and breathing exercises then either have the patient identify catstrophizing cognitions or the therapist might try and induce these feelings, third step is to practice relaxation while having these feelings, fourth step is to recognize cognitive distortions, 5th step can be to use systematic desensitization
47
Social anxiety disorder
People become so anxious in social situations and are afraid of being judged or humiliated from others, leads to avoiding social situations
48
Course of social anxiety
Usually childhood or early adolescence