Tendons and their conditions Flashcards

1
Q

What is a tendon? What is a ligament?

A

Tendons connect muscles to bones
- Allows for movement
- only stretch a bit
- transmit forces from muscles to bones
- E.g.biceps contraction the force is transmitted to the humerus and the forearm and results in elbow flexion

Ligament connect bone to bone
-maintains joint stability by preventing bones from moving apart
- have a limited stretching ability thus limit how much a joint moves, & protects against injury

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2
Q

What is the function of tendons?

A

Bind structures & keep them stable

Elastic properties allow them to store energy & release it to muscle w/ no additional work
- results in smooth movement
- Reorientation of collagen type i fibres
- Straightening of the wavy fibrils
- Sliding between adjacent collagen fibrils and fibres

Transmit the mechanical force of muscle contraction to the bones

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3
Q

What is the structure of tendons?

A

Made up of mostly collagen type 1 & proteoglycan

Parallel rows of fibroblasts (tenoblasts) are organised into microfibrils & wavy fibrils
- provide non-linear strength
- fibroblasts are spindle-shaped immature tendon cells that give rise to tenocytes. highly proliferative & involved in synthesis of collagen
- tenocytes (fibrocytes)= mature tendon cells. Responsible for turnover of maintenance of the extracellular matrix & respond to mechanical load of tendon & make adaptations.
- both reside in tendon

collagen molecules consist of polypeptide chains
- 3 chains combine together to form a densely packed helical tropocollagen molecule.
- 5 of these combined together form a microfibril.

Primary collagen fibres
consist of many collagen fibrils
- fibrils have crimpled structure

Primary fibres are bunched together into primary fibre bundles (subfasicles)
- bundle is surrounded by a sheath of connective tissue called endotenon
- allows gliding of bundles against one another during tendon movement w/ little resistance
- primary fibres have crimped waveform

Groups of primary fibre bundles form secondary fibre bundles (fasicles).
- bundle surrounded by endotenon

Multiple secondary fibre bundles form tertiary fibre bundles
- bundle surrounded by endotenon

Groups of tertiary fibre bundles form the tendon unit.
- covered in connective tissue called epitenon
- Epitenon contains nerves & blood vessels which provide neurovascular supply to tendon.

Epitenon is covered in a connective tissue layer called paratenon
- allows tendon to move against neighbouring tissues.

Tendon is attached to bone by collagenous fibres (Sharpey fibres) that continue into the matrix of the bone.

In situations where tendon require extra freedom of movement, tendon separated from surroundings by synovial sheath

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4
Q

Vascular supply of tendons?

A

Sparse

Small arterioles from adjecent muscle tissue branch
- rterioles accompanied by vein, & lymphatic vessel
- Augmeneted by small vessels from addjacent loose connective tissue

Vessels rarely pass between bone & tendon except at achilles tendon
- Receives blood supply across osteotendinous junction

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5
Q

Nerve supply of tendons

A

innervation of tendons start from cutaneous, peritendinous & muscular nerves.

Nerve fibres cross & enter endotenon & paratenon.
- These fibres then enter & innervate the epitenon.

Nerves terminate & reside on surface of the tendon.

Largely sensory

no evidence of any capacity for vasomotor control

Myelinated nerve fibers are specialized mechanoreceptors (Golgi tendon organs) sensing tension & pressure in the tendon
- located close to the muscle

Unmyelinated fibres are responsible for sensing & transmitting pain (nocioceptors)

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6
Q

What determines the thickness and strength of tendons?

A

Thickness depends on size & strength of associated muscle

AND Degree of pennation of the muscle

Tendons can adapt their strength & stiffness to match mechanical demands - but process is slow & incomplete
- Deform slowly when exposed to external force
- Once the deforming force has been removed it returns to its original shape
- If you pull fast - snaps
- If you pull slow - stretches

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7
Q

How do muscles insert into bones?

A

Enthesis/Osteotendinous junctions = where bone & tendon meet
- Can be Fibrocartilaginous or fibrous

Fibrocartilaginous:
- no sharp boundaries between zones
- proportions of each component vary between entheses
- usually found where a tendon approaches bone at high angle
- Found at attachment of tendon to metaphysis or diaphysis

Fibrous Entheses
- found on shafts of long bones, & small bones of hands & feet
- tendon approaches bone at acute angle & merges at periosteum
- connects w/ dense fibrous conective tissue
- attach to greater area of bone compared to fibrocartilaginous entheses

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8
Q

What are the three main pathologies that affect tendons?

A
  1. tendinopathy- injury to tendon or overuse
    - Or specifically enthesitis- inflammation of enthsis
  2. tenosynovitis- Inflammation of synovial lining of the tendon sheath
  3. tendon rupture

Others:
- Tendonitis= inflammation of tendon
- Tendinosis= chronic degeneration of tendon without inflammation, due to failed healing of repeated minor injuries

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9
Q

Pathophysiology of Tendinopathy

A

Occurs as part of systemic inflammatory condition

Due to injury/overuse

Idiopathic

Mainly caused by
- Trauma
- Inflammatory Arthritis

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10
Q

Presentation of tendinopathy/ enthesitis

A

Pain
- Worse on active movement
- increased when active movement is performed against resistance

Tenderness at ethesis

Rubor, calor, dolor, tumor

Soft tissue swelling - not always present

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11
Q

Investigations for enthesitis

A

X ray - may see clacifiation at enthesis

Ultrasound scan - may see oedema, calcification, tears

MRI - - may see oedema, calcification, partial thickness tears

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12
Q

Give examples of tendiopathy/ enthesitis and their clinical presentation

A

Tennis elbow
- i.e. lateral epicondylitis
- inflammation of tendons & forearm muscles due to overuse i.e. playing tennis
- muscles & tenon responsible for extension of wrist & finger
- Most common tendon injured =
extensor carpi radialis brevis i.e. at common extensor region
- Pain + tenderness + stiffness + weakness + tingling
- pain on resisted extension

Golfer elbow
- i.e. medial epicondylitis
- less common
- inflammation of tendon due to overuse- flexion of wrist
- Pain + tenderness + stiffness + weakness + tingling
- Most common injured = flexor carpi radialis & pronator teres i.e. at common flexion region
- pain on resisted flexion

Jumpers knee
- i.e. patella tendinitis
- Pain at inferior pole of patella
or can also be at insertion of quads
- Overuse of patella tendon e.g. when jumping or running constant stress
- patella tendon role= extension of leg
- pain (when straightening leg or bending) + tenderness + swollen knee + stiffness
- different to Osgood Schlatter

Rotator cuff- specifically supraspinatus tendonitis
- caused by overload of rotator cuff tendon- due to repeated overhead activities e.g. weight lifting, swimming, tennis
- In subacromial space
- When subacromial space becomes narrowed = supraspinatus irritation
- area of poor blood supply
- Angiogenesis can occur whilst attempting to repair- causes congestion & pain
- pain at greater tuberosity+ pain radiates up neck & down deltoid + stiffness + inflammation + loss of strength
- Painful arc + weak shoulder abductions + drop arm sign

Achillies tendonitis:
- achilles attaches gastrocnemius & soleus to calcaneus
- Help go on tip toes
- injury due to inability to adapt to strain & overuse e.g. running & football
- Pain + morning stiffness

Bicep tendonitis

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13
Q

How is tendinopathy managed?

A

RICE

Avoid applying pressure

NSAID

Physio

Analgesics - paracetamol

NSAIDs

Local corticosteroid injection

Surgery

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14
Q

What is Osgood Schlatter disease?

A

Osgood Schlatter:
- occurs in children
- during contact sport e.g. rugby
- inflammation & irritation of patella tendon where it attaches to the growth plate of tibia
- As quadricep muscle stretches, it pulls on patellar tendon
- Repeated tension & irritation causes pain, swelling, inflammation, & formation of bump right below knee

Different to patella tendonitis:
- irritation originating where patella inserts onto growth plate of knees- Osgood-Schlatter
- Injury to patellar tendon- occurs slightly higher that OS- patella tendinitis

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15
Q

What are the main causes of tenosynovitis?

A

Inflammatory arthritis

Trauma
-Result of repetitive or
- unaccustomed movement

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16
Q

Clinical presentation of tenosynovitis
- How does trigger finger occur?

A

Pain

Swollen & tender tendon

Crepitus may be felt on palpation

May result in trigger finger or thumb (tenosynovitis of the flexor tendons)
- constriction of the tendon sheath
- results in formation of tendon
- Nodule catches as it enters or leaves the flexor tendon pulleys
- Flexors stronger than extensors, so finger gets stuck in flexion

17
Q

What are the commons sites of tenosynovitis?

A

Abductor pollicis longus

Extensor pollicis brevis tendons (de quervain’s tenosynovitis - pain on radial aspect of wrist)

Finger flexors

18
Q

How is tenosynovitis managed?

A

Rest

Splinting

Local corticosteroid injections

Surgical decompression of the tendon sheath

19
Q

What is the pathophysiology of a tendon rupture?

A

May result from:
- chronic inflammation
- degeneration
- trauma

E.g. rupture of the extensor tendons of the finger commonly seen in RA

20
Q

Clinical presentation of tendon rupture

A

Loss of movement at joint to which the tendon provides power

Deformity

Swelling

Bruising

snap sound when it occurs

Inability to weight bear

Crepitus

E.g. achilles tendon rupture

21
Q

How are tendon ruptures managed?

A

Sometimes no intervention is required
- E.g. long head of biceps tendon rupture
- Because function is preserved w/ other muscles (short head of biceps, brachialis, supinator)

Splintage e.g. mallet splint

Surgery is often required to restore function

May required direct repair of the tendon/ tendon transfer

22
Q

How does tendon repair occur?

A

Initial proliferation of fibroblasts

Interstitial deposition of new collagen fibres

Complete remodelling of the tissue e.g. doesn’t happen in adult tendons - so healing tendons never recover to their original strength

23
Q

What is an avulsion?

A
  • Injury to bone at site of tendon insertion – avulsion fraction = tendon (or ligament) pulls of a piece of bone