Teeth Development Flashcards
Disturbances in size
- Microdontia
- Macrodontia
Microdontia - Small
Macrodontia - Large (Don’t confuse with fusion)
Gemination
Attempted division of tooth germ with incomplete formation of 2 teeth, usually single root.
Fusion
Union of two tooth germs by dentin. Often with individual roots.
Concrescence
Form of fusion where teeth joined by cementum, ↑↑ MxMs
Dilaceration
Bend or curve in tooth or root
Possibly due to trauma
Problems with RCT or extraction
Dens‑in‑dente
(Dens invaginatus)
“tooth within a tooth”
invagination in enamel organ before calcification
↑↑ MxLa I, can be bilateral
sequela of periapical infection
prophylactic restoration of pit
Dens evaginatus
tooth-like structure in central fossa. ↑↑ MdBis, may contain pulp tissue
- interferes with occlusion and there is often a pulp horn inside the projection
Taurodontism
block-shaped teeth with large pulps (“bull-like teeth”)
Elogated pulp chambers
one or multiple teeth
isolated (phenotypic expression of gene pool) or with syndromes,
esp. Klinefelter’s Syndrome (polyploidy of X chromosome, eg XXXY)
Enamel Pearl
Excess of enamel at bi or trifurcation of molars at CEJ
Oligodontia / Anodontia
A few or no tooth development
Anodontia invariably associated with systemic problem, most common, hereditary ectodermal dysplasia - if missing most teeth
evolutionary trend toward fewer teeth
Oligodontia - a few teeth
Anodontia - no teeth
Supernumerary teeth
extra teeth
90% in Mx
_**Mesiodens_, between MxCeIs, most common
- Gardner’s Syndrome (see also Bone, Neoplasms)
Often die of colon cancer
- Cleidocranial Dysplasia
Disturbances in structure
Enamel - Amelogenesis imperfecta
Hereditary
Amelogenesis imperfecta (Hereditary) (inherited defect of ameloblast)
autosomal dominant, recessive, sex-linked
12 - 15 types
clinically produces hypoplasia (quantitative defects)
hypocalcification
hypomaturation
Hereditary Environmental
All teeth Some teeth
Both dentitions One dentition
Family history No family history
Disturbances in structure
Enamel -
Environmental enamel defects
Hypoplasia / Hypocalcification
(1) Rickets
(2) Exanthematous diseases - often viral diseases affecting epithelium
(3) Congential Syphilis
a. Hutchinson’s incisors
b. Mulberry molars
(4) Turner’s tooth - Local infection or trauma
(5) Fluorosis - Mottled enamel, generalized, > 1ppm discoloration due to other elements, ie Fe
(6) Tetracycline
(7) Idiopathic
Disturbances in structure
Dentinogenesis Imperfecta
Dominantly inherited genetic defect of dentin independent or with osteogenesis imperfecta (due to mutation of dentin sialophosphoprotein DSPP) both dentitions, all teeth affected, mom or dad should have this. Autosomal dominant.
osteogenesis imperfecta - brittle bones ± blue sclerae ± DI (not mediated through DSPP)
Teeth - grayish to yellowish-brown, translucent, enamel fractures from dentin (lacks scalloping), exposed dentin prone to attrition
Radiographic findings: Obliteration of pulp chambers and canals ± attrition, ± root fractures
Treatment: Crowns to prevent attrition
Disturbances in structure
Dentinal dysplasia
Autosomal dominant condition characterized by pulpal obliteration with abnormal dentin, defective root formation and tendency for periapical pathology.
Clinically affects all teeth and both dentitions but teeth clinically appear normal
Radiographic findings: Obliteration of pulp, short underdeveloped roots, periapical radiolucencies