Teeth Flashcards
Silica
Abrasive
Sodium bicarbonate
Abrasive
Sulfur
Bactericidal
Calcium
Inhibits demineralization
Fluorine
Anticariogenic
Chloride
Inhibits calculus formation
Benzyl isothiocyanate
Antiocariogenic
Goadby
Acidogenic and proteolytic bacteria but no specific organism of dental decay and poly microbial disease
Clarke
Introduced microbial succession. Lactobacillus found in established Caries
Van houte found organisms involved were
Low ph non mutans streptococci but later identified atypical strains of plaque
Bifidobacteria
Aka gut inhabitants, Anaerobic and produce lactic acid
Beneficial species
Defectiva, parasanguins, Mitis, Oralis, sanguins
Saliva has immunoglobulins
IgG, IgM, IgA
Stimulation of parasympathetic nerve causes
Watery saliva with less organic material and vasodilation of the gland
Sympathetic results in
Mucous saliva and Inhibition of glands
Serous glands
Parotid - watery amylase rich - glossopharyngeal
Lingual - watery lipase - IX
Mixed glands
Subamandibular - mucin- facial
Sublingual - mucin - facial
Retromolar - mucin- VII and IX
Seromucous glands
Buccal and labial - mucin - VII
Saliva secretion steps
Primary fluid production - isotonic in acini
Saliva modification- hypotonic in ducts less electrolytes
Saliva contains
Cells
Bacteria
Calcium
K 2X than plasma
Enzymes
Increasing salivary flow rate results I
Increase in na cl and hco3 and reduction in phosphate and K
Clearance is delayed when
Unstimulated flow rate is low
Residual volume is high
V max is high
2 digestive enzymes
Lingual lipase - by glands on tongue
Amylase - salivary glands
Lactoferrin
Bacteria static and binds Fe
Lysozyme
Attacks bacterial walls
Proline rich proteins
Protect enamel
Mucin functions
Tissue coating - role in enamel Pellicle, anti-microbial
Lubrication
Aggregation of bacterial cells
Statherins
Produced acinar cells Prevent crystallization of calcium phosphate in ductal saliva and oral fluid
PRP
Like statherin, very asymmetrical
Inhibit Calcium phosphate growth
Found in mature enamel pellicle
Strong promoters of bacterial adhesion
SP produced by
Acinar cells of parotid and present in submandibular saliva
Readily adsorbed
Myeloperoxidase
Derived from leukocytes gingival crevice antibacterial
Lactoferrin
Nutritional immunity by iron starvation of bacteria
Using enterochelins better than Lactoferrin
Degraded by proteases
Lysozyme aka muramidase
Hydrolysis of pepidoglycan later
Cystatins
Protective against unwanted proteolysis
Affects calcium phosphate precipitation
Major buffer components
Phosphate
Bicarbonate
Salivary proteins
Phosphate system
Phosphate concentration decreases with increasing flow rate
Best at 6.8 pH
Major in unstimulated saliva
Bicarbonate buffer
Best at 6 pH
50% unstimulated and 90% stimulated due to flow dependent variAtions
Salivary proteins
Best at 5 or less pH
Good for high and low pH
Stephan Curve
Shows effects on plaque pH after cariogenic challenge
Dental Carie’s
Acid dissolution of tooth mineral started by acidogenic bacteria in dental plaque which has been exposed to fermented sugars
CaF has
Lower solubility product than HA so it’s more stable
Salivary hyper function is called
Sialorrhea
Ethology of hypersalivation
Neurological
Salivary
Anatomy of hard or soft tissue
Normal saliva and hypo rates
Unstimulated .3-.5 ml/min .1 or less
Stimulated 1-1.5, .5 women .7 men
Xerostomia tx
Pilocarpine, bethanechol, sialor
CHX
2X daily inhibits dental biofilm accumulation almost completely
With thymol is good varnish 40%
CPC
Antibacterial but cleared quickly
No anti cariogenic
CPP ACP
At night without brushing off
Ca and phosphate ion saturation
Iodine
Antibacterial
Broad spectrum
Good for mutans
Arginine
Buffer dental plaque due to ionization of ammonium
Cationic as anti septic
Better with Fl
DEF index ____
Only captured cavitated lesions
ICDAS
Measure surface changes and depth of Carie’s
PUFA
Stages severe Carie’s
CAST
Stages early and severe caries
CCS is based on
Tooth surface
Presence or absence of Caries
Site of origin
Severity of change
Estimation of lesion activity
CCS examination done on a
Clean tooth
Compressed air
Adequate lighting
Rounded explorer or ball end probe
Affected dentin?
Dentin exposed to bacterial acids but has not been infected by cariogenic bacteria
CCS assessment process includes
Identification and classification of lesions
Recent restorations due to Carie’s
Cavitated lesions
Radiolucency
Enamel hypoplasia
Results from disrupted amelogenesis
Halitosis bacteria
S. Moorei
How do they cause odour
Microbes interacts with amino acids and they’re transformed in to volatile compounds and volatile sulfur compounds
Three major VSC
Methyl mercaptan CH3SH
Hydrogen sulphide H2S2
Dimethyl sulphide C2H6S
Drugs cause malodour
diuretics
Antihistamine
Metronidazole
Systemic causes of halitosis
Chronic bronchitis
Pneumonia
TB
Diagnosis of halitosis
Subjective assessment
Organoleptic - gold standard
Halimeter - only detects VSC
Gas chromatography - quantitative
Unstimulated saliva can be tested
Calculus Formed by
Binding of calcium ions to carbohydrates protein complexes of the organic matrix and precipitation of crystalline calcium phosphate salts
Factors in calculus formation
Plaque thickness
Rate of salivary sugar clearance
Salivary film velocity and acid clearance
Effect of salivary urea on plaque ph
Diagnodent by kavo
Laser fluorescence 655 to 680 nm
Inspektorpro Qlf
Quantitative laser fluorescence
CariVu, microlux
Transillumination technology
Cariescan pro
Electric conductivity
Canary system
Photothermal radiometry and luminescence