TBI and concussion Flashcards
why do we oxygenate TBI patients even if sats are good
don’t want to cause secondary damage via hypoxia, pre oxygenation for RSI if really sick.
over oxygenation is only really a concern with high flow over a long timeframe, not acutely an issue
what is the target etC02 for a TBI patient
30-35, C02 is a potent vasodilator, thus the target in TBI is lower because want to decrease further bleeding
what causes concussion
rapid movement to the head/impact to the skull, causing the brain to move within the skull
sx of concussion
headache, confusion, memory loss, dizziness
what patients can be treated under the concussion guideline
for patients that can obey commands and have a mechanism of injury consistent with a TBI
patient has a concussion if…
there are abnormal vital signs, there is abnormality in memory or coordination or balance
LOC or abnormal vitals in a patient with a head strike is an indicator off..
intercranal bleed
a concussion is a TBI with what characteristics?
no detectable signs of bleeding/blood clot/brain tissue swelling on a CT but signs and symptoms of altered brain function
a CT of someone with a TBI will show….
signs of bleeding/blood clot/brain tissue swelling
why are the elderly more susceptible to inter-cranial bleeding
cerebral atrophy exposing veins, thus more likely to be damaged in even minor falls
what is the criteria to use the severe TBI guidelines
Use this guideline for patients who cannot obey commands and have a mechanism of injury consistent with traumatic brain injury
fluid management in TBI
Administer 0.9% sodium chloride IV if there are signs of hypovolaemia, or the systolic blood pressure is less than 120 mmHg in an adult, or less than the normal predicted systolic blood pressure in a child:
500 ml for an adult.
10 ml/kg for a child.
Repeat as required, minimising the total volume.
management of seizure in TBI
treat under seizure guideline with midaz plus give prophylactic levetiracetam to minimise secondary injury
what are the goals of management of a severe TBI
identify TBI
minimise secondary damage
treat distracting injuries
what is secondary injury and list some common causes
further physiological insult to brain after primary injury including hypoxia, hypoventilation, hypotension and hypovolemia
management of patient with ? TBI but suspected drug and alcohol on board
assume severe TBI until proven otherwise
why do we have a high threshold for sedation in TBI
because sedation will likely contribute to secondary injury
raised ICP post TBI is associated with
poor prognostic outcomes
list some indicators of increased ICP
unilateral pupil dilation
posturing
severe HTN, with either Brady or tachy
irregular resps