Task 3 - Social Anxiety Disorder and Panic Attacks Flashcards

1
Q

What is social anxiety disorder?

A

People with social anxiety disorder become so anxious in social situations and are so afraid of being rejected, judged, or humiliated in public that they are preoccupied with worries about such events to the point that their lives may become focused on avoiding social encounters

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2
Q

Prevalence of social anxiety disorder

A
  • 1-7% internationally
  • 12% in the US
  • women tend to have more severe social fears than men
  • develops in early preschool years or adolescence
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3
Q

Comorbidities of social anxiety disorder

A
  • mood disorders
  • other anxiety disorders
  • 70% comorbidity
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4
Q

DSM-5 Criteria social anxiety disorder

A

A Marked fear or anxiety about one or more social situations in which the individual is exposed to possible scrutiny by others –> social interactions, being observed, performing in front of others
B The individual fears that he or she will act in a way or show anxiety symptoms that will be negatively evaluated
C The social situation almost always provoked fear or anxiety
D The social situations are avoided or endured with intense fear or anxiety
E The fear or anxiety is out of proportion to the actual threat posed by the social situation and to the sociocultural context
F The fear, anxiety, or avoidance is persistent, typically lasting for 6 months or more

Performance only: if the fear is restricted to speaking or performing in public

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5
Q

Social performance deficits in SAD

A

Patients with SAD show performance problems during a conversation

  • a conversation is unstructured, bidirectional, and requires interaction
  • a conversation asks for more interpersonally sensitive social behavior –> making contact, listening, showing interest, being responsive, and interacting
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6
Q

interpretation bias and judgmental bias

A

interpretation bias = the interpretation of ambiguous events as negative

judgmental bias = the overestimation of the costs and/or probability of a negative event

  • SPs interpret social events as more negative and judge social events as more threatening
  • interpretation bias of social phobia is a negative interpretation of all social events, irrespective of valence
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7
Q

Clark and Wells Model

A

People with social anxiety disorder interpret social situations in a threatening way because of a range of dysfunctional beliefs they have about themselves, others, and the social world

  • use of safety behaviors
  • pre-event rumination
  • post-even rumination
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8
Q

pre-event rumination

A

thoughts regarding past failures partly account for SAD maintenance and lead to anticipatory anxiety and predictions of poor performance

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9
Q

past-event rumination

A

typically begins after the social situation and revolves around intrusive images and thoughts regarding perceived failures and of adverse images of themselves that were experienced during the event

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10
Q

Rapee and Heimberg Model

A
  • when a social situation is anticipated or encountered, people with social anxiety disorder form a mental representation of their external appearance, based on memories of past social situations and internal and external cues
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11
Q

Hoffman Model

A
  • people with social anxiety disorder are apprehensive of social situations due to having unrealistic social standards and poorly defined social goals

–> combines the Clark/Wells Model and the Rapee/Heimberg model and expands them further

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12
Q

Causes SAD: Genetic

A

brain areas of the amygdala, hippocampus, and prefrontal cortex are involved

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13
Q

Causes SAD: Cognitive

A
  • excessively high standards
  • focus on negative aspects of interactions
  • harsh evaluation of themselves
  • rumination about one’s performance
  • family environment

–> safety behaviors

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14
Q

safety behaviors

A

= coping mechanisms used to reduce anxiety and fear when the individual feels threatened

–> engaging in safety behaviors increases self-focused attention, reduces the ability to attend to objective social information, and may cause the feared result

  • avoidance of social situations
  • escaping social situations
  • trying not to attract the gaze of others
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15
Q

Treatment SAD: Cognitive Behavioral Therapy

A

Behavioral Component:
- exposing clients to social situations that make them anxious (from least to most)
- role-playing, relaxing techniques, eliminating safety behaviors, experiments

Cognitive component:
- identifying negative cognitions clients have about themselves and about social situations and teaching them how to dispute these cognitions

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16
Q

Treatment SAD: Medication

A

Selective serotonin reuptake inhibitors (SSRIs) and serotonin-norepinephrine reuptake inhibitors (SNRIs)
- efficacious in reducing symptoms
- symptoms return when medication is stopped

Benzodiazepines
Antiepileptics
Antipsychotics
Oxytocin

17
Q

Treatment SAD: Mindfulness-Based Interventions

A
  • teach individuals to be less judgmental about their own thoughts and reactions and more focused on, and relaxed in, the present moment

Acceptance and Commitment Therapy (ACT):
- builds on CBT techniques to emphasize mindfulness, acceptance, and values

–> third-wave approaches of CBT

18
Q

What is Panic Disorder?

A

Most commonly, panic attacks arise in certain situations but not every time: They are terrifying experiences, causing a person intense fear or discomfort, the physiological symptoms of anxiety, and the feeling of losing control, going crazy, or dying

19
Q

Prevalence of panic disorder

A
  • 3-5% will develop panic disorder at some time
  • onset is usually between late adolescence and the mid-thirties
  • more common in women
  • chronic
20
Q

Comorbidities of panic disorder

A
  • chronic generalized anxiety disorder
  • depression
  • alcohol abuse
  • increased risk of suicide
21
Q

DSM-5 Criteria Panic Disorder

A

A Recurrent unexpected panic attacks. A panic attack is an abrupt surge of intense discomfort that reaches a peak within minutes, and during which time four (or more) of the following symptoms occur:
1. Palpitations, pounding heart, or accelerated heart rate
2. Sweating
3. Trembling or shaking
4. Sensations of shortness of breath or smothering
5. Feelings of choking
6. Chest pain or discomfort
7. Nausea or abdominal distress
8. Feeling dizzy, unsteady, light-headed, or faint
9. Chills or heat sensations
10. Paresthesias (numbness or tingling sensations)
11. Derealization (feelings of unreality) or depersonalization (being detached from oneself)
12. Fear of losing control or “going crazy”
13. Fear of dying

B At least one of the attacks has been followed by 1 month (or more) of one or both of the following:
1. Persistent concern or worry about additional panic attacks or their consequences
2. A significant maladaptive change in behavior related to the attacks

22
Q

Catastrophic misinterpretation in PD

A

= autonomic arousal/bodily sensations are interpreted as more dangerous than they are

  • patients interpret ambiguous stimuli as threatening, particularly when the stimuli are related to symptoms of bodily arousal
  • the actual or presumed presence of bodily changes is associated with danger schemas
23
Q

Catastrophic cognitions in PD

A

= catastrophic cognitions with themes of danger are experienced as a result of misinterpretation

  • themes of danger, disaster, and physical or mental catastrophe are more marked during elevated anxiety and panic
24
Q

Cognitive Mediation via Biological Challenge Tests

A

Biological challenge tests = pharmacological and physiological procedures can reliably induce panic attacks in panic disorder patients

Biological challenge tests that influence the panic response:
- inhalation of CO2
- infusion of soduim lactate
- voluntary hyperventilation

Cognitive modifications that guide the panic response:
- subjects’ expectations about the distress and anxiety they might experience
- interpretation of sensations
- perceived control over the procedure

25
Q

Causes of PD: Biological

A
  • heritability: 43-48%
  • poorly regulated fight-or-flight response
    –> poorly regulated neurotransmitters:
    norepinephrine, serotonin, GABA, CCK
  • triggers: hyperventilation, carbon dioxide, caffeine, sodium lactate
  • limbic system that is involved in the stress response: amygdala, hippocampus, and hypothalamus
  • dysregulation of locus ceruleus: pathways to the limbic system
    –> poor regulation may cause panic attacks
26
Q

Causes of PD: Cognitive

A

anxiety sensitivity: unfounded belief that bodily symptoms have harmful consequences

interoceptive awareness: a heightened awareness of bodily cues that may signal a coming panic attack

interoceptive conditioning: bodily cues are conditioned stimuli signaling new attacks

27
Q

Conditioned avoidance response

A

By avoiding places/situations that are associated with certain symptoms of panic, they reduce their symptoms, thereby reinforcing their avoidance behavior

28
Q

Agoraphobia

A

confining to safe places and avoiding a wide range of places that one feels are unsafe

29
Q

Treatment PD: Biological

A
  • SSRIs, SNRIs, and tricyclic antidepressants –> affecting serotonin and norepinephrine systems
  • benzodiazepines suppress the CNS and influence functioning in the GABA, norepinephrine, and serotonin systems
    –> work quickly to reduce panic attacks
    –> addictive

–> relapse of symptoms when drug therapies are discontinued

30
Q

Treatment PD: CBT

A

Clients confront the situations or thoughts that arouse anxiety

  • relaxation and breathing exercises
  • identifying the catastrophizing cognitions
  • systematic desensitization therapy –> gradual exposure to the feared situation