Tachycardia lecture and module Flashcards

1
Q

What causes a wide tachycardia?

A

Electrical signal has to travel through the slowly conducting ventricular cells, which takes a longer time

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2
Q

How does increased automaticity work?

A

Decreased parasympathetic drive
Increased phase 4 slope
The threshold is more negative, and the resting membrane potential is more positive – more easy to get an AP

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3
Q

Atrial fibrillation

A

Most common sustained arrhythmia
Atria depolarize at 400-600 BPM with irregular conduction to the ventricles
Irregular RR intervals
No P waves (can’t see atrial activity)

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4
Q

What is triggered activity

A

During the partial refractory period, oscillations in the membrane potential can trigger abnormal APs which can lead to a tachy

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5
Q

Atrial flutter

A

Re-entry circuit that occurs entirely in the R atria
Around 300 BPM
Can be regular or irregular (usually conducts about 2:1)
Sawtooth pattern
Characteristics: > 100BPM, regular rhythm, sawtooth P waves

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6
Q

Orthodromic vs antidromic

A

Ortho: conduction down the AV node is normal
Anti: conduction is backwards through the AV node

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7
Q

Wolf-Parkinson-White

A

Palpitations + pre-excitation
Congenital accessory path (antegrade AV conduction) and tachycardia episodes
Accessory pathway is the Bundle of Kent (antegrade and retrograde)

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8
Q

Pre-excitation

A

Presence of an antegradely conducting accessory path that can pre-depol the ventricles
Will see a delta wave on ECG
Shortened PR interval and widened QRS complex

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9
Q

Pre-excited atrial fibrillation

A

Atrial rate 400-600 BPM
Ventricular rate dictated by the refractory period of the accessory path
Can be so fast that it results in cardiac risk

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10
Q

Symptoms associated with tachycardia

A
Palpitations
Pre-syncope
Syncope
Fatigue
Poor exercise tolerance
Chest pain
Sweating (diaphoresis)
Shortness of breath
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11
Q

Everyone with SVT should have what investigation?

A

Echocardiogram

Want to rule out mitral valve prolapse and congenital valve abnormalities

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12
Q

5 waves to evaluate/measure tachycardia

A
ECG
Holter monitor
Event monitor
Implantable cardiac monitors
Echocardiogram
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13
Q

Treatment during a tachy episode

A
Valsalva
Carotid sinus massage
Diving reflex (face in cold water)
Adenosine
IV AV nodal blocker (beta blocker, calcium channel blocker)
Cardioversion (extreme circumstances)
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14
Q

How to prevent tachy episodes

A

Nothing and treat as they come
AV nodal blocker (beta blocker, calcium channel blocker)
Ablation

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15
Q

3 general mechanisms of tachycardia

A

Re-entry
Automaticity
Triggered activity

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16
Q

Where is the origin of impulse in
1. Narrow
2. Wide
QRS complexes?

A
  1. Supraventricular

2. May be ventricular

17
Q

3 types of supraventricular tachycardias

A

Ectopic atrial tachycardia
AVNRT
AVRT

18
Q

Ectopic atrial tachycardia

A

A form of tachyarrhythmia originating from within the atria, but outside of the SA node
Usually due to a single ectopic focus
Characteristics: HR > 100, regular rhythm, abnormal P wave morphology (not from the SA node)

19
Q

Multifocal atrial tachycardia

A

More than one ectopic focus of automaticity outside the SA node
At least 3 ectopic foci of automaticity evident on ECG
Characteristics: HR > 100, irregular rhythm, 3 or more unique P waves

20
Q

AVNRT

A

Both the slow and fast pathways are located in the AV node

Only the AV node is an obligate part of the circuit

21
Q

AVRT

A

2 arms of the re-entry circuit
One is the AV node, the other is an accessory pathway
The AV node, atrium, and ventricles are all a part of the pathway
P waves can be buried in the QRS complexes (atria being depolarized at the same time as the ventricles)

22
Q

What does it mean if P waves come after the QRS

A

They are retrograde P waves that come from the bottom of the atrium (not the SA node)