Tachyarrhythmias Flashcards

1
Q

Definition of narrow complex tachycardia?

A

Rate >100bpm
QRS complex <120ms
- narrow QRS complxes maen that the ventricles are being depolarised by normal conduction pathway

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2
Q

Causes of regular narrow complex tachycardia

A

Sinus tachycardia
Focal atrial tachycardia
Atrial flutter
Atrioventricular re-entry tachycardia e.g. WPW syndrome
Atrioventricular nodal re-entry tachycardia (SVT)

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3
Q

What are the causes of sinus tachycardia

A
Infection
Pain
Anxiety
Exercise
Bleeding
Dehydration 
Sepsis - due to systemic vasodilation (to keep BP up)
Drugs - caffeine, nicotine, salbutamol
Anaemia
Pregnancy 
CO2 retention 
Autonomic neuropathy 
Fever
Pulmonary embolism
Hypertyroidism
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4
Q

What is focal atrial tachycardia + ECG changes

A

A group of atrial cells acting as the pacemaker, and out-pacing the sinoatrial node >150bpm
P wave becomes superimposed onto the preceding T-wave

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5
Q

What is atrial flutter

A

Rate >250bpm in the atria, leading to a saw-tooth baseline
- ?P waves can’t be seen
AV node passes on some of the impulses, so the ventricular rate is a multiple of the atria rate
- e.g. atria 300bpm = 100bpm (3:1 AV block)`

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6
Q

What is atrioventricular re-entry tachycardia

A

An accessory pathway that allows electrical activity from the ventricles to pass to the resting atrial monocytes, creating a circuit
e.g. Wolff-Parkinson-White syndrome

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7
Q

What is atrioventricular nodal re-entry tachycardia + ECG signs

A

New circuits form within the AV node, which constantly stimulate the ventricles to contact
- causes p waves to be very close to the QRS/not visible

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8
Q

What are the irregular narrow-complex tachycardias

A

Sinus arrhythmia
Atrial fibrillation
Atrial flutter with variable block
Multi-focal atrial tachycardia

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9
Q

What is a sinus arrhythmia

A

A normal variant - rate changes on inspiration and expiration
- sinus tachycardia + ectopic beats

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10
Q

What is multi-focal atrial tachycardia

A

Like focal atrial tachycardia, but with multiple groups of cells taking it in turns to initiate cardiac cycle
- associated with COPD

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11
Q

How are narrow-complex tachyarrhythmias managed

A

Oxygen, 12-lead ECG and IV access
No adverse signs
- if irregular rhythm; probably AF
- regular rhythm; by definition is an SVT, and vagal manoeuvres should be attempted
- adenosine if this doesn’t work
- bet-blockers for possible atrial flutter if this fails
Adverse signs
- get help
- sedation and DC cardioversion (up to 3)
- correct electrolytes
- amiodarone and repeat shock as needed

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12
Q

Describe the use of adenosine in SVT

A

6mg bolus IV with 0.9% saline flush while recording a 12-lead rhythm strip
12mg bolus IV after 2 mins if initial dose unsuccessful
Max one more 12mg dose before attempting verapamil

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13
Q

What are the side effects of adenosine?

A

Chest tightness
Dyspnoea
Headache
Flushing

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14
Q

Contraindications of adenosine

A

Asthma
2nd/3rd degree heart block
Sinoatrial disease

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15
Q

Definition of broad-complex tachycardia?

A

Heart rate >100bpm
QRS >120ms
- no clear QRS complexes indicates VF or asystole

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16
Q

Possible causes of a broad-complex tachycardia

A

Ventricular fibrillation
Ventricular tachycardia
Any cause of narrow-complex tachycardia + BBB/metabolic causes of a wide QRS
Antidromic AVRT

17
Q

Describe ventricular fibrillation + ECG

A

Ventricles attempt to contract at rates up to 500bpm, this leads to a loss of cardiac output
Chaotic and no discernible pattern on ECG

18
Q

Describe ventricular tachycardia + ECG

A

Foci in ventricles discharge at a high frequency, causing an abnormal spread or charge through the ventricles = wide and abnormal QRS
Broad and regular QRS complex and no P waves

19
Q

What is Torsade de pointes

A

Polymorphic ventricular tachycardia - VT with a varying axis

- looks like ventricular fibrillation

20
Q

What in the history and examination could help you differentiate an SVT + BBB and VT?

A

Recent MI - most likely VT as it can cause ventricular damage
12-lead ECG
Response to certain medications

21
Q

What ECG findings suggest a patient is experiencing VT, not SVT + BBB?

A

Positive or negative QRS concordance in all leads
QRS >160ms
Marked left axis deviation
AV dissociated (p waves are independent of the QRS complexes)
Fusion or capture beats
RSR’ pattern - where R is taller than R’

22
Q

How is a broad-complex tachycardia managed?

A

Oxygen, 12-lead ECG and IV access
No adverse signs
- correct electrolyte abnormalities
- give amiodarone if rhythm is regular (adenosine instead if known history of SVT + BBB)
- seek expert help and give IV magnesium
- if none of this is successful; sedation and shock is required
Averse signs
- sedation and DC cardioversion (up to 3)
- correct electrolytes
- amiodarone and further cardioversion as needed

23
Q

How should the patient be managed once the VT is corrected?

A

Establish the cause
Maintain anti-arrhythmics (if MI was the cause) for 12-24 hours
- solatol or amiodarone
ICD or surgical isolation of arryhythmic area if required

24
Q

Why does VF require a non-synchronised DC shock?

A

Synchronised shocks use the R wave to trigger defibrillation - and VF doesn’t have an R waves

25
Q

What are the adverse signs in a broad complex tachycardia?

A

Low cardiac output - clammy, low blood pressure, decreasing consciousness
Oliguria
Angina
Pulmonary oedema

26
Q

What are the adverse signs in a narrow complex tachycardia?

A

Shock
Chest pain/ischaemia
Heart failure
Syncope