Tachyarrhythmias Flashcards
Classification
- Supraventricular and ventricular
- HR greater than 100bpm for 3 beats or more
Sinus tachycardia
- narrow QRS complex
- HR 150-180/min
Tachyarrhythmias result from:
Enhanced automaticity
Reentry or trigged activity
Supraventricular tachycardia
-reentry tachycardia and WPW symptoms
- palpitations
- pounding sensations in neck
- fatigue
- chest pain
- dyspnea
- dizziness
- sweating
Supraventricular tachycardia
- narrow QRS tachycardia
- wide QRS tachycardia
- if not hemodynamic stable –> vagal manouevers –> if not effective –> adenosine –> if not effective –> verapamil or diltiazem or beta blockers –> if ineffective –> synchronized cardioversion
if hemodynamic stable –> synchronized cardioversion
- if not hemodynamic stable –> vagal manouevers –> if not effective –> adenosine (if not evidence of pre-exictation on resting ECG) –> if not effective –> procanamide (?) and ? –> if not effective –>synchronized cardioversion
if hemodynamic stable –> synchronized cardioversion
Ventricular premature beat
- Common among healthy people, often asymptomatic and benign
- Avoid caffeine, stress, hypoxia…
- Reassurance, B blocker, ICD (implantable cardiac device)
Ventricular tachycardia
- Three or more consecutive PVC
- Sustained VT: persists >30s, severe symptoms such as syncope, required cardioversion, degenerates to VF
- Non-sustained: shorter and self-terminated
- Monomorphic vs polymorphic
Ventricular tachycardia
-symptoms
- palpitations
- chest pain/pressure
- dyspnea
- dizziness, hypotension, syncope, cardiogenic shock signs of reduced cardiac output
- cardiac arrest
Ventricular tachycardia
-treatment of sustained VT
hemodynamically unstable
- pulseless: PCR
- with pulse: polymorphic (defibrillate), monomorphic (cardioversion)
hemodynamically stable -Pharmacological conversion with antiarrhythmics: QT prolongation present: amiodarone No QT prolongation: procainamide Refractors VT: electrical cardioversion
Ventricular tachycardia
-Treatment of unsustained VT or resolved sustained VT
Identifiable reversible cause
- Correct any electrolyte imbalances
- Stop drugs that prolong QT
- Consider digoxin immune fab for digoxin toxicity
No identifiable cause
-Long term management with antiarrhythmics (b blockers), device therapy or ablation
Torsade de pointes
-treatment:
Type of polymorphic VT, can progress to VF
- Hemodynamically unstable: defibrillation plus CPR
- Hemodynamically stable: IV magnesium sulfate
- Identify and treat underlying etiology
Ventricular flutter
cardioversion
Ventricular fibrillation
electrical defibrillation
survivors: ICD