Table 52-6 Flashcards

1
Q

Hyponatremia

Risk factors:

A
  • loss of sodium
    • gastrointestinal fluid loss
    • sweating
    • use of diuretics
  • gain of water
    • hypotonic tube feedings
    • excessive drinking of water
    • excess IV D5W administration (dextrose in h2o)
  • syndrome of inappropriate ADH
    • head injury, aids, malignant tumors
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2
Q

Hyponatremia

Clinical manifestations:

A
  • lethargy, confusion, apprehension
  • muscle twitching
  • abdominal cramps
  • anorexia, nausea, vomiting
  • headache, seizures, coma

Lab findings:
Serum sodium < 135 mEq/L
Serum osmolality <280 mOsm/kg

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3
Q

Hyponatremia

Nursing interventions:

A
  • Assess clinical manifestations
  • monitor fluid intake and output
  • monitor lab data
  • assess client closely if administering hypertonic saline solutions
  • encourage food and fluid high in sodium
  • limit water intake as indicated
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4
Q

Hypernatremia

Risk factors:

A

-Loss of water
-insensible water loss
(hyperventilation or fever)
-diarrhea
-water deprivation
-Gain of sodium
-parental administration of saline solutions
-Hypertonic tube feedings w/o adequate water
-excessive use of table salt (1 tsp contains 2,300 mg of sodium
- conditions such as : diabetes insipidus, heat stroke

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5
Q

Hypernatremia

Clinical manifestations:

A

-thirst
-dry, sticky mucous membranes
-tongue dry, red, swollen
-weakness
-severe hypernatremia (fatigue, restlessness, decreased level of consciousness, disorientation, convulsions)
Lab findings:
Serum sodium >145mEq/L
Serum osmolality >300 mOsm/kg

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6
Q

Hypernatremia

Nursing interventions:

A
  • Monitor fluid intake and output
  • Monitor behavioral changes
  • Monitor lab findings
  • Encourage fluids as ordered
  • Monitor diet as ordered (restrict intake of salt/sodium)
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7
Q

Hypokalemia

Risk factors:

A
  • Loss of potassium
    • vomiting and gastric suction
    • diarrhea
    • heavy perspiration
    • use of potassium-wasting drugs
    • poor intake of potassium ( w/ debilitated clients, alcoholics, anorexia, nervosa)
    • hyperaldosteronism
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8
Q

Hypokalemia

Clinical manifestations:

A

-Muscle weakness, leg cramps
-Fatigue, lethargy
-anorexia, nausea, vomiting
-decreased bowel sounds, decreased bowel motility
-cardiac dysthymias
-depressed deep-tendon reflexes
-weak, irregular pulses
Lab findings:
Serum potassium < 3.5mEq\L
Arterial blood gases may show alkalosis
T-wave flattening & ST- segment depression on ECG

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9
Q

Hypokalemia

Nursing interventions:

A
  • Monitor heart rate and rhythm
  • Monitor clients receiving digitalis (digoxin) bc hypokalemia increases risk of digitalis toxicity
  • administer oral K+ with food or fluid to prevent gastric irritation
  • administer IV K+ at a rate no faster than 10-20mEq/h never administer undiluted K+ through IV
  • Monitor pain & inflammation in injection site
  • teach pt about K+ rich foods
  • teach pt how to prevent excess loss of K+ (diuretics)
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10
Q

Hyperkalemia

Risk factors:

A
  • decreased potassium excretion
    • renal failure
    • hypodosteronism
    • K+ conserving diuretics
  • high K+ intake
    • excessive use of K+ containing salt substitutes
    • excessive or rapid IV infusion of K+
    • K+ shift out of the tissue cells into the plasma (infections, burns, acidosis)
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11
Q

Hyperkalemia

Clinical manifestations:

A

-Gastrointestinal hyperactivity, diarrhea
-irritability, apthay, confusion
-cardiac dysrhythmias or arrest
-muscle weakness, areflexia (absence of reflexes)
-decreased heart rate, irregular pulse, paresthesias and numbness in extremities
-lab findings:
Serum potassium > 5.0 mEq/L
Peaked T-wave, widened QRS on ECG

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12
Q

Hyperkalemia

Nursing interventions:

A
  • closely monitor cardiac status and ECG
  • administer diuretics and other meds such as glucose and insulin
  • hold K+ supplements and K+ conserving diuretics
  • monitor K+ levels carefully, a rapid drop may occur as K+ shifts into cells
  • teach pt to avoid foods high in K+ and salt substitutes
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13
Q

Hypocalcemia

Risk factors:

A

-Surgical removal of the parathyroid glands
-conditions such as:
hypoparathyroidism, acute pancreatitis,
hyperphosphatemia, thyroid carcinoma
-inadequate vitamin D intake
-malabsorption
-hypomagnesemia
-alkalosis
-sepsis
-alcohol abuse

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14
Q

Hypocalcemia

Clinical manifestations:

A

-numbness, tingling of the extremities and around the mouth
-muscle tremors, cramps; if severe can progress to tetany and convulsions
-cardiac dysrhythmias ; decreased cardiac output
-positive trousseaus and chvosteks signs; confusion, anxiety, possible psychoses, hyperactive deep-tendon reflexes
Lab findings:
Calcium <8.5 mg/dL (total) or 4.5 mEq/L (ionized)
Lengthened QT intervals
Prolonged ST segments

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15
Q

Hypocalcemia

Nursing interventions:

A
  • Closely monitor respiratory and cardiovascular status
  • take precautions to protect a confused client
  • administer oral or parenteral calcium, when administering IV closely monitor cardiac status and ECG during infusion
  • Teach pt’s at high risk for osteoporosis: dietary sources rich in calcium, recommend 1000-1500 mg of calcium per day, calcium supplements, reg exercise, estrogen replacement therapy
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16
Q

Hypercalcemia

Risk factors:

A

-prolonged immobilization

Conditions as such: hyperparathyroidism, malignancy of the bone, Paget’s disease

17
Q

Hypercalcemia

Clinical manifestations:

A

-lethargy, weakness
-depressed deep tendon reflexes
-bone pain
-anorexia, nausea, vomiting
-constipation
-polyuria, hypercalciuria
-flank pain secondary to urinary calculi (kidney infection)
-dysrhythmias, possible heart block
Lab findings:
Calcium > 10.5 mg/dL total or 5.5 ionized
Shortened QT intervals, ST segments

18
Q

Hypercalcemia

Nursing interventions:

A
  • increase pt movement and exercise
  • encourage fluids to dilute urine
  • teach pts to limit intake of food and fluid high in calcium
  • encourage ingestion of fiber to prevent constipation
  • protect a confused pt; monitor for pathological fractures in pts w/ long term hypercalcemia
  • encourage intake of acid-ash fluids (prune juice) to counteract deposits of calcium salts in urine
19
Q

Hypomagnesemia

Risk factors:

A
  • excess loss from GI tract
  • long term use of certain drugs (diuretics )
  • conditions such as : chronic alcoholism, pancreatitis, burns
20
Q

Hypomagnesemia

Clinical manifestations:

A

-neuromuscular irritability w/ tremors
-increased reflexes, tremors, convulsions
-positive chvosteks and trousseaus signs
-tachycardia, elevated blood pressure, dysrhythmias
-disorientation & confusion
-vertigo, anorexia, dysphagia
-respiratory difficulties
Lab findings:
Magnesium <1.5mEq/L, prolonged QT intervals, depressed ST segments, broad flattened T waves, prominent U waves

21
Q

Hypomagnesemia

Nursing interventions:

A
  • Assess pts receiving digitalis for dig toxicity
  • hypomagnesemia increases the risk of toxicity
  • take protective measures when there is a possibility of seizures
    • assess the pts ability to swallow water prior to initiating oral feeding
    • initiate safety measures to prevent injury during seizure activity
    • carefully administer magnesium salts as ordered
  • encourage pts to eat magnesium rich foods
  • refer pts to alcohol treatment programs as needed
22
Q

Hypermagnesemia

Risk factors:

A

Abnormal retention of magnesium, as in :
Renal failure
Adrenal insufficiency
Treatment w/ magnesium salts

23
Q

Hypermagnesemia

Clinical manifestations:

A

-peripheral vasodilation, flushing
-nausea, vomiting
-muscle weakness, paralysis
-hypotension, bradycardia
-depressed deep tendon reflexes
-lethargy, drowsiness
-respiratory depression, coma
-respiratory and cardiac arrest if hypermagnesemia is severe
Lab findings:
Magnesium >2.5 mEq/L
ECG w/ prolonged PR interval, widened QRS, tall Twave

24
Q

Hypermagnesemia

Nursing interventions:

A
  • Monitor VS and level of consciousness when pts at risk
  • if patellar reflexes are absent, notify PHCP
  • advise pts who have renal disease to contact their provider before taking OTC meds