T&O - Fractures Flashcards

1
Q

Principles of Fracture Management

Reduce
Immobilise (Hold)
Rehabilitate

A

1.) Reduce - restore anatomical alignment
- usually requires reduction and counter traction
- ↓swelling, ↓nerve damage, restores blood supply
- local anaesthetic or conscious sedation needed

2.) Immobilise - simple splints or plaster casts
- plasters are not circumferential in first 2 weeks to allow the fracture to swell, preventing compartment syndrome
- if axial instability, plaster should cross the joint above and below e.g. ‘above knee/elbow’ plasters
- need thromboprophylaxis if unable to weight bear
- safety net for compartment syndrome

3.) Rehabilitate
- intensive period of physiotherapy
- patients should move unaffected joints

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2
Q

Open Fractures

Clinical Features
Investigations
Management
Rehabilitation
Outcomes/Complications of an Open Fracture x4

A

1.) Clinical Features
- pain, swelling, deformity, overlying wound/punctum
- check neurovascular status and overlying skin
- skin loss: plastic surgery input identified early

2.) Investigations
- routine bloods: inc clotting and G/S
- neuro examination should be repeated multiple times
- photograph, X-ray, CT (complex fractures)

3.) Management
- resuscitation, analgesia, anti-emetics
- realignment, wound dressings, and splinting
- IV antibiotic cover and tetanus vaccine (if needed)
- surgical debridement and fracture irrigation in theatre
- external fixation is used in widespread soft tissue damage whilst you wait for the skin and fracture to heal
- internal fixation once fracture has healed sufficiently
- soft tissue coverage must be obtained within 72hrs

4.) Rehabilitation
- after internal fixation: immobilised in splint/sling until fracture heals
- antibiotics to prevent infection
- healing time is variable and physio is needed

5.) Outcomes/Complications of an Open Fracture
- infection, neurovascular compromise, non-union
- compartment syndrome
- fat embolism (long bone fractures): up to 3 days post-trauma, fever, SOB, retinal haemorrhages, confusion, petechial rash

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3
Q

Classification and Severity Scores for Open Fractures

Gustilo-Anderson Classification x3(5)
Mangled Extremity Severity Score

A

1.) Gustilo-Anderson Classification - wound size
- 1: <1cm + clean, 2: 1-10cm + clean
- 3A: >10cm, high energy, good soft tissue coverage, OR farm injury
- 3B: >10cm , high energy, bad soft tissue coverage
- 3C: any injury with vascular injury
- 3A ortho only, 3B need plastics, 3C needs vascular

2.) Mangled Extremity Severity Score - used to predict necessity of amputation after lower extremity trauma
- skeletal/soft tissue injury: low energy (1-4) vs high energy
- limb ischaemia: no ischaemia (0-3) severe (no CRT)
- shock: stable (0-2) persistent hypotension
- age: <30 (0-2) >50
- score >7/11 predictive of amputation

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4
Q

Pathophysiology of Compartment Syndrome

Increase in Intra-Compartmental Pressure (ICP)
Increase in Hydrostatic Pressure
Compression of Traversing Nerves
Ischaemia

A

1.) Increase in Intra-Compartmental Pressure (ICP)
- due to fluid being deposited in fascial compartments which are closed and cannot be distended

2.) Increase in Hydrostatic Pressure
- due to venous compression because of ↑ICP
- causes fluid to move out of veins which increases intra-compartmental pressure even further

3.) Compression of Traversing Nerves
- causes a sensory +/- motor deficit distally
- means paraesthesia is a common symptom

4.) Ischaemia - when arterial inflow is compromised
- occurs when ICP reaches diastolic BP
- late sign of missed compartment syndrome
- acute arterial insufficiency (6Ps): pain, paraesthesia, pallor, perishingly cold, paralysis, pulselessness

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5
Q

Compartment Syndrome

Mechanism x3
Clinical Features
Investigations
Initial Management
Definitive Management

A

1.) Mechanism - main 3: high energy trauma, crush injuries, fractures causing vascular injury
- others: iatrogenic vascular injury, tight casts/splints, DVT, post-reperfusion swelling

2.) Clinical Features - within hrs or up to 4hrs post-injury
- severe pain, disproportionate to the injury (excessive use of analgesia)
- not improved with initial measures
- pain worsened by passive stretching of the muscle bellies of the area
- distal paraesthesia w/ evolving neurology
- tense fascial compartment (minimally distensible)
- 5Ps: if compartment syndrome is missed

3.) Investigations - clinical diagnosis
- elevated/rising CK may aid diagnosis
- intra-compartmental pressure monitor is only used in clinical uncertainty or if the patient is unconscious/intubated: >20 mmHg is abnormal whilst >40mmHg is diagnostic
- no visible pathology on an X-ray

4.) Initial Management
- aggressive IV fluids (AKI risk), high flow oxygen, IV analgesia
- keep limb at neutral level (DO NOT elevate or lower)
- remove all dressing/splints/casts

5.) Definitive Management - urgent fasciotomy
- skin incisions left open for 24-48hrs to assess for any dead tissue which will need to be debrided, wound is closed once remaining tissue are healthy
- monitor renal function due to the potential effects of rhabdomyolysis or reperfusion injury or myoglobinuria after a fasciotomy
- death of muscle groups may occur within 4-6 hours
- consider debridement and amputation if there are necrotic muscle groups at fasciotomy

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6
Q

Tibial Shaft Fractures

Mechanism
Clinical Features
Investigations
Management
Surgical Management

A

1.) Mechanism - vulnerable to both direct (fall, direct blow) or indirect injuries (twisting, bending)
- ↑risk of open fractures and compartment syndrome due to lack of significant soft tissue envelope

2.) Clinical Features
- severe pain w/ inability to weight bear
- may be a clear deformity and swelling and bruising
- full neuro exam needed, check skin for open fracture

3.) Investigations
- urgent bloods: inc clotting and G/S
- X-ray (AP+lateral) of tibia and fibula inc knee+ankle
- CT: intra-articular extension, spiral fracture of distal tibia, fracture of posterior malleolus

4.) Management
- reduction: MUA, re-X-ray, reassess neuro
- immobilise: above knee back slab, elevate
- non-operative: Sarmiento cast in closed stable tibial fractures can be an alternative to operation

5.) Surgical Management - required most of the time
- intramedullary (IM) nailing most commonly used for fixation, patients usually full weight bear immediately
- ORIF: may be needed for intra-articular extension
- temporary external fixation (for multiple injuries)
- fibula usually heals alone once tibia is stabilised
- can take 15-30 weeks (4-8mths) for union of the fracture

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7
Q

Neck of Femur Fracture (#NOF)

Mechanism
Avascular Necrosis
Clinical Features
Investigations
Post-Op Management

A

1.) Mechanism
- low energy fall in frail older patients
- high energy injury e.g. RTC or fall from height

2.) Avascular Necrosis - of the femoral head in a displaced intracapsular #NOF
- due to retrograde blood supply from MCFA

3.) Clinical Features
- pain in the hip or referred pain to the groin, thigh, or knee
- difficulty/no weight-bearing
- leg is shortened and externally rotated (gluteus medius)
- tenderness on palpation of the greater trochanter
- limited straight leg raise
- distal neurovascular deficits are rare (but must be assessed)

4.) Investigations
- X-ray: hip (AP and lateral), pelvis (AP), femur, can draw Shenton’s line to assess whether neck of femur fracture
- routine bloods: inc lactate, clotting, G/S, CK
- urine dip, CXR, ECG, AMTS esp for elderly

5.) Post-Op Management
- immediate post-operative weight bearing
- LMWH 6-12hrs after surgery for 28 days or 10 days followed by aspirin for further 28 days
- repeat bloods, X-ray, analgesia, IV fluids
- early rehab w/ physios and occupational therapists

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8
Q

Classification and Scoring Systems for #NOF

Garden Classification
Nottingham Hip Fracture Score
Mortality Figures

A

1.) Garden Classification - for intracapsular fractures
- I: incomplete #NOF, II: complete # but non-displaced
- III: complete #, partial displacement
- IV: complete # and fully displaced

2.) Nottingham Hip Fracture Score - 30 day mortality risk using:
- age, gender, AMTS, Hb on admission, where they live
- co-morbidities, active malignancy last 20 yrs
- raised serum lactate also indicator of ↑mortality

3.) Mortality Figures
- mortality: 10% in 1mth, 20% in 3 mths, 30% in 1 yr

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9
Q

Surgical Management of #NOF

Displaced Intracapsular
Non-Displaced Intracapsular
Inter-Trochanteric
Sub-Trochanteric

A

1.) Displaced Intracapsular - cemented hemiarthroplasty
- replace femoral head and neck
- total hip replacement (inc acetabulum) for patients who walk independently and not cognitively impaired
- can have aseptic loosening of a total hip replacement needing revision, presents with hip/groin pain radiating to the knee

2.) Non-Displaced Intracapsular - INTERNAL FIXATION with cannulated hip screws (for younger patients to prevent having prosthesis)
- three parallel screws in inverted triangle formation
- can consider hemiarthroplasty if unfit

3.) Inter-Trochanteric - dynamic hip screw (DHS)
- for fixation, dynamic lag screw provides compression and primary healing of the bone

4.) Sub-Trochanteric: intramedullary femoral nail
- titanium rod through medullary cavity for stabilisation
- also reduces pain

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10
Q

Types of Distal Radius Fractures

Pathophysiology
Colles Fracture
Smith’s Fracture
Barton’s Fracture

A

1.) Pathophysiology
- FOOSH causes forced supination or pronation of the carpus, increasing impaction load of the distal radius
- ↑risk w/ age/osteoporosis, children 5-15 also prone

1.) Colles Fracture - extra-articular fracture of the distal radius w/ dorsal angulation and displacement
- body load forces the wrist into supination
- typically occurs as fragility fracture in osteoporosis
- most common wrist fracture (90%)
- leads to dinner-fork deformity
- complications: median nerve injury (most common), compartment syndrome, malunion, vascular compromise, rupture of EPL tendon
- late complications: OA, complex regional pain syndrome

2.) Smith’s Fracture - extra-articular fracture of the distal radius w/ volar/palmar angulation +/- displacement
- falling backwards causing forced pronation
- leads to garden-spade deformity

3.) Barton’s Fracture - intra-articular fracture of the distal radius with dislocation of the radio-carpal joint
- can be volar (more common) or dorsal

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11
Q

Distal Radius Fracture

Clinical Features
Investigations
Management
Surgical Management
Complications x3

A

1.) Clinical Features
- traumatic episode with immediate pain +/- deformity and sudden swelling around the fracture site
- must assess evidence of neurovascular compromise (CRT, pulses, median/ulnar/radial nerve)

2.) Investigations
- X-ray (gold): radial height <11mm, radial inclination <22°, radial tilt >11°
- CT/MRI: if complex or for operative planning

3.) Management
- reduction: traction and MUA(haematoma/Biers block)
- immobilise: below-elbow backslab cast
- rehabilitation via physiotherapists

4.) Surgical Management - unstable fractures or intra-articular step of the radiocarpal joint >2mm
- ORIF with plating or K-wire fixation

5.) Complications
- malunion: poor realignment –> shortened radius causing ↓ROM, wrist pain, ↓forearm rotation
- MN compression: ↑ in malunion
- osteoarthritis

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12
Q

Classification of Ankle Fractures

Definition
Anatomical Classification
Weber Classification
Lauge-Hansen Classification

A

1.) Definition - fracture of any malleolus (lateral, medial, posterior) +/- disruption to the syndesmosis

2.) Anatomical
- isolated lateral/medial malleolar fractures
- bimalleolar and trimalleolar (lateral/medial/posterior)

3.) Weber - classifies lateral malleolus fractures
- type A: below syndesmosis, B: level of syndesmosis
- type C: above the level of the syndesmosis
- higher injury –> ↑likelihood of ankle instability
- type C fractures always need surgical fixation

4.) Lauge-Hansen - based on ankle position at time of injury and the deforming force involved
- more widely used and more detailed than Weber’s

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13
Q

Ankle Fracture

Clinical Features
Ottawa Ankle Rules
Investigations
Management
Surgical Management

A

1.) Clinical Features - traumatic episode followed by:
- ankle pain +/- associated deformity e.g. dislocation
- open fractures may have neurovascular compromise

2.) Ottawa Ankle Rules - X-ray requirements (near 100% sensitivity)
- bony tenderness at posterior edge/tip of LM OR MM
- inability to weight bear for 4 steps

3.) Investigations
- X-ray: mortise view (modified AP, internal rotation by 10-20°) + lateral view, taken in ankle dorsiflexion since the talus can appear translated when plantarflexed
- CT for complex fractures for surgical planning

4.) Management
- reduction: manage open fracture accordingly
- hold: below knee back slab, post-reduction neuro exam, repeat X-ray to ensure reduction is adequate
- conservative: non-displaced MM, Weber A, Weber B w/out talar shift, unfit for surgical intervention
- weight bear as tolerated in a CAM (controlled ankle motion) boot for 6 weeks

5.) Surgical Management - ORIF
- displaced bi/trimalleolar fractures, open fracture
- Weber C fracture, Weber B w/ talar shift
- talar shift is widening of the medial clear space which is suggestive of greater ankle instability

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14
Q

Fracture Healing

Pathophysiology of Fracture Healing
Factors affecting Fracture Healing
Ultrasound Stimulating Fracture Healing (EXOGEN)

A

1.) Pathophysiology of Fracture Healing - 5 steps
- haematoma –> inflammation –> callus formation –> consolidation –> bone remodelling

2.) Factors affecting Fracture Healing
- local: blood supply, denervation, infection, necrotic tissue, foreign bodies, surgical techniques
- systemic: age, anaemia, hypoxia, hypovolaemia, obesity, diabetes, malignancy, malnutrition, drugs

3.) Ultrasound Stimulating Fracture Healing (EXOGEN)
- stimulates production of GFs and proteins that ↑ the removal of old bone, ↑the production of new bone
- used to treat long bone fracture w/ non-union or delayed healing
- adv: quicker healing, avoid surgery, self-administered

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15
Q

Scaphoid Fracture

Pathophysiology
Clinical Features
Investigations
Management

A

1.) Pathophysiology - a type of wrist fracture, often due to a FOOSH causing axial compression of the scaphoid with the wrist hyperextended, and radially deviated
- often occurs during contact sports or RTAs
- proximal injuries can cause avascular necrosis due to the retrograde blood supply from the radial artery

2.) Clinical Features
- pain at the base of the thumb
- maximal tenderness over the anatomic snuffbox and tenderness of the scaphoid tubercle
- loss of grip/pinch strength
- wrist joint effusion: less likely if hyperacute or delayed
- pain during ulnar deviation of the wrist

3.) Investigations
- wrist X-Ray: AP, lateral, ‘scaphoid views’. can be missed within the first week of the injury
- MRI: technically first-line but X-Rays still often used
- CT: only to plan operative Mx or fracture union

4.) Management - referral to orthopaedics
- immobilisation w/ a (futuro) splint and X-ray
- another X-ray after 1 week if the first X-ray is normal
- displaced or proximal pole fractures: needs surgical fixation
- undisplaced fractures: below-elbow cast for 6-8 weeks
- complications: avascular necrosis, non-union → pain and early osteoarthritis

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16
Q

Paediatric Fractures

Fracture Types
Growth Plate Fractures
Non-Accidental Injury (NAI)

A

1.) Fracture Types
- greenstick fracture: unilateral cortical breach only
- complete fracture: both sides of cortex are breached
- toddlers fracture: spiral or oblique undisplaced fracture of the distal shaft of the tibia with an intact fibula
- plastic deformity: stress on bone resulting in deformity without cortical disruption
- buckle (‘torus’) fracture: periosteal haematoma due to incomplete cortical disruption

2.) Growth Plate Fractures - Salter-Harris Classification
- I: physis only (x-ray often normal), II: physis + metaphysis
- III: physis + epiphysis (inc joint), IV: physis+metaphysis+epiphysis
- V: crush injury involving the physis (x-ray may resemble type I)
- III, IV and V will usually require surgery

3.) Non-Accidental Injury (NAI)
- multiple injuries, injuries at sites not commonly exposed to trauma
- lack of concordance between proposed and actual mechanism of injury
- delayed presentation, delay in reaching milestones
- children on the at risk register