T&O - ankle and foot Flashcards

1
Q

What makes up the ankle joint?

A
  • Talus bone articulates with the mortise (medial and lateral malleolus)
  • Tibia and fibula join at the syndesmosis - a strong sutrcutre comprised of anterior inferior tibiofibular ligament and posterior inferior tibiofibular ligament
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2
Q

How are ankle fractures classed anatomically?

A
  • Isolated lateral malleolar
  • Isolated medial malleolar
  • Bimalleolar (medial + lateral)
  • Trimalleolar (medial + lateral + posterior malleolar)
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3
Q

How can lateral malleolar fractures be further classified?

A

Weber Classification

  • Type A - below the syndesmosis
  • Type B - at the level of the syndesmosis
  • Type C - above the level of the syndesmosis
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4
Q

How does the location of lateral malleolar fractures affect the management?

A
  • More proximal fractures have highter chance of ankle instability
  • Therefore type C fractures almost always need surgical fixation
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5
Q

How do patients present if they have an ankle fracture?

A
  • Traumatic injury followed by ankle pain
  • May be an associated deformit
  • May have neurovascular compromise if very deformed
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6
Q

What are the Ottawa ankle rules?

A

If there is any uncertainty e.g. no deformity but cannot monbilise ankle use these rules. Presence of any of the features means plan radiographs must be taken

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7
Q

In what situations can the Ottawa ankle rules not be applied?

A
  • Intoxicated/ Uncooperative
  • Other distracting painful injuries
  • Diminished leg sensation
  • Gross swelling
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8
Q

How are suspected ankle fractures investigated?

A

Plain radiograph AP and Lateral

Ankle must be in full dorsiflexion when the X-Ray is taken

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9
Q

Which patients are managed conservatively for ankle fractures?

A
  • Non-displaced medial malleolus fractures
  • Weber A or B fractures without talar shift
  • Patients unfit for surgery
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10
Q

When is ORIF indicated for ankle fractures?

A
  • Displaced bimalleolar or trimalleolar fractures
  • Weber C classifications
  • Weber B classifications with talar shift
  • Open fractures
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11
Q

What complications can follow an ankle fracture?

A
  • Post traumatic arthritis (Rare if appropriately reduced and fixated)
  • If undergone ORIF:
    • Surgical site infection
    • DVT
    • PE
    • Neurovascular injury
    • Non union
    • Metalwork prominence
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12
Q

Which tarsal bone is the most commonly fractured?

A

Calcaneum

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13
Q

By what mechanism do calcaneal fractures occur?

A

Usually a fall from high with significant axial loading onto the bone

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14
Q

How are calcaneal fractures divided on the basis of subtalar involvement?

A
  • Intra-articular (75% of calcaneal fractures)
    • involves articular surgace of the subtalar joint
  • Extra-articular (25% of calcaneal fractures) \
    • These are commonly avlusion fractures includin avulsion of the calcaneal tuberosity by the achilles tendon
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15
Q

How do calcaeneal fractures appear clinically?

A
  • Recent trauma eg fall from height or RTA
  • Pain and tenderness around calcaneal region
  • Inability to weight bear
  • O/E : swollen, bruised with potential shortened and widened heel
  • May have varus deformity
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16
Q

What imaging should be done in suspected calcaneal fracutres?

A
  • Plain radiograph: AP, lateral and oblique views
  • CT imaging is the gold standard and should be done in any syspected case
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17
Q

How are calcaneal fractures managed?

A

Majority intra-articular fractures require surgical intervention unless <2mm displaced or a near normal Bohler’s angle

Extra-articular fractures are treated non-operatively unless there is significant displacement. Cast + NWB for 10-12 weeks

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18
Q

What is achilles tendonitis?

A

Inflammation of the achilles tendon

Prevalent in those who engage in high intensity activities causing the tendon to chronically overload

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19
Q

Describe the pathophysiology behind achilles tendonitis?

A
  • Repetitive action of the tendon causes microtears leading to localised inflammation
  • Overtime the tendon becomes thickened and loses elasticity
  • Rupture can aoocur when a sudden force is aplplied across the tendon
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20
Q

What are some of the risk factors for achilles tendonitis or rupture?

A
  • Unfit individuals who have a sudden increase in exercise frequency
  • Poor footwear
  • Male
  • Obesity
  • Recent floroquinolone use
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21
Q

What are the clinical features of achilles tendonitis?

A
  • Gradual onset of pain and stiffness at the posterior ankle
  • Worse with movement
  • Can imprve with mild exercise or heat application
  • O/E there is tenderness over the tendon, usually worst 2-6cm above insertion site
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22
Q

What are the clinical features of an achilles tendon rupture?

A
  • Sudden onset severe pain in posterior calg
  • Audible popping sound
  • Loss of power on ankle plantarflexion
23
Q

What clinical test can be done to indicate achilles tendon rupture?

A

Simmond’s Test

24
Q

How can achilles tendonitits or tendon rupture be investigated?

A
  • Both are usually clinical diagnoses
  • If unsure can use ultrasound
25
Q

How is achilles tendonitis managed?

A
  • Stop precipitating exercise
  • Ice the area
  • Anti-inflammatory medication
  • Rehabilityion and physio in chronic cases
26
Q

How is achilles tendon rupture managed?

A
  • Initial (acute partial thickness or full thickness)
    • analgesia
    • immobilisation splinted in plaster with ankle and toes fully pointed
    • crutches and no weight bearing for 2 weeks
  • Then ankle is brought to a partial part for 4 weeks
  • Then brought to a neutral positio for 4 weeks
27
Q

When are surgical fixations (end to end tendon repairs) performed in the case of ruptured achilles tendons?

A
  • Delayed presentations (>2 weeks)
  • Re-rupture
28
Q

By what mechanism for talar fractures occur?

A

High energy trauma e.g. fall from height or RTA when the ankle is forced into dorsiflexion

29
Q

Where do the majority of talar fractures occur anatomically?

A
  • 50% are through the talar neck
  • Less commonly through the body, lateral or posterior process
30
Q

Why are talar fractures at high risk of avascular necrosis?

A

The talus relies on extraosseous arterial supply which is highly susceptible to interruption when fractured

31
Q

How should the ankle be posistioned when assessing for talar fractures?

A

Take in both dorsiflexion and plantarflexion

32
Q

How are talar fractures classified?

A

Hawkins Classification

33
Q

How does the risk of AVN relate to the Hawkins Classification

A
  • Type 1: 0-15% risk of AVN
  • Type 2: 20-50% risk of AVN
  • Type 3: 90-100% risk of AVN
  • Type 4: 100% risk of AVN
34
Q

How are talar fractures managed?

A

Type 1 fractures: conservatively, plaster and non-weight bearing crutches for 3 months

Type 2-4 fractures: attempt closed reduction and cast

Definitive surgical fixation is required the next day if reduction is unsuccessful

35
Q

What are some of the complications of talar fractures?

A
  • Avascular necrosis
  • Osteoarthritis secondary to AVN or malunion
36
Q

What is a talar pilion fracture?

A

Severe injury to the distal tibia that are articularly impacted and severly comminuted

37
Q

How do Talar Pilon fractures appear on examination?

A
  • May have obvious ankle deformity
  • Associated significant swelling and burising
  • Skin blistering may occur after several hours
  • Look for evidence of open fracture
  • Look for signs of compartment syndrome
  • Check peripheral pulses
38
Q

How are Pilon fractures classified?

A
39
Q

How are talar pilon fractures managed?

A
  • Initial management: realign the limb and apply below-knee backslab and repear neurovascular assessment
    • Elevate limb, keep NBM to prep for surgery
  • Majority are treated operatively unless in cases where surgery is high risk
    • reconstruct the articular surgact and restore alignement of ankle mortise
    • usually done in a staged approach
    • Temporary external fixator then definitive ORIF 7-14 days later
40
Q

What complications can occur following a talar pilon fracture?

A
  • Compartment syndrome
  • Wound infection or dehisence
  • Delayed or non union
  • Post traumatic arthritis
41
Q

What are Lisfranc injuries?

A

Severe injury to the tarsometatarsal joints can be solely ligamentous or involve the bony structures of the midfoot

42
Q

What are the clinical features of a Lisfranc injury?

A
  • Hx of severe torsional or translational forces through a plantar flexed foot
  • Severe pain - provoked by stressing the midfoot
  • Difficulty weight bearing
  • Swelling and tenderness over the midfoot
  • Plantar bruising
  • Piano key sign (prominence of metatarsal bones)
43
Q

How are Lisfranc injuries managed?

A
  • Ensure haemodynamic stability
  • Closed reduction in A&E to correct any cross deformity
  • Conservative management can be done if there is no significant displacement (cast immobilisation + non weight bearing for 6-12 weeks)
  • Surgical fixation usually a screw between medial cuneiform and 2nd metatasal if there is clear displacement
44
Q

What is Hallux Valgus?

A

Deformity of the 1st metatarsophalangeal joint with medial deviation of the 1st metatarsal and associated joint subluxation

45
Q

What are the main risk factors for developing hallux valgus?

A
  • Female
  • Connective tissue disorder
  • Hypermobility syndromes
  • Anatomical variants predispose to mal-alignment of 1st MTP
46
Q

What are the clinical features of hallux valgus?

A
  • Painful medial prominence
    • aggravateed by walking or weight wearing activities
    • aggravated by narrow toes shoes
  • Contracture of the extensor hallucis longus may be visible in long standing joint subluxation
  • Excessive keratosis may indicate abnormal weight distribution
47
Q

What is the main investigation for hallux valgus?

A

Plain radiographs to measure the degree of lateral deviation and join subluxation

48
Q

How can hallux valgus be managed?

A
  • Analgesia
  • Adjust footwear
  • Physiotherapy
  • Surgery
49
Q

What are the basic principles of surgery for Hallux Valgus? When is it offered?

A

Offered if QoL is significantly impacted by the condition

Surgery either involved osteotomy of the first metatarsal or fusion of metatarsal to the medial cuneiform

50
Q

What is plantar fasciitis? Describe the pathophysiology

A

Inflammation of plantar fascia - connective tissue from medial process of calcaneal tuberosity to the proximal phalanges causing infracalcaneal pain

Theory: micro-tears to the plantar fascia occur causing chronic breakdown of the structure

51
Q

Give some of the risk factors of plantar fasciitis

A
  • Anatomical factors - excessive prontation or high arches
  • Weak plantar flexors or tight gastrocnemius or soleus
  • Prolonged standing
  • Excessive running
  • Leg legnth discrpancy
  • Obesity
  • Unsupportive footwear
52
Q

How does plantar fasciitis present clinically?

A
  • Sharp pain on plantar aspect of foot - mainly at the heel
  • Can radiate to the arch distally
  • Tends to be worse with the first few steps of the day or after periods of inactivty then eases off
  • O/E: look for over pronation, high arches, leg legnth discrepance, femoral anteversion, infracalcaneal region often tender on palpation
53
Q

How is plantar fasciitis diagnosed?

A

Clinically - investigations only done if there is a doubt in the diagnois

Plain X-rays can be done to exclude bony injury if there is a history of trauma - may see a heel spur in 50% of plantar fasciitis cases

54
Q

How is plantar fasciitis managed?

A
  • Activity moderation
  • Regular analgesia (typically NSAIDs)
  • Adjust footwear - well cushioned heel
  • Physiotherapy
  • Corticosteroid injections can be trialled if no improvement
  • Plantar fasciotomy can be considered if all else fails