SYNCOPE Flashcards
DDX
Basic Things:
Arrythmia
Ischemia
Exotic Things:
Arrythmogenic Right Ventricular Dysplasia (epsilon waves, flipped t waves V1-V3)
Brugada (RBBB, STE, V1-V3)
Left Ventricular Hypertrophy (marked high voltage)
Pulmonary Embolism (Right axis shift, S1Q3T3, RBBB, Deep flipped t waves V1-V3)
QT too long / too short (Qt > 1/2 RR)
Wolf-Parkinson White
HOCM
Bifascicular Block
Brugada Sydrome: Criteria
Management
ECG CRITERIA:
“RBBB with STE in V1-V3”
RBBB in V1-V3
J-point elevation
Saddle shaped or sloped ST segment elevation
Inverted t wave
MANAGEMENT:
Stable: close outpatient follow up with cardiologist
Unstable or drop attacks: admission and monitoring with cardiology consultation
Strict instructions to aboid QT prolonging drugs
Long-QT Syndrome: Criteria
QT Greater than 470 ms in men
QT greater than 480 ms in females
QTc increases the risk of arrythmia: > 500 ms
HOCM
Large amplitude QRS
Left ventricular hypertrophy with increased precordial voltages
non-specific ST segment and T-wave abnormalities
Deep, narrow (“dagger-like”) Q waves in lateral (I, aVL, V5-6) and/or inferior (II, III, aVF) leads
Pulmonary Embolism
1) Sinus Tachycardia
2) Right Axis Deviation
3) Incomplete or complete RBBB
4) Right Ventricular Strain Pattern:
T wave inversion inferior and anteroseptal leads
5) S1, Q3, T3 Pattern:
Large S wave in lead I
Small Q wave in lead III
Inverted T wave in lead III
QT too long / too short (Qt > 1/2 RR)
Wolf-Parkinson White
HOCM
LVH
Sokolov-Lyon criteria:
S wave depth in V1 + tallest R wave height in V5-V6 > 35 mm
Non-Voltage Criteria:
STE Concave limited to V1 – V3
STE is modest in relation to large voltages
STD and strain (T Wave Inversion) lateral leads (V5, V6, I, aVL)
Changes to LBBB over time
STD are downslope due to subendocardial ischemia
Precordial Leads:
R wave in V4, V5 or V6 > 26 mm
Limb Leads:
R wave in lead I + S wave in lead III > 25 mm
R wave in aVL > 11 mm
R wave in aVF > 20 mm
S wave in aVR > 14 mm
Wolff-Parkinson-White
Triad of:
PR Interval <120 msec
Delta Wave (Initial Slurring of the QRS ) in at least 2/3 orthogonal leads (I, aVF, V2)
Prolonged QRS complex >110 msec
a/w:
Discordant ST and T Changes (inverted T Waves) in opposite direction of the QRS complex
MANAGEMENT:
UNSTABLE:
Synchronized cardioversion (150-200 J)
STABLE:
Vagal maneuvers
Adenosine
Procainamide: 15 to 17 mg / kg IV over 20-30 min until 50% QRS widening
DO NOT USE NODAL BLOCKING AGENTS INCLUDING BETA BLOCKERS OR CCB’S
BIFASCICULAR BLOCK
RBBB plus either LAD or RAD