Surgical Infections Flashcards
Temperature
<36 C or >38 C
HR
> 90 beats per minute
RR
> 20 breaths per minute
PaCO2
< 32 mmHg
WBC Count
<4000 or >12000 cells/mm3 or >10% immature forms
UTI
3-5 days
Pneumonia
7-10 days
Bacteremia
7-14 days
Penetrating gastrointestinal trauma
12-24 hours
Perforated or gangrenous appendicitis
3-5 days
Peritoneal soilage secondary to perforated viscus with moderate contamination
5-7 days
Extensive peritoneal soilage (feculent peritonitis) in the immunocompromised host
7-14 days
Only skin microbiota
Class I: Clean
Opened w/o significant spillage
Class II: Clean contaminated
Open accidental wounds
Class III: Contaminated
Traumatic wound + significant treatment delay
Class IV: Dirty
Necrotic tissue, purulent discharge
Class IV: Dirty
Introduction of bacteria due to major breaks in sterile technique (open cardiac massage)
Class III: Contaminated
Perforated viscus high degree of contamination
Class IV: Dirty
Hernia repair
Class I: Clean
Perforated diverticulitis
Class IV: Dirty
Breast biopsy specimen P device (mesh, valve)
Class I: Clean
Cholecystectomy
Class II: Clean contaminated
Elective GI surgery (not colon)
Class II: Clean contaminated
Penetrating abdominal trauma
Class III: Contaminated
Necrotizing soft
tissue infections
Class IV: Dirty
Large tissue injury
Class III: Contaminated
Enterotomy during bowel obstruction
Class III: Contaminated
Occurs when microbes invade normally sterile peritoneal cavity via hematogenous dissemination from distant source of infection or direct inoculation
Primary microbial peritonitis
Common among patients who retain large amounts of peritoneal fluid due to ascites and those treated for renal failure via peritoneal dialysis
Primary microbial peritonitis
Diffuse tenderness
Primary microbial peritonitis
Guarding without localized findings
Primary microbial peritonitis
Absence of pneumoperitoneum
Primary microbial peritonitis
Occurs subsequent to contamination of the peritoneal cavity due to perforation or severe inflammation and infection of an intra-abdominal organ
Secondary microbial peritonitis
Poorly understood entity that is more common in immunosuppressed patients
Tertiary (Persistent) Peritonitis
Peritoneal host defences do not effectively clear or sequester the initial secondary microbial peritoneal infection
Tertiary (Persistent) Peritonitis
Develops in the absence of original visceral organ
Tertiary (Persistent) Peritonitis
Caused by manipulation of the biliary tract to treat a variety of diseases but nearly 50% of patients have no identifiable cause
Pyogenic liver abscess
Should be sampled and treated with a 4 to 6 week course of antibiotics
Small (< 1 cm), multiple abscesses
Parameters for antibiotic therapy and drain removal for larger abscesses:
- Clear evidence cavity collapse
- Output <10 - 20 mL/d
- No evidence of ongoing source of contamination
- Clinical condition improved
Dosage of metronidazole for amoebic liver abscess:
750 mg TID x 10 days
Current care includes staging with dynamic, contrast material enhanced helical CT scan to evaluate the extent of pancreatitis coupled with the use of one of several prognostic scoring systems.
Severe Acute Pancreatitis
Placed past the ligament of Treitz, associated with decreased development of infected necrosis → decreased gut translocation of bacteria
Nasojejunal feeding tubes
Dilation of the retroperitoneal drain tract
VARD (Video-Assisted Retroperitoneal Drainage)
Debridement of pancreatic bed
VARD (Video-Assisted Retroperitoneal Drainage)