Stuttering Midterm Flashcards

1
Q

Stuttering definition

A

a communication disorder in which the flow of speech is broken by repetitions, prolongations, or abnormal stoppages of sounds and syllables; defined by symptoms rather than the underlying etiology

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2
Q

Stuttering diagnosis

A
  • presence of core behaviors
  • occurs at the level of the syllable
  • > 3 disfluencies per 100 syllables
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3
Q

Developmental stuttering

A

the most common form of stuttering that begins in childhood during intense speech and language acquisition

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4
Q

Neurogenic stuttering

A

rare syndrome in which stuttering symptoms result from a head injury or stroke

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5
Q

Psychogenic stuttering

A

rare syndrome in which stuttering symptoms arise while a person is coping with mental illness

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6
Q

Cluttering

A

an uncommon developmental disorder in which language planning is disorganized, resulting in disfluencies

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7
Q

Worldwide prevalence

A

70 million people worldwide (1% of the population)

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8
Q

Onset

A

usually begins between 2 and 3 when children are developing complex syntax and lasts 1-2 years; decreases toward school age

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9
Q

Treatments for adults

A

combine approaches to increase communication and decrease impacts

  • stuttering modification
  • fluency shaping
  • speech easy (DAF technology included in a unilateral hearing aid)
  • pharmaceuticals (negative side effects)
  • cognitive treatments
  • counselling
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10
Q

Incidence

A

how many people have ever stuttered or the number of new cases during a given time period; estimated around 5%
- higher than prevalence because some children will recover

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11
Q

Prevalence

A

how many people stutter at a given time; estimated ~1%

- does not vary across countries

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12
Q

Probable risk factors

A
  • heritability
  • age
  • biological sex
  • bilingualism (if genetic)
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13
Q

Heritability

A

the only confirmed risk factor; people who have a close family member who stutters have a much higher probability for stuttering than people who don’t

  • 20-75% of people who stutter have a positive family history of stuttering (~10% in normally fluent people)
  • females who stutter are more likely to have family members who stutter (likely to pass it on)
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14
Q

You can induce fluency by

A

a) Speaking in a non-habitual manner
b) Choral reading
c) Shadowing
d) Singing
e) Using a metronome
f) Masking noise
g) DAF
h) Slow speech
i) Monotone

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15
Q

Why induce fluency?

A

1) Demonstrates that fluency is possible
2) Probing severity of stuttering
3) Testing therapy approaches
4) Differential diagnosis

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16
Q

Adaptation effect

A

stuttering decreases over successive readings of a passage

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17
Q

Consistency effect

A

stuttering occurs on the same words while reading a passage more than once

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18
Q

Adjacency / clustering effect

A

stutters occur on nearby words that have also been stuttered on

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19
Q

Anticipation effect

A

tendency to predict the words on which a person is going to stutter

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20
Q

Natural recovery

A

Recovery without intervention that occur within 2-3 years after onset

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21
Q

Estimated percentage of recovery

A

50-80%

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22
Q

Earlier onset age is better/worse for natural recovery?

A

Better

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23
Q

Repetitions are more common to the _______ group

A

Recovery

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24
Q

Blocks are more common to the _______ group

A

Persistent

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25
Stage models of stuttering suggest that
Stuttering begins as slow and easy tempo, becomes more tense, and then results in social impact and fear
26
Cluttering characteristics
1) Fast or blurred speech 2) irregular rate (robo, jerky, choppy) 3) Many disfluencies 4) Inadequate self-monitoring 5) Language planning and organization problems
27
Cluttering is fast or slow speech?
Fast
28
Fluency assessment of cluttering (3 things)
1) Maze material 2) Revisions 3) Incomplete phrases
29
Rate assessment of cluttering (2 things)
1) Objective counts (syllable/min) | 2) Perceptual rate on scale
30
Clarity assessment of cluttering (3 things)
1) Collapsed words 2) Omitted syllables 3) Slurred sound
31
Treatment hierarchy for cluttering
1) Slow rate 2) Increase self-awareness 3) Reduce disfluencies 4) Improve clarity 5) Improve language organization
32
Hypothesis
Testable question
33
Levelt model
Message is conceived and goes through language formation and delivery process (need picture)
34
Onset types and time frames
Sudden - one day (40.7%) Intermediate - over 1 -2 weeks (32.1%) Gradual - over 3-6 weeks (27.1%)
35
Secondary behaviors
1) Eye blinking 2) Hand or finger movements 3) Nods
36
Is phonological ability related to stuttering severity?
Evidence indicates that it is not
37
Does speech difficulty increase with age for those with stutters?
Yes
38
Children stutter more on ______ words than content words.
Function words (because their language is weighted more toward this)
39
Adults stutter more on ______ words than function words.
Content words (because their language is weighted more toward this)
40
Males who stutter have a
22% risk of having a son who stutters 9% risk of having a daughter who stutters
41
Females who stutter have
36% risk of having a son who stutters 17% risk of having a daughter who stutters
42
Best fitting genetic model
Environmental factors, interacting with many genes, and a single major locus
43
Gender risk factors (child and adult)
4: 1 for teens and adults 2: 1 for children
44
Stuttering is specifically onset at this ages (in months)
30-33
45
Guitar's perspective
Environmental influences such as criticism/stressful events can exacerbate and perpetuate persistent stuttering
46
Borderline stuttering | Guitar
- 10 stutters in 100 words - Little awareness/embarrassment - Might not be clinical stuttering
47
Brown's factors (loci of stuttering)
1) Consonants 2) Word initial sounds 3) Connective speech 4) Content words 5) Longer words 6) Utterance initial words 7) Stressed syllables
48
Breakdown model
Stuttering results from a momentary breakdown in speech motor coordination
49
Multifactorial model
Stuttering is the result of dynamic changing interactions of factors that reside in both the individual and the environment
50
Shifting Perspective Theory #1. Diagnosogenic theory
Inappropriate labelling of normal disfluencies as stuttering by parents and parent behavior reinforces and worsens disfluencies
51
Shifting Perspective Theory #2. Psychoanalytic theory
Stuttering is the result of unresolved infantile psychosexual need and conflict
52
Shifting Perspective Theory #3. Guitar integrating theories
Primary stuttering: involves genetics and brain structure Secondary: involves temperament and emotional responses interacting with learning *reactive temperament makes stuttering worse (but the problem is that well adjusted people still stutter)
53
Stuttering Moment Theory #1. Demands and capacities model
Language capacities are overwhelmed by the environment
54
Stuttering Moment Theory #2. Covert repair model
Stuttering occurs from internal monitoring and repairing what a person means to say
55
Stuttering Moment Theory #3. Breakdown model
A breakdown in speech-motor coordination
56
Stuttering Moment Theory #4. Reduced capacity for internal modeling
Stuttering results from a weak internal model. Their model is inaccurate when comparing input to desired output
57
Stuttering Moment Theory #5. Central processing - concurrent attention demanding tasks (Bosshardt)
Phonological and articulatory systems are more prone to interference by concurrent processing (dual-tasks mess PWS up)
58
Cause of Stuttering Theory #1. Cerebral dominance
PWS have not developed LH dominance
59
Cause of Stuttering Theory #2. Communicative failure and anticipatory struggle
Anticipation of difficult speak triggers more disfluencies
60
Cause of Stuttering Theory #3. Multi-factorial model
Stuttering is the result of dynamic, changing interactions that reside in both the environment and the individual
61
Cause of Stuttering Theory #4. Disorder of brain organization
RH might be overdeveloped to compensate for underdeveloped LH