Stroke, COPD, MS Flashcards

1
Q

What is a stroke?

A

Interruption of perfusion to the brain that results in infarction (brain death). Severity depends of location/extent of brain involved.

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2
Q

Time of neurological metabolism, metabolism, and cell death altered because of stroke?

A

NM- 30 seconds
M- stops in 2 minutes
CD- 5 minutes

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3
Q

What is a transient ischemic attack? (TIA)

A

Brief episode of neurological dysfunction (usually less than 1 hr). Caused by ischemia but no cell death. Includes visual/mobility/sensory/perception/speech symptoms. It is an intermittent blockage and treated as a stroke.

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4
Q

Symptoms of TIA?

A

Slurred speech (muscle paralysis of speaking), vertigo, aphasia. Symptoms will resolve.

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5
Q

What is an ischemic stroke and types?

A

Caused by a clot. 2 types are thrombotic and embolic.

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6
Q

What is a thrombotic stroke?

A

Narrowing of artery by fatty deposits of plaque. Causes a clot to form which will block entry of blood through the artery.

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7
Q

Embolic stroke?

A

Embolus is a blood clot that circulates in blood and moves. It blocks blood flow to brain. Can be caused by atrial fibrillation.

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8
Q

What is atrial fibrillation?

A

Irregular heart rhythm so blood will pool in atrium and form a clot that is released into blood stream.

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9
Q

What is a hemorrhagic stroke?

A

Bursted blood vessel allow blood to seep into and damage brain tissues until clotting shuts off the leakage. Described as the worst headache of their life. There’s 3 types (aneurysm, ateriovenous malformation (born with it, blood vessel weak and at risk for breaking open).

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10
Q

Risk factors for stroke?

A

Smoking, obesity, hypertension, diabetes, high cholesterol, family hx of atherosclerosis.

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11
Q

History (things we want to know for a stroke)?

A

Want to know time last seen normal, patients LOC, medications, hx of atrial fibrillation, have they experienced these symptoms before, any impaired cognition/mobility/sensory perception.

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12
Q

What do we do for health promotion for stroke?

A

Modify risk factors, control BP, use aspirin, smoking cessation, and manage cholesterol.

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13
Q

Lab/assessments for stroke?

A

No lab assessments confirm a stroke by APTT gives us a baseline for anticoagulation therapy. CT is a 3D image of brain that tells us type of stroke they’re having. We want to get them in CT scanner within first 60 minutes.

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14
Q

What does fast stand for?

A

F- face (is it drooping)
A- arms (can you raise both)
S- speech (is it slurred or jumbled)
T- time, call 911 right away and act fast

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15
Q

What is the Canadian neurological scale?

A

Assess LOC/orientation/speech (give commands and ask questions, they answer correctly). Motor (assess face, arms, legs). Test expression by give people items and ask patient to identify them.

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16
Q

Characteristics of symptoms with stroke?

A

Symptoms usually don’t differ between types of strokes, they are related to the location of stroke, and many body functions are affected.

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17
Q

Goal of stroke therapy?

A

Re-establish blood flow.

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18
Q

What is fibrinolytic therapy?

A

Busts the clots and its given IV. Do CT scan first the find out type of stroke. Rule out hemorrhagic stroke and this therapy is only given to ischemic stroke. Give 4.5 hrs after onset of symptoms (less effective after 4.5 hrs because damage has already occurred). Want to reverse and minimize obstruction effects.

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19
Q

Embolectomy?

A

Remove a clot by surgically pulling it out.

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20
Q

What is a carotid artery angioplasty/stent?

A

Open up the artery by putting in a balloon or stent.

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21
Q

What medications can you use to treat a stroke?

A

Anti platelet agents (aspirin or clopidogrel). Anticoagulants (prevents further episodes of stroke, more for ischemic strokes). Nimodipine administered to hemorrhagic stroke to relax blood vessel of cerebrosystem. Stool softeners/analgesics/anti-anxiety meds all reduce inter cranial pressure.

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22
Q

What is aphasia?

A

Loss of comprehension/use of language or inability to communicate.

23
Q

What is expressive aphasia (broca’s aphasia)?

A

Difficulty expressing their thoughts through speech/writing. They understand what’s being said but unable to answer.

24
Q

What is receptive aphasia (wenickes aphasia)?

A

Difficulty understanding spoken/written language. Able to speak but words they say might be used incorrectly.

25
Q

What is dysarthria?

A

Difficulty with motor function to tongue/muscles of speech (pronunciation, articulate, phonate- act of producing sound/voice).

26
Q

Communication strategies for stroke?

A

Writing pad, simple commands, body language (gesturing), speak slow/calm.

27
Q

What is COPD?

A

Preventable, progressive, respiratory disorder that causes systemic symptoms with increasing frequency/severity of exacerbations. Major cause is smoking. Partially reversible airway obstruction/lung hyperinflation. Causes chronic inflammation in airways/bronchioles/alveoli. Mucus hypersectrion is normal.

28
Q

What is emphysema?

A

Alveoli problem. They become damaged and there’s limited gas exchange. Air sits in alveoli and lungs loose elasticity. Bronchioles collapse so it’s harder to exhale and get rid of CO2 (can inhale easier).

29
Q

What is bronchitis?

A

Inflammation of bronchioles. Airway is more narrow and its more challenging to exhale. There’s excess mucous production and cilia become damaged/need to cough out sputum.

30
Q

Causes/risk factors of COPD?

A

Smoking, genetics (alpha 1 antitrypsin causes damage to lungs), chemical/dust exposure, infections, age (alveoli start to decease as we age).

31
Q

Lab assessments for COPD?

A

Arterial blood gases, lower O2 levels/higher CO2 levels, sputum samples, WBC count-hemoglobin.

32
Q

Imaging assessment to do for COPD?

A

Chest x ray. It’ll show hyperinflation of the lungs.

33
Q

What is #1 way to diagnose COPD?

A

Pulmonary function test/spirometry. Measure volume/speed they blow out CO2. COPD people not able to exhale same volume inhaled.

34
Q

S+S of COPD?

A

Progressive dyspnea, SOB with extortion, cough last >3 months with sputum, lung infection, wheezes/decreased breath sounds, fatigue, weight loss (feel full early), pursed lip breathing, cyanosis, tripod position, nail clubbing, accessory muscle use, hypoxemia, and barrel chest.

35
Q

Positioning for COPD?

A

Upright/tripod position will enable effective coughing to remove secretions.

36
Q

What is a bronchodilator and pronchodilator?

A

B (short acting)- PRN, can take every 4-6 hrs
P (long acting)- take once or twice/day

37
Q

O2 therapy levels for COPD?

A

Between 88-92% is our goal.

38
Q

Hydration to help with COPD?

A

2 L/day to help thin secretions.

39
Q

What are inhaled corticosteroids and oral therapies for COPD?

A

IC- take 1-2 times/day and rinse out mouth after to prevent thrush
OT- some decrease mucous production or bronchodilate

40
Q

How to improve endurance and prevent respiratory infection?

A

IE- pace activities, conserve energy, encourage independence, assists during acute exacerbations
RI- increased risk for lung infections because of excessive secretions, vaccines recommended (for pneumonia, COVID, influenza)

41
Q

What is multiple sclerosis?

A

Chronic progressive/degenerative autoimmune disease of the CNS. No cure. T lymphocytes attack myelin/break it down which results in scar tissue. Damage is patchy and in various areas.

42
Q

Symptoms of MS?

A

Vary depending on where damage is. Once axons are damaged and there’s a permanent disability.

43
Q

What is relapsing remitting MS?

A

Clearly defined relapses, most cases, period of disease activity and then no activity. Remission- recovery is nearly complete.

44
Q

What is primary progressive MS?

A

Slow accumulation of disability, steady/gradual deterioration, without relapses/remissions.

45
Q

What is secondary progressive MS?

A

Follows relapsing remitting, district releases and remission is less apparent, increasing levels of disability.

46
Q

What is progressive relapsing MS?

A

Symptoms will steadily get worse.

47
Q

Cause of MS/risk factors?

A

Genetics (some more likely to develop MS), environment (exposed to something that triggers abnormal immune response), immune system (has abnormal responses). Affects more women than men, race (caucasians of northern european descent), family hx, environment (cool climate with little sunshine), diet (low Vitamin D).

48
Q

What to assess for history wise MS?

A

Vision, mobility, sensory perception changes, aggravation of symptoms, personality changes, family hx.

49
Q

What will aggravate symptoms of MS?

A

Hot baths, temperature extremes, stress, fatigue.

50
Q

What diagnostic test are used for MS?

A
  1. MRI- 3D image of soft tissues, can see damage to CNS
  2. Evoked potential testing- measures speed of transmission time
    For diagnosis they need evidence of disease activity over time in different areas.
51
Q

S+S of MS?

A

Vary depending on area of CNS affected. Fatigue, weakness, depression, shaking/tremors, loose coordination, cognitive impairment, pain, bladder/bowel dysfunction, vision loss, double vision, sexual dysfunction, dysarthria, dysphasia, spasticity. Also have increased risk of infections so avoid large crowds/HH.

52
Q

Symptom management meds for pain, spasticity, fatigue, bowel dysfunction?

A

Pain- neuropathic pain (gabapentin), analgesics, tricyclic antidepressant, cannabinoids
Spasticity- muscle relaxant
Fatigue- CNS stimulant
Bowel dysfunction- laxatives, stool softener, urine flow promotion

53
Q

Mobility therapy for MS?

A

Refer to physio or occupational therapy, exercise program (ROM, stretching, strengthening), assistive devices.