States of Shock Flashcards

1
Q

What is the MAIN problem of hypovolemic shock?

A
  • decreased preload (less volume the heart is pumping)
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2
Q

Signs and symptoms hypovolemic shock

A
  • cool and clamped down vasculature
  • tachycardia (to get any little vol around the body)
  • low urine output (not enough vol or pressure and kidneys wont work do to not enough circulating volume)
  • dry mucous membranes
  • narrowed pulse/BP
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3
Q

cause and characteristics of hypovolemic shock

A
  • dehydration ( diarrhea, burns, salt-wasting)
  • fluid loss
  • dilated veins
  • hemorrhage (blood loss) with trauma or bleedings
  • increased afterload (veins constrict to increase vascular resistance to get blood and oxygen to where it needs to be)
  • decreased cardiac output (not enough to pump)
  • low volume circulation
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4
Q

What is hypovolemic Shock?

A

volume depletion

intravascular volume down

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5
Q

distributive shock: preload, afterload, contractility, BP, HR

A
  • low preloads (venous dilation and 3rd spacing so not in vasculature)
  • low afterload due to massive arterial dilation
  • increase contractility initially and decrease in late-stage
  • low BP (vasodilation)
  • increase HR
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6
Q

cardiogenic shock: what happens to Preload, afterload, contractility, BP, HR

A
  • increase preload (high fluid retention and vasoconstriction)
  • increase afterload to compensate by vasoconstriction
  • decreased contractility (pump failure)
  • low BP (not pump as efficiently)
  • high to low depends on the cause
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7
Q

hypovolemic: what happens to Preload, afterload, contractility, BP, HR

A

decreased preload
increased afterload (vasoconstrictor to shunt blood to vital organs)
contractility decrease (less preload = less stretch myocytes)
decreased or normal BP
increase HR (to get more vol around body)

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8
Q

the goal of treatment and types of treatment for distributive shock

A
  • move fluid into vasculature so the heart has something to pump
  • Fill large empty space in the vasculature (correct 3rd spacing and fill with fluid)
  • normal saline and L.R.
  • with infection treat the cause with antibiotics ➡ maintain BP
  • consider vasopressor and inotropes (epi) increase flight or fight to slam close vasculature
  • isotonic fluids
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9
Q

Signs and symptoms of distributive shock

A
  • flushed
  • peripherally dilated
  • tachycardia
  • concentrated urine
  • hypotensive
  • high temp
  • warm skin

Anaphylaxis: edema, flushed and warm
Septic: depends on the presentation

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10
Q

Types of distributive shock (3) and their characteristics:

A
  • Septic shock (infectious): massive inflammatory response leading to massive vasodilation
  • Anaphylactic (autoimmune): mass inflammatory response, immune system
  • Neurogenic (spinal trauma): loss of sympathetic NS simulations lead to an inability to vascoconstrict
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11
Q

Characteristics of distributive shock

A
  • decreased preload (fluid not where it needs to be (dilated veins and 3rd spacing)
  • decreased afterload due to dilated arterie
  • decreased contractility
  • cardiac output is high (heart pumping into nothing),
  • leaky blood vessels
  • inability to maintain vascular tone (vasodilation)
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12
Q

What is distributive shock and what is the main problem?

A
  • change in fluid distribution

- decreased afterload (due to dilated arteries)

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13
Q

Cardiogenic shock treatment and reason for this treatment

A
  • Decrease the workload of the heart and increase contractility (squeeze)
  • decrease preload (less volume pumping)
  • decrease work of heart with fluid removal Lasix
  • decrease afterload (dilate arteries)
  • increase contractility with inotropes
  • get rid of built-up fluid, albumin pull fluid back into circulation
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14
Q

Signs and symptoms of cardiogenic shock

A
  • cool and clamped down vasculature
  • mottled skin
  • crackles in lung
  • peripheral edema
  • perfusion problem
  • contractility issues
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15
Q

Characteristics of cardiogenic shock

A
  • decreased cardiac output (not working pumps)
  • decreased blood pressure
  • decreased tissue perfusion
  • increased catecholamine release
  • RAAS activation (hold on to Na+ and H2O, blood shunting to vital organs, not circulating as efficiently
  • increased afterload (pump what it can with less efficient pumping mechanism with same volume as before )
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16
Q

What is Cardiogenic shock? main problem.

A
  • pump failure with no volume loss

- contractility issue

17
Q

Hypovolemic shock treatments

A
  • Replace fluid (isotonic, L. R., normal saline),

- hemorrhagic give blood (Replace blood loss)

18
Q

Signs and symptoms hypovolemic shock

A
  • cool and clamped down vasculature
  • tachycardia (to get any little vol around the body)
  • low urine output (not enough vol or pressure and kidneys wont work do to not enough circulating volume)
  • dry mucous membranes
  • narrowed pulse/BP
19
Q

Signs and symptoms of cardiogenic shock

A
  • cool and clamped down vasculature
  • mottled skin
  • crackles in lung
  • peripheral edema
  • perfusion problem
  • contractility issues