SPVSC - Buzzwords! Flashcards

1
Q

Immune stuff

  • Decreased resistance
  • antibody production
A

Neuroimmune hypothesis

  • Neurodystrophy
  • Neuroimmunomodulation
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2
Q

Stress induces altered neuroendocrine function –> Immune suppression

A

Neuroimmune hypothesis

  • Neurodystrophy
  • Neuroimmunomodulation
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3
Q

Hans Selye’s work

A

Neuroimmune hypothesis

  • Neurodystrophy
  • Neuroimmunomodulation
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4
Q

Work on stress & immune function, GAS

A

Neuroimmune hypothesis

  • Neurodystrophy
  • Neuroimmunomodulation
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5
Q

SNS and PNS Balance

A

Somatoautonomic Reflex (SAR)

  • Langworthy, Smith, and Paxson
  • widely endorsed and accepted explanation of CMT efficacy on organ systems and total health
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6
Q

Immune stuff

A

Neuroimmune hypothesis

  • Neurodystrophy
  • Neuroimmunomodulation
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7
Q

altered neuroendocrine function

A

Neuroimmune hypothesis

  • Neurodystrophy
  • Neuroimmunomodulation
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8
Q

Neurodystrophy AKA

A

Neuroimmune hypothesis

  • Neurodystrophy
  • Neuroimmunomodulation
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9
Q

Critical to health, scientific and medical support

-SNS and PNS Balance

A

Somatoautonomic Reflex (SAR)

  • Langworthy, Smith, and Paxson
  • widely endorsed and accepted explanation of CMT efficacy on organ systems and total health
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10
Q

Central Modulation

A

Somatoautonomic Reflex (SAR)

  • Langworthy, Smith, and Paxson
  • widely endorsed and accepted explanation of CMT efficacy on organ systems and total health
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11
Q

Descending inhibition regulated dorsal horn input

A

Somatoautonomic Reflex (SAR)

  • Langworthy, Smith, and Paxson
  • widely endorsed and accepted explanation of CMT efficacy on organ systems and total health
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12
Q

Central modulation

-Descending inhibition regulated dorsal horn input

A

Somatoautonomic Reflex (SAR)

  • Langworthy, Smith, and Paxson
  • widely endorsed and accepted explanation of CMT efficacy on organ systems and total health
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13
Q

GAS =

A

General adaptation syndrome

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14
Q

General adaption syndrome was created by

A

Hans Selye

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15
Q

Peripheral Modulation

A

Somatoautonomic Reflex (SAR)

  • Langworthy, Smith, and Paxson
  • widely endorsed and accepted explanation of CMT efficacy on organ systems and total health
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16
Q

somatovisceral reflexes

A

Somatoautonomic Reflex (SAR)

  • Langworthy, Smith, and Paxson
  • widely endorsed and accepted explanation of CMT efficacy on organ systems and total health
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17
Q

PNS dominance

A

Somatoautonomic Reflex (SAR)

  • Langworthy, Smith, and Paxson
  • widely endorsed and accepted explanation of CMT efficacy on organ systems and total health
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18
Q

Somatic structures

A

Spinal reflexes

  • Somatoautonomic (SAR)
  • Somatovisceral
  • Somatosomatic
  • viscerosomatic
  • viscerovisceral
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19
Q

Somatoautonomic reflex

A

Spinal reflexes

  • Somatoautonomic (SAR)
  • Somatovisceral
  • Somatosomatic
  • viscerosomatic
  • viscerovisceral
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20
Q

Somatovisceral reflex

A

Spinal reflexes

  • Somatoautonomic (SAR)
  • Somatovisceral
  • Somatosomatic
  • viscerosomatic
  • viscerovisceral
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21
Q

somatosomatic reflex

A

Spinal reflexes

  • Somatoautonomic (SAR)
  • Somatovisceral
  • Somatosomatic
  • viscerosomatic
  • viscerovisceral
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22
Q

viscerosomatic reflex

A

Spinal reflexes

  • Somatoautonomic (SAR)
  • Somatovisceral
  • Somatosomatic
  • viscerosomatic
  • viscerovisceral
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23
Q

viscerovisceral reflex

A

Spinal reflexes

  • Somatoautonomic (SAR)
  • Somatovisceral
  • Somatosomatic
  • viscerosomatic
  • viscerovisceral
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24
Q

bones, muscles, ligaments, joint capsules arising from the same somite (embryology)
-somatic structures

A

Spinal reflexes

  • Somatoautonomic (SAR)
  • Somatovisceral
  • Somatosomatic
  • viscerosomatic
  • viscerovisceral
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25
Q

VBAI

A

Vertebral Basilar artery insufficiency (VBAI)

-procedure if compromise detected –> Stop, observe, refer (and describe)

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26
Q

Drop attacks

A

Vertebral Basilar artery insufficiency (VBAI)

-procedure if compromise detected –> Stop, observe, refer (and describe)

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27
Q

Signs and symptoms: 5 D’s, 3 N’s

A

Vertebral Basilar artery insufficiency (VBAI)

-procedure if compromise detected –> Stop, observe, refer (and describe)

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28
Q

No clear list of risk signs

A

Vertebral Basilar artery insufficiency (VBAI)

-procedure if compromise detected –> Stop, observe, refer (and describe)

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29
Q

CVA’s

  • never give second adjustment
  • most pt.’s bet 31-35 yo
A

Vertebral Basilar artery insufficiency (VBAI)

-procedure if compromise detected –> Stop, observe, refer (and describe)

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30
Q

Known for GAS

A

Hans Selye

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31
Q

UMNL sign’s and Sx’s

A

Myelopathy

  • Compressive myelopathy
  • Cord compression (B.J. Palmer)
32
Q

SIDS

A

Myelopathy

  • Compressive myelopathy
  • Cord compression (B.J. Palmer)
33
Q

Incr. susceptibility to URI’s

A

Myelopathy

  • Compressive myelopathy
  • Cord compression (B.J. Palmer)
34
Q

inversion of the atlas

A

SIDS

35
Q

inversion of the atlas

A

Myelopathy

  • Compressive myelopathy
  • Cord compression (B.J. Palmer)
36
Q

Dentate ligament

A

Dural Torque

-alternative upper cervical technique

37
Q

Meningeal torsion

A

Dural Torque

-alternative upper cervical technique

38
Q

Mechanoreceptor funk

A

Proprioceptive insult

-now considered a component of the SAR, but has been considered and tested separately in the past

39
Q

Biomechanical abberation

A

Proprioceptive insult

-now considered a component of the SAR, but has been considered and tested separately in the past

40
Q

Surgical recovery

A

Proprioceptive insult

-now considered a component of the SAR, but has been considered and tested separately in the past

41
Q

congenital anomalies

A

Proprioceptive insult

-now considered a component of the SAR, but has been considered and tested separately in the past

42
Q

Nansel and Szlazak

A

Somatic Visceral Disease Mimicry Hypothesis

43
Q

Alternative to CMT affecting visceral function

A

Somatic Visceral Disease Mimicry Hypothesis

44
Q

Acute - SHLRP

A

Inflammation hypothesis

-Charles (Skip) Lantz

45
Q

Immobilization –> Inflammation

A

Inflammation hypothesis

-Charles (Skip) Lantz

46
Q

Pre stage spinal lesions

A

Inflammation hypothesis

-Charles (Skip) Lantz

47
Q

VSC Phase I

A

Inflammation hypothesis

-Charles (Skip) Lantz

48
Q

Langworthy, Smith, Paxson - 1906

A

Segmental Dysfunction (SDF)

-All subsets lead to segmental facilitation

49
Q

Neurobiological Models - non inflammatory/inflammatory

A

Segmental Dysfunction (SDF)

-All subsets lead to segmental facilitation

50
Q

Korr: Fixation model

A

Segmental Dysfunction (SDF) - non inflammatory

-All subsets lead to segmental facilitation

51
Q

Sympathecotonia

A

Segmental Dysfunction (SDF) - Non inflammatory

-All subsets lead to segmental facilitation

52
Q

Spinal Learning

A

Segmental Dysfunction (SDF) - Non inflammatory

-All subsets lead to segmental facilitation

53
Q

Patterson - Steinmetz

A

Segmental Dysfunction (SDF) - Non inflammatory

-All subsets lead to segmental facilitation

54
Q

Dvorak

A

Segmental Dysfunction (SDF) - Inflammatory

-All subsets lead to segmental facilitation

55
Q

Gatterman - Goe

A

Segmental Dysfunction (SDF) - Inflammatory

-All subsets lead to segmental facilitation

56
Q

Mense

A

Segmental Dysfunction (SDF) - Inflammatory

-All subsets lead to segmental facilitation

57
Q

Minimal energy hypothesis

A

Segmental Dysfunction (SDF) - Inflammatory

-All subsets lead to segmental facilitation

58
Q

Hypomobility of motor unit –> Hypermobility above and below –> Segmental facilitation –> Post. horn flooded –> Lat. horn fires up (sympathecotonia) –> sympathetic response, adherent reflexes

A

Fixation theory - Irwin Korr

-Subset of SDF

59
Q

Motion Palpation analysis and technique

A

Henri Gillet - Fixation Theory

-mentioned in SAR

60
Q

Joint Play

A

Henri Gillet - Fixation Theory

-mentioned in SAR

61
Q

X-ray criteria (medical)

A

Instability Hypothesis

BJ pioneered radiography

62
Q

Prolonged immobilization leads to permanent impairment

A

Immobilization Degeneration

  • Leach & Lantz
  • Use it or lose it
63
Q

VSC Phase 2

A
  • Instability Hypothesis

- VBAI

64
Q

Kirkaldy - Willis

A

Instability Hypothesis

65
Q

Etiology: numerous postural and traumatic insults

A

Instability Hypothesis

66
Q

VSC Phase 3

A

Immobilization Degeneration

  • Leach & Lantz
  • Use it or lose it
67
Q

Immobilization leads to premature degeneration

A

Immobilization Degeneration

  • Leach & Lantz
  • Use it or lose it
68
Q

Via subluxation, DJD (Osteophytes) Etc.

A

Neuropathology Hypothesis

-Neural Compression
-Nerve Compression
DD Palmer
–Original “bone out of place”
–Incr. and Decr. Body Tonus

69
Q

electrical stuff

A

Neuropathology Hypothesis

-Neural Compression
-Nerve Compression
DD Palmer
–Original “bone out of place”
–Incr. and Decr. Body Tonus

70
Q

decrease nerve transmission

A

Neuropathology Hypothesis

-Neural Compression
-Nerve Compression
DD Palmer
–Original “bone out of place”
–Incr. and Decr. Body Tonus

71
Q

Acute: Incr. signals, irritation / excitation, HYPERESTHESIA, spasms

A

Neuropathology Hypothesis

-Neural Compression
-Nerve Compression
DD Palmer
–Original “bone out of place”
–Incr. and Decr. Body Tonus

72
Q

Chronic: Decrease signals, ANESTHESIA / HYPOESTHESIA, atone, flaccid paralysis, paresis

A

Neuropathology Hypothesis

-Neural Compression
-Nerve Compression
DD Palmer
–Original “bone out of place”
–Incr. and Decr. Body Tonus

73
Q

Anesthesia/Hypoesthesia

A

Neuropathology Hypothesis

-Neural Compression
-Nerve Compression
DD Palmer
–Original “bone out of place”
–Incr. and Decr. Body Tonus

74
Q

Hyperesthesia

A

Neuropathology Hypothesis

-Neural Compression
-Nerve Compression
DD Palmer
–Original “bone out of place”
–Incr. and Decr. Body Tonus

75
Q

LMNL Signs and symptoms

A

Neuropathology Hypothesis

-Neural Compression
-Nerve Compression
DD Palmer
–Original “bone out of place”
–Incr. and Decr. Body Tonus

76
Q

Protein Stuff

A

Axoplasmic Aberration (AXT)

-plausible explanation of why pain seems to move as healing occurs