Sports (2008-2019) Flashcards

1
Q
  1. 4 contraindications to an HTO (2012)
A
  • JAAOS 2005 - High Tibial Osteotomy
    • Disease of the contra-lateral compartment
    • Absent contra-lateral meniscus
    • Symptomatic patellofemoral disease
    • Inflammatory arthritis
    • Severe angular malalignment
    • Non-concordant pain pattern
    • Older physiologic age (better suited to TKA)
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2
Q
  1. List 4 advantages of doing a medial opening wedge HTO over a lateral closing wedge. (2016)
A

JAAOS 2005 - HTO/JAAOS 2011

  • Easy to control correction (can dial it in)
  • Less extensive surgical dissection/no disruption to proximal tib/fib joint
  • Less proximity to peroneal nerve
  • Can be combined with PF procedures
  • No loss of lateral bone stock
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3
Q
  1. List 5 features of traumatic proximal tibia fibula dislocation. (2013)
A

JAAOS 2003 - Instability of the Proximal Tibiofibular Joint

  • Pain over lateral knee, radiates up biceps femoris
  • Prominence of fibular head
  • Pain aggravated by ankle ROM
  • Instability to weightbear
  • Peroneal nerve symptoms
  • Posterolateral corner instability
  • LCL instability
  • Pain and loss of knee ROM
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4
Q
  1. In regards to an acute, traumatic proximal tibia fibula dislocation (2013):
  • Explain the mechanism of injury, including position of limb and knee position. (2 marks)
  • Two features seen on examination
  • Explain your reduction technique (2 marks)
A
  • Explain the mechanism of injury, including position of limb and knee position. (2 marks)
    • Hyperflexed knee, plantarflexed and internally rotated foot
  • Two features seen on examination
    • Prominence of fibular head and lateral knee
    • Instability of posterolateral corner
  • Explain your reduction technique (2 marks)
    • Knee at 80-110o of flexion (relax biceps femoris)
    • Ankle dorsiflexed, foot everted and externally rotated (relax peroneals)
    • Pressure to fibular head in appropriate direction
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5
Q
  1. List 4 things that can cause loss of extension after ACL reconstruction? (2012, 2016)
A

JAAOS 1999 - Loss of Extension after Reconstruction of the ACL

  • Cyclops lesion
  • Capsulitis leading to arthrofibrosis
  • Post-operative immobilization
  • Lack of post-operative rehabilitation
  • Non-anatomic graft placement
  • Infection
  • Trauma - bucket handle of meniscal tear
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6
Q
  1. What are 4 surgical techniques for ACL reconstruction in an 11 year old female athlete? (2012, 2013)
A
  • Iliotibial band extra-physeal reconstruction (modified McIntosh)
    • IT band remains attached to Gerdy’s tubercle, passes over femoral condyle and under intermeniscal ligament to suture under flaps of anterior tibial periosteum
  • All Epiphyseal Reconstruction
    • Anderson technique: 4 strand hamstring all epiphyseal suspensory technique on femur, epiphyseal tibial tunnel exiting above physis but interference screw inserted below physis to tether graft
    • Ganley technique - 4 string hamstring graft with epiphyseal femoral interference screw and epiphyseal vertical tibial interference screw
    • Cordasco-Green - epiphyseal tunnels in femur and tibia; suspensory fixation for both
  • Partial trans-physeal
    • Epiphyseal femoral tunnel and trans-physeal tibial tunnel
  • Trans-physeal reconstruction
    • I would not answer this on an exam for an 11yo if I could avoid it; if I use it I would specify partial transphyseal with an epiphyseal femoral tunnel and a vertical, central, small diameter tibial tunnel
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7
Q
  1. When performing the Thesally physical exam on the knee, what are 3 conditions that can give you a false positive. (2011, 2013)
A
  • OA
  • Osteochondral Injury
  • Collateral Ligament Injury
  • Posterolateral Corner Injury
  • Associated ACL Injury
  • Tibial Plateau #
  • Loose body
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8
Q
  1. What are 6 risk factors for recurrent patellar dislocation? (2014, 2015)
A

JAAOS 1997 - PF Instability

  • Previous patellar instability
  • Increased ligamentous laxity
  • Patella alta
  • Trochlear dysplasia/Hypoplastic lateral condyle
  • Patellar dysplasia
  • Malalignment (increased femoral anteversion, genu valgum, increase ER of tibia)
  • Increased Q Angle
  • VMO Atrophy
  • Female gender (AAOS COR)
    • Not a risk factor for initial dislocation, but risk factor for recurrence
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9
Q
  1. List 3 prognostic factors for healing of OCD of the knee. (2015)
A
  • Status of physis
  • Size of lesion (>15mm)
  • Chronicity of lesion
  • Integrity of cartilage
  • Mechanical symptoms/loose fragment
  • Atypical location (patella, lateral femoral condyle)
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10
Q
  1. Patient with a PCL and Posterolateral corner injury:
  2. Increased external rotation of tibia with knee at 30 degrees but not at 90 degrees
  3. Increased external rotation of tibia with knee at 30 degrees and at 90 degrees
  4. Increased laxity with varus stress
  5. Pivot shift positive
A

ANSWER: B (increase ER or Dial test at 30 AND 90 degrees)

2013, 2015

  • JAAOS 2016 - Management of Posterior Cruciate Ligament Injuries
    • A positive result occurring only at 30o of knee flexion indicates a PLC injury, and 90o of knee flexion indicates a combined PCL and PLC injury. 
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11
Q
  1. Which of the following are not part of the posterolateral corner
  2. LCL
  3. PCL
  4. Popliteofibular ligament
  5. Popliteus
A

ANSWER: B (not PCL)

2013, 2014

Covey (JBJS 2001) Injuries of the posterolateral corner of the knee

  • Static:
    • LCL
    • Popliteus Tendon
    • Popliteofibular ligament
    • Lateral Capsule
    • Variable –> arcuate ligament, fabellofibular ligament
  • Dynamic Structures:
    • Biceps femoris
    • Popliteus muscles
    • IT band
    • Lateral head of gastrocs
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12
Q
  1. What vessel provides blood supply to the PCL?
  2. Inferior geniculate
  3. Superior geniculate
  4. Middle geniculate
A

ANSWER: C (middle geniculate)

2011,2016

COR2

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13
Q
  1. Late complaint after PCL insufficient Knee?
  2. Medial compartment OA
  3. Instability
  4. Lateral Compartment OA
A

ANSWER: A (AnteroMEDIAL)

  • 2009
  • Old Evidence:
    • “The natural history of the PCL-deficient knee leads to increased contact pressures in both the medial and PF compartments”
  • ACL - posteromedial
  • PCL – anteromedial
  • Both medial compartment
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14
Q

14.All of the following are risk factors for ACL tear except:

  1. Female
  2. Cavovarus
  3. Increased Q-angle
  4. Increased BMI
A

ANSWER: B (Not cavovarus)

  • 2016
  • Really not the greatest evidence for BMI available
  • JAAOS 2000 - Noncontact ACL Injuries: Risk Factors and Prevention Strategies
    • Environmental:
      • Equipment
      • Shoe-surface interaction
    • Anatomic:
      • Knee angle
      • Hip angle
      • Laxity
      • Notch size
      • Hormonal
    • Biomechanical:
      • Muscular strength
      • Skill level
      • Neuromuscular control
      • Body movement
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15
Q
  1. When treating an infection post-ACL reconstruction, all are true EXCEPT: 
  2. Blood cultures are usually not positive
  3. Every reasonable effort should be made to retain the graft and hardware
  4. Acute infections have the same prevalence as delayed infections
  5. Arthrocentesis is the diagnostic modality of choice
A

ANSWER: C

2016

  • JAAOS 2013 - Management of Septic Arthritis Following ACL Reconstruction
    • “Blood cultures, which are often ordered at presentation, are not reliable markers of infection because they are often negative or prone to contamination from skin flora”
    • “All reasonable attempts are initially made to preserve the reconstructed graft and associated hardware”
    • “Typically presents either acutely (<2 weeks from surgery) or subacutely (2 weeks to 2 months). Late presentation is relatively infrequent”
    • “Arthrocentesis remains the standard of care for diagnosis of septic arthritis following ACL reconstruction”
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16
Q
  1. Where is the posterolateral bundle of the ACL tightest?
  2. Extension
  3. 30 degrees flexion
  4. 90 degrees flexion
  5. Same in all degrees flexion/extension
A

ANSWER: A

  • 2014
  • Amis (JBJS Br 1991) Functional anatomy of the ACL 
  • Anterior bundles tight in FLEXION
  • Posterior bundles tight in EXTENSION
    *
17
Q
  1. What is true regarding ACL tunnel Placement
  2. Tibial tunnel is most important
  3. If femoral tunnel is too posterior, graft lengthens in flexion
  4. If femoral tunnel too anterior, graft lengthens in flexion
  5. Over the top femoral placement of graft does not change in extension
A

ANSWER: C

  • 2008, 2012
  • Depends on whether tunnel is anterior in femur or tibia
  • Etiologic Factors That Lead to Failure After Primary Anterior Cruciate Ligament Surgery. Jan 2017 Clin Sports Med
  • TRICK: femur rotates during flexion, what is posterior becomes anterior and vis versa
18
Q
  1. Chronic ACL all associated except:
  2. Increased risk of OA
  3. ACL recon has not been shown to decrease OA
  4. More chance of damaging cartilage and meniscus
  5. More likely to damage lateral as opposed to medial meniscus
A

ANSWER: D (chronic ACL = posteroMEDIAL OA)

  • 2013 (two variants)
  • Cipolla M (KSSTA 1995) Different patterns of meniscal tears in acute Acl ruptures and in chronic ACL deficient knees
    • Retrospective study of 1103 reconstructions
    • “Acute injuries show a higher rate of lateral meniscus tears, chronic laxities are very frequently associated with severe medial meniscal lesions”
  • Gillquist J (Sports Med 1999) ACL reconstruction and the long-term incidence of gonarthrosis
    • 10-20% incidence of arthrosis
    • 10x higher than age matched controls
    • Menisectomy doubles risk (50-70% incidence at 15-20 years)
    • Not necessarily symptomatic
  • Lohmander LS (AJSM 2007) The long-term consequence of ACL and meniscus injuries: osteoarthritis
    • “there is lack of evidence to support a protective role of repair or reconstructive surgery of the ACL or meniscus against osteoarthritis development”
    • 50% of patients with ACL/meniscus injuries have OA at 10-20 years
  • Oiestad BE (AJSM 2009) Knee OA after ACL injury: a systematic review
    • Review of 7 prospective and 24 retrospective
    • 0-13% incidence of OA for ISOLATED ACL injury at at least 10 years
    • Combined injury 21-48%
19
Q
  1. What is not associated with a poor prognosis after ACL injury?
  2. Hearing a pop at time of injury
  3. Smoking
  4. Gain 15lbs in first year
  5. Wearing a brace for 1 year post-op
A

ANSWER: D

  • 2010
  • OKU 9:
    • Independent poor predictors after ACL reconstruction:
      • Smoking
      • Recollection of hearing a pop at injury
      • Weight gain of 15lbs
      • No change in educational level since injury
  • Spindler K (JBJS 2005) Clinical outcome at a minimum of five years after reconstruction of the ACL
    • Independent predictors of a worse outcome…..included the patient’s recollection of hearing or feeling a pop, a weight gain of >15lbs, and no change in educational level since surgery
20
Q
  1. What is true regarding pediatric ACL reconstruction:
  2. Transphyseal tibial tunnel is an option
  3. Bone-patellar-tendon-bone graft is a good option
  4. Screw placement across the physis is acceptable
  5. Non-operative treatment has good outcomes
A

ANSWER: A

2015 

JAAOS - ACL in the Skeletally Immature

  • “Conservative management of skeletally immature patients with complete ACL injuries has been less successful. The high non-compliance rate is associated with secondary damage of the menisci and/or articular cartilage, and a large percentage of patients eventually require surgical reconstruction”

2018 IOC consensus on pediatric ACL:

  • OK to go transphyseal BUT need to be central, vertical and no hardware crossing physis
  • Non-operative
    • Kocher found that partial tears-1/3 of them required surgery for persistent instability
    • 50% will drop out of sports b/c of instability
    • Increased rate of meniscal and chondral damage
21
Q
  1. Hamstrings tendon grafts in ACL reconstruction, all except:
  2. Previous MCL injury is a contra-indication to using hamstring tendon
  3. Ligamentous laxity is a contraindication to using hamstring tendon
  4. Sprinter is a contraindication to using hamstring tendon
  5. Previous hamstring injury is a contra-indication to using hamstring tendon
A

ANSWER: A

2008

JAAOS 2005 - Graft Selection in ACL Reconstruction

  • Contra-indications to hamstring autograft:
  • Generalized ligamentous laxity
  • Competitive sprinters
  • Previous hamstring injury
  • Acute MCL
22
Q
  1. In performing an opening wedge HTO, all of the following are true except:
  2. In an ACL deficient knee, placing the bone wedge posteromedially will decrease the tibial slope and decrease anterior translation
  3. In a PCL deficient knee, placing the bone wedge anteromedially will increase the tibial slope and increase posterior translation
  4. Placing the bone wedge direct medial will not affect the slope
  5. Smaller anterior gap with a larger posterior gap will preserve the native tibial slope
A

ANSWER: B (best answer) but C also not true

2015

Charles: C is also NOT true

  • JAAOS 2011 – Role of the HTO in the varus knee
    • “The tibia has a triangular shape and to preserve the anatomic slope, the anterior opening shoulder be smaller than the posterior medial opening.
    • My understanding is that you are opening more lateral posteriorly (base of triangle) and therefore are opening LESS central posterior then central anterior.

Increased tibial slope will DECREASE posterior translation

JAAOS 2011 - Role of the HTO in the varus knee

  • ACL deficiency –> decrease tibial slope
  • PCL deficiency –> increase tibial slope
23
Q
  1. When doing a proximal tibial osteotomy for a varus knee, what is the difference between doing a fibular osteotomy and a partial resection of the fibular head
  2. Increased peroneal nerve injury with osteotomy
  3. Increased non union
  4. Difficult to do the tibial osteotomy
  5. Increased risk of popiteal artery injury
A

ANSWER: C

2008

  • JAAOS 2005 - HTO
    • Incidence of peroneal nerve palsy after lateral closing wedge 0-20%
    • Osteotomy in vicinity of the fibular head associated with higher incidence of peroneal nerve palsy than fibular osteotomy >15cm distal to head
24
Q
  1. What is true regarding ACL graft choices:
  2. Single bundle reconstruction restores normal knee kinematics
  3. Posterolateral bundle is shorter
  4. Double bundle reconstruction has better clinical outcomes
  5. Can regain hamstring strength following hamstring graft harvest with appropriate PT
A

ANSWER: B

2015

  • JAAOS 2008 - Controversies in Soft Tissue ACL Reconstruction
    • No difference in clinical outcomes between single and double bundle
  • Fu F () Gross, arthroscopic and radiographic anatomies of the ACL: Foundations for ACL Surgery
    • AM bundle is 38mm in length, PL bundle is 17.8mm
  • Tiamklang T (Cochrane 2012) Double-bundle versus single-bundle reconstruction for ACL rupture in adults
    • Comparison of 17 trials
    • No statistical or clinical difference between groups in IKDC, Tegner, Lysholm scores in immediate or long follow up
  • Ardern CL (Arthroscopy 2010) Hamstring strength recovery after hamstring tendon harvest for ACL reconstruction
    • No differences between semi-T/gracilis and Semi-T alone)
    • All had 3-27% strength deficit compared to contra-lateral side
  • Battaglia TC - Strength and regrowth of hamstring tendons after hamstring autograft ACL reconstruction
25
25. Which of the following is true about cruciate synovial cysts? 1. Cause pain during deep squatting 2. Are very rare and have no classification system 3. Are impossible to distinguish from synovial sarcoma on MRI/"are confused with" 4. Have a high risk of recurrence following arthroscopic excision
ANSWER - A 2014 Cysts about the Knee: Evaluation and Management. JAAOS August 2013 * B - Untrue: Prevalence of 0.2 to 1.9%, and classification is anterior to ACL, between ACL and PCL and posterior to PCL * A - True: Pain can be intermittent or constant, and often worse with exercise especially squatting (squeezing the cyst) * D - Untrue: Arthroscopic excision is the treatment of choice with “no” symptomatic recurrences * C - Untrue: Although the differential includes other ganglion cysts, hemangioma, synovial sarcoma and villonodular synovitis these pathologies should have unique MRI features that should lead to proper diagnosis
26
26. Post ACL reconstruction. What will predict arthritis? 1. degree of pre-op AP laxity 2. intra-op findings of a chondral lesion 3. use of a hamstring graft 4. Wrestling with Brendan oneil early
ANSWER: B 2011 * Oiestad (AJSM 2009) Knee osteoarthritis after anterior cruciate ligament injury * Predictors of OA: * Obesity * Chondrosis in medial compartment \> grade 2 * Prior medial menisectomy * Longer duration of follow up * Female * PF chondrosis * Prior lateral menisectomy
27
27. Proper placement of ACL tibial tunnel to prevent graft impingement is achieved by? 1. Placement of tunnel in the center of the ACL footprint 2. Placement of tunnel posterior to the ACL footprint, 6mm anterior to PCL 3. Placement of tunnel in front of the ACL footprint 4. Placement of tunnel posterior to ACL foot print
ANSWER: A 2011 JAAOS 1999 - Loss of extension after reconstruction of the ACL * Ideal place for the tunnel is 3mm posterior to the center of the origin of the ACL * Orthobullets * Tibial tunnel should be 10-11mm in front of the anterior border of PCL insertion
28
28. What position do you tighten your ACL graft? 1. full extension 2. 30 degrees of flexion 3. 90 degrees of flexion 4. ryan martin is gay
ANSWER: B 2011 JAAOS 1999 - Loss of extension after reconstruction of the ACL * Some biomechanical studies that there is a loss of extension with tightening in 30o * Areneja (Arthroscopy 2009) * No difference in a systematic review of papers tightening in full extension, 30o flexion, 10o flexion JAAOS 2012 – Graft tensionin during knee ligament reconstruction: * Some biomech studies say decrease extension if overtension at 30 degrees (most studies look at 30 degrees) * Consensus is that you can tension at full extension or 30 degrees (more common at Western) * Maximum tensioning at full extension OR submaximum loading/tensioning at 30 degrees (which is why we use the tensionometer)
29
29. Which position of the knee places the ACL at its greatest strain ? 1. Hyperextension 2. 30 deg flexion 3. 90 deg flexion 4. hyperflexion
ANSWER: A 2008 * Wheeless: * Hyper-extension * At 5o of hyperextension the ACL ligament forces range between 50 and 240 Newtons * Hyperextension of the knee develops much higher forces in ACL than in the PCL * ACL strain at 30o of knee flexion are significant higher than at 90o where the ligament remains unstrained * Frank (JBJS 1997) Current Concepts Review * Extensive in vitro and in vivo evidence has shown that the circumstances that cause the highest loads and strains on the ACL during daily function are quadriceps-powered extension of the knee, moving it from approximately 40o of flexion to full extension; hyperextension of the knee; excessive internal tibial rotation; or excessive varus or valgus stress on the tibia * 2018 - A Review on Biomechanics of Anterior Cruciate Ligament and Materials for Reconstruction * the highest shear forces on the anterior cruciate ligament occur during hyperextension/low flexion angles of the knee joint
30
30. Which of the following activities places the greatest peak strain on the ACL : 1. Ascending stairs 2. Ascending a ramp 3. Descending stairs 4. Descending a ramp
ANSWER: C 2008 Frank (JBJS 1997) Current Concepts Review * ®Frank, JBJS, Current Concepts Review, 1997 - highest loads and strains during daily function on ACL are during quad powered extension from 40° to hyperextension * PCL * - Level Walking = 352 N * - Ascending Stairs = 641 N * - Descending Stairs = 262 N * - Ascending Ramp = 1215 N * - Descending Ramp = 449 N * ACL * - Level Walking = 169 N * - Ascending Stairs = 67 N * - Descending Stairs = 445 N * - Ascending Ramp = 27 N * - Descending Ramp = 93 N
31
31. Which of these is true about ACL injuries? 1. MRI shows bruise on the Posterior Medial Femur 2. 40% of pts with ACL tear show bruising on MRI 3. Visible on scope 4. Full thickness injury to cartilage on histology
ANSWER: C (prob best answer) or D (also say histologic evidence of chondrocyte damage in 35%) * A: False Bone bruising on lateral side of femur and tibia. Middle 1/3 of lateral femoral condyle and posterior 1/3 of lateral tibial plateau. The plateau comes forward under the femur during injury and impact in the middle third of the femur. * B: False M- ore than 80% * Graf, Am J Sp Med, 1993 - middle third of the lateral femoral condyle and the posterior third of the lateral tibial plateau, no correlation with position or location of meniscus injury, 48% had bone bruising * Treatment of ACL Injuries, Part I; Vol.33,No.10,2005; p.1583 “Several investigators reported 80% or more of ACL injuries were associated with bone bruises in the lateral compartment.” * Mair, Am J Sp Med, 2004 – PCL injury with 83% isolated injury of PCL and at least one bruise * Costa-paz, Arthroscopy, 2001 – f/u MRI with ACL and bone bruise after recon, 24 to 64 months post op, 71% gone, 29% retained suggesting OC injury * Davies, Clinical Radiology, 2004 – bone bruises persist longer than 12 to 14 weeks, more than previously thought, 67% had ACL injuries, 23% collateral injuries, 10% no assoc lig injury, no correlation in bruise size or location with type of or lack of injury
32
32. Grade III MCL & ACL rupture. What to do? 1. Rehab as per ACL 2. Brace in extension x 2-3wks 3. Reconstruct ACL, fix MCL, brace 30-90deg 4. Reconstruct ACL, brace 30-90deg
ANSWER: B Grade 3 MCL gets residual laxity, not a functional problem. Controversial what to do for combined but some suggest trial nonop. 2008 * Combined anterior and posterior cruciate and medial collateral ligament injury: nonsurgical and delayed surgical treatment (ICL 2003) * "anterior cruciate ligament injuries in combination with medial collateral ligament and/or PCL injury can initially be treated nonsurgically and reconstructed later as dictated by patient symptoms and activity level." * Medvecky M (Sports Med and Arthrosc Revew 2015) Management of Acute combined ACL-Medial and posteromedial instability of the knee * There is fairly uniform consensus in the literature that non-operative management of first-degree and second-degree injuries is appropriate. With regard to acute third-degree medial-sided injuries, some controversy does exist regarding non-operative versus operative intervention. However, most studies advocate non-operative treatment of medial-sided injury * Recommend short term immobilization in extension * Previous answer (2008 toronto and 2008 UWO) is grade 3 MCL gets residual laxity. All agree that isolated grade 3 gets nonop initially but highly controversial if treat nonop or op for combined ACL and grade 3. Would try non-op and brace first.
33
33. A 20yo male football player sustains an isolated, grade III MCL tear. Which of the following is true? 1. Operative repair leads to earlier healing and return to play 2. Non-operative treatment leads to return to play in 3 weeks 3. You should perform arthroscopy to detect any intra-articular pathology and to determine treatment plan 4. The MCL usually tears from the femoral side
ANSWER - D * Treatment of Medial Collateral Ligament Injuries. JAAOS 2009 * Most commonly a rupture at the femoral insertion, however an avulsion with bony fragment is an indication for acute fixation. * Pellegrini-Stieda Syndrome is recalcification at the femoral insertion * Return to Sport: * Serscheid and Garrick (AJSM 1981) * Grade II: In high school football series average return in 19.5 days * Grade I: In high school football average return in 10.6 days * Jones (Clin Orthop 1986): * Grade III injuries in high school players average return in 34 days * ACL tears comprise up to 95% of associated injuries; 20% are with grade I MCL injuries, 52% are with grade II MCL injuries, 78% are with grade III injuries * Should be able to pick up on MR or clinical exam * Kim C (Clin Sport Med 2016) Return to Play after MCL Injury * Not great evidence, review article * Conservative management - 5-7 weeks to return to play * Operative - 6-9 months * Definitely some injury severity bias * JAAOS 2017 - Physical examination of knee ligament injuries * Agree MCL usually tears from femoral side - check for point tenderness at medial epicondyle * Valgus stress should be done at both 0deg and 30deg; 30deg helps isolate sMCL
34
34. What type of fibres in the meniscus resist hoop stresses? 1. Circumferential 2. Crossing 3. Radial 4. Vertical
ANSWER - A 2011, 2014 * From: The knee meniscus: Structure-function, pathophysiology, current repair techniques, and prospects for regeneration. E.A. Makris et al. / Biomaterials 32 (2011) 7411-7431 * “The biomechanical properties of the knee meniscus are appropriately tuned to withstand the forces exerted on the tissue. Many studies have helped to quantify the properties of the tissue both in humans and in animal models. According to these studies, the meniscus resists axial compression with an aggregate modulus of 100-150 kPa. The tensile modulus of the tissue varies between the circumferential and radial directions; it is approximately 100-300 MPa circumferentially and 10-fold lower than this radially.” * From The meniscus: Review of basic principles with application to surgery and rehabilition (J Athl Train 2001) * Three collagen fiber layers are specifically arranged to convert compressive loads into circumferential or “hoop” stresses (Figure (Figure4).4). In the superficial layer, the fibers travel radially, serving as “ties” that resist shearing or splitting. In the middle layer, the fibers run parallel or circumferentially to resist hoop stress during weight bearing. Lastly, there is a deep layer of collagen bundles that are aligned parallel to the periphery * 70% water, 30% organic; Type II collagen is main component, GAGs 1%