Spine Peds Flashcards

1
Q

What is the pseudoluxation of the cervical spine?

A

A physiologic (normal) pseudosubluxation of cervical vertebrae of up to 40% (or 4 mm) seen in young children.

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2
Q

In who do we see it more frequently?

A

seen in children less than 8 years

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3
Q

What is the location?

A

C2 on C3 is most common

C3 on C4 is second most common

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4
Q

What is the Pathophysiology?

A

◦ caused by the horizontal nature of the facet joints at younger ages

facet joints become more vertical with age

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5
Q

What are the recommended views and Findings?

A

1- Lateral radiograph with flexion and extension

2- reduction of subluxation with extension xrays

3- absence of anterior soft-tissue swelling (usually seen with traumatic cause)

4- Swischuk’s line

Spino-laminar line drawn from spino-laminar point on C1 to C3

Spino-laminar point on C2 should be within 1.5 mm of spino-laminar line

helpful to differentiate pseudo-subluxation from true injury

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6
Q

DDx Pseudosubluxation

A

Hangman Fx

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7
Q

Normal findings in children cervical spine

A
  • Pre-vertebral swelling < 2/3 of adjacent vertebral width
    • Smooth contour lines of
    • anterior vertebral bodies
    • posterior vertebral bodies
    • Spino-laminar line (inside lamina)
    • tips of spinous process
  • Parallel facet joints
  • Normal Retro-Pharyngeal space
    • < 6 mm at C2
      • Retro-tracheal space < 14 mm
  • Atlanto-Dens interval < 5 mm in children and < 3 mm adolescents
  • Absent vertebral body wedging : 7% of normal children have a wedge shaped C3 vertebral body
  • Absence of cervical lordosis: loss of cervical lordosis may be found in 14% of normal children
  • C2-3 or C3-4 pseudo-subluxation < 4mm: considered normal as long as the posterior laminar line is contiguous
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8
Q

Cervical immobilization peds partiuclarity

A

using an adult backboard for pediatric patients creates a dangerous level of cervical flexion

transporting patients less than 8 years of age requires a spine board with occipital depression or enough thoracic elevation to align the cervical and thoracic segments of the spine

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9
Q

Collar immobilization: indications

A

▪ stable odontoid fractures

Atlanto-axial instability

acute atlanto-axial rotatory displacement (AARD)

stable subaxial cervical spine trauma

Modalities: rigid collar vs. soft collar (depends on injury, often controversial)

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10
Q

Recommendations regarding car seating for children according to age

A
  • Rear-facing car seat in the rear center seat until at least 1 years of age and weighing at least 20lbs.
  • Front-facing car seat in the rear center seat until at least 4 years of age or weighing at least 40lbs.
  • This child should be in more than a simple seat belt restraint. The child meets criteria to ride in a booster seat, (over 40 pounds and older than 4 years of age), and should be seated in the rear center seat.
  • Riding in the front seat is typically not recommended until the age of 13 with the caveat of any child under 80 pounds in the front seat should have the airbag turned off.
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11
Q

Position of cervical immobilization in children and adults

A
  • Cervical spine injuries should be immobilized in a postion of relative extension in both children and adults.
  • Applying cervical traction with the external auditory meatus in-line with the shoulders can serve as a guideline.
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12
Q

What is the most common cause of infantile torticolis?

A

Congenital Muscular Torticolis

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13
Q

What are the risk factors for congenital muscular torticolis?

A
  • Oligo-hydramnios
  • first pregnancy (limited intrauterine space)
  • traumatic delivery
  • breech delivery
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14
Q

What is the Pathophysiology of congenital muscular torticolis?

A

Contracture of the sternocleidomastoid (SCM)

Cervical rotational deformity with chin rotation away from the affected side and head tilt towards the affected side

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15
Q

What are the Associated conditions with congenital muscular torticolis?

A

◦ associated with other packaging disorders

  • DDH
  • metatarsus adductus
  • calcaneovalgus feet
  • plagiocephaly (asymmetric flattening of the skull)occurs on contralateral side
  • congenital atlanto-occipital abnormalities
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16
Q

What is the Prognosis of congenital muscular torticolis?

A

◦ typically resolves with stretching within the first year

if left untreated

permanent rotational deformity

positional plagiocephaly

craniofacial deformities

facial asymmetry

facial hemihypoplasia

compensatory scoliosis

17
Q

What are the clinical findings in congenital muscular torticolis and what test should we order?

A
  • Palpable neck mass

US

CT dynamic for AARD

MRI for central causes of torticolis

18
Q

What is the DDx of congenital muscular torticolis?

A
  1. AARD: painful (compared to painless for congenital muscular torticollis)post-traumatic or post-infectious (Grisel’s disease)
  2. Klippel-Feil syndrome classic triad: short webbed neck; low posterior hairline; limited cervical range of motion
  3. Ophthalmologic and vestibular conditions
  4. Lesions of central and peripheral nervous system
19
Q

What is the TREATMENT of congenital muscular torticolis?

A

Nonoperative:

passive stretching

condition present for less than 1 year

less than 30° limitation in ROM

outcomes: 90-95% respond to passive stretching in the first year of life

Operative:

bipolar release of SCM or Z-lengthening

indications

failed response to at least 1 year of stretching

outcomes: good outcomes (92% success), even in older children

facial asymmetry can improve as long as release done prior to 10 years of age

20
Q

What are the COMPLICATIONS of congenital muscular torticolis?

A

1- Permanent rotational deformity

risk factors: left untreated or unnoticed

2- Positional plagiocephaly

risk factors: left untreated or unnoticed

3- Craniofacial deformities

facial asymmetry

facial hemi-hypoplasia

4- Compensatory scoliosis

21
Q

What is AARD?

A

C1-C2 rotatory instability (fixed rotation of C1 on C2) caused by subluxation or facet dislocation

22
Q

What are the common causes of AARD?

What are the associated conditions with AARD?

A
  1. Infection (~35%): may have history of pharyngitis or otitis media; Grisel’s disease is the condition of AARD following a respiratory infection or retro-pharyngeal abscess; thought to be linked to lymphatic edema in area of cervical spine
  2. trauma (~24%)
  3. recent head or neck surgery (~20%)
  4. idiopathic

Associated conditions:

  • Down’s syndrome
  • rheumatoid arthritis
  • tumors
  • congenital anomalies
23
Q

What is the diagnostic gold standard of AARD?

How to do it and what do you see?

A

Dynamic CT

take CT with head straight forward, and then in maximal rotation to right and left

will see fixed rotation of C1 on C2 which does not change with dynamic rotation

24
Q

Clinical exam of AARD?

A

ipsilateral rotation and contralateral tilt of the head in relation to the lateral mass of C1

chin rotated to the side opposite the facet subluxation (e.g. right sided facet subluxation will have chin rotated to the left)

contra-lateral sternocleidomastoid may be spasticsternocleidomastoid (SCM) spasm occurs on the SAME side as the chin (e.g. right sided facet subluxation will have chin rotated to the left, and left SCM will be spastic)

this protective spasticity occurs to reduce further subluxation

C1-C2 subluxation (and resultant chin position) is primary, SCM spasm is secondary/reactive

25
What is the treatment algorithm of AARD?
**_1- subluxation present for \< 1 week (traumatic or Grisel's disease)_** **--\> _Nonoperative_**: soft collar, NSAIDS, exercise program indications many patients probably reduce spontaneously before seeking medical attention **_2- subluxation persists \> 1 week_** head halter traction, NSAIDS, benzodiazepines, then hard collar x 3 months indications: persistent torticollis in spite of soft collar (above) x 2 weeks technique: small amount (5 lbs.) usually enough either in hospital or at home; muscle relaxants and analgesics may be needed **_3- subluxation persists \> 1 mos._** **_or failed halter traction x 2 weeks (above)_** --\> halo traction, then halo vest x 3 months indications 4- **subluxation persists \> 3 mos** **neurologic deficits present** **failed halo traction x 2 weeks** **recurrent subluxation** --\> Operativeposterior C1-C2 fusion indications