Spinal trauma Flashcards

1
Q

List two patient groups at inc’d risk of c-spine injury.

A
  • Down’s Syndrome - inc’d risk atlanto-occipital dislocation
  • Rheumatoid arthritis - inc’d risk of rupture of C2 transverse ligement
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2
Q

Outline the NEXUS Low-risk Criteria for c-spine injury.

A
  • Absence of posterior midline cervical tenderness
  • No abnormal neurologic findings
  • Normal level of alertness
  • No evidence of intoxication
  • No painful distracting injuries
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3
Q

What is the name and classification system of C1 #’s?

What is the mechanism associated with each type and state whether they are stable or unstable.

A
  • Jefferson #
    • Type 1 - Posterior arch # - Hyperextension - stable
    • Type 2 - Anterior arch # - Hyperflexion - stable
    • Type 3 - Type 1 + Type 2 #s - Axial load -> burst fracture - stable if transverse ligament intact, otherwise unstable
    • Type 4 - # through the lateral mass - Axial load + rotational force - unstable
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4
Q

How is a Jefferson’s # id’d on odontoid plain film?

A

Lateral displacement of the lateral masses of C1 with respect to the body of C2 - ie, they should line up.

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5
Q

What is a Hangman’s #?

List the types.

A

Traumatic anterior spondylolisthesis of C2. Best seen on lateral views (CT or XR).

  • Levine & Edwards
    • Type 1: <3mm horizontal displacement of C2 on C3
    • Type 2: >3mm w/ >11deg angulation
    • Type 2a: No displacement but severe angulation
    • Type 3: Severe angulation + displacement + facet dislocation
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6
Q

Outline the classification system of odontoid #s.

What are the mechanisms of this type of #?

A

Anderson and D’Alonso System:

  • Type 1: Tip of odontoid
  • Type 2: Base of odontoid
  • Type 3: Through the lateral masses
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7
Q

List 8 unstable C-spine injuries.

Mnemonic?

A

Jumping Off Awnings Nearly Warrants Frank Spinal Trauma!

  • Jefferson’s
  • Odontoid
  • Atlanto-occipital dislocation
  • Neural arch
  • Wedge #
  • Facet dislocation
  • Spinal subluxation
  • Teardrop
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8
Q

What is the Canadian C-Spine Rule? What is its sens + spec?

A

Sens 99.7% Spec 50%

  • Applies to patients with:
    • GCS 15 AND
    • Head or neck pain OR
    • No neck pain PLUS
    • Injury above the clavicles AND non-ambulatory AND dangerous mechanism

​STEP 1: Are there ANY high-risk factors? If yes -> CT. If no -> STEP 2

  • Age >65
  • Dangerous mech’m
  • Extremity paraesthesia

​STEP 2: Are there ANY low-risk factors. If no -> CT. If yes -> STEP 3

  1. Simple rear-end MVA
  2. Sitting in ED
  3. Ambulatory at any time
  4. Delayed onset of neck pain
  5. Absence of midline c-spine tenderness

STEP 3: Able to actively rotate the neck 45deg bilaterally? If yes, C-spine cleared. If no -> CT

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9
Q

Compare the NEXUS C-spine Rule and the Canadian C-Spine Rule.

A
  • Similar, very high sensitivity - 99.6 & 99.7%
  • CCSR has better specificity - 50% vs 12%
  • Nexus is simpler one-step process - if all criteria are met, no CT
  • CCSR is a three-step process - Presence of high risk factors -> CT. Presence low risk factors -> range neck. If OK, no CT
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10
Q

List the 3 most common incomplete spinal cord syndromes. What is the common mechanism that results in each?

A
  1. Anterior cord syndrome - hyperflexion
  2. Central cord syndrome - hyperextension
  3. Brown-Sequard - hemisection of cord due penetrating trauma or mass
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11
Q

Describe the central cord syndrome.

A
  • Damage to the centre of the spinal cord, usually from hyperextension injuries
  • Results in:
    • Motor weakness and sensory loss in all extremities
    • Upper extremities affected more than lower - see cord anatomy pic
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12
Q

Describe the anterior cord syndrome.

A
  • Damage to the anterior segment of the spinal cord, usually as the result of hyperflexion injuries -> retropulsed fragments.
  • Results in:
    • Compression of the anterior spinal artery
    • Paraesthesia
    • Hyperalgesia
    • Preservation of dorsal columns -> proprioception, vibration
  • NROS emmergency due irreversible damage after 24h
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13
Q

Describe Brown-Sequard syndrome.

A
  • Hemisection of the cord, usually as the result of penetrating trauma or a mass.
  • Results in:
    • ipsilateral loss of dorsal columns -> position, vibration and motor.
    • Contralateral loss of pain and temperature.
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14
Q

Describe Wallenberg’s Syndrome - why does it occur in spinal trauma?

Mnemonic?

A

LeMI PICcup MIself, eye am tIPSI and my other side is numb.

  • LeMI: Lateral Medullary Infarct
  • PIC: PICA occlusion
  • MI: Miosis
  • eye: ptosis (Miosis + ptosis = Horner’s)
  • and my other side is numb: contralateral paraesthesia of body and limbs w/ ipsilateral facial paraesthesia

In high cervical spinal trauma, injuries can damage the vertebral artery which supplies PICA -> Wallenberg’s or Lateral Medullary Syndrome

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15
Q

Describe Horner’s Syndrome. What causes it?

A

Horny PAM

Horner’s Syndrome:

  • Ptosis
  • Anhydrosis
  • Miosis

Occurs due to ipsilateral sympathetic cervical chain injury in spinal and neck trauma.

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16
Q

Give four signs of Cauda Equina Syndrome.

A
  • Lower limb weakness
  • Bowel and/or bladder dysfunction
  • Loss of anal tone
  • Saddle paraesthesia
17
Q

What does preservation of the bulbocavernous reflex indicate?

What is it?

A
  • Squeezing of the penis (or clitoris) -> inc in anal tone
  • Suggests spinal shock rather than complete cord transection
  • Better prognostic sign as spinal shock should recover
18
Q

List causes of Horner’s syndrome.

A
  • Carotid artery dissection
  • Wallenberg’s (Lateral Medullary) syndrome
  • Neck trauma
  • Pancoast tumour
  • Thyroid tumour or resection
  • Sympathectomy
  • MS
  • Encephalitis
19
Q

List trauma-related indications for CTA neck.

A
  • High cervical injury - C1-3
  • Cervical spine ligamentous injury
  • Facet joint dislocation
  • Any vertebral body #
  • Transverse foramen #
20
Q

Are steroids indicated in spinal cord injury?

A
  • Controversial
  • D/w NROS
21
Q

What is neurogenic shock?

A
  • Better thought of as neurogenic hypotension
  • Spinal cord injury -> loss of autonomic function:
    • Vasodilatation (loss of vasomotor tone)
    • Bradycardia (loss of reflex tachycardia
  • Consider in patient with:
    • Flacidity
    • Areflexia
    • Bradycardic
    • Peripherally warm
    • Hypotenisive
    • Other causes of shock (ie hypovolaemic, obstructive) already excluded
22
Q

What are the Denver Criteria for Blunt Cerebrovascular Injuries?

A

Signs and symptoms:

  • Focal neurologic deficit
  • Arterial Hemorrhage
  • Cervical Bruit or Thrill (<50yo)
  • Infarct on Head CT
  • Expanding Neck Hematoma
  • Neuro exam inconsistent with Head CT

Risk factors:

  • Midface Fractures
  • Cervical Spine Injuries
  • Basilar Skull Fracture
  • GCS<8
  • Hanging with Anoxic Brain Injury
  • Seat belt abrasion or other soft tissue injury of the anterior neck resulting in significant swelling or altered mental status
  • Isolated seatbelt sign without other neurologic symptoms has not been identified as a risk factor
23
Q

What is the managment of BCVI?

A
  • Heparinisation
  • NROS and IR consult
24
Q

What is the normal prevertebral soft tissue distances in adults?

A
  • <7mm at C2, and
  • <22mm at C6