Spinal Column Disorders Flashcards
(71 cards)
Cervical Strain
● Although the term “strain” generally refers to injury of
muscle tissue, the term “Cervical Strain” includes
ligamentous injury of the facet joints and/or
intervertebral disks, in addition to muscle injury.
○ “Neck sprain” is synonymous
Common etiology of cervical strain
whiplash injury
Cervical Strain pathophysiology
○ Whiplash injuries occur with rapid
acceleration and/or deceleration,
which results in rapid flexion and
extension of the cervical spine.
○ The velocity of the movements can
stretch and tear muscles, disrupt
ligaments, dislocate facet joints, and
cause spinal fractures.
○ Cervical instability can develop.
Cervical Strain Clinical Presentation
○ Cervical pain that begins after an accident or trauma, but can start spontaneously (even during sleep).
○ The pain does not radiate into the arms or down the spine.
○ Diffuse pain anywhere from the base of the skull to the cervicothoracic junction is most common.
○ Pain is often increased with motion and may be accompanied by muscular spasm in the sternocleidomastoids, trapezius muscles, or paraspinals.
○ Occipital headaches may occur and may persist for months
Cervical Strain PE
○ Exam may reveal tenderness to palpation of the sternocleidomastoids,
trapezius muscles, or paraspinals secondary to muscle spasm.
○ Tenderness may be noted over the spinous processes and facets
Cervical Strain diagnosis
○ Pain often limits range of motion.
○ The neurologic examination should be normal
■ No Lhermitte’s sign (electrical pulse through body with neck flexion)
■ No Spurling sign (radiating pain down arm with lateral bending)
■ No pain or paresthesias into the arms or down the spine AP, Lateral, and Odontoid (open-mouth) X-rays should be obtained if there is a history of trauma, associated neurologic symptoms, or if the patient is elderly
○ If severe pain, Flexion and Extension films (aka
Flex/Ex films) should be obtained, but should be
ordered by a consulting specialist
Cervical Strain Managemen
○ Most patients return to normal within 4-6 weeks
○ Acetaminophen and NSAIDs are preferred, although a short course (no
more than 1 week) of narcotic may be needed.
○ 1-3 weeks in a Soft Cervical Collar is often beneficial.
○ 1-2 weeks of Muscle Relaxants can be beneficial.
○ Manipulation of the cervical spine is contraindicated
in patients with acute cervical spine injuries!
Acute Low Back Pain
● Acute LBP is more of a symptom than a diagnosis. However, 80%+
of LBP patients are not able to be given a more specific diagnosis.
○ Acute = LBP of less than 3 months in duration.
● Lifetime prevalence in the US is 60-80%.
● Of all cases of acute LBP, 70% are secondary to
lumbar sprain or strain
Acute Low Back Pain pathophuysiology
○ Lumbar strain is an injury to the paravertebral
spinal muscles.
○ Lumbar sprain refers to ligamentous injury of
the facet joints or annulus fibrosus,
transvertebral or other spinous ligaments.
○ Age-related facet or vertebral arthropathy can flare if provoked acutely by certain movements or lifting
Acute Low Back Pain presentation
○ Patients often report an acute onset of LBP after lifting or twisting
○ Low back pain may radiate down into the buttocks.
○ Even though lifting or twisting is a common
cause, patient who are sedentary also
frequently experience LBP (poor fitness,
sitting at computer).
○ May have difficulty standing up straight
and transitioning from position to position.
○ Patients often have diffuse tenderness to palpation in the lumbar region and/or sacroiliac area
○ Lumbar range of motion is often decreased
secondary to pain.
Acute Low Back Pain diagnosis
○ The goal is to identify the serious causes of low back pain and know when it is necessary to order radiologic imaging.
○ Because most have strain or sprain, plain X-rays are usually not helpful and need not be ordered for all cases of LBP
○ If significant pain at rest, pain at night, or history of trauma,
ordering X-rays (AP and Lateral) would be appropriate.
Acute Low Back Pain management
○ As long as the patient has no sciatic symptoms or significant neurologic deficit,
treatment of acute LBP (most commonly strain or sprain) has 2 phases:
“Essentials of Musculoskeletal Care,” 5e. Armstrong, Hubbard.
■ Phase 1- Focus on Symptomatic Relief
■ Phase 2- Focus on Return to Activity
Chronic Low Back Pain
● Chronic LBP is defined as LBP that lasts for 3+ months.
● Symptoms are most commonly recurrent and episodic,
but for some the pain is unrelenting
Once Chronic LBP has been identified, a thorough evaluation should occur, ruling out the following:
○ Cancer (metastatic or primary)
○ Osteoporosis with fractures
○ Osteomyelitis and discitis (infection)
○ Abdominal pathology (aneurysm, retroperitoneal tumor)
Chronic Low Back Pain pathophysiology
○ The most common cause of Chronic LBP is
degenerative disc disease.
○ Other causes include:
■ Lumbar stenosis
■ Ankylosing Spondylitis
■ Old vertebral fractures
Chronic Low Back Pain clinical presentation
○ Chronic low back pain that radiates to one or both buttocks.
○ Pain is aggravated by activity (lifting, bending, twisting, etc).
○ Patient may have history of intermittent sciatica (radiating down the back of the leg), but back pain is the predominant symptom.
○ Rest helps to relieve the pain for most patients.
■ Some have difficulty sleeping because of the back pain
○ Progressively worsening back pain with weight loss is a red flag for cancer and should not be ignored
Chronic Low Back Pain evaluation
○ On examination, there may be diffuse
tenderness to palpation in the lumbar and
sacroiliac regions
○ Neurologic exam of the LEs should be normal.
○ Range of motion may be restricted.
○ X-rays (AP and Lateral) will reveal age-related
degenerative changes.
X-ray findings of chronic low back pain
■ Osteophytes with decreased disc height
■ May see spondylosis/spondylolisthesis
Narcotic abuse or misuse is a large concern in this group
Chronic Low Back Pain
Spinal Fractures pathophysiology
○ High-velocity impact, hyperextension,
hyperflexion, or hyper-rotation can all lead
to vertebral fracture
○ If the central canal or neural foramen are
compromised, neural tissue may be injured
(spinal cord or spinal nerves).
○ Osteoporosis and bony metastasis can
result in “insufficiency” fractures.
Spinal Fractures clinical presentation
○ Spinal pain is generally severe, even at rest.
○ Other symptoms depend on presence or absence of neural tissue compression
■ Spinal cord damage will result in paresthesias
and loss of motor control below the level of
the injury.
■ Nerve root impingement will result in pain and paresthesias along the associated dermatome (potentially motor loss)
Spinal Fractures evaluation
○ Evaluate motor and sensory function throughout all cervical, thoracic, and
lumbar dermatomal/myotomal distributions.
○ Assess deep tendon reflexes for presence and symmetry.
○ Examine for swelling and contusions over the spinal column.
○ Palpate for point tenderness and step-off lesions (posterior ligament injury)
Radiologic Imaging of Spinal Fractures
■ Plain X-rays are an option and often reveal the injury.
■ However, if significant trauma or abnormal examination, CT scan is the initial imaging modality of choice in most trauma centers.
● Provides better images with more definitive diagnosis (and faster)
■ MRI is obtained after the CT if there is a concern for neural tissue compression and ligamentous injury
Spinal Fractures management
○ If imaging studies reveal no evidence of fracture, and neurologic exam is
normal, spinal precautions may be lifted after confirmation by a
neurologic or orthopedic specialist.
○ If a spinal fracture is identified, urgent consultation by a specialist is
standard of care