Sonography Theory Exam Question Pool Flashcards
According to the electrocardiogram (EKG), electrical systole is:
Onset of the QRS to the onset of the T wave
Onset of the T wave to the onset of the P wave
Onset of the QRS complex to the end of the T wave
End of the T wave to the onset of the QRS complex
Onset of the QRS complex to the end of the T wave
All of the following are components of a pulsed-wave Doppler of a pulmonary vein EXCEPT:
E
S2
S1
AR
E
All of the following are considered a part of normal ventricular diastole EXCEPT:
Ventricular depolarization
Isovolumic relaxation
Early passive filling
Atrial systole
Ventricular depolarization
All of the following are true statements concerning the left ventricle EXCEPT:
Heavily trabeculated
Contains two papillary muscle groups
Bullet shaped (truncated ellipsoid)
Top normal thickness is approximately 1.0 cm
Heavily trabeculated
All of the following are true statements concerning the right ventricle EXCEPT:
Most anterior positioned cardiac chamber
Normal wall thickness is 0.3 to 0.5 cm
Heavily trabeculated
Normally forms the cardiac apex
Normally forms the cardiac apex
All of the following left ventricular wall segments may be evaluated in the parasternal long-axis view EXCEPT:
Cardiac apex
Basal anterior interventricular septum
Mid-anterior interventricular septum
Cardiac apex
All of the following left ventricular wall segments may be evaluated in the parasternal short-axis of the left ventricle at the level of the papillary muscles EXCEPT:
Anterolateral
Anterior interventricular septum
Anterior wall
Cardiac apex
Cardiac apex
All of the following structures are located in the right atrium EXCEPT:
Crista terminalis
Thebesian valve
Moderator band
Eustachian valve
Moderator band
All of the following ventricular wall segments may be supplied by the right coronary artery EXCEPT:
Basal and mid-inferior walls of the left ventricle
Basal and mid-anterior interventricular septum
Basal and mid-inferolateral walls of the left ventricle
Lateral wall of the right ventricle
Basal and mid-anterior interventricular septum
All of the following wall segments may be visualized in the apical four-chamber view EXCEPT:
Lateral wall of the right ventricle
Anterolateral wall
Cardiac apex
Anterior interventricular septum
Anterior interventricular septum
All of the following wall segments may be visualized in the apical two-chamber view EXCEPT:
Cardiac apex
Right ventricular outflow tract
Inferior wall
Anterior wall
Right ventricular outflow tract
Normal pressure values in millimeters of mercury (mm Hg) for the listed cardiac chambers or great vessels include all of the following EXCEPT:
Right atrial pressure: 2 to 8 mean
Right ventricle: 15 to 30 systolic; 2 to 8 diastolic
Pulmonary artery: 15 to 30 systolic; 2 to 12 mean diastolic
Aorta: 100 to 140 systolic; 3 to 12 end-diastolic
Aorta: 100 to 140 systolic; 3 to 12 end-diastolic
Structures of the mitral valve apparatus include all of the following EXCEPT:
Chordae tendineae
Mitral valve annulus
Papillary muscles
Sinuses of Valsalva
Sinuses of Valsalva
The Chiari network is found in the:
Left ventricle
Right ventricle
Right atrium
Left atrium
Right atrium
The boundaries of the functional left ventricular outflow tract are best described as extending from the:
Anterior aortic valve annulus to the posterior aortic valve annulus
Anteromedial position of the tricuspid valve annulus to the pulmonic valve annulus
Free edge of the anterior mitral valve leaflet to the aortic valve annulus
Tips of the left ventricular papillary muscles to the edge of the anterior mitral valve leaflet
Free edge of the anterior mitral valve leaflet to the aortic valve annulus
The coronary sinus can be differentiated from the descending thoracic aorta with pulsed-wave Doppler because coronary sinus flow is predominantly diastolic while aortic flow is:
Phasic
Predominantly diastolic
Equiphasic
Predominantly systolic
Predominantly systolic
The correct order for the branches of the transverse aorta (aortic arch) is:
Left subclavian, right subclavian, left common carotid
Right brachiocephalic; left brachiocephalic, left common carotid
Sinus of Valsalva, right innominate, left innominate
Right brachiocephalic, left common carotid, left subclavian
Right brachiocephalic, left common carotid, left subclavian
The crista terminalis is found in the:
Right atrium
Left atrium
Left ventricle
Right ventricle
Right atrium
The eustachian valve is found in the:
Left ventricle
Right atrium
Left atrium
Right ventricle
Right atrium
The imaginary boundaries that define the mid-left ventricle are the:
Mitral annulus to the tip of the papillary muscles
Base of the papillary muscles to the cardiac apex
Tip of the papillary muscles to the base of the papillary muscles
Aortic annulus to the edge of the mitral valve
Tip of the papillary muscles to the base of the papillary muscles
The left anterior descending coronary artery supplies blood to all of the following EXCEPT:
Anterior wall of the left ventricle
Inferior wall of the left ventricle
Anterior interventricular septum
Apical cap
Inferior wall of the left ventricle
The moderator band is always located in the:
Right atrium
Left ventricle
Right ventricle
Left atrium
Right ventricle
The most likely explanation of main pulmonary artery dilatation is:
Truncus arteriosus
Bicuspid aortic valve
Pulmonary hypertension
Carcinoid heart disease
Pulmonary hypertension
The name of the aortic segment located between the left subclavian artery and the insertion of the ligamentum arteriosum is the:
Aortic root
Transverse aorta
Aortic isthmus
Sino-tubular junction
Aortic isthmus
The names of the two left ventricular papillary muscle groups are:
Anterior; posterior
Medial; lateral
Superior; inferior
Anterolateral; posteromedial
Anterolateral; posteromedial
The most common cause of chronic tricuspid regurgitation is:
Tricuspid valve prolapse
Ebstein’s anomaly
Rheumatic heart disease
Pulmonary hypertension
Pulmonary hypertension
The most common etiology of pulmonary regurgitation is:
Infective endocarditis
Pulmonary hypertension
Rheumatic heart disease
Carcinoid heart disease
Pulmonary hypertension
The most common etiology of tricuspid stenosis is:
Right atrial myxoma
Carcinoid heart disease
Rheumatic fever
Infective endocarditis
Rheumatic fever
The murmur of tricuspid regurgitation is best described as a:
Pansystolic murmur heard best at the lower left sternal border
Pansystolic murmur heard best at the cardiac apex with radiation to the axilla
Systolic ejection murmur heard best at the upper right sternal border
Holodiastolic murmur heard best at the lower left sternal border
Pansystolic murmur heard best at the lower left sternal border
The pulmonary vein atrial reversal wave may be _________ in peak velocity and duration in a patient with severe acute aortic regurgitation.
Reversed
Decreased
Increased
Unchanged
Increased
The severity of aortic regurgitation may best be determined with color flow Doppler by all of the following methods EXCEPT:
Measuring the vena contracta in the parasternal long-axis view
Comparing the aortic regurgitation jet width with the left ventricular outflow tract width in the parasternal long-axis view
Measuring the aortic regurgitation jet aliasing area in the parasternal long-axis view
Determining the presence of holodiastolic flow reversal in the descending thoracic aorta and/or abdominal aorta
Measuring the aortic regurgitation jet aliasing area in the parasternal long-axis view
The typical two-dimensional echocardiographic findings in rheumatic tricuspid stenosis include all of the following EXCEPT:
Right atrial dilatation
Diastolic doming of the anterior tricuspid valve leaflet
Systolic bowing of the posterior tricuspid valve leaflet
Leaflet thickening especially at the leaflet tips and chordae tendineae
Systolic bowing of the posterior tricuspid valve leaflet
When two-dimensional evaluation of a systolic ejection murmur reveals a thickened aortic valve with normal systolic excursion and a peak velocity across the aortic valve of 1.5 m/s. The diagnosis is most likely aortic valve:
Sclerosis
Prolapse
Stenosis
Regurgitation
Sclerosis
A tricuspid regurgitation peak velocity of 3.0 m/s is obtained. This indicates:
Pulmonary hypertension
Severe tricuspid regurgitation
Mild tricuspid regurgitation
Moderate tricuspid regurgitation
Pulmonary hypertension
All of the following are cardiac Doppler findings for tricuspid valve stenosis EXCEPT:
Decreased tricuspid valve area
Increased tricuspid valve E wave velocity
Increased mean pressure gradient
Decreased pressure half-time
Decreased pressure half-time
All of the following are considered useful quantitative measurements to determine the severity of aortic regurgitation EXCEPT:
Effective regurgitant orifice
Peak velocity of aortic regurgitation
Regurgitant fraction
Regurgitant volume
Peak velocity of aortic regurgitation
All of the following are dilated in significant chronic tricuspid regurgitation EXCEPT:
Right atrium
Inferior vena cava
Pulmonary veins
Hepatic veins
Pulmonary veins
All of the following color flow Doppler findings indicate significant pulmonary regurgitation EXCEPT:
Wide jet width at origin
Holodiastolic flow reversal in the main pulmonary artery
Peak velocity of < 1.0 m/s
Jet width/Right ventricular outflow tract width > 70%
Peak velocity of < 1.0 m/s
An intracardiac pressure that may be determined from the continuous-wave Doppler tricuspid regurgitation signal is:
Mean pulmonary artery pressure
Systemic vascular resistance
Pulmonary artery end-diastolic pressure
Systolic pulmonary artery pressure
Systolic pulmonary artery pressure
Cardiac Doppler findings associated with significant chronic tricuspid regurgitation include all of the following EXCEPT:
Increased E velocity of the tricuspid valve
Systolic flow reversal in the hepatic vein
Systolic flow reversal in the pulmonary vein
Concave late systolic configuration of the regurgitation signal
Systolic flow reversal in the pulmonary vein
Causes of anatomic tricuspid regurgitation include all of the following EXCEPT:
Ebstein’s anomaly
Pulmonary hypertension
Infective endocarditis
Carcinoid heart disease
Pulmonary hypertension
Echocardiographic evidence of severe acute aortic regurgitation includes all of the following EXCEPT:
Premature opening of the aortic valve
Premature closure of the mitral valve
Premature opening of the mitral valve
Reverse doming of the anterior mitral valve leaflet
Premature opening of the mitral valve
Holodiastolic flow reversal in the descending thoracic aorta and/or the abdominal aorta may be present in each of the following EXCEPT:
Severe aortic regurgitation
Patent ductus arteriosus
Severe mitral regurgitation
Aortopulmonary window
Severe mitral regurgitation
In a patient with severe acute aortic regurgitation the left ventricular end-diastolic pressure increases rapidly. This pathophysiology will affect which of the following?
Systolic ejection period
Closure of the mitral valve
Left ventricular dimension
Closure of the pulmonary valve
Closure of the mitral valve
In significant chronic aortic regurgitation, M-mode and two-dimensional evidence includes all of the following EXCEPT:
Hyperkinesis of the interventricular septum
Left ventricular dilatation
Paradoxical interventricular septal motion
Hyperkinesis of the posterior (inferolateral) wall of the left ventricle
Paradoxical interventricular septal motion
M-mode and two-dimensional echocardiographic findings for chronic tricuspid regurgitation include:
Right ventricular hypertrophy
Protected right ventricle
Paradoxical interventricular septal motion
Left ventricular volume overload
Paradoxical interventricular septal motion
Methods for determining the severity of tricuspid regurgitation with pulsed-wave Doppler include all of the following EXCEPT:
Laminar flow of the tricuspid regurgitant jet
Holosystolic flow reversal of the hepatic vein
Peak velocity of the tricuspid regurgitant jet
Increased E wave velocity of the tricuspid valve
Peak velocity of the tricuspid regurgitant jet
Possible echocardiographic and cardiac Doppler findings in a patient with carcinoid heart disease include all of the following EXCEPT:
Tricuspid regurgitation
Tricuspid stenosis
Pulmonary regurgitation
Tricuspid valve prolapse
Tricuspid valve prolapse
Posterior displacement of the aortic valve leaflet(s) into the left ventricle outflow tract during ventricular diastole is called aortic valve:
Sclerosis
Prolapse
Stenosis
Perforation
Prolapse
Premature closure of the mitral valve is associated with all of the following EXCEPT:
Loss of sinus rhythm
Acute severe aortic regurgitation
First-degree atrioventricular block
Acute severe mitral regurgitation
Acute severe mitral regurgitation
Severe aortic regurgitation is diagnosed with continuous-wave Doppler by all of the following criteria EXCEPT:
A pressure half-time of < 200 msec
A maximum velocity of 4 m/s
Steep deceleration slope
Increased jet density
A maximum velocity of 4 m/s
Significant chronic pulmonary regurgitation is associated with:
Right ventricular volume overload
Right ventricular hypertrophy
Right atrial hypertrophy
Left ventricular volume overload
Right ventricular volume overload
Signs of significant tricuspid regurgitation include all of the following EXCEPT:
Jugular venous distention
Pulsus paradoxus
Hepatomegaly
Right ventricular heart failure
Pulsus paradoxus
The M-mode finding that indicates severe acute aortic regurgitation is premature aortic valve:
Mid-systolic closure
Systolic flutter
Opening
Closure
Opening
The M-mode/two-dimensional echocardiography parameters that have been proposed as an indicator for aortic valve replacement in severe chronic aortic regurgitation are left ventricular:
End-systolic dimension ≥ 55 mm and fractional shortening of ≤ 25%
End-diastolic dimension ≥ 55 mm and fractional shortening ≤ 25%
End-diastolic dimension ≤ 55 mm and fractional shortening of ≥ 25%
End-diastolic dimension ≥ 70 mm and left atrial dimension ≥ 55 mm
End-systolic dimension ≥ 55 mm and fractional shortening of ≤ 25%
The continuous-wave Doppler signal of aortic regurgitation may be differentiated from the continuous-wave Doppler signal of mitral stenosis by the following guideline:
If the diastolic flow pattern commences after mitral valve opening then the signal is due to aortic regurgitation
If the diastolic flow pattern commences before mitral valve opening then the signal is due to aortic regurgitation
Cannot be differentiated by continuous-wave Doppler.
The Doppler flow velocity pattern of mitral valve stenosis is laminar while the Doppler flow pattern of aortic regurgitation is turbulent.
If the diastolic flow pattern commences before mitral valve opening then the signal is due to aortic regurgitation
The mitral valve pulsed-wave Doppler flow pattern often associated with severe acute aortic regurgitation is grade:
II (pseudonormal)
III or IV (restrictive)
Normal for age
I (impaired relaxation)
III or IV (restrictive)
All of the following represents possible etiologies for acute aortic regurgitation EXCEPT:
Aortic dissection
Infective endocarditis
Aortic valve sclerosis
Trauma
Aortic valve sclerosis
An effect of significant aortic valve stenosis on the left ventricle is:
Concentric left ventricular hypertrophy
Eccentric left ventricular hypertrophy
Asymmetrical septal hypertrophy
Protected in significant aortic valve stenosis
Concentric left ventricular hypertrophy
Aortic valve with reduced systolic excursion. On physical examination there was a crescendo-decrescendo systolic ejection murmur and a diastolic decrescendo murmur heard. The most likely diagnosis is aortic valve:
Flail
Stenosis and regurgitation
Regurgitation
Stenosis and mitral valve prolapse
Stenosis and regurgitation
Cardiac Doppler parameters used to assess the severity of valvular aortic stenosis include all the following EXCEPT:
Aortic velocity ratio
Mean pressure gradient
Peak aortic valve velocity
Aortic pressure half-time
Aortic pressure half-time
Cardiac magnetic resonance imaging provides all of the following information in a patient with aortic regurgitation EXCEPT:
Regurgitant volume
Left ventricular volumes
Detailed resolution of the aortic valve
Effective regurgitant orifice
Detailed resolution of the aortic valve
In the parasternal long-axis view, severe aortic valve stenosis is defined as an aortic valve leaflet separation that measures:
≥ 14 mm
≤ 12 mm
≤ 8 mm
≤ 10 mm
≤ 8 mm
Of the transvalvular pressure gradients that can be measured in the echocardiography laboratory, the most useful in examining aortic valve stenosis is probably:
Mean systolic gradient
Peak instantaneous pressure gradient
Peak-to-peak gradient
Mean diastolic gradient
Mean systolic gradient
Pathologies that may result in a left ventricular pressure overload include all the following EXCEPT:
Mitral valve stenosis
Valvular aortic stenosis
Discrete subaortic stenosis
Systemic hypertension
Mitral valve stenosis
Possible two-dimensional echocardiographic findings in significant aortic valve stenosis include all the following EXCEPT:
Post-stenotic dilatation of the descending aorta
Left ventricular hypertrophy
Post-stenotic dilatation of the ascending aorta
Aortic valve calcification
Post-stenotic dilatation of the descending aorta
Reverse diastolic doming of the anterior mitral valve leaflet is associated with:
Rheumatic mitral valve stenosis
Papillary muscle dysfunction
Flail mitral valve
Severe aortic regurgitation
Severe aortic regurgitation
Secondary echocardiographic findings associated with severe valvular aortic stenosis include all the following EXCEPT:
Left ventricular hypertrophy
Post-stenotic dilatation of the ascending aorta
Right ventricular hypertrophy
Decreased left ventricular systolic function (late in course)
Right ventricular hypertrophy
The Doppler maximum peak instantaneous pressure gradient in a patient with aortic stenosis is 100 mm Hg. The cardiac catheterization peak-to-peak pressure gradient will most likely be:
Higher than 100 mm Hg
Dependent upon respiration
Equal to 100 mm Hg
Lower than 100 mm Hg
Lower than 100 mm Hg
The LEAST common valve regurgitation found in normal patients is:
Pulmonary regurgitation
Mitral regurgitation
Tricuspid regurgitation
Aortic regurgitation
Aortic regurgitation
The aortic valve area considered severe aortic valve stenosis is:
≤ 1.0 cm^2
< 2 cm^2
< 3 cm^2
< 1.5 cm^2
≤ 1.0 cm^2
The characteristic M-mode findings for aortic valve stenosis include all the following EXCEPT:
Thickening of the aortic valve leaflets
Diastolic flutter of the aortic valve leaflets
A lack of systolic flutter of the aortic valve leaflets
Reduced leaflet separation in systole
Diastolic flutter of the aortic valve leaflets
The characteristic feature of the murmur of chronic aortic regurgitation is a:
Harsh systolic ejection murmur heard best at the right upper sternal border
Diastolic crescendo-decrescendo murmur heard best along the left upper sternal border
Diastolic rumble following an opening snap
Diastolic decrescendo murmur heard best along the left sternal border
Diastolic decrescendo murmur heard best along the left sternal border
The echocardiographer may differentiate between the similar systolic flow patterns seen in coexisting severe aortic valve stenosis and mitral regurgitation by all the following EXCEPT:
Aortic ejection time is shorter that the mitral regurgitation time
Since both are systolic flow patterns, it is not possible to separate mitral regurgitation from aortic valve stenosis.
Mitral regurgitation flow always lasts until mitral valve opening, whereas aortic valve stenosis flow does not.
Mitral diastolic filling profile should be present during recording of the mitral regurgitation, whereas no diastolic flow is observed in aortic valve stenosis.
Since both are systolic flow patterns, it is not possible to separate mitral regurgitation from aortic valve stenosis.
The hallmark M-mode finding for aortic regurgitation is:
Chaotic diastolic flutter of the mitral valve
Systolic flutter of the aortic valve
Coarse diastolic flutter of the anterior mitral valve leaflet
Fine diastolic flutter of the anterior mitral valve leaflet
Fine diastolic flutter of the anterior mitral valve leaflet
The most common etiology of chronic aortic regurgitation is:
Marfan’s syndrome
Dilatation of the aortic root and aortic annulus
Trauma
Infective endocarditis
Dilatation of the aortic root and aortic annulus
The murmur associated with severe aortic regurgitation is:
Austin-Flint
Graham-Steell
Still’s
Carvallo’s
Austin-Flint
The murmur of aortic stenosis is described as:
Systolic ejection murmur heard best at the right upper sternal border
Holosystolic murmur heard best at the cardiac apex
Diastolic rumble
Holodiastolic decrescendo murmur heard best at the right sternal border
Systolic ejection murmur heard best at the right upper sternal border
The onset of flow to peak aortic velocity continuous-wave Doppler tracing in severe valvular aortic stenosis is:
Increased with inspiration
Decreased
Decreased with expiration
Increased
Increased
The pulse that is characteristic of significant valvular aortic stenosis is:
Pulsus bisferiens
Pulsus alternans
Pulsus paradoxus
Pulsus parvus et tardus
Pulsus parvus et tardus
The severity of aortic valve stenosis may be underestimated if only the maximum velocity measurement is used in the following condition:
Doppler intercept angle of 0°
Low cardiac output
Anemia
Significant aortic regurgitation
Low cardiac output
The two-dimensional view which best visualizes systolic doming of the aortic valve leaflets is the:
Subcostal short-axis view of the aortic valve
Parasternal short-axis view of the aortic valve
Parasternal long-axis view
Apical five-chamber view
Parasternal long-axis view