SMT-1 (Part 3) Flashcards

1
Q

What is the only study showing that a “cracking” sound is unnecessary?

A

Just one study published twice by Flynn et. al (2003) & Flynn et al. (2006)

It was using the Chicago HVT targeting SIJ.

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2
Q

What study shows that OA HVLAT increases activity and improves mouth opening?

A

Masilla-Ferragut et al (2009) JMPT

n=37 women with chronic (>6 months) bilateral mechanical neck pain and active month opening of less than 40 mm.

Results: A single session of HVLAT to the right and left OA joint immediately significantly increased active mouth opening by 3.5 mm.

Explanation: Reduction of reflex inhibition, allowing the masseter muscle to relax and thus increase in mouth opening ROM.

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3
Q

What is the main reason why OA HVLAT increases activity and improves mouth opening?

A

Reduction of reflex inhibition allows the masseter muscle to relax and thus increases mouth opening ROM.

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4
Q

What do the results of Koppenhaver et al. (2011) show?

A

The thickness of the Contracted Transverse Abdominus (TrA) and Internal Oblique (IO) musculature decreased immediately after the initial spinal manipulation technique (SMT). Still, it was NOT significantly different from baseline after 3-4 days or at one week.

*Lumbar Multifidus (LM) thickness significantly increased at 3 to 4 days immediately after two sessions of SMT, but no differences were found immediately after one session of SMT or a 1-week follow-up (following two sessions of SMT)

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5
Q

What was the only statistically significant result from the Koppenhaver et al. (2011) show?

A

TrA and IO, muscle thickness changes, occurred from baseline to 3-4 days immediately after session two of SMT in the LM muscle at L5-S1. Changes to LM were only present for a few seconds at L5-S1 immediately after the second session of SMT on day 3 or 4.

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6
Q

What are the results of Boal & Gillette (2004) show?

A

*SMT may alter afferent input to the central nervous system and, as a result, alter reflex mechanisms and ascending and descending pain-modulation elements of the pain system.

  • SMT causes the co-activation of both large-diameter A-beta and small-diameter A-delta, c-fiber mechanosensitive afferents, providing a counterirritant like tens, acupuncture, and vigorous deep massage.
  • SMT as an effect on the dynamic cellular mechanism within neurons-Neuronal plasticity.
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7
Q

What is Neuronal plasticity?

A

*Behavior changes in the dorsal horn of neurons are influenced by previous events that contribute to persistent pain states (Lont long-term potentiation LTP or central sensitization) or the reverse (Long Term Depression LTD).

*Provides a mechanism that contributes to the amplification of synaptic transmission in nociceptive circuits (central sensitization), leading to central neuronal elements overreacting to normal input with persistent hyperexcitability.

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8
Q

What is the post-cavitation refractory period?

What study hold support for this?

A

Once the joint has been cavitated, it cannot be recavitated for 15-30 minutes.

  1. Unsworth et. al (1971)
  2. Sandoz (1996)
  3. Mierau et. al (1989)
  4. Watson et. al (1989)
  5. Brodeur (1995)
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9
Q

Importance of the Boal & Gillette (2004) study?

A

*Spinal Manipulation Technique reverses long-term potential with an already sensitized pain signaling system.

*SMT may preferentially activate small diameter Alpha delta fibers in the skin and deep tissue, providing input to the spinal cord neurons, causing “spinal” LTD (Long-Term Depression) or suppressed neuronal discharge in the dorsal horn.

  • SMT provides sufficient forces to the paraspinal tissues to trigger activation of both A-delta and c-fibers mechanosensitive afferents, thus initiating LTD, which is responsible for reversing ongoing LTP (central sensitization) in dorsal horn that is likely participating in the generation of low back pain.

*LTD occurs quickly (within seconds to minutes) after afferent stimulation (SMT), and it has been observed to last for hours.

*The Long Term Depression mechanism is the explanation for the antinociceptive effect of various manual therapies, including SMT.

*LTD necessary to allow structures to assume and/or sustain normal biomechanics post-SMT.

*SMT may allow for normal (neuronal) activity, creating afferent input that decreases LTP and/or stimulates LTD.

*Spinal (dorsal horn cells) and supraspinal (brain)/ descending mechanisms appear to play upon and down-modulate nociceptive processing from LBP

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10
Q

The George, Bialosky & Bishop study (2006-2011) shows?

A

“temporal sensory summation” model, explaining the hypoalgesic mechanism post SMT.

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11
Q

Why 15-30 min post-cavitation refractory period?

A

Due to the presence of collapse gaseous fluid, which remains unresorbed in the joint. Upon attempted cavitation, this gas expands to prevent a sudden drop in intra-articular pressure. Thus, the cracking sound does not occur until all the gas in the join is completely resorbed (Sandoz 1969).

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12
Q

In what study investigated the side of the cracking with C3-4 HVLAT manipulations?

A

Bolton et al. (2007)

N of 20, by putting microphone in the skin

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13
Q

What are the results from the Bolton et al. (2007) study investigating the side of the cracking with C3-4 HVLAT manipulations?

A
  1. With C3-4 with the primary lever of ROTATION, it is more likely to occur on the CONTRALATERAL side of the applicator.
  2. C3-4 HVLAT with the primary lever of SIDE-BENDING, the popping was more likely to occur on the ipsilateral than the contralateral side to the applicator.
  3. The patient can’t even ID the side of cavitation.
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14
Q

Dunning et. (2013) al follow the Bolton et.al (2007) study:

What are the results from this study investigating the side of the cracking with C1-42 HVLAT manipulations?

A

Microphones were put on the C1 level.

Results:
1. HVLAT cavitations were bilateral event

  1. Cavitations were no more likely to occur ipsilateral than contralateral with respect to the primary application.
  2. Mean number of pops after bilateral HVLAT= 6.95
  3. Neither the patient nor the therapist could reliably determine the side of the cavitations.
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15
Q

What are the results from the study by Dunning et al. (2014) on the side and the number of cracks post CTJ post manipulation?

A

4:1 cracks ipsilateral: Contralateral post-CTJ manipulation

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16
Q

What was the validate study for CS pre-manipulation testing:

A

Lateral Glide Test (PAIVM) in the cervical spine.
-Fernandez De Las Penas et.al (2005)

N=25 patients with mechanical pain
Method: 2 PA cervical X-rays taken on each patient at the maximum end range of right and left lateral flexion.

Radiology results compared with blinded results of PT using C3-7 cervical lateral glide (PAIVM) test to diagnose the “hypermobile” segment and side of dysfunction.

The study has better reliability on C3 and C4 segments

Results: Intervertebral radiological motion at the hypermobile side was 3.44 mm less than the contralateral side at the dysfunctional segment

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17
Q

Main Result and Conclusion from Fernandez De Las Penas et.al (2005) study:

A

Results: Intervertebral radiological motion at the HYPERMOBILE side was 3.44 mm less than the contralateral side at the dysfunctional segment

Conclusion: The first study to provide evidence that the lateral gliding test for the cervical spine is comparable with radiological assessment for the diagnosis of hypomobility fromC3-7

Validation of the lateral glide test. Is possible to diagnose ZJ hypomobility with the test

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18
Q

Diagnostic criteria to justify a cervical manipulation.

What are the clinical criteria justifying spinal manipulative therapy for neck pain?

Based on Smith and Boton (2013) systematic review

A

Based on Smith and Boton (2013)

  • 30 RCT’s involving mobilization or manipulation for neck pain.

*SUbjects:
-43% of patients treated had Acute and chronic mechanical neck pain
-10% of studies included with CGH

*Clinical criteria used to determine the need for neck manipulation in over half (63%) of the RCT.
1-Exclusion of serious conditions
2-Manual examination for tenderness on palpation
3-Altred vertebral motion in the neck or upper thoracic region is known to lack validity.

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19
Q

Conclusion of the Smith and Boton (2013) systematic review?

A

Highlights the Absence of reliable and valid diagnostic protocols to determine the need for spinal manipulation in persons presenting with non-serious, idiopathic, or whiplash-associated (grade II) neck pain.

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20
Q

What is the purpose of the study by Cook & Hegedus (2011)

A

Purpose:
*Diagnostic utility of physical clinical test for spinal dysfunction. Examine sensitivity and specificity, +LR and -LR of 14 stand-alone physical clinical tests.

*Test for SCREENING (Ruling out disorder). A cut-off sensitivity of -LR <0..20 is useful.
-LR= how much the odds of the disorder decrease when the test is negative

*Test for DIAGNOSIS (ruling in disorders). A cut-off +LR> or = 5.0 is useful. +LR= how much the odds of the disorder increase when the test is positive.

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21
Q

What are the results of the study by Cook & Hegedus (2011)?

A

^Results:

A. For TS: No test with adequate +LR and -LR

B. For CS:
-Diagnostic test (+LR) (ONLY): Lateral glide test for C2-3 facet dysfunction

-Screening test (-LR) (ONLY);
1. Prone unilateral/ central PA’s for fascet dysfunction
2. Spurling’s test for radiculopathy

-Neither screening nor diagnostic test
1. Upper limb tension test (ULTT)
2. C5-6 lateral glide test

C. For LS:
-Diagnostic test (+LR)
1. Centralization for discogenic pain
2. PAIVM’s and PPIVM for radiologic instability
3. Percussion and supine signs for compression fractures

-Screening test (-LR)
1. Extension-rotation test for facet pain
2. Straight Leg Raise for Nerve root compression

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22
Q

What are the conclusions of the study by Cook & Hegedus (2011)?

A

Conclusions:

Stand-alone physical clinical tests provide only marginal value in diagnosing spinal conditions.

Clusters of tests appear to provide more promising findings and assist clinical decision-making.
(example: Laslett CPR for SI Pain)

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23
Q

Should we abandon cervical spine manipulation for mechanical neck pain?

A

NO!

*Cassidy et al. (2012): when risk, benefit, and patient preference are considered, there is currently no preferred first-line therapy and NO evidence that mobilization is safer or more effective than manipulation.

*Hurwitz et. al (2008):
The international multidisciplinary task force endorsed manipulation as one of several first-line treatments for neck pain, whiplash, and related headaches.

*Leaver et al. (2010)
Manipulation, multimodal PT, neck exercise, and drugs (paracetamol) all had significant short-term effects on pain compared with placebo. Acupuncture and manipulation had significant short-term effects on disability compared with placebo.

*Bronfort et. al (2012)
Spinal manipulation is more effective for acute & subacute neck pain (short & long-term) than management with NSAIDs or paracetamol.

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24
Q

This is a study that determines the cost-effectiveness of SMt compared to other treatment options for people with spinal problems of any duration.

A

Michaleff et. al (2012) Systematic Review

  • Results: SMT was superior to general practitioner (GP) care for neck pain regarding recovery and QOL. SMT was associated with lower total costs and higher rates of recovery. (Korthals de Bos et. al 2003)

*Two studies found SMT to be a more effective treatment option than an exercise program in terms of pain, recovery, and QOL. (Bosman et. al 2011, Korthals de Bos et. al, 2003)

*SMT is dominant compared with exercises for neck pain.

*Both trials found that SMT plus GP, rather than GP alone, was a more cost-effective treatment for LBP than GP care alone. (UK Beam trial, 2004; Williams et al., 2004)

*SMT plus GP care was a more cost-effective treatment than GP care plus exercise. (UK BEAM trial, 2004).

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25
Q

If I rotate the CS to the right, what side will it close?

A

The right side will close.
The same side will close with the rotation.

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26
Q

If I rotate the TS or LS to the right, what side will it close?

A

The right side will open.
Opposite of the CS.
The same side of the rotation will be open.

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27
Q

The Canadian Cervical Spine Rule has been develop to determine when radiographs of the CS are indicated in individuals with neck pain following trauma.

It has highly….

A

High Sensitivity. 100% Sensitive, lack specificity just 43%

It’s a most use tool for when evaluation an older individual with neck pain caused by trauma, PT should be alert for the presence of CS fx’s.

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28
Q

What was the Ross & Cheeks (2008a) case report about?

A

Detecting CS fracture and the Canadian CS rule:

In patients with neck pain caused by trauma, PT should be alert for the presence of cervical spine fractures, even if the initial radiographs are negative for a fracture.

  • In the case report, did A-P, lateral, and open-mouth X-ray views. The patient declines any medical and PT intervention. After eight months, f/u patient had No pain, AROM was WNL pain-free, and no neurologic deficits.

*Importance of screening for underlying cervical spine fractures in patients with neck pain following trauma-acute and subacute (8 weeks post-injury).

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29
Q

4 Risk Factors for using the Canadian Cervical Spine Rule to mandate X-rays:

A
  1. > 65y/o
  2. Paresthesia in the extremities & Dangerous MOI (fall from >1m or 5 stairs, axial load to the head, high-speed MVA (>100 km/h, rollover or ejection), bicycle collision, motorized recreational vehicle accident.
  3. Can the patient rotate the neck 45 degrees in both directions? If not, radiographs are required.
  4. Hx of osteoporosis, constant pain that was worse at night
30
Q

Three types of odontoid fractures:

A
  1. Oblique fracture in the upper portion of the odontoid.
  2. Fracture at the base of odontoid as it attaches to the body of C2 (most common type).
  3. The fracture line extends through the body of the axis.
31
Q

What things to ask for a proper neurological exam after CS trauma injury:

A
  1. Changes in sensation
  2. Strength deficits
  3. Gait
  4. Bowel & Bladder function
  5. UMN & LMN testing (DTRs, clonus, Babinski, Hoffman)
32
Q

What does that data show when testing the upper cervical ligament in a patient with Os odontoideum presenting with CGH.

A

Mintken et.al (2008)

-There is very little data to inform practitioners on the appropriate use of ligamentous stability tests when assessing the upper cervical spine. No evidence exists to support the use of the transverse ligament test (anterior shear test) as a valid test in clinical practice.

33
Q

What is an Os Odontoideum condition?

A

Where the dens are separated form the body of the axis od C2 vertebrae

34
Q

What is a Klippel-Feil congenital fusion?

A

Rare bone disorder that causes the abnormal fusion of two or more vertebrae in the neck (cervical spine

35
Q

Symptoms of Cranio-vertebral Instability:

A
  1. Occipital numbness
    Suggestive of compromised transverse ligament; thus excessive C1 anterior translation on C2, this irritates C2 spinal nerve, resulting in occipital numbness
  2. Neck-Tongue syndrome
    -Paraesthesia of ipsilateral ½ of tongue with contralateral rotation (suggestive of compromised alar ligament)
  3. Stretching of the C2 ventral Rami, which includes afferents from the Lingual nerve
    -FOR EXAMPLE: Right neck-tongue syndrome (right side of tongue numb) with LR
  4. Headache with sustained flexion
    -May be indicative of a compromised transverse ligament
  5. Lump in throat during flexion (gagging, clunk)
  6. Cord Signs
36
Q

What are you looking for with open mouth XR?

A
  1. Is the spinous process of C2 midline?
  2. Are the periodontoid spaces equal?
  3. Are the articular margins of C1 and C2 lined up?

***IMPORTANT: Don’t have to have all 3 features above to have an alar ligament tear

37
Q

Why NOT use the craniovertebral instability tests?

What do Osmotherly and Rivett (2011) show?

A

There is little to no evidence to establish the validity of the use of clinical stability tests of the upper cervical spine (mintken et. el 2008)

The inter-examiner reliability of the transverse ligament (anterior drawer test), alar ligament (lateral displacement) test, and sharp purser test have all been found to be “poor to unacceptable.” (Cattrysse et al. 1997)

38
Q

What is Cervico Genic Headaches (CGH)

A

Based on the International Headache Society:

“Pain refers to a source in the neck & perceived in one or more regions of the head and/or face.”

39
Q

What are the dominant features of CGH:

A

Unilateral head pain without side shift, combined with neck pain and limited neck range of motion (SJaastad et al. 1998)

40
Q

Diagnostic criteria for cervicogenic headache by (SJaastad et al. 1998)

Symptoms & signs of neck involvement

A

Main Two:
*1. Head pain by neck movement and sustaining awkward head positioning
*2. Ipsilateral neck, shoulder, and arm pain (vague nonradicular nature, occasionally arm pain of a radicular nature).

  1. By External pressure over the upper cervical or occipital region
  2. Confirmatory evidence by diagnostic anesthetic block
  3. Unilaterality of the head pain, without side shift
  4. Head pain characteristics
    -Mod to severe, non-throbbing, and nonlacinating pain, usually starting in the neck. Episodes vary in duration, fluctuation, and continuous pain.
  5. Other characteristics:
    - Only marginal effect or lack of effect from indomethacin, ergotamine or sumatriptan
    -female
    -history of head or neck trauma
41
Q

What of the CS is the primary pain generator for CGH:

A

C1-C2

80% of the CGH subjects (Hall & Robinson, 2004)

72% of CGH subjects by (Zito et.al 2006)

63% of CGH subjects (Hall et.al 2010)

42
Q

What is the pathogenesis of CGH:

A

Convergence of afferent of the TRIGEMINAL & UPPER THRE CERVICAL SPINE NERVES onto the second-order neurons in the TRIGEMINO-CERVICAL NUCLEUS in the upper cervical spine cord is likely to lead to headache (Bogduk ,2004)

43
Q

What does Bogduk’s (2005) study show?

A

C0-C3 joints, discs, ligament, muscle, dura.

Local arteries

44
Q

C1-2 Flexion-Rotation Test (FRT) unilateral ROM normative values in asymptomatic have been established by:

A

Hall & Robinson (2004): 45 deg was normal

Amiri et.al (2003); 42 deg was normal

Ogince et.al (2007): 39 deg was normal

45
Q

How sensitive and specific is the Flexion-Rotation Test (FRT)?

A

Sensitivity: 91%
Specificity: 90%

46
Q

What was the average PROM for the Flexion-Rotation Test (FRT) with CGH patients?

A

20 degrees of right and left rotation

47
Q

What was the average PROM for the Flexion-Rotation Test (FRT) with migraine patients with aura or asymptomatics?

A

39 deg

48
Q

What was the “cut-off” for a positive Flexion-Rotation Test (FRT) in the Ogince et.al 2007 study?

A

32 deg

49
Q

What is the conclusion of Ogince et.al 2007?

A

FRT is an accurate and reliable clinical measure in identifying C1/2 dysfunction and the differential diagnosis of cervicogenic headache.

50
Q

What is the best specific and reliable test for CGH:

A

Flexion-Rotation Test (FRT)

51
Q

What is the CN V:

A

Trigeminal Nerve:
1. Opthalmic
2. Maxilary
3. Mandibular

52
Q

The main function of the Trigeminal Nerve:

A

-The main sensory nerve of the scalp, face, cornea, oral and nasal cavities and cranial dura mater

-Proprioceptive pathway for the muscle of mastication and TMJ- auriculotemporal branch from the mandibular branch of the trigeminal nerve supplies TMJ

-Motor supply to the muscle of mastication by mandibular branch (masseter, temporalis, medial & lateral pterygoids.

53
Q

Main Trigeminal nerve branch involved with CGH pain:

A

The more dense projection from the C1-3 is the ophthalmic branch.

54
Q

The most common complaints in patients with CGH :

A

Orbitofrontal and parietal regions pain based on Bogduk 2005

55
Q

What CNs are involved with ear pain, throat pain, and swallowing?

A

CN VII, IX, X

56
Q

What are the three trigeminocervical nuclei:

A
  1. Mesencephalic nucleus: Proprioception
  2. Main Sensory Nucleus: Touch/Pressure
  3. Spinal Nucleus: Pain/Temperature
57
Q

What did the (Jull et.al 2002) study show:

A

100% in the manipulative therapy group got better.

58
Q

Evidence for the use of SMT in CGH:

What did the Cochrane Systematic Review (Bronfort et.al 2005) show:

A

*SMT and low-intensity endurance training are effective for CGH.

*SMT may be effective for migraine and has a similar effect to amitriptyline

*SMT is less effective than amitriptyline for TTHs

59
Q

What did the Zito et. al (2006) show

A

Validity of pain provocation, AROM, and PPIVM testing in diagnosing CGH.

*All hypomobile upper cervical joints were not necessarily painful, but all painful upper cervical joints were Hypomobile.

The principal segment was C1/2 segments.

Painful C0-3 segmental dysfunction was not found in migraine or control subjects.

60
Q

Studies that show the pain referred to the head from the C-spine:

A

Bogduk and Govind (2009) or Jull (1997)

61
Q

CGH diagnostic sign and sx’s Based on Sjaastad et.al (1998):

A

-Unilateral head pain
-W/o side shift
-Neck pain (with movement & palpation)
-Limited cervical ROM

62
Q

What did Zito et. al (2006) show between cervicogenic vs migraine headaches?

A

Manual examination and pain provocation of the upper C-spine can distinguish cervicogenic headaches from migraine headaches: 80% sensitivity.

63
Q

The study that validates the lateral glides and puts them as good as radiological assessment for C3-7?

A

Fernandez-de-las Penas (2005)

64
Q

The study finds that 70% of the inter-examiner agreement of C0-C3 agreement followed lateral glide testing.

A

Jull et.al (1997)

65
Q
  1. Study that shows that CGH C2/3, the most likely source of pain via Diagnostic injections :
  2. Study that shows that CGH, C1/C2 is the primary pain generator via Manual examination:
A
  1. (Bagduk 2005)
  2. Hall and Robinson (2004) & Jull et. al (1997)
66
Q

*Ogince et al. 2007 show that C1-C2 FRT for 39 deg, 32 deg, and 20 deg was:

A

39 deg: Average Passive Cervical rotation for patients with migraine headaches.

32 deg: Cutt-off for a passive flexion rotation test

20 deg: average passive cervical rotation of patients with cerviogenic headaches

67
Q

What did the C1-C2 FRT study from Hall and Robinson (2004) show:

A

The greater the restriction in the flexion rotation test, the more severe the headaches.

68
Q

The importance of the Nier & Robinson (1997) study:

A

Following SMT, HA Frequencies, Durations, and intensities significantly reduced, and improvements were 50% better.

69
Q

What is the most valuable indicator of treatment effect when treating CGH?

A

HA frequency

70
Q

What are three things to look for when treating HA to measure successful outcomes:

A

Frequencies
Intensity
Duration