Small Bowel obstruction, large bowel obstruction Flashcards

1
Q

acute colonic pseudoobstruction (Ogilvie syndrome)

A

dilated colon without mechanical observation; unclear etiology from it but likely from combination of autonomic nervous systems or electrolyte abnormalities

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2
Q

causes of acute colonic pseudoobstruction (Ogilvie’s)

A

major surgery, traumatic injury, severe infection
electrolyte derangement (low K, low Mg, low Ca)
medications (opiates, anticholingerics)
neurological disorders (stroke, dementia)

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3
Q

clinical findings of acute colonic pseudoobstruction

A

abdominal distention, pain, obstipation, vomiting
tympanic to percussion, decreased bowel sounds
if perforation, guarding, rigidity rebound tenderness

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4
Q

Imaging findings of acute colonic pseudoobstruction

A

Xray: colonic dilation, normal haustra, non dilated small bowel

CT scan with colonic dilation without anatomical obstruction

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5
Q

Management of acute colonic pseudoobstruction

A

NPO, nasogastric or rectal tube decompression

supportive care
neostigmine if no improvement 48 hrs or if cecal diameter is >12 cm.

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6
Q

who gets acute colonic pseudoobstruction (Ogilvie syndrome)?

A

institutionalized men age>60 or hospitalized.

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7
Q

who should not get neostigme?

A

those with recent MI, bradycardia and beta blocker therapy or active bronchospasm or PUD.

It can lead to bradycardia, bronchospasm, hypotension.

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8
Q

if pt fails neostigme for acute colonic pseudoobstruction what to do next

A

may need colonscopic or surgical decompression including a percutaneous tube cecostomy

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9
Q

do we ever use methylnatrexone for acute colonic pseudoobstruction treatment?

A

no because it can increase risk for bowel perforation.

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10
Q

crampy periumbilical abd pain with nausea and vomiting without radiatio nto the back and history of abdominal surgery

A

likely proximal small bowel obstruction.

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11
Q

complete bowel obstruction characteristics:

A

obstipation and can’t pass flatus

generally starts 12 to 24 hrs after onset of obstruction.

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12
Q

what is the next step to do after suspected bowel obstruction?

A

get CXR upright and supine abdominal films to diagnose SBO

do not need further imaging.

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13
Q

what is seen on XR for SBO?

A

see multiple air-fluid levels with distended small bowel loops. air in colorectal area rules out complete obstruction

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14
Q

when to order CT abd with oral and IV contrast?

A

when CXR and AB XR are non diagnostic for SBO

CT scans are better at looking at SBO etiology, evidence of ischemia or closed loop obstructions.

But if CT abd is non diagnostic then order a small bowel series (as they are as great as CT scans).

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15
Q

non operative management of SBO is:

A

NPO, fluid resuscitation and electrolyte repletion and serial abdominal examinations and strict monitoring of UOP and NG output.

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16
Q

when do you know SBO is improved?

A

decreased abdominal distension, passage of flatus and stool and decreased NG output

17
Q

what can also be used to help diagnose and treat SBO?

A

water soluble gastrografin contrast study with CT scan can help assess strangulation and help with therapy.

18
Q

why is gastrografin contrast study helpful for treatment of SBO?

A

gastrografin is hypertonic and draws water into the intestinal lumen and decreases wall edema abd stimulates intestinal peristalsis and can lead to improved bowel function with faster symptom resolution

Presence of contrast in COLON 4-24 hrs after administration is a good predictor of SBO resolution and lack of this may mean that surgery is needed.

19
Q

when to get surgery for SBO

A

worsening clinical symptoms, complete obstruction and strangulation or necrosis.
Seen about 25% of pts with SBO.

20
Q

in SBO what happens to lipase

A

may be slightly elevated.

Some other causes of non pancreatic elevations for lipase are renal insufficiency, DKA, and SBO or ileus.