Sleep Disorders Flashcards

1
Q

sleep

A
  • a physiological process
  • the body’s rest cycle
  • associated with recumbency and immobility
  • lacks conscious awareness but easily awakened
  • essential for healthy functioning and survival
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2
Q

sleep

A
  • state in which an individual lacks conscious awareness of environmental surroundings but can be easily aroused
  • insufficient sleep: obtaining less than the recommended amount of sleep, most adults require 7-8 hrs of sleep in a 24 hr period
  • fragmented sleep: frequent arousals or actual awakenings that interrupt sleep
  • nonrestorative sleep: sleep that is of adequate duration but does not result in the individual feeling refreshed and alert the next day
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3
Q

sleep disturbances and disorders

A
  • sleep disturbance: conditions of poor sleep quality
  • sleep disorders: abnormalities unique to sleep
    – insomnia
    – narcolepsy
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4
Q

sleep-wake cycle

A
  • controlled by the brain
  • wake behavior
    – RAS and various neurotransmitters: reticular activating system helps control sleep-wake cycle
    – orexin (hypocretin): helps regulate sleep-wake cycle, helps keep people awake, low level of orexin = prone to narcolepsy
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5
Q

reticular activating system (RAS)

A
  • sensory stimuli within cerebral cortex
  • regulates sleep-wake cycle
  • 4 functions:
    – motor
    – sensory
    – visceral: normal functioning of the organs, like breathing
    – consciousness
    (activation of RAS causes alertness and attention, when RAS is activated it affects the 4 functions)
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6
Q

circadian rhythm

A
  • managed by the suprachiasmatic nucleus (SCN) in hypothalamus
  • synchronized through light detectors in retina
  • light is the strongest time cue
    (circadian rhythm: biologic rhythms of behavior and physiology within a 24 hr period, strongly linked to light,
    time cue: body meant to be awake when it is light, and asleep when it is dark,
    ICU psychosis: pt not being able to distinguish day and night (bc almost never dark and lots of noise in ICU)
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7
Q

stages of sleep - chart

A
  • NREM stage 1: 5%
  • NREM stage 2: 50% (most)
  • NREM stage 3: 15%
  • REM: 25%
  • wake after sleep onset: 5%
    (majority of time in non REM sleep)
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8
Q

phases of sleep

A
  • sleep latency: time it takes for a person to fall asleep
  • NREM (non-rapid eye movement): 3 stages
  • REM (rapid eye movement)
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9
Q

sleep latency

A
  • starts when eyes are closed for sleep
  • ends when non-REM sleep is entered
  • time varies: usually 10-40 minutes
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10
Q

NREM sleep

A
  • 75% to 80% of sleep time
  • divided into 3 stages
    – stage 1: slow eye movement (person can be easily awakened)
    – stage 2: HR and temperature decrease (spend most of our night’s sleep)
    – stage 3: deep or slow wave sleep (SWS); delta waves, parasomnias (difficult to awaken, decreases with age - older we get, less deep sleep we get)
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11
Q

parasomnias

A
  • unusual and often undesirable behaviors while falling asleep, transitioning between sleep stages, or during arousal from sleep
  • due to CNS activation
  • examples:
    – sleepwalking
    – sleep terrors
    – nightmares
    – sleep paralysis
    – sleep hallucinations
    (common in stage 3 - slow wave sleep)
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12
Q

REM sleep

A
  • 20-25% of sleep cycle
  • occurs 3-4 times a night (cycle through sleep cycle 3-4 times a night)
  • greatly reduced skeletal muscle tone
  • period when most vivid dreaming occurs
    (brain is very active but mentally restful)
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13
Q

stages of sleep - chart (she didn’t talk about)

A
  • NREM stage N1: falling asleep
    – heartbeat and breathing slow down
    – muscles begin to relax
    – lasts: a few min.
  • NREM stage N2: light sleep
    – heartbeat and breathing slow down further
    – no eye movements
    – body temperature drops
    – brain produces “sleep spindles”
    – lasts: about 25 min.
  • NREM stage N3: slow wave sleep
    – deepest sleep state
    – heartbeat and breathing are at their slowest rate
    – no eye movements
    – body is fully relaxed
    – delta brain waves are present
    – tissue repair and growth, and cell regeneration
    – immune system strengthens
  • REM stage R:
    – primary dreaming stage
    – eye movements become rapid
    – breathing and heart rate increases
    – limb muscles become temporarily paralyzed
    – brain activity is markedly increased
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14
Q

aging and sleep

A
  • middle age:
    – more stage shifts: down in NREM3 and REM
    – resistant to sleep deprivation: can function on lower amount of sleep fairly well whereas younger person needs more sleep to focus
    – increased awakenings
    – changes in sleep efficiency
  • older adults:
    – phase changes: go to bed earlier and arise earlier
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15
Q

which statement is true regarding REM sleep? select all that apply.
a. muscle tone is greatly reduced
b. it occurs only once at night
c. it is separated by distinct physiologic stages
d. the most vivid dreaming occurs at this phase

A

a. muscle tone is greatly reduced
d. the most vivid dreaming occurs at this phase

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16
Q

what is the best description for sleep?
a. quiet state in which there is little brain activity
b. loosely organized state similar to coma
c. state in which pain sensitivity decreases
d. state in which the individuals lack conscious awareness of the environment

A

d. state in which the individual lacks conscious awareness of the environment

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17
Q

effects of sleep deprivation and sleep disorders - picture

A
  • neurologic:
    – cognitive impairment
    – behavioral changes (e.g., irritability, moodiness)
  • immune:
    – impaired function
  • respiratory:
    – asthmas exacerbated during sleep
  • cardiovascular:
    – heart disease (hypertension, dysrhythmias)
    – increased BP in people with hypertension
    – stroke
  • gastrointestinal:
    – increased risk for obesity
    – increased gastroesophageal reflux disease (GERD)
  • endocrine:
    – increased risk for type 2 diabetes
    – increased insulin resistance
    – decreased growth hormone
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18
Q

sleep disturbances in the hospital

A
  • hospitalization associated with decreased sleep time
    – environmental sleep-disruptive factors
    – psychoactive medications
    – acute and critical illness
    (want to have quiet time, turn TV off, turn off lights so that pt has better sleep time)
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19
Q

insomnia

A

symptoms include:
- difficulty falling asleep
- difficulty staying asleep
- waking up too early
- complaints of waking up feeling unrefreshed

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20
Q

insomnia

A

acute insomnia:
- difficulty falling asleep or remaining asleep for at least 3 nights/week for less than a month
chronic insomnia:
- same symptoms as acute
- daytime symptoms that persist for 1 month or longer

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21
Q

insomnia

A

aggravated by inadequate sleep hygiene
- stimulants
- medications
- using alcohol to induce sleep (reduces REM sleep, more insufficient sleep)
- irregular sleep schedule
- nightmare
- exercising near bedtime (not good within 6 hrs of bedtime)
- jet lag

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22
Q

chronic insomnia

A

etiology:
- often no known cause
- stressful life event
- psychiatric illness or medical condition
- medications or substance abuse

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23
Q

insomnia

A

clinical manifestations:
- difficulty falling asleep (long sleep latency)
- frequent awakening (fragmented sleep)
- prolonged nighttime awakenings
- feeling unrefreshed on awakening (nonrestorative sleep)
- fatigue, trouble with concentration
- forgetfulness, confusion
- anxiety

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24
Q

insomnia

A

diagnosis:
- self-report
- actigraphy
- polysomnography (PSG): sleep study
– at home sleep study: thing on forehead and nasal cannula type thing
– traditional sleep study: done in office with the full stuff (EEG, EOG/ECG, oronasal air flow, chin EMG, position sensor, pulse ox, thoracic and abdominal movements, control unit)
(most common to self-report,
1st thing when might have insomnia is to keep a sleep log (time went to bed, time woke up, did they wake up during the night, did they feel refreshed?) keep sleep log for 2 weeks)

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25
Q

actigraphy

A

watch like device, worn on wrist, that can determine sleep and wake over a 14 day period

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26
Q

insomnia

A

interprofessional care
- education
- track sleep
- sleep hygiene (table 7-3)
- cognitive-behavioral therapy for insomnia
- complementary and alternative therapies (alt ex: aromatherapy)
- drug therapy
(since insomnia can be bc of stress and mental health, therapy is 1st line of treatment (bc it is least invasive and may solve problem without using meds)

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27
Q

patient teaching - chart

A

yes, no???? _________slide 32

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28
Q

insomnia

A

causes:
- psychiatric
- medical illness, medications
- stress: finances, employment, school, life
- substances: caffeine, alcohol, nicotine
- exercise
- age, gender (men sleep better than women)
- other factors: travel

29
Q

insomnia

A

treatment:
- begin with least invasive
- cognitive behavior therapy
- therapist: counseling

30
Q

insomnia - drug therapy

A
  • benzodiazepines
  • benzodiazepine-receptor like agents
  • melatonin-receptor agonist
  • antidepressants
  • antihistamines
  • alternative therapies
    (melatonin - hormone that helps induce sleep, designed for short term use so won’t have same chemical effect if used longer than 2 weeks-month, not intended for daily use,
    sleep is very important for those in mental health crisis)
31
Q

insomnia - drug therapy

A

sedative-hypnotic drugs
- drugs that depress CNS function
- primarily used to treat anxiety and insomnia
- antianxiety agents or anxiolytics
- distinction between antianxiety effects and hypnotic effects depends on dosage

32
Q

insomnia - drug therapy

A

benzodiazepines
- used to treat anxiety and insomnia
- used to induce general anesthesia
- used to manage seizure disorders, muscle spasms, panic disorder, and alcohol withdrawal
- potential for abuse (have to see provider every 3 months)
- can produce physical dependence

33
Q

insomnia - drug therapy

A
  • benzos used specifically for sleep
    – temazepam (Restoril)
    – triazolam (Halcion)
  • common benzos (for anxiety, seizures, panic disorders) (will make a person sleepy but not commonly prescribed for sleep)
    – diazepam (Valium)
    – lorazepam (Ativan)
    – alprazolam (Xanax)
34
Q

Benzos

A

pharmacologic effects
- CNS: reduce anxiety and promote sleep
- cardiovascular system: oral vs. intravenous
- respiratory system: weak respiratory depressants
therapeutic effects:
- anxiety
- insomnia
- seizure disorders, muscle spasm
- ETOH withdrawal, perioperative applications
(resp. depression = resp. rate down, resp. depth down,
benzos will cause some resp. depression,
alcohol withdrawal - causes tremors, delusion tremors, so benzos can calm them down,
perioperative - to calm patient down before procedure)

35
Q

benzos

A

adverse effects
- CNS depression
- amnesia
- sleep driving
- paradoxical effects
- respiratory depression
- abuse

36
Q

benzos

A
  • acute toxicity
  • oral overdose: drowsiness, lethargy, and confusion
  • intravenous toxicity: life-threatening reactions, profound hypotension, respiratory arrest, and cardiac arrest
    (dangerous drugs, don’t want them on benzos for too long)
37
Q

benzos

A
  • general treatment measures
    – oral: gastric lavage, activated charcoal, and dialysis
  • treatment with flumazenil (Romazicon)
    – competitive benzodiazepine receptor agonist
    – reverses sedative effects if benzodiazepines but may not reverse respiratory depression
    – monitor for seizures when benzodiazepine stopped
    – effects fade an hour after administration: monitor for sedation
    (general treatment measures: used to get benzos out of their system)
38
Q

benzos-receptor-like agents

A

Zolpidem (Ambien)
- sedative-hypnotic
- short-term management of insomnia
- side effects: daytime drowsiness and dizziness
(causes strange things like sleep driving, sleep shopping, sleep baking)

39
Q

benzos-receptor-like agents

A

Zaleplon (Sonata)
- approved for short-term management of insomnia
- most common side effects: headache, nausea, drowsiness, dizziness, myalgia, and abdominal pain

40
Q

benzos-receptor-like agents

A

Eszopiclone (Lunesta)
- approved for treating insomnia
- no limitation on how long it can be used
- generally well tolerated
- adverse effect: bitter aftertaste, headache, somnolence, dizziness, and dry mouth
(better than ambien - less side effects, better tolerated)

41
Q

antidepressants

A
  • trazodone (Oleptro)
    – atypical antidepressant with strong sedative actions
    – can decrease sleep latency and prolong sleep duration
    – does not cause tolerance or physical dependence
  • doxepin and amitryptyline
    – old tricyclic antidepressant with strong sedative actions
    – used to treat patients who have trouble staying asleep
42
Q

antihistamines

A
  • diphenhydramine (Benadryl)
    – may be added to nighttime cold/pain preparations
  • doxylamine (Unisom)
    – can be purchased without prescription
    – less effective
    – tolerance develops quickly (1-2 weeks)
    – adverse effects: daytime drowsiness and anticholinergic effects
    – not intended for long term use
43
Q

alternative medicines

A

complementary and alternative therapies
- melatonin: effective related to jetlag and shift work
- valerian root, chamomile, passionflower, lemon balm, lavender: have very mild sedative effects, proof of benefits in insomnia is lacking
- white noise and relaxation strategies

44
Q

insomnia - nursing management

A

nursing assessment:
- sleep history
- assess diet, caffeine, and alcohol intake
- ask about sleep aids
- sleep diary for 2 weeks
- medical history: factors that affect sleep

45
Q

insomnia - nursing diagnoses

A
  • sleep deprivation
  • disturbed sleep pattern
  • readiness for enhanced sleep
46
Q

insomnia - nursing implementation

A
  • assume primary role in teaching sleep hygiene
    – decrease caffeine intake
    – bedtime routine
    – decreased blue light before bedtime
    – reduce light and noise
  • teach patient about sleep medications
47
Q

Epworth Sleepiness Scale

A

situations
- sitting and reading
- watching television
- sitting inactive in a public place (e.g., theater or class)
- as a passenger in a car for an hour w/o a break
- sitting and talking to someone
- sitting quietly after lunch w/o alcohol
- in a car, while stopped for few minutes in traffic
rate situations: 0 - never doze, 1 - slight chance of dozing, 2 - moderate chance of dozing, 3 - high chance of dozing
1-6: getting enough sleep
7-8: average score
9+ very sleepy and should continue to seek sleep assistance

48
Q

sleep apnea occurs

A

bc airway gets closed
tongue shifts back, larger neck circumference causes tongue and epiglottis to shift back and airway is closed

49
Q

sleep apnea

A

symptoms:
- loud snoring
- excessive day time sleepiness
- frequent episodes of obstructive breathing during sleep
- morning headache
- unrefreshing sleep
- increased irritability
treatments:
- non-surgical:
– change sleep position
– decrease weight
– CPAP (continuous positive airway pressure)
– drug therapy for underlying causes
- surgical:
– adenoidectomy
– uvulectomy
– remodeling posterior oropharynx
– bariatric surgery to decrease weight (less weight = less pressure that closes airway)

50
Q

respiratory and sleep problems

A
  • sleep apnea
  • snoring and hypoventilation
  • obesity hypoventilation syndrome
  • reduced chest wall compliance
  • more work of breathing
  • less total lung capacity and functional residual capacity
  • also called obstructive sleep apnea-hypopnea syndrome (OSA)
  • partial or complete upper airway obstruction during sleep
  • apneic period may include hypoxemia and hypercapnia
    complications can result in:
  • hypertension
  • cardiac changes
  • poor concentration/memory
  • impotence
  • depression
51
Q

clinical signs and symptoms

A
  • apnea is cessation of spontaneous respiration for longer than 10 seconds
  • each obstruction may last from 10-90 seconds
  • apnea and arousal cycles occur repeatedly, as many as 200-400 times during 6-8 hours of sleep
  • frequent arousal during sleep
  • insomnia
  • excessive daytime sleepiness
  • witnessed apneic episodes
  • loud snoring
  • morning headache
  • irritability
52
Q

risk factors

A

sleep apnea occurs in 2-10% of americans but is considered to be underreported
- obesity (BMI > 28kg/m2)
- age > 65 yrs
- neck circumference > 17 inches
- craniofacial abnormalities that affect the upper airway, and acromegaly
- smokers are more likely to have OSA
- OSA is more common in men than in women until after menopause, when the prevalence of the disorder is the same in both genders. women with OSA have higher mortality rates.

53
Q

lab and diagnostic testing

A

may take 1 or 2 nights for complete diagnosis depends
- polysomnography aka sleep study

54
Q

treatment

A

mild sleep apnea:
- sleeping on one’s side
- elevating head of bed
- avoiding sedatives and alcohol 3-4 hrs before sleep
- weight loss
- oral appliance
severe (>15 apnea/hypopnea events/hr):
- CPAP (continuous)
– possible compliance issues
- BiPAP (one pressure on inspiration, one pressure on exhalation, more complicated bc has to sense when inhaling/exhaling)
- surgery
– uvulopalatopharyngoplasty (UPPP or UP3)
– genioglossal advancement and hyoid myotomy (GAHM)
(bring BiPAP or CPAP when traveling and in hospital (bc you have what is comfortable for you))

55
Q

patient teaching

A

if patient on CPAP or BiPAP remind them to always take with them if overnight trips are planned.
- explain benefit of loosing weight
- explain benefit of sleeping on side
- explain the need to avoid sedatives or alcohol 3-4 hrs prior to bedtime
- stress reduction
- stress importance of exercise
- avoid smoking
- self imagine disturbance

56
Q

discharge planning

A
  • case manager: need for equipment
  • nutritional consult
  • spiritual consult
57
Q

nursing diagnosis

A
  • anxiety
  • insomnia
  • imbalanced nutrition more than body requires
  • knowledge deficit
58
Q

CPAP with nasal mask - picture

A

management of sleep apnea often involves sleeping with a nasal mask in place. the pressure supplied by air coming from the compressor opens the oropharynx and nasopharynx.
other options are:
- CPAP with nasal pillow

59
Q

narcolepsy

A
  • brain unable to regulate sleep-wake cycles normally
  • causes uncontrollable urges to sleep, often go directly into REM sleep
  • unknown cause
    – associated with destruction of neurons that produce orexin
    – orexin: neuropeptide that regulates sleep/wake cycles
    – low levels of orexin lead to difficulty staying awake
    (onset: adolescence to early in third decade - could occur anytime)
60
Q

narcolepsy

A

2 types:
- type 1: with cataplexy
- type 2: without cataplexy
symptoms:
- sleep paralysis (mind awake, body asleep, can’t move)
- cataplexy (brief and sudden loss of skeletal muscle tone that can manifest as muscle weakness or complete collapse and falling, can be strongly linked to emotions)
- fragmented nighttime sleep

61
Q

narcolepsy - nursing and interprofessional management

A
  • teach about sleep and sleep hygiene
  • take naps (take 3 or more short 15 mins naps throughout the day)
  • avoid heavy meals and alcohol
  • ensure patient safety (no driving or swimming)
  • lifestyle changes
62
Q

narcolepsy - drug therapy

A
  • Modafinil (Provigil)
  • Armodafinil (Nuvigil)
  • both are wake-promotion drugs
63
Q

gerontologic considerations

A

older age is associated with:
- overall shorter total sleep time
- decreased sleep efficiency
- more awakenings (use bathroom, drink water, pain, resp. issues may keep them from breathing well during sleep)
- insomnia symptoms
(they need the same amount of sleep it is just hard to get it)

64
Q

gerontologic considerations

A

sleep:
- awakenings during the night increases risk for falls
- medications used by older adults can contribute to sleep problems
- avoid long-acting benzos (benzos can cause daytime sleepiness)

65
Q

nurse fatigue

A

(can be physically exhausted or mentally (emotionally) exhausted)
- inadequate sleep
- extended work hours
- increased risk for errors

66
Q

nurse fatigue

A
  • patients are at risk
  • awake for 17 hrs = same cognition as blood alcohol level 0.05%
  • awake for more than 24 hrs = blood alcohol level 0.10%
67
Q

national academy of medicine

A
  • formerly institute of medicine: nonprofit organization devoted to safety and evidence-based practice in healthcare
  • recommendations:
    – no more than 12 hrs work in 24 hr period
    – limit to 60 hrs of work in a 7 day period
  • do:
    – take at least one break plus a lunch break
    – use caffeine therapeutically as a stimulant to stay awake
    – nutrition: complex carbs and protein
    – exercise
  • don’t:
    – do not drink alcohol (depressant)
68
Q

shift work sleep disorder

A

characterized by:
- insomnia
- excessive sleepiness
symptoms:
- fatigue or malaise
- difficulty paying attention or concentrating
- memory impairment
- mood disturbance or irritability
- excessive daytime sleepiness
- hyperactivity, impulsivity, aggression, and other behavioral problems
- reduced motivation, energy, or initative
- higher risk of errors or accidents
- feelings of sleep dissatisfaction

69
Q

special sleep needs of nurses

A
  • nurses on rapidly rotating (some days, some nights) shifts are at increased risk of experiencing shift work sleep disorder
  • use strategies to help reduce distress associated with shift work
    – on-site napping
    – consistent sleep-wake schedule