Sleep Flashcards
Why?
Prunes + regulates synaptic connections, emotion and social function
Nir + Tononi 2010
PET reduced activity in brain stem and thalamus, like anaesthetics. SWS reduced activity in cortical areas, still emotion. Dolphins, dream in all stages
Siegel 2008
Against SWS, so much variation across species
Pare + Linas 1995
Cognition is unconscious, thalamocortical disabled in REM, dreams are unconscious or pre-conscious
Hennevin et al. 2006
Dreaming is unconscious - neuronal signals serve no function
Hobson & Voss 2011
Primary vs. Secondary. AIM model, PGO as input. Dreaming provides a model of psychosis
Voss et al. 2009
Lucid dreaming shows global activity, hybrid state - AIM isn’t static
Solms 2000
Psychodynamic theory: REM = pons, dreams = higher cortical and OTPJ. Dreaming isn’t neural, associated with goal-direction and dopamine libidinal drive
Leslie et al. 2007
Dreams are reported during anaesthesia
Gernnaro et al. 2012
V1 lesions don’t stop dreaming. Dreams rely on higher order cortex bottom-up
Domhoff & Fox 2015
Dreaming reflects DMN, mind-wandering. DMN still active during light sedation
Review of PET studies, reduced activity subcortically
Nir + Tononi 2010
Variation across species with SWS
Siegel 2008
Cognition is unconscious, thalamocortical systems are disabled in REM, dreams are preconscious
Pare + Linas 1995
Dreams are not conscious - neuronal signals have no functional relevance
Hennevin et al. 2006
Primary vs. secondary: AIM model. PGO input - dopamine, dreaming is a model of psychosis
Hobson + Voss 201`1
Lucid dreaming
Voss et al. 2009
Psychodynamic theory of dreaming
Solms 2000
Dreams after surgery
Leslie et al. 2007
V1 lesions don’t block dreams: not bottom-up
Gennaro et al. 2012
Default mode network: min-wandering. Persistent with light sedation
Domhoff + Fox 2015
