Skin infections/infestations Flashcards

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1
Q

What is erysipelas/cellulitis?

A

Both are spreading bacterial infections of the skin
Cellulitis involves the deep subcutaneous tissue
Erysipelas is an acute superficial form of cellulitis and involves the dermis and upper subcutaneous tissue

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2
Q

What are the causes of erysipelas/cellulitis?

A

strep pyogenes

and staph aureus

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3
Q

What are the risk factors for erysipelas/cellulitis?

A
Immunosuppression 
Wounds 
Leg ulcers 
Toeweb intertrigo (burning sensation between toes)
Minor skin surgery
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4
Q

How does erysipelas/cellulitis present?

A
Swelling (tumor) 
Erythema (rubor) 
Warmth (Calor) 
Pain (Dolor) 
May be associated with lymmphangitis (inflammation of the lymphatic system)
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5
Q

How is erysipelas distinguished from cellulitis?

A

Erysipelas will have a well defined, red raised border

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6
Q

What is the management of cellulitis?

A

Antibiotics- flucloxacillin or benzylpenicillin

Supportive care- rest, leg elevation, sterile dressings, analgesia

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7
Q

What are the complications of erysipelas/cellulitis?

A

Local necrosis
Abscess
Septicaemia

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8
Q

What is the cause of staphylococcal scalded skin syndrome?

A

Production of a circulating epidermolytic toxin from phage group 2, benzylpenicillin- resistant (coagulase positive) staphylococci

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9
Q

What is the presentation of staphylococcal scalded skin syndrome?

A

Develops within a few hours to a few days, may be worse over the face, neck, axillae, or groins
A scald like skin appearance will be followed by a large flaccid bulla
Perioral crusting is typical
There is intraepidermal blistering in this condition
Lesions are very painful
Sometimes the eruption is more localised
Recovery is usually within 5-7 days

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10
Q

How would you treat staphylococcal scalded skin syndrome?

A

Abx- flucloxacillin, erythromycin or an appropriate cephalosporin
Analgesia

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11
Q

What is a superficial fungal infection?

A

A common and mild infection of the superficial layers of the skin, nails and hair
Can be severe in immunocompromised individuals

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12
Q

What are the three main groups of superficial fungal infection?

A

Yeasts (candidiasis)
Dermatophytes (tinea/ringworm)
Moulds (aspergillus)

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13
Q

What is the general presentation of a fungal infection?

A

Presentation varies with the site of the infection, it is usually unilateral and itchy.

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14
Q

What is tinea corporis?

A

This is tinea infection of the trunk and the limbs

It presents with itchy, circular or annular lesions with a clearly defined raised and scaly edges is typical

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15
Q

What is tinea cruris?

A

Tinea infection of the groin and natal cleft

Very itchy and similar to tinea corporis

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16
Q

What is tinea pedis?

A

Also known as athlete’s foot

Moist scaling and fissuring in toewebs, spreading to the sole and dorsal aspect of the foot

17
Q

What is tinea manuum?

A

Tinea infection of the hand

Scaling and dryness in the palmar creases

18
Q

What is tinea capitis?

A

This is scalp ringworm

You get patches of broken hair, scaling and inflammation

19
Q

What is tinea unguium?

A

This is tinea infection of the nail- yellow discolouration thickened and crumbly nail

20
Q

What is tinea incognito?

A

This is inappropriate treatment of tinea infection with topical or systemic corticosteroids
You get ill defined and less scaly lesions

21
Q

What is candidiasis? How does it present?

A

This is a candidal skin infection (yeast)

Causes white plaques on mucosal areas, erythema with satellitle lesions in flexures

22
Q

How does pityriasis/tinea versicolor present?

A

This is an infection with Malassezia furfur

Presents as scaly pale brown patches on the upper trunk that fail to tan on sun exposure, they are usually asymptomatic.

23
Q

What is the management of fungal infections?

A

You have to establish the correct diagnosis by skin scrapings, hair or nail clippings (for dermatophytes) or skin swabs (for yeasts)

Topical antifungal agents eg: terbinafine cream

Oral antifungals: itraconazole

Correct predisposing factors where possible- moist environment, underlying immunosuppression