Shoulder Pathology Flashcards
Frozen shoulder
condition characterized by functional restriction of both active and passive shoulder motion for which radiographs of the GH joint are essentially unremarkable except for the possible presence of osteopenia or calcific tendonitis
what pattern does frozen shoulder follow?
loss of ROM often in capsular pattern (Cyriax)
loss of ER > ABD > IR
capsular endfeel
frozen shoulder: shortening contracture of?
anterio-inferior capsule, rotator interval, coracohumeral ligament
frozen shoulder types
Primary: adhesive capsulitis (etiology unknown)
Secondary: linked cause
adhesive capsulitis
- etiology unknown
- regional ischemia of the shoulder soft tissues from autonomic sympathetic dysfunction?
- some genetic tendencies
- females>males
- peak incidence in early 50’s
defined clinical course of adhesive capsulitis
4 stages: 1=acute 2=freezing 3=frozen 4=thawing
- typically takes 1-3 years to run course
- important that tx be individualized according to stage
acute stage- adhesive capsulitis
0-3 months
- pathology=acute synovitis
- pain on AROM and PROM
- empty endfeel
- ROM is normal (anesthesia)
freezing stage- adhesive capsulitis
3-9 months
- pathology= hypertrophic hypervascular synovitis, proliferation of scar tissue
- pain on AROM and PROM
- empty end feel, pain before
- ROM becomes severely limited
*the shorter the acute & freezing (inflammatory) phase, the shorter the overall course
frozen stage- adhesive capsulitis
5-9 months
- pathology= dense mature scar tissue, decreased capsular volume (reduction of redundant fold), contractures of coracohumeral ligament, subscapularis, subacromial bursa
- no pain on AROM and PROM
- capsular end feel
- ROM severely limited
- prolonged loss of joint ROM causes changes in muscle- loss of sarcomeres
thawing stage- adhesive capsulitis
15-24 months
- pathology= restoration of capsular volume
- no pain on AROM and PROM
- capsular endfeel
- ROM gradually improving
stage 1 adhesive capsulitis treatment
GOAL: interrupt pain and inflammation, promote relaxation, educate
modalities: as needed for: pain, inflammation, relaxation
strengthening: early closed chain
ROM: AAROM, pain free ROM, gentle PROM, pendulum
stage 2 adhesive capsulitis treatment
goal: minimize pain, inflammation, capsular adhesions, and ROM restriction; posture HEP
modalities: as needed to: decrease pain & inflammation, improve tissue extensibility
strengthening: more advanced: scapular training- specific rotator cuff strengthening
ROM: AROM, PROM
stage 3 & 4 adhesive capsulitis treatment
Goal: increase ROM; posture HEP
modalities: to promote: relaxation, tissue extensibility, reduce tx discomfort
strengthening: more specific: scapular training to reestablish force couples, continued rotator cuff strengthening
ROM: more specific: AROM to reestablish scapular and GH mechanics; more aggressive stretching (PNF, STM, low load prolonged stretch)
secondary frozen shoulder
Loss of ROM: underlying or associated condition can be identified
Intrinsic
Extrinsic
Systemic
Intrinsic secondary frozen shoulder
related directly to the GH joint
rotator cuff disorders, bicep tendonopathy
extrinsic secondary frozen shoulder
remote from the GH joint
cervical radioculopathy, breast surgery, humeral or clavical fx, AC DJD
systemic secondary frozen shoulder
DM, hyper/hypothyroidism, hypoadrenalism
rotator cuff tears/impingement
Intrinsic/Primary
Extrinsic/Secondary
Intrinsic/Primary rotator cuff tears/impingement
=subacromial space issues
- abnormally shaped acromion (hook shaped); rough undersurface
- degenerative changes in the AC joint
- decreased vascularity (critical zone)
Extrinsic/Secondary rotator cuff tears/impingement
=stength/environment
- GH force couple dyskinesia
- ST force couple dyskinesia
- posture
- excessive overhead use of arm
- posterior capsule shortening
GH force couple
- deltoid elevates the arm but also produces superior translation of humeral head
- inferior & medial forces of rotator cuff offset superior translation of deltoid (specifically infraspinatus, teres minor and subscap)
- RC also assists in limiting anterior/posterior translation of humeral head
ST force couple
rotation of scapula is provided by trapezius force couple (upper, mid, lower) and serratus anterior
ST muscle balance
- efficient forces depend on stability of origins of the scapula
- scapular position affects length-tension properties of rotator cuff
- scapular upward rotation, posterior tilt, lateral rotation- NECESSARY to maximize subacromial space
integrated RC, GH, and ST force couples
scapular rotation during arm elevation adds to total ROM
lack of scapular rotation leads to impingement
- scapular rotation is necessary to keep acromion moving away from deltoid insertion
- lack of scapular rotation-head of humerus translates superiorly
failure of scapular adduction-head of humerus translates anteriorly
neer stage 1
- edema and hemorrhage
- minimal weakness
- excessive overhead use
- usually <25 y/o
neer stage 2
- fibrosis and tendonitis of cuff and bursa following repeated mechanical inflammation
- usually 25-40 y/o
neer stage 3
- bone spurs
- incomplete and complete tears of cuff and biceps tendon
- degeneration of remaining tendons
- usually >40 y/o
- common 5-40% > 60 y/o have evidence of full thickness tears
treatment principles for RC dysfunction
1: conservative rx for 6 months
2: surgery for RC pathology
conservative tx for RC dysfunction
- inflammation in acute phase
- manual therapy and exercise to address impairments in posture, weakness and stabilization
- DO NOT ignore the cervico-thoracic spine!
surgery for RC dysfunction
Primary impingement:
- subacromial decompression
- acromioplasty
Primary & secondary impingement:
-capsular repair
-post-op rehab:
modalities for pain relief, inflammation
initial protection from active & passive ms force
PROM->AAROM->AROM
gentle UE closed chain, stabilization ex at 3 wks
thoracic outlet syndrome
=mechanical, non-traumatic compression of the neurovascular bundle
- largest nerves affected first: sensory first, then motor
- poorly localized aching pain
- need to rule out CTS, radiculopathy, distal nerve compression
areas at risk for thoracic outlet syndrome
1: superior thoracic outlet
2: scalene groove
3: costoclavicular space
4: infracoracoid space
TOS: superior thoracic outlet
- cervical rib or long C7 TP
- often ulnar nerve distribution
- ^ symptoms with altered posture: forward head; protracted shoulders
TOS: scalene groove
- between ant & middle scalene
- scalene hypertrophy or tightness
- forward head posture
- symptoms ^ w/ overhead tasks and some cervical positions
**soft tissue release and posture correction
TOS: costoclavicular space
- between clavicle and 1st rib
- elevated ribs
- depressed, retracted shoulders
- backpacks, carrying heavy loads
- symptoms ^ with military postures
**1st rib, posture (scap elevation)
TOS: infracoracoid space
- beneath coracoid between pec minor and ribs
- tight pec minor
- symptoms ^ with overhead activity
**strengthen scap stabilizers, stretch pec minor
hypermobility/instability of GH joint
-GH stability involves articular geometry, the static capsulo-ligamentous complex, dynamix muscular stabilizers and NM control
most common abnormal GH motions
- excessive anterior translation during lateral rotation and abduction
- excessive anterior translation during medial rotation
- potential for axillary nerve damage
continuum of shoulder stability
Normal: normal congruity and loading
Lax/hypermobile: congruity maintained, but joint is unloaded
Subluxed: partial contact of articular surfaces- congruity lost
Dislocated: no contact of articular surfaces- congruity lost
contribution of shoulder musculature to joint stability
- passive muscle tension from bulk effect of rotator cuff
- rotator cuff contraction- compression of articular surfaces
- joint motion that secondarily tightens passive ligamentous restraints
- barrier or restrain effect of contracted rotator cuff muscle
- redirection of joint force to center of glenoid surface by coordination of forces from GH and ST joints
acute GH dislocation
up to 96% are trauma induced, TUBS injury, requiring surgery
- traumatic-unidirectional-Bankart-surgery
- > 20% successful without surgery: high re-dislocation rate
associated injuries:
- Bankart- injury to the glenoid
- Hill-sachs deformity- humeral head
in general, dislocations with HIll-sachs lesion and/or bankart lesion commonly experience chronic instability (commonly associated with traumatic dislocation)
Bankart
=injury to the glenoid
- Soft: avulsion of ant int GH ligament and labrum from anterior rim of glenoid
- Hard: fx of the glenoid rim
chronic GH dislocation
=progression from instability/subluxation
- usually due to increased passive laxity
- instability -> subluxation -> dislocation
- success rate w/out surgery >80%
- AMBRI: rarely requires inferior capsular shift
AMBRI
Atraumatic
Multidirectional
Bilateral
Rehabilitation Indicated
*rarely requires inferior capsular shift
GH dislocation presentation
Presentation (after reduction):
- (+)apprehension sign, anterior tenderness
- RTC weakness (if tear)
- deltoid weakness and/or lateral shoulder sensory loss if axillary nerve injured
- acute -> UE in ER with anterior prominence of humeral head
conservative treatment of GH dislocation
- improve dynamic stability/ proprioception of GH joint
- immobilize for up to 3 weeks? in IR or ER
- avoid forceful ER; no PROM/stretching
- focus on neuromuscular coordination/ re-education
surgical treatment for GH dislocation
anterior capsular shift or anterior capsulo-labral reconstruction if Bankart present
Predictors:
- if 40 y/o minimize immobilization, look for RTC tears if no response to tx after 2 wks
AC injury
trauma
disruption of AC ligs
no dynamic stability possible
AC trauma
- direct blow to lateral shoulder
- FOOSH driving humeral head into acromion
disruption of AC ligaments
1st deg: no instability
2nd deg: AP instability
3rd deg:: gross instability, distal clavicle high riding
AC joint treatment
- pain control, protected ROM, isometrics
- progress to strengthening ex, dynamic strengthening, sport/occupation specific activities
- perform ex sidelying, seated, or standing. avoid supine- scap pinned, results in greated clavicular rotation at AC
SC injury
- blunt force to sternum or clavicle
- lateral compression from clavicle
- usually dislocate anterior/inferior
- posterior more serious-can compromise NV, breathing/swallowing problems
- rare, less than 3% of shoulder injuries
labral tears
mechanism of injury
- FOOSH
- consequence of dislocation
- strong bicep contraction
- range from minor fraying to Bankart to SLAP lesions
- stable (pain but no locking/clicking) to unstable (pain with locking/clicking)
- symptoms often similar to AC joint pathology
SLAP lesions
4 types:
1: rough edge
2: labrum torn off glenoid (common)
3: bucket handle
4: tear includes bicep tendon
PT can treat symptoms and rebalance muscles
stable labral tears
stable=pain but no locking/clicking
- NSAID’s/ cortisone injection
- scapular stabilizer and RTC re-training
- limit strengthening to <90%
unstable labral tears
- conservative tx is rarely successful
- arthroscopic debridement and stabilization of unstable tears