Shoulder Biomech Flashcards
Joint Stabilization
shoulder joint capsule is relatively lax and must rely on muscles for active stabilization.
How does the shoulder maintain PASSIVE stability?
When the arm hangs freely to the side (at neutral) there is little or no active contraction of the deltoid or rotator cuff muscles b/c:
a. ) The way the articulating joint surfaces are situated – glenoid cavity is facing lateral, forward, and superior creating a lip for the humeral head.
b. ) The superior joint capsule and the coracohumeral ligament hold the humerus in the glenoid fossa against gravity. This is achieved b/c the superior joint capsule and coracohumeral ligament are usually taut creating an opposing force to the vertical weight of the humerus. This causes the pull the head of the humerus in against the upward facing glenoid cavity
How does the shoulder maintain ACTIVE stability?
When the shoulder is raised in any plane away from the side of the body, the superior joint capsule becomes lax. Therefore it is the responsibility of the dynamic ligaments – the rotator cuff muscles - to maintain congruency and stabilization
What two conditions cause abnormal alterations and compromises to shoulder stability?
- ) Thoracic kyphosis- this situates the scapula in a downward rotation. This takes away the stabilizing ‘lip’ of the glenoid fossa and places the humerus into a pseudo-abduction in reference to the newly positioned scapula. In addition, the normally taut superior joint capsule and coracoacromial ligaments become lax. Therefore, the dynamic ligments (rotator cuff) take over with ACTIVE stabilization. This may lead to impingement syndrome because of the constant, increased tone in the rotator cuff group.
- ) The same occurs in a patient with muscle paresis. Weakness or imbalance may cause the scapular muscles to orient the scapula in the same forward rotation as thoracic kyphosis. Depending on the severity of the muscle paresis, inferior dislocation or subluxation may occur if the rotator cuff muscles are affected.
What influence does the GH joint capsule have on the shoulder during movement?
The fibres of the GH joint capsule face anteriorly and medially. As the arm abducts this twist in the joint capsule increases and pulls the head of the humerus into the glenoid cavity.
This increasing tension furthers abduction because the medial fibres become taught. This causes the capsule to pull the humerus into external rotation – untwisting of the joint capsule.
This also allows for further abduction b/c the external rotation also helps to prevent the greater tubercle from colliding with the acromial arch.
What is the force couple relationship between the deltoid and the rotator cuff muscles during elevation?
The strong multi-pennate fibres of the deltoid act below the centre of rotation causing an upward and outward force on the humerus – this is for movement and elevation.
The rotator cuff muscles act above the centre of rotation causing an inward, downward force on the humerus – this is to help maintain the congruency of the humeral head in the glenoid cavity. These muscles oppose each other & create a force coupling necessary for shoulder elevation.
What are other force couple relationships in the shoulder?
- The three parts of the trapezius muscle and serratus anterior share a force coupling relationship to help with the rotation of the scapula. With the upward rotation of the glenoid fossa, the scapula needs to track superiorly, anteriorly and laterally.
- The long head of the biceps helps to depress the head of the humerus with abduction of the arm in external rotation b/c of the pulley system it creates in the bicipital groove.
What occurs with the FIRST 15-30˚ of shoulder movement?
During the first 15-30˚ of abduction, most movement occurs at the GH joint.
During this phase the scapular muscles stabilize the scapula against the thoracic wall
What occurs after 15-30˚ of shoulder movement?
At this point the scapula begins to contribute to shoulder abduction by rotating superiorly, elevating and moving forward on the chest wall.
Scapular rotation puts tension on the conoid ligament and causes a posterior rotation of the clav (after 90* of ABD), allowing the SC and AC joints to contribute to shoulder abduction.
What is the Scapulohumeral rhythm of the shoulder complex?
During 180˚ of abduction, there is a 2:1 ratio of movement of the humerus to the scapula.
Phase 1- first 30˚ is performed by the GH, 0-5˚ clavicular elevation, scapula is stabilized against the thorax and has minimal or no movement. The scapula does not move; therefore, there is no ratio.
Phase 2 – next 60˚ of elevation the scapula begins to rotate (20˚) and there is a 2:1 scapulohumeral rhythm. The clavicle continues to elevate (15˚)
Phase 3 – last 90˚ of elevation there continues to be a 2:1 ratio of scapulohumeral movement. In this final stage the clavicle rotates posteriorly (b/c of the pull on the conoid ligament) and the humerus laterally rotates 90˚ to clear the acromial arch.
What are the osseous structures involved to achieve full arm elevation and what movements must they perform?
Movement of the shoulder girdle isn’t limited to only the GH, AC, SC joints and the scapulothoracic mechanism. In fact, these combined joints contribute about 160˚ to the full ROM of abduction. The remaining movement (approx. 20˚) come from other osseous components.
Upper T/S – EXT; SB and Rotation (Ipsilateral)
Ribs – 1st and 2nd bodies – descend and move posteriorly
Manubrium – SB and Rotate (Ipsilateral)
Lower T/S – SB (contralateral)
The above is for unilateral abduction. Bilateral abduction causes extension. Fixed spinal deformities cannot perform full elevation.
History
Location:* WHERE?
– ant brachial pain may indicate bicipital tendonitis or Adhesive Capsulitis
– Lat brachial pain may indicate SupraSpin Tendonitis or bursitis
– Sup/lat pain may indicate an AC sprain
– L/R/B?
History
Mechanism of Injury:* HOW?
– RSI: bicipital tendonitis, supraspin tendonitis
– Trauma: bursitis, AC sprain, dislocation, fracture
– Insidious: Adhesive Capsulitis
History
Nature
Intermittent or Constant?
History
Onset:* WHEN?
Acute, Subacute, or Chronic