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Cardio > Shock Drugs > Flashcards

Shock Drugs Flashcards

1
Q

first line for shock

A

dopamine and norepinephrine

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2
Q

dopamine dosing

A

1-3 mcg/kg/min
3-10 mcg/kg/min
10-20 mcg/kg/min

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3
Q

low dopamine dosing receptors

A

mainly DA

some B1

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4
Q

low dopamine MOA

A

vasodilation of arteries (renal, mesenteric, cerebral, and coronary)
natriuretic response

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5
Q

low dopamine monitoring

A

evidence does not support use for renal protection

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6
Q

med dose dopamine receptors

A

mostly B1

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7
Q

med dose dopamine MOA

A

increase contractility and HR

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8
Q

med dose dopamine monitoring

A

can induce tachyarrhythmias

may complement vasoconstrictive effects of NE

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9
Q

high dose dopamine receptors

A

mostly a1

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10
Q

high dose dopamine moa

A

increase SVR

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11
Q

high dose dopamine monitoring

A

renal blood flow effects lost

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12
Q

NE dosing

A

2-20 mcg/min

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13
Q

NE receptors

A

mostly a1

some b1

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14
Q

NE moa

A

increase SVR

small increase in HR and contractility

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15
Q

NE monitoring

A

decrease renal perfusion

CO and LV function decrease at high doses

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16
Q

Epi dosing

A

1-10 mcg/min

17
Q

epi receptors

A

works in both a1 and b1

18
Q

epi moa

A

increases SVR, HR, and contractility

19
Q

epi monitoring

A

watch for tachyarrhythmias

20
Q

phenylephrine dosing

A

30-300 mcg/min

21
Q

PE receptors

A

only a1

22
Q

pe moa

A

increased svr

23
Q

pe monitoring

A

minimal cardiac activity

preferred in patients w history of arrhythmias

24
Q

vaso dosing

A

0.03-0.04 units/min

25
Q

vaso receptors

A

NONE

26
Q

vaso moa

A

non-adrenergic

increases SVR by stimulation of V1 smooth muscle receptors (causes vasoconstriction)

27
Q

vaso monitoring

A

do not titrate above state dose (too much vasoconstriction)

effective in acidosis and hypoxia

28
Q

dobutamine dosing

A

2-20 mcg/kg/min

29
Q

dobutamine receptors

A

mostly b1

30
Q

dobutamine moa

A

increase HR and CO

31
Q

dobutamine monitoring

A

primarily used for cardiogenic shock

can induce tachyarrhythmias

32
Q

milrinone dosing

A

0.35-0.75 mcg/kg/min

33
Q

milrinone receptors

A

NONE

34
Q

milrinone moa

A

inhibits PDE in cardiac and vascular tissue
decreases preload and afterload which..
increases contractility and CO

35
Q

milrinone monitoring

A

inodilator
renal function should be monitored
can cause arrhythmias

36
Q

AT 2 dosing

A

20-40 ng/kg/min

37
Q

at 2 receptors

A

NONE

38
Q

at 2 moa

A 
causes vasoconstriction
increases aldosterone (increases BP)
39
Q

at 2 monitoring

A

thrombotic event

patient should respond within minutes

Cardio (4 decks)

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