Shock Flashcards
What are the types of shock?
- Hypovolemic
- Distributive
- Cradiogenic
- Mechanical
What are the 3 main initial responses to hypovolemic shock?
- Baroreceptors
- RAAS
- Internal transfusion: Increasing the TPR decreases hydrostatic pressure which means more fluid will flow into capillaries
What is decompensated shock in hypovolemia?
Decompensated shock is the signs/symptoms experienced when the initial bodily responses to fix shock failed and have become excessive
What are the 4 primary signs of decompensated shock?
- Excessive sympathetic activity: sweaty, clammy, tachycardic
- RAAS can cause excessive vasoconstriction which leads to a very high TPR: causing cold and clammy peripheries, pallor, weak peripheral pulses and prolonged capillary time
- Decreased perfusion to the brain
- Tachypnoea: the body is trying to breathe faster to emit the lactic acid that is forming as a result of decreased perfusion and oxygen
What are the causes of mechanical shock?
List 2 major causes, the path of each and what the capillary refill time would be
Failing to fill the ventricles either due to an obstruction or a restriction
Obstruction Eg. A massive, proximal PE can occlude a large pulmonary artery which means…
RV cannot empty ➞ reduced return of blood/filling of left heart ➞ reduced (left) EDV ➞ reduced SV ➞ fall in CO ➞ fall in MABP
Filling restriction Eg Cardiac tamponade where blood/fluid build up in pericardial space restricts filling of the heart, which results in…
Reduced EDV ➞ reduced SV ➞ fall in CO ➞ fall in MABP + high CVP
Capillary refill time would be prolonged as there is more blood in the peripheries that can’t circulate
What is Beck’s triad?
A sign of cardiac tamponade: low BP, raised JVP, muffled heart sounds
What is the cause of cardiogenic shock? What are some causes and the inevitable outcome?
The heart cannot contract enough to empty the ventricles, could be due to an MI, HF, arrhythmia and causes a reduced SV and CO –> lowering the BP and the CVP
What is characteristic of distributive shock and are the two causes?
Profound peripheral vasodilation, anaphylactic shock and septic shock
What happens in septic shock and what is the clinical picture?
Bacteria release endotoxins that cause:
1. vasodilation lowering the TPR
2. increased vascular permeability
The Baroreceptors sense this and trigger an increase in HR
Clinical picture: tachycardia, hypotension, warm/red extremities
What happens in anaphylactic shock and what is the clinical picture?
A severe allergic reaction causes MAST cells to degranulate abundant histamine causing extreme vasodilation. The hypersensitive reaction can cause increased permeability - leading to laryngeal edema, bronchospasms and difficulty breathing
Clinical picture: tachycardia, hypotension, warm/red extremities, airway inflammation