Shock Flashcards

1
Q

define shock

A

inadequate perfusion to sustain normal organ function

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2
Q

what is hypovolaemic shock and what typically causes it

A

loss of intravascular volume so loss of forward flow and SBP
vomiting, diarrhoea, third spacing, renal fluid loss, blood loss, severe dehydration

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3
Q

clinical features of mild hypovolaemic shock

A

normal/raised pulse pressure
tachycardia
sweating

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4
Q

clinical features of large volume hypovolaemic shock

A
narrow pulse pressure 
raised CRT 
hypotension 
tachycardia 
anxiety/confusion 
decreased urine output
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5
Q

describe the action of the baroreceptor reflex in maintaining blood pressure

A

reduced stretch in carotid sinus and aortic arch

decreased afferent input to the medullar CV centre and increases SNS output and inhibition of PNS from solitary nucleus

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6
Q

what nerve mediates afferent CV centre input from aortic arch

A

X

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7
Q

what nerve mediates afferent CV centre input from the carotid sinus

A

IX

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8
Q

describe how starling forces can be of use in hypovolaemic shock

A

reduced pressure in capillaries reduces capillary hydrostatic pressure, and shifts fluid flow INTO the capillary

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9
Q

describe the action of the SNS to increase blood pressure

A

release of adrenline and NA to cause chronotropy and inotropy
raised SVR and redirection of blood from periphery

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10
Q

what part of the SNS response to hypovolaemic shock may lead to decompensation

A

secretion of vasodilators, if left untreated this will decompensate the system and kill the patient

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11
Q

where is renin released from?

A

juxtaglomerular apparatus

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12
Q

action of renin in low blood pressure

A

stimulation of AT II to secrete ADH/aldosterone and subsequent vasoconstriction

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13
Q

what is the frank starling law of the heart

A

greater loading volume of the ventricle in diastole leads to increased ventricular ejection in systole

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14
Q

what is cardiogenic shock and causes

A

failure of the heart to meet circulatory demand
MI most common
myocarditis, acute mitral prolapse, cardiomyopathy, myocardial contusion

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15
Q

clinical signs of cardiogenic shock

A
hypotension 
fatigue 
syncope 
raised JVP
hepatic congestion 
pulmonary oedema
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16
Q

what drugs may be used as inotropes in cardiogenic shock

A

adrenaline, dobutamine
dopamine, dopexamine
milrinone, levosimer

17
Q

action of intra-aortic balloon pump

A

increases diastole pressure by inflation to better perfuse coronary arteries and delaftes in systole to reduce afterload

18
Q

cause of obstructive shock

A

PE
cardiac tamponade
tension pneumo

19
Q

management of cardiac tamponade

A

needle pericardiocentesis or thoracostomy as emergency evacuation

20
Q

what may be used to diagnose tamponade or PE in an emergency situation in an unstable patient

A

USS

21
Q

what is the cause of distributive shock

A

vasodilation that is inappropriate
anaphylaxis
sepsis
neurogenic

22
Q

what causes vasodilation in anaphylactic shock and how can it be managed

A

histmine

adrenaline stabilises mast cells and causes vasoconstriction

23
Q

true/false - serum mast tryptase levels can be used to diagnose anaphylactic shock

A

true, but only retrospectively, they should never dictate immediate resus

24
Q

what may be used in septic shock to diagnose/treat

A

rising lactate in a SIRS/septic patient

early pressors

25
Q

cause of neurogenic shock and management

A

spinal/central trauma
loss of thoracic SNS outflow leading to hypotension
dopamine and other pressors

26
Q

shockable rhythms?

A

pulseless VT

VF

27
Q

non-shockable rhythms

A

PEA

Asystole