Ses 4 Foot And Ankle Disorders And Gait Msk Flashcards

1
Q

Ankle Fractures notion
Trauma

A

Discontinuation in cortex of tibia, fibula or talus.
Usually an inversion (medial malleolus and lateral ligament) or eversion injury ( lateral malleolus and medial ligaments).
Unstable fractures - talar shift due to disruption of any 2 of syndesmosis, medial or lateral ligaments.

Consider their co-morbidities eg diabetics have longer healing time for bones.
Have to wait for fracture blisters to heal.

Treatment - Open fractures where skin is breached needs urgent surgery with extensive irrigation and debridement to reduce risk of osteomyelitis(infection).
Stable = non-operative -cast

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2
Q

5th metatarsal fracture notion
Trauma

A

Avulsion fracture = bone breaks due to excessive tension through the inserting tendon.

Fibularis brevis muscle and plantar aponeurosis insert causing significant tension during forced inversion

Typical mechanism of injury: stepping on a curb or climbing steps

Make sure it’s not infused apophysis

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3
Q

sprained ankle - which ligament is most at risk
Trauma

A

Partial or complete tear of one or more ligaments of the
ankle joint.

conservative management (non-operative).

Forced eversion: medial ligament sprain = deltoid ligament

Forced inversion: lateral ligament sprain

Most common mechanism of injury is an inversion injury affecting a plantar-flexed and weightbearing foot.
In this injury, the anterior talofibular ligament is most at risk of sprain.

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4
Q

Hallux (big toe) Valgus(distal goes lateral)

A

Lateral (valgus) deviation of hallux while there is medial deviation (varus) of metatarsal.
Joint subluxation.

Causes: - poorly-understood. Exacerbated by tight-fitting shoes and the line of pull of the extrinsic tendons (e.g. EHL tendon).
Most common cause of a ‘bunion’.

Risk factors: Female Age >65 years
Connective tissue disorders that cause ligamentous laxity eg ethers-danlos
Trauma, gout, RA

Management: Analgesia, Supportive footwear, Surgical correction(metatarsal osteotomy and realigning the fragments).

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5
Q

OA of ankle

A

Causes - Usually secondary:
Post- traumatic eg fracture, severe sprain
Inflammation eg RA
Joint stress eg ballet
Obesity
Primary - idiopathic - better outcome

Joints affected:
Talocrural joint
Subtalar joint

Management:
Arthodesis - good outcome
Arthoplasty - risky

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6
Q

Achilles tendinopathy(causes, where it occurs, risk factors, symptoms and treatment) and rupture(causes, symptoms, diagnosis, treatment)
Notion

A

Rupture causes:
Overstretching the tendon by falling from height, stepping off kerb, falling with foot outstretched and dorsiflexed, making a push-off with an extended knee

Symptoms/signs:
Sudden pain at back of ankle
Loud pop
Palpable gap in tendon
Pain, swelling, bruising
Cannot tip toe or push off while walking

Diagnosis -Thompson’s test – Calf squeeze and foot plantar flexes.
Ultrasound/ MRI to show gap.

Treatment - conservative, maybe surgical (ends frayed)

Tendinopathy – Repetitive action causing microtears within the tendon - degenerative
Occurs at calcaneal tuberosity or vascular area within tendon.

Risk factors: Trauma, Poor footwear, Obesity, diabetes, exercise frequency eg runners

Symptoms - worse pain in morning and after exercise, swelling, bone spur, thickening of tendon

Treatment: Physio

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7
Q

Lesser toes
Claw toe + Hammer toe

A

What is claw toe:
Affect all four of the small toes at the same time.
The toes are hyperextended at the MTPJ and flexed at the PIP joint.

Causes:
Muscle imbalance causes ligaments and tendons to become unnaturally tight - neurological damage - stroke, diabetes, cerebral palsy.
Trauma, RA.

Hammer toe what is it:
deformity in which the toe is flexed at the PIPJ.

Causes:
Toe in a flexed position for too long, the muscles contract and shorten, over time, the muscles cannot extend the toe -tight shoe/adjacent hallux valgus.

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8
Q

Flat foot
Pes planovalgus

A

Flat foot is a loss of the medial longitudinal arch.
Valgus angulation of the hindfoot.
Only abnormal from adolescence upwards.
Rigid(no arch at all) and flexible(normal when standing on tip toes).

Causes:
Adolescents- tarsal coalition
Adult acquired - dysfunction of the tibialis posterior tendon - stretching of the spring ligament - talar head displaced inferomedially, flattening the medial longitudinal arch and producing lateral deviation of the hindfoot.

Risk factors:
Genetics – Marfans Syndrome and Downs Syndrome
Adult - Ligament laxity in pregnancy, hypertension, obesity, Diabetes

Treatment:
Adolescence- not orthotics
Adult - orthotics, physio for muscle strength, surgical reconstruction

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9
Q

Diabetes and the foot

A

Conditions:
Peripheral vascular disease causes ischaemia
Peripheral neuropathy causes loss of sensation
Immunosuppression due to poor glycaemic control

Symptoms:
Deformity
Pain & stiffness
Loss of sensation
Ulcers

Prevention:
Annual diabetic foot check
Self care advice – good fitting shoes, checking the sole of the foot regularly
Tight glycaemic control

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10
Q

Charcot arthropathy

A

What is it? - Progressive destruction of the bone and soft tissue of the foot.
Characterised by multiple joint dislocations, fractures and deformities.
Usually ankle and foot.

Causes:
Neuropathy - reduced ability to detect touch, temp, pain so keep walking on foot - Repeated microtrauma causing fractures, inflammation causing osteolysis.

Presentation:
rocker-bottom foot

Treatment: Treat the underlying condition
optimisation of glycaemic control and reduction of the load placed on the affected joints.

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11
Q

Compartment Syndrome

A

Increase in pressure within a CLOSED fascial compartment that compromises the neurovascular
bundle.

6P ’s - Pain (Out of proportion from injury), Paraesthesia, Pulselessness, Perishingly cold, Paralysis, Pallor

Rhabdomyolysis due to muscle necrosis due to ischaemia.
Acute kidney injury due to this.
Volkmann’s ischaemic contracture

Diagnosis:
Can measure compartment pressures - exceeding systolic arterial pressure -loss of peripheral pulses and increased capillary refill time.

Causes: trauma- haemorrhage/oedema

Fascial compartments:
Thigh x3
Leg x4
Arm x2
Forearm x2
Treatment: Fasciotomy

Complications: Operative - Poor surgical technique, necrotic tissue, nerve and vascular injury Post-operative: Inability to close, infection, failure of treatment

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12
Q

Hallux rigidus

A

OA of 1st MPTJ
Causes: force equivalent to twice the body weight passes through this small joint with each step.
Secondary - gout and septic arthritis.

Symptoms- pain when walking and on attempted dorsiflexion of the toe.
Patients walk on lateral border of foot.
Osteophyte may develop on joint.

Treatment- activity modification, analgesia, orthotics(prevents movement of joint), intra-articular steroid injections, arthodesis, arthoplasty

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13
Q

Surgical management of OA terms

A

• Arthroplasty = joint replacement
• Arthrodesis = joint fusion
• Excision arthroplasty = surgical removal of the joint with interposition of
soft tissue (e.g. a rolled-up tendon, between the bone ends)
• Osteotomy = surgical cutting of a bone to allow realignment (to take the load off the affected part of the joint)

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14
Q

Define gait and gait cycle

A

Gait is the mechanism by which the body is transported using co-ordinated movements of the major lower limb joints.

Gait cycle is the period of time from initial contact to next initial contact on the same side of the body.

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15
Q

Describe the phases of the gait cycle notion

A

60%
Initial contact – heel strikes ground - double support
Loading response - deceleration - shock of the impact is absorbed by the knee and ankle joints.
The person also rocks forwards on their heel to put foot flat in next step.
Mid-stance – the foot is flat on the ground and the centre of gravity of the
body is shifted to front of the foot, ready for forward propulsion.
Terminal stance – Ankle plantarflexes
Pre-swing – The metatarsophalangeal joints flex to give a ‘push off’ by the toes - double support.

40%
Initial swing – the knee flexes - double support
Mid-swing – the hip flexes and the pelvis ‘swings’ forwards. Foot off the ground and not plantarflexed - ankle neutral.
Terminal swing – the knee extends, and the foot is prepared for heel strike.

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16
Q

Define step, stride and cadence

A

Stride: The distance from initial contact with one leg to the next
initial contact with the same leg

Step: The distance from initial contact with the one leg to initial
contact with the opposite leg

Cadence: The number of steps per minute

17
Q

Describe the differences between walking and running

A

Double support replaced by double float
Sprinting = 40% stance and 60% swing

18
Q

Identify the muscles used in normal gait

A

Tibialis anterior prevents slapping of foot to ground during heel strike. It eccentrically contracts to lower it gently.
Gastrocnemius and soleus do concentric contraction for pla

19
Q

Trendelenburg gait

A

In the stance phase of the Trendelenburg gait, the pelvis drops on the unaffected side. The patient then tries to compensate by swinging their torso over towards the affected side. The resulting gait is ‘waddling’.
Causes:
SGN leisons
Trauma eg dislocation or fracture of GT where G.medius attaches
Muscle pain and inhibition of function (e.g. in hip OA)
Biomechanical hip instability (e.g. developmental dysplasia of the hip).

20
Q

Hemiplegic gait

A

A hemiplegic gait is due to paralysis of one side of the body.
Lean towards the unaffected side of the body then circumduct (swing) the paralysed leg.
The patient cannot bear much weight on the paralysed leg so can only take a short step with the unaffected leg.
Gait = short step with unaffected leg followed by circumduction of the affected leg.

Causes - trauma to CNS eg stroke, head injury, spinal injury

The patients have spasticity (continuous contraction) of the affected side of the body. The spasticity is most severe in the flexor muscles of the upper limb and the extensor muscles of the lower limb as these are the dominant muscle groups = flexed upper limb and an extended lower limb.

21
Q

Diplegic gait

A

spasticity affects both lower limbs.
Feet close together - narrow gait + forefoot makes the initial contact with the ground

Spasticity In:
Hip adductors so scissoring
Hamstrings so knee flexed
gastrocnemius + soleus so plantarflexion

Causes:
Cerebral palsy

22
Q

High-steppage gait

A

Swing phase - the absence of dorsiflexion so under gravity is plantarflexed.
Patient has to flex the hip much more than usual in order to lift their foot high off the ground and stop their toes from dragging along the floor.
Heel strike - eccentric contraction of the tibialis anterior muscle is absent so foot slaps.
If superficial peroneal nerve isn’t damaged will compensate with eversion flick.

Causes:
Common peroneal nerve palsy e.g. trauma to the nerve following a fracture of the neck of the fibula
Sciatica (L4 myotome dorsiflexes the ankle)
Neuromuscular disease

23
Q

Parkinsonian gait

A

Patients with Parkinson’s Disease find it difficult to initiate movement. To counteract this they flex their neck and trunk forwards to move their centre of gravity in front of their lower limbs.
They also take very short steps, known as a shuffling gait. Festinant gait, which is the tendency to take accelerating steps.
Loss of arm swing whilst walking.

24
Q

Ataxic gait

A

Clumsy, staggering movements with feet wide apart.
Patients often hold their arms outwards to help them to balance.
Whilst standing still, the patient’s body may sway back and forth and from side to side – this is called titubation.
Patients will not be able to walk in a straight line.

Causes:
Proprioceptive
Cerebellar disease - inherited or acquired eg stroke or acute alcohol intoxication
Vestibular - damage to balance organs

25
Q

Antalgic gait

A

Want to reduce pain in affected limb
So shorten stance phase on affected and shorten swing phase on unaffected limb - this is uneven.
Walking stick used in the hand opposite the painful limb. They can lean
towards the walking stick, shift their centre of gravity away from
the painful limb and therefore reduce the load through it during the
stance phase.