Sensory - Module 9 Flashcards

1
Q

Meniere’s Disease

A

Abnormal inner ear fluid balance caused by malabsorption in the sac or blockage from the duct.

  • Unknown cause – no cure
  • Feeling of aura (pressure/fullness in ear)
  • Episodic incapacitating vertigo with tinnitus (or roaring sounds)
  • Attacks increase in frequency and symptoms
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2
Q

S&S of Meniere’s Disease

A
  • sudden attacks of vertigo
  • tinnitus
  • hearing loss
  • N/V

After attack:

  • vertigo 2-4 hours
  • dizziness
  • unsteadiness
  • gait changes
  • depression
  • moody
  • VS within normal limits
  • hearing loss
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3
Q

Management of Meniere’s Disease

A
  • quiet, dark room
  • low salt diet
  • fluids 2500 cc/day
  • avoid ETOH and caffeine
  • safety: home, work, driving
  • prevent attacks: avoid sudden head movement and flashing lights
  • transmission of air waves into the fluid in the inner ear is having some success
  • watch for potential hearing loss
  • F&E balance
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4
Q

Medication for Meniere’s Disease

A
  • sedations/tranquilzers
  • antihistamines (AntiVert)
  • vasodilators
  • diuretics
  • anti-seizure drugs
  • ototoxic
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5
Q

Diagnosis of Meniere’s Disease

A
  • Check symptoms
  • perform Weber’s test
  • Rule out other dz
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6
Q

Mastoiditis

A

Inflammation of the mastoid resulting from a middle ear infection (otitis media)

Acute mastoiditis is rare r/t antibiotics

Chronic mastoiditis leads to cholesteatoma, which is an ingrowth of skin of the external layer of the eardrum into the middle ear. – use Otoscope.

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7
Q

Clinical Picture of Mastoiditis

A
  • postauricular pain and tenderness
  • otorrhea: drainage
  • mastoid area red and edematous
  • fever, H/A
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8
Q

Treatment for Mastoiditis

A
  • Diagnosis is related to symptomology
  • Meds - Antibiotics
  • Surgery - myringotomy, mastoidectomy
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9
Q

Tympanoplasty

A
  • Common surgery for otitis media
  • Surgical reconstruction of a perforated tympanic membrane, can also restore inner ear structures
  • done Outpatient
  • Improves hearing
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10
Q

Otosclerosis

A
  • New abnormal bone fixates the stapes
  • The stapes cannot vibrate and sound cannot be conducted
  • Common in white women and worsened by pregnancy
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11
Q

Clinical Picture of Otosclerosis

A
  • Progressive conductive hearing loss
  • Tinnitus
  • Bone conduction is better than air on Rinne test
  • Audiogram indicates conductive hearing loss
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12
Q

Treatment for Otosclerosis

A
  • Fluoride supplement may mature spongy bone growth
  • Amplification: hearing aids
  • Surgical: stapledectomy with graft or prosthesis
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13
Q

Acoustic Neuroma

A
  • Slow growing benign tumor of the 8th cranial nerve
  • In the internal auditory canal extending to press on the brain stem
  • Equal in men and women at middle age
  • Usually unilateral
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14
Q

Treatment for Acoustic Neuroma

A
  • Diagnosis by MRI or CT
  • Surgical removal
  • Preserve facial nerve function
  • Approach depends on if there is hearing loss
  • Complications: facial nerve damange, cerebrospinal fluid leak, infection, cerebral edema
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15
Q

Cochlear Implant

A
  • Profound sensorineural bilateral hearing loss
  • Stimulates the auditory nerve
  • The mic and signal processor is outside the body - implanted electrodes
  • 1 year, adult with profound hearing loss, extensive rehab
  • MRI will inactivate the device
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16
Q

Presbycusis

A
  • Hearing loss due to aging.

- Caused by exposure to noise, diseases, poor nutrition, ototoxic drugs.

17
Q

Conductive hearing loss

A

occurs in other and middle ear. Sound is impaired from outer to inner ear.

Most common cause: otitis media.

Hears better in noisy environment.

18
Q

Sensorineural loss

A

Impairment of the vestibulocochlear nerve (CN 8)

  • hereditary, noise trauma, syphilis, TB
  • High pitch ability decreases first (so don’t shout)
  • Hearing aids may not always be helpful
19
Q

Nursing considerations for Presbycusis

A
  • speak normally and slowly
  • do not exaggerate facial expressions
  • use simple sentences
  • rephrase if not understood with different words
  • do not shout
  • do not cover mouth when speaking
  • maintain eye contact
20
Q

Rosacea

A
  • affects middle age adults
  • marked by erythemia (cheeks) and pustules
  • have more sebaceous glands

Treat:

  • topically hydrocortisone (MetroGel)
  • PO tetracycline
21
Q

Psoriasis

A
  • chronic noninfectious inflammatory disease
  • increase in epidermal cell production
  • 15 - 50 years
  • can be on scalp, elbows, knees, back and genitilia
  • bilateral symmetry
  • stress aggravates
22
Q

Clinical Picture of Psoriasis

A
  • red raised patches
  • may or may not itch
  • medical management: remove scales, emollient creams
  • corticosteriods, occlusive dressings and VITAMIN D
  • intralesional injections
  • systemic methotrexate, cytotoxic
  • UV light helps shedding
23
Q

Nursing management for Psoriasis

A
  • Anthran leaves brownish stains
  • avoid sun exposure
  • national psoriasis foundation
  • self acceptance
  • disease can generally be controlled but NOT CURED
24
Q

Eczema/Dermatitis

A
  • many variant forms
  • hereditary, allergies
  • any age
  • lesions spread
  • avoid irritants, moisturizers
  • Complications: secondary infections and pruritis
25
Q

Acne vulgaris

A
  • androgens stimulate the sebaceous glands, accumulated sebum plugs the pilosebaceous ducts
  • white heads (closed)
  • black heads (open)
  • papules, pustules, nodules, and cysts
  • 12-35 age, puberty, family history.
26
Q

Mangement of Acne vulgaris

A
  • reduce bacterial colonies
  • salicylic acid and benzoyl peroxide, Vit A acid, tetracycline, clindamycin and erythromycin
  • Cryosurgery, dermabrasion scarring
  • Diet restrictions not recommended
  • Wash twice/day - avoid over scrubbing, oil free cosmetics, no popping or squeezing