SEM 1 EXAM Flashcards

1
Q

AETIOLOGY OF PHOBIAS

A
  • heritable

- classical conditioning

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2
Q

treatment phobias

A

exposure-based

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3
Q

panic disorder/ agoraphobia treatment

A
  • cbt
  • medication
  • psychoeducation
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4
Q

aetiology of GAD

A
  • info processing model
  • meta cognitive model
  • avoidance theory
  • intolerance of uncertainty
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5
Q

treatment GAD

A
  • meds
  • cbt
  • Interpersonal psychotherapy
  • mindfulness meditation
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6
Q

epidemiology depression

A

3.1 men and 5.1 women %%

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7
Q

problems ass with depression

A

suicide
co-morbid anxiety disorders
impaired social and occupational functioning
physical health probs

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8
Q

aetiology of depression

A
  • genetic
  • polymorphism on 5-HTTLPT gene
  • neurotransmitter imbalance
  • hyperactivity in HPA
  • structure abnormalities pre- frontal cortex, hippocampus, anterior cingulate cortex and the amygdala
  • environment
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9
Q

theories: aetiology depression COGNITIVE

A

cognitive:
- depressive attributional style seeing negative events as due to internal,
global, and stable factors
- Beck’s negative cognitive triad – depressed people hold a negative view of the self, the world and the future, and this view is maintained by cognitive distortions

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10
Q

theories: aetiology depression: BEHAVIORUAL

A
  • Focus on contingencies associated with depressed and non-depressed
    behaviours
  • Highlight the role of poor coping skills
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11
Q

theories aetiology depression PSYCHOANALYTIC

A

depression is a form of pathological grief

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12
Q

treatment depression: pharma and physical approaches

A

– Medication
– Non-medical…
– Repetitive transcranial magnetic stimulation
– Vagus nerve stimulation
– Bright light therapy for seasonal affective disorder – Electroconvulsive therapy (for severe depression)

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13
Q

ABC METHOD

A

Activating event
Beliefs
Consequences/feelings/behaviours

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14
Q

treatment depression (psychological approaches)

A
  • CBT
  • interpersonal psychotherapy
  • psychodynamic therapy
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15
Q

problems with BPD

A

anxiety
substance misuse
social/eco costs
suicide

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16
Q

bpd and creativity

A

association

shared vulnerability model: vulnerability of creativity share factors such as cognitive disinhibition..

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17
Q

aetiology bpd

A

biological
stressful life events
psychological factors

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18
Q

treatment bpd

A

mood stabilising medication

  • cbt
  • IPSRT
  • Hospitalisation
  • mindfulness based cbt
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19
Q

aetiology ocd

A
  • neuropsychological model; failure of inhibitory pathways

- cognitive model

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20
Q

treatment OCD

A
  • meds

- cbt

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21
Q

Aetiology of PTSD

A
  • cognitive models
  • learning accounts (CC)
  • biological accounts (extreme arousal at time of trauma)
  • avoidance
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22
Q

treatment PTSD

A
  • meds

- cbt

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23
Q

AETIOLOGY anorexia

A
  • moderate genetic
  • interaction g and env
  • high levels of serotonin
  • abnormal function of neuroendocrine system (fullness/hunger)
  • brain abnormalities
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24
Q

treatment anorexia/ bulimia

A
  • cbt
  • motivational enhancement therapy (MET)
  • family therapy/ maudsley model
  • pharma approaches
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25
aetiology bullimia
- genetic - reduced serotonergic function - familial predis to obesity, substance use and mood disorders - epigenetic factors: exposure to maternal stress - under/over nutrition in utero
26
aetiology bullimia/binge eating - dual pathway model
dietary restriction -> negative affect -> binge eating episode
27
aetiology binge eating
- genetic - serotonin dysfucntion - hormonal disturbances - many develop depression b4
28
treatment binge eating
same as other disorders - behavioural weight loss pharma: SSRIs anticonvulsants
29
substance use epi
alcohol most used in AUS - comorbidity major challenge in treatment - 35% have other mental issues - earlier first use - increase risk of substance use disorder
30
aetiology of substance use disorders
- loss of control - choice theory - strong genetic component - reward systems: dopaminergic system & endogenous opioid system - inhibition dysregulation argues result of a failure of inhibitory system - learning, CC, inventive sensitisation theory
31
personality theories: SU
interaction b/w novelty seeking, harm avoidance, and reward dependence
32
cognitive theories: SU
- outcome expectancy theory: individual’s expectation of positive consequences from substance use increases propensity to use - relapse prevention theory -CBT of AU
33
motivation: SU
``` PRIME theory Plans Teaches Accounts Adresses Acknowledges ```
34
social and cultural factors: SU
- family function, modelling, monitoring, permissive or too harsh - peers - substance use in marginalised communities cultural: availability, cost, social acceptability
35
treatment SU
- goals - detoxification (1st step) - medications - motivational interviewing - CBTraining - recovery models
36
aetiology of gambling
serotonin, dopamine and noradrenaline -> inhibitory control, reward mechanisms and arousal - personality: impulsivity classical and operant conditioning parental modelling and early neg childhood experiences - cultural attitudes - integrated pathways model (behaviourally conditioned, emotionally vulnerable, biologically based problem
37
treatment gambling
public health model - prevention - gamblers anonymous - self-help - pharma, SSRIs, opioid antagonists, mood stabilisers, - primal addiction - cbt
38
1. factor approaches to personality disorders | 2. becks cognitive model
1. degree to which person demonstrates certain traits and combinations of traits 2. Beck’s cognitive model: role of dysfunctional core beliefs about themselves, others and the world
39
Youngs schema therapy for treatment of personality disorders
schema therapy: EMSs and Modes 1. Rigid and resistant to change. 2. Educate about schemas and use cognitive and behavioural techniques to modify them Early maladaptive schemas (EMSs / life traps) - Unrelenting Standards: (achievement and morality) - All disorders Modes: - More state like, but very consistent - E.g., Angry Child (“feels intensely angry, enraged, infuriated, frustrated, impatient because the core emotional (or physical) needs are not being met”) - For Borderline and Narcissism
40
dialectical behavioural therapy (personality disorder)
1. Linehan 2. For Borderline personality disorder but also for others (e.g., antisocial, substance abuse, and eating) 3. Disturbances emotional regulation 4. Biosocial model: due to interaction between biologically-based vulnerability and ‘invalidating’ environments 5. Group (skills and intensive) as well as individual 6. Clinically useful
41
treatment personality disorder (ryle and cognitive analytic therapy)
1. Links cognitive psychology with object relations approach 2. Reciprocal role procedures: complimentary patterns regarding how individual enacts in relationships 3. Therapy: helping the person to develop an understanding of these reciprocal role procedures
42
cluster A disorders (aetiology and treatment)
aetiology: genetic neurological abnormalities with certain enviro conditions treatment: - difficult - trust and intimacy issues cbt, medication
43
cluster B disorders (aetiology and treatment)
aetiology of antisocial PD - interaction: genetic vulnerability and adverse environmental conditions treatment - mentalisation-based treatment - antidepr & lithium and antipsychotic medication (debate)
44
cluster B pd aet & treatment
borderline PD aetiology - genetic & biological influences - strong association with psychosocial factors (childhood trauma) ``` treatment DBT psychodynamic cbt scheme therapy cognitive analytic therapy ```
45
key considerations in childhood disorders
- age/devel. level - dependence on parents - internal experiences - adults reactions/ interpretations
46
3 subtypes of ADHD
- inattentive - hyperactive/impulsive - combined
47
treatment ADHD
- behavioural interventions | - psychopharmacology + psychological treatments together most effective
48
diagnosing autism
- deficits in social-emotional reciprocity - deficits in nonverbal communicative behaviours - deficits in relationships - sameness, receptiveness, sensory issues
49
what is Theory of Mind
ability to attribute mental states— beliefs, intents, desires, emotions, knowledge, etc.—to oneself, and to others, and to understand that others have beliefs, desires, intentions, and perspectives that are different from one's own.
50
Separation Anxiety
excessive worry about separation from home or attachment figures, losing them, refusal to go places, fear of being alone - CBT
51
Oppositional defiant disorder
- angry/irritable mood - argumentative/defiant behaviour - vindictiveness
52
aetiology: Oppositional defiant disorder
alterations in androgen - differences in frontal brain activation - brain injury - parental role
53
Conduct disorder?
aggression to people or animals - societal norms violated persistently and repetitive (age-appropriate)
54
Enuresis what is it?
emptying of bladder Two categories: • Primary enuresis, where the child has never been dry • Secondary enuresis, where the child has had a period of dryness for at least six months - grow out of it
55
what is encopresis
Repetitive soiling in inappropriate places at least once a month for three months
56
Course of psychotic disorders
1. Premorbid phase—presence of risk factors prior to the onset of any symptoms 2. Prodromal phase—preliminary period of decline in mental state and functioning prior to onset 3. Acute phase—active positive and negative symptoms 4. Early recovery phase—associated with depression and anxiety 5. Later recovery phase—challenges with reintegrating into social, recreational and vocational pursuits
57
aetiology of psychosis
``` diathesis-stress model vulnerability factors: neurotransmitters abnormalities - psychosocial - triggering factors (drugs/alc, stress, both) ```
58
Hallucinations causes
Dysfunction in auditory imagery theory: Hallucinating individuals them for actual sounds. • Refined auditory imagery theory: Hallucinating cannot tell the difference between actual and hallucinated sound. • Dysfunction in verbal self monitoring: Breakdown in ability to monitor one’s intention to make internal speech • Hallucinations and cognitive deficit: Increased susceptibility to intrusive and unwanted cognitive activity
59
Delusions
- jumping to conclusion bias - blame others - spreading activation hypothesis
60
somatic and dissociative disorders? what are they?
somatic - prominent anxiety about health (illness anxiety disorder) dissociative - loss of identity
61
what is conversion disorder?
disturbance in motor or sensory functioning
62
What is munchausens syndrome
Factitious disorder imposed on another (previously called Munchausen syndrome by proxy) is when someone falsely claims that another person has physical or psychological signs or symptoms of illness, or causes injury or disease in another person with the intention of deceiving others.
63
aetiology of somatic disorders
``` biological: - under-activity of the HPA axis - neurobiological modles trauma personality: development of alexithymia, can predispose) ```
64
treatment of sexual dysfunction
CBT - Behaviour therapy: combo of education, sensate focus exercises, communication skills - internet based treatment - meds
65
What is paraphilic sexual activity
atypical sexual activities (children, non-consenting adults, non-human objects, humiliation of others or self)
66
Examples of paraphilic disorders
* Exhibitionistic—exposing genitals to an involuntary observer * Fetishistic—use of nonliving object for sexual gratification * Frotteuristic—touching or rubbing against a non-consenting person for sexual gratification * Paedophilic—sexual activity with children * Sexual masochistic and sexual sadism—experience of sexual stimulation through the infliction of pain or humiliation on another person * Transvestic—cross-dressing * Voyeuristic—looking at unsuspecting individuals as they undress
67
what is gender dysphoria
gender expression different from what assigned at birth
68
emotional wellbeing and ageing
improve with increasing age | better coping strategies
69
Social changes with age: Carstensens socioemotional selectivity theory:
- Greater focus on meaningful relationships | - Perceived received social support predicts relationship satisfaction
70
What is Baltes theory of selection, optimisation and compensation (SOC)
- Active choices to adapt to events that might limit pursuit of goals
71
What is alzheimers disease
- most common dementia - causes: neurofibrillary tangles, neuritic changes, produced by a toxic molecule known as amyloid beta - cognitive dysfunction - memory impairment - personality changes, behaviour problems, - hallucinations
72
aphasia
language disturbance
73
apraxia
inability to carry out motor activities
74
agnosia
failure to recognise objects as well as decline in exec functioning - planning, organising (not agnosia)
75
What is vascular dementia
2nd or third most common form of dementia Causes: multiple strokes - single infarct (stroke) - small vessel disease in the brain
76
What r the other forms of dementia
Frontotemporal dementia Lewy body dementia
77
Treatment of dementia
psych and beh interventions Vaccine to slow progression? - Optimal care of individuals with dementia - Lifestyle: mental and physical activity
78
What is the self regulation theory in health psychology
Individuals work towards goals to maintain emotional comfort decrease the effect of a health event on their lives - Response to health threat based on cognitive and emotional representations of the illness
79
Five key components of the self-regulation theory
ICTCC Identity, Cause, Timeline, Consequences, Control - Emotional representation of the illness such as fear, anxiety or resignation • Emotional responses impact on behaviour by motivating responses that reduce negative emotions and increase positive emotions
80
What are the levels of intervention by O'Connell et al., (2009)
``` bottom-up Universal preventive interventions Selective preventive interventions Indicated preventive interventions Treatment ```
81
Relationship b/w stress & disease
- can disturb the immune system - impact on cancer in contradictory and unclear But goes through 3 stages: Stage 1 Alarm reaction (sympathetic dominance, arousal) Stage 2 Resistance (Endocrine releases stress hormones ) Stage 3 Exhaustion Adrenal gland loses ability to function normally
82
What to morse & Johnson (1991) propose (health psych)
levels of anxiety can be high | role of positive affect and coping in response to illness - important
83
Problem solving in stress management?
Describe and explore options, and then look at pros & cons of options before deciding
84
maladaptive stress?
Maladaptive – Problem focused when many of the elements are essentially uncontrollable – Emotion-focused when the stress has many controllable elements