Self-harm and suicide Flashcards
Suicide is highest amongst
Males
social class I and V
middle aged 40-54
over 80 years old
Suicide and self-harm aetiology
Genetics
- FHx self-harm and suicide
- 43% heritability
Childhood and life experiences
- childhood abuse, neglect or bullying
- subcultures LGBTQIA+, goth
- stressful life events in adulthood (e.g. domestic violence)
- complete suicide = loss events
Social isolation
- living alone, unemployed, divorced, widowed, single
- connectedness is a protective factor
Occupation
- access to lethal
- manual, male-dominated trades
Physical illness
- chronic, painful and terminal
- high in neurological (e.g. epilepsy) and stigmatised conditions (e.g. HIV)
Hx self-harm
- increased risk of future self-harm
- self-harm with suicidal intent is the strongest predictor of eventual suicide
Self-harm and suicide aetiology: psychiatric illness
Depression = may increase as severe depression lifts
BPAD
Personality disorders = dissocial, disinhibited and borderline traits
Schizophrenia = command hallucinations to self-harm in young, high-functioning, recently diagnosed people, with insihgt into the severity of illness
Anxiety disorders
Substane use disorders = intoxication exacerbeted distress and impulsivity , alcohol use
Theories: mentalisation
the ability to reflect on, process, and manage opinions
when childhood traumatic, abusive, neglectful -> little opportunity for reflection
learn behaviours to manage their mood/ventilate emotions (e.g. shopping, punching walls)
mentalisation underdeveloped = self-harm to deal with difficult emotions like anger, sadness and anxiety
Theories: self-preservation
coping strategy = attack only a part of the body to secure survival of whole purson
for some, painful non-lethal self-harm protects them from suicide
Suicide prevention strategies at a national level
limit paracetamol pack sizes and numbers purchased
install barriers, free telephones, and helpline posters at suicide hotspots
mandatory catalytic oconverters to cut CO in car exhaust fumes
media guidance for reporting: limited detail to prevent copying
Clinical presentation: preparation
research on methods, obtaining equipment, visitng potential locations, ad rehearsal, putting affairs in order (last acts) e.g. writing a will, rehoming pets, seeing friends, apologising to enemiesn
Clinical presentation: method
explore what the patient did in detail (location, timing, precautions taken against discovery e.g. locking doors, turning off phones), suicide notes (or signing off online), and drug and alcohol use
Methods:
- scratching, punching, head-banging
- cutting, burning, interfering with wounds
- self-poisoning
- inhaling toxic fumes or inert gases
- swallowing or inserting items
- self-stabbing
- jumping from a height or in front of trains/vehicles
- hanging asphyxiation
- shooting
- drowning
- self-immolation
clincial presentation: self-harm functions (and lethality)
coping strategy
relieve/regulate strong emotions
feel something instead of numbness
exchange emotional pain for physical pain
escape consciousness e.g. overdose to sleep
self-punishment
communicating distress
changing intolerable situations e.g. relationship issues
suicide
Clinical presentation: discovery
calling an ambulance, messaging a friend less concerning than if they were accidentally discovered (and resisted assistance)
clinical presentation: self-cutting
commonly knife/razorblade on concealed parts of ars or thights
relieve emotional tension and replace with calmness or mild elation
safety measures to prevent serious harm suggests lower risk of suicide e.g. cleaning blades, cutting safer fleshy areas, bandaging wounds
changed cutting pattern very worrying
clinical presentation: overdose
sx = unexplained sedation, confusion/coma, arrhythmia, brady/tachycardia, hypo/hypertension, respiratory depression
accidental OD may be deliberate
paracetamol
- N+V
- untreated - fulminant liver failure, jaundice, abdo pain, confusion, coma, death
- staggered OD (excess tablets taken over more than 1 hour) dangerous = blood paracetamol levels exceredingly low, damage is cumulative
NSAIDs
- headache, N+V, drowsiness, dizziness, blurred vision, sometimes tinnitus
- large OD = AKI, seizures, hepatic dysfunction, cardiovascular collapse, and coma
Mx: biological
Lacerations = suture under LA and closed, deep cuts needs surgical closure
OD = depends on substance, physical obs, blood tests, ECGs, sometimes over several hours, ToxBase for mx guidance for rarer overdoses
Antidotes for OD
- paracetamol = N-ac
- benzodiazepines = flumazenil
- Insulin = glucagon
- opiates = naloxone
- digoxin = digoxin-specific antibody fragments (Digibind)
- iron salts = DFO mesylate
Activated charcoal = binds many poisons e.g. anti-depressants, useful only within 1 hour
Active elimination = haemodialysis (e.g. lithium, salicylates, valproate), urine alkalinisation (e.g. salicylates), activated characoal (carbamazepine)
Gastric lavage (stomach pumping) = wash out stomach contents within 1 hour of ovderdose, rarely used and never for corrosive substances
transfer to IC for respiratory depression especially
NAC and paracetmol overdose
Mx: RA
once physical consequences dealt with, psychiatrist/psychiatric liaison nurse -> psychosocial assessment. Includes:
- details of current and previous self-harm
- social circumstances and stressors, and triggers for self-harm
- diagnosis of underlying mental illness and its relationship to self-harm
- evaluation of hopelessness, suicidal intent, and persistent plans to self-harm
- strengths and coping strategies
- risk and protective factors for future self-harm/completed suicide