Sedation + Behavior Guidance + Abuse/Neglect + Pain Management Flashcards

1
Q

Goals of procedural sedation

A
  • Promote patient welfare + safety
  • Facilitate provision of quality care
  • Minimize extremes of disruptive behavior
  • Promote positive psychological response to tx
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2
Q

Child has ~__ alveoli at birth which grows to ~__ by adulthood

A

Child has ~20 million alveoli at birth which grows to ~300 million by adulthood

Respiratory system is the last to develop

Young children have less surface area for gas exchange

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3
Q

Hypercarbia + children

A

Children tolerate greater incidences of hypercarbia, unlike adults, where hypercarbia drives immediate increased respiratory response.

Hypercarbia may be associated w/ onset of cardiac arrhythmias

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4
Q

Pediatric diaphragms

A

Pediatric diaphragm is less capable of displacing large volumes in vertical dimensions.

Children have relatively small functional reserve capacity.

Likewise, children have relatively larger post-expiration volume.

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5
Q

Pediatric vs. adult airways

A
  • ⇡airway resistance
  • Glottis (vocal cords) are positioned more anteriorly + cephalad
  • Narrowest portion of the upper airway is at level of cricoid cartilage (below vocal cords)
  • A relatively larger tongue + epiglottis
  • A larger head to body size ratio
  • Mandible is less developed
  • Potential for significant lymphoid tissue obstructing:
    • Nasopharynx
    • Oropharynx
    • Larygopharynx
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6
Q

What affects medication regimen selection in sedation?

A
  • Extent of tx
  • Child temperament
  • Age/parental expectations
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7
Q

Patient selection for sedation

A
  • Traditional techniques have been unsuccessful in managing behavior
  • Patient ASA I or ha a medical condition that is well-controlled ASA II
  • Patient is pre- or uncooperative
  • Extent of tx: ultimately determined by amount of LA allowed by weight
  • Needle phobic; excessively fearful older child
  • Older child w/ poor experiences or coping abilities
  • Distance traveled even for patients w/o behavioral problems
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8
Q

Prematurity + sedation

A
  • Reduced surfactant; alveoli less patent
    • Less surface area for qualitative gas exchange
  • Poor qualitative + quantitative gas exchange
  • ⇡incidence of early life intubation
  • Altered neural reflex pathways; ⇡gag reflex
  • Potentially at greater risk for laryngospasm
  • Hospitalization; ventilator use
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9
Q

Atopic triad

A
  • Asthma
  • Eczema
  • Food allergy

May suggest IgE hyper-responder caution

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10
Q

C-spine precautions w/ sedation

A

Down syndrome

Arnold-Chiari malformation

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11
Q

ASA pre-procedure fasting guidelines for sedation

A
  • No solid foods, non-human milk, and infant formula up to 8hr
  • Clear liquids = 2hr minimum fasting period
    • For children +6mo
    • Clear liquids include anything w/o pulp/particulate
  • Breast milk = 4hr minimum fasting period
  • Infant formula = 6hr minimum fasting period
  • Non-human milk = 6hr minimum fasting period
  • Light meal = 6hr minimum fasting period
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12
Q

ADD/ADHD medications + sedation

A
  • Should be taken following NPO guidelines
  • Bladder empty prior to giving meds
  • Baseline vital signs
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13
Q

LA toxicity

A

Initial signs of mild LA toxicity may mimic effects of N2O/O2 or may be misinterpreted as painful response/maladaptive behavior

  • Lidocaine: CNS + CV effects
    • 4.4mg/kg w/ or w/o epi
  • Septocaine: CNS + CV + immune effects
    • 4-5mg/kg (7mg/kg listed by manufacturer but also noted that should not be used on children <4yo; 5mg/kg noted for children)
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14
Q

Causes + effects of LA toxicity

A
  • Causes of OD
    • Intravascular injection
    • Excess dosage delivered to patient
  • Effects
    • CNS excitement followed by depression, seizures, disorientation, loss of consciousness
    • CV system depressed
  • ⇣myocardial contractility
  • ⇣cardiac output
  • CV collapse
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15
Q

Minimal level of sedation: Cognitive Function, Physiological Function, Monitor, Personnel

A
  • Cognitive Function: May be impaired
  • Physiological Function: Not affected
  • Monitor: Observation only; intermittent
  • Personnel: Not specified
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16
Q

Moderate level of sedation: Cognitive Function, Physiological Function, Monitor, Personnel

A
  • Cognitive Function:
    • Depression
    • Responds to light tactile stimulation
  • Physiological Function:
    • Patent, self-correcting airway
    • Ventilation + CV function is adequate
  • Monitor:
    • O2 sat
    • HR
    • Intermittent BP + RR (no designated period of recording)
    • Capnography recommended if patient is not capable of appropriate bi-directional verbal interactions
    • EKG + defib available (should)
  • Personnel:
    • Person responsible for monitoring other than operator
    • May do other tasks
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17
Q

Deep level of sedation: Cognitive Function, Physiological Function, Monitor, Personnel

A
  • Cognitive Function:
    • Depression
    • Not easily aroused
  • Physiological Function:
    • Potential loss of airway reflexes
    • CV may be affected
  • Monitor:
    • O2 sat
    • HR
    • EKG
    • Capnography recommended
    • Intermittent BP + RR (recorded q5min)
  • Personnel:
    • Person responsible solely for monitoring
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18
Q

Sedation - fatal trio

A
  • Hypovolemia = Patients are NPO, verify “not over NPO” but as close to guidelines as possible
  • Hypoxia = Recognize respiratory obstruction/distress immediately
  • Hypercapnia = If hypoxia persists, hypercapnia may result + make patient more prone to cardiac arrhythmias
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19
Q

How often do you calibrate inhalation equipment (sedation)

A

Annually

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20
Q

Where do you do submucosal injection

A

B/w 1st and 2nd primary molar in maxillary vestibule

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21
Q

N2O-O2 induction + recovery

A
  • Induction = 5min
  • Recovery = 10min
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22
Q

Relative contraindications for N2O-O2 (may be used following med consult)

A
  • Wheezing (moderate-severe asthma)
  • Nasopharyngeal obstruction
  • Tuberculosis, cystic fibrosis
  • Sickle cell disease (due to lowered oxygen tension in blood)
  • Acute otitis media
  • Methionine synthetase deficiency
  • 1st trimester pregnancy

Other contraindications: COPD, narrow angle glaucoma, pneumothorax, small obstruction, middle ear surgery, retinal surgeries

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23
Q

Chronic exposure/abuse of N2O-O2

A

May result in peripheral neuropathies

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24
Q

Chloral hydrate

A
  • Sedative/hypnotic
  • CNS depression – minimal CV or respiratory effect
  • Mucosal irritant
  • Gastric irritation a side effect; unpleasant taste
  • Onset: 30-60min
  • Peak: 60min
  • Duration: 5hr
  • Working time: up to 60min
  • 10-50mg/kg PO to 1g max (as sole agent; reduced doses if used in combination w/ other sedatives)
  • Metabolized to trichlorethanol in liver; excreted by kidney
  • Arrhythmias in higher doses (usually >75mg/kg)
  • No reversal agent
    • When combined w/ other sedates, this is why CH dose should be loweer
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25
Q

Diazepam

A
  • Sedative/hypnotic
  • CNS depression – minimal CV or respiratory effect
  • Amnesia, ataxia (acts in cortex, limbic system thalamus, hypothalamus)
  • Onset: 45-60min
  • Peak: 60min
  • ½ life: 20-40hr
  • 0.25-0.3mg/kg PO (<10mg total dose)
  • Reversal: Flumazenil (0.01mg/kg and can be repeated up to a total of 1mg)
    • IV preferred, but can be IM or submucosal – onset of reversal doesn’t occur for up to 1-2min assuming circulation is occurring
  • Contraindications: Narrow angle glaucoma
  • Half-life 20-40hr w/ sedative metabolite
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26
Q

Midazolam

A
  • Sedative/hypnotic
  • CNS depression – minimal CV or respiratory effect
  • Anterograde amnesia potential feature
  • Onset: 15min
  • Working time: 30-40min
  • 0.5-0.75mg/kg to 15mg total – orally
  • 0.2-0.3mg/kg – nasally
  • Reversal: Flumazenil (not to exceed a cumulative dose of 0.05mg/kg or 1mg, whichever is lower)
    • IV preferred, but can be IM or submucosal – onset of reversal effect does occur for up to 1-2min
  • 3-4x potency of diazepam
  • Respiratory depression w/ higher doses or rapid IV bolus
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27
Q

Hydroxyzine

A
  • Antiemetic/antihistamine
  • CNS depression – anxiolytic, bronchodilator
  • Analgesia – dry mouth
  • Onset: 15-30min
  • Duration: 2-4hr duration
  • 0.5-1mg/kg orally
  • Used often w/ chloral hydrate, meperidine or midazolam
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28
Q

Meperidine

A
  • Narcotic
  • CNS, CV, respiratory depression
  • Reversal: Naloxone (0.1mg/kg; max 2.0mg)
    • IV preferred, but can be IM or submucosal – onset doesn’t occur for up to 1-2min
  • Sedation, analgesia, lowers seizure threshold
  • Caution in patients w/
    • Pulmonary complications
    • Head trauma
    • Seizures
    • Hepatic/renal disease
    • Airway obstruction
    • Concomitant LA dose consideration is very important
  • Onset: 30min
  • Peak: 1-2hr
  • Duration: 2-4hr
  • 1.0-2.0mg/kg PO
  • 50mg max PO, SM, IM
  • Metabolized by liver, excreted by kidney
  • Side effects:
    • Dizziness
    • Xerostomia
    • Sweating
    • Nausea/vomiting
    • Seizures
    • Respiratory depression
  • NOT to be used in patients w/ h/o asthma → may shift production in respiratory tree from prostaglandins to leukotrienes which are bronchial irritants
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29
Q

Physiologic monitoring based on behavior: Screaming/yelling vocalizations

A
  • Clinical signs:
    • Few tears
    • Controlled breathing
    • Struggling against immobilization
  • Pre-cordial: Take earpiece out
  • Capnogaph: Not needed
  • Pulse ox:
    • keep stabilized
    • Set upper HR limits to 230bpm
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30
Q

Physiologic monitoring based on behavior: Mild crying

A
  • Clinical signs:
    • Some tears, eyelids open, some ptosis
  • Pre-cordial: Take earpiece out
  • Capnogaph: Not needed
  • Pulse ox:
    • keep stabilized
    • Set upper HR limits to 230bpm
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31
Q

Physiologic monitoring based on behavior: Quiet (responsive)

A
  • Clinical signs:
    • Eyes closed, open when requested or mildly stimulated
    • Breathing WNL
    • Possible occasional crying
  • Pre-cordial: Earpiece in + listening, attentive to gurgling or snoring (adjust head tilt)
  • Capnogaph:
    • Place/monitor probe
    • Monitor adequacy of ventilation paying attention to head position
  • Pulse ox:
    • Heightened awareness for incidence of desaturation (changing pitch of pulse ox)
    • Assess ventilation + head position
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32
Q

Physiologic monitoring based on behavior: Quiet (unresponsive)

A
  • Clinical signs:
    • Eyes closed, partial ptosis w/ possible divergent eyes
    • Does not open eyes on command
    • Breathing shallow
    • Subtle respiratory movements
  • Pre-cordial:
    • Earpiece in + listening, attentive to gurgling or snoring (adjust head tilt)
    • Keen focus on airway sounds, indication of obstruction or restriction or airway
  • Capnogaph:
    • Be cognizant of frequency of breathing, expired concentrations of CO2 + indications of apneic episodes
  • Pulse ox:
    • Heightened awareness for incidence of desaturation (changing pitch of pulse ox)
    • Assess ventilation + head position
  • Alter passive immobilization to allow patient assessment
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33
Q

Basic schema for a deteriorating patient under procedural sedation

A
  • Problem w/ ventilation – respiratory distress
  • Problem w/ oxygenation – hypovolemia, hypoxia, hypercapnia
  • Persistent respiratory distress + hypotension → CV arrhythmia (bradycardia)
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34
Q

Signs of respiratory distress in a child – sedation

A
  • Chest breathing – non-diaphragmatic
  • Nasal flaring
  • Platysma flaring
  • Grunting
  • Rocking horse/see-saw respiratory patterns
  • Use of intercostal muscles for breathing
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35
Q

Potential emergencies associated w/ sedation

A
  • Respiratory rate depression or arrest
  • Upper airway obstruction including laryngospasm
  • Allergic reaction/overdose
    • If more than 1 organ system involved (e.g. respiratory + dermatologic) epi instead of diphenhydramine
  • Bradycardia and/or hypotension
  • Hypoglycemia
    • Ensure glucose preoperatively if concern (diabetic)
    • Have source of glucose (orange juice, frosting) on hand
  • Vomiting
    • Right mainstem bronchus is straight compared to left mainstem and more likely to aspirate
  • Seizures
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36
Q

SOAPME

A

Commonly used acronym useful in planning + preparation for a procedure.

  • S – size appropriate suction catheters
  • O – Adequate oxygen supply + functioning flowmeter or other devices to allow delivery
  • A – Size appropriate airway equipment, including but not limited to: BMV, OPA, LMA, NPA, facemasks, endotracheal tubes
  • P – Pharmacy to include basic life supporting drugs for emergency
  • M – Monitors (functioning + appropriate for one level deeper than intended sedation)
  • E – Special equipment such as defibrillator/AED
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37
Q

Nitrous usage is a safe, effective technique for:

A
  • Reducing anxiety
  • Producing analgesia
  • Enhancing effective communication between patient + healthcare provider
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38
Q

Nitrous oxide: Properties

A
  • Analgesic and anxiolytic
  • Causes CNS depression and euphoria
  • Little effect on respiratory system
  • Colorless and odorless gas w/ faint sweet smell
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39
Q

Nitrous oxide: Safety

A
  • Patient can respond normally to verbal commands
  • Causes minimal impairment of any reflexes, thus protecting the cough reflex
    • No risk of losing protective reflexes
  • Pt returns to preprocedure mobility after usage
  • No recorded fatalities or cases of serious morbidity when used within recommended concentration
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40
Q

Nitrous oxide: MOA

A
  • Absorbed quickly → allows for rapid onset and recovery [2-3 min]
  • Analgesic effect
    • Initiated by neuronal release of endogenous opioid peptides
    • These activate opioid receptors and descending gamma-aminobutyric acid type A (GABAA) receptors and the noradrenergic pathways that modulate nociceptive processing at the spinal level
  • Anxiolytic effect
    • involves activation of the GABAA receptor either directly or indirectly through the benzodiazepine binding site
  • Lungs
    • Rapid uptake of nitrous → absorbed quickly from alveoli and held in a simple solution in the serum
    • Relatively insoluble → passes down a gradient into other tissues and cells in the body like the CNS
    • Excreted quickly from the lungs
  • Cardiac
    • Causes minor depression in cardiac output while peripheral resistance is slightly increased → thereby maintaining the blood pressure
      • Advantage when treating patient w/ cerebrovascular system
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41
Q

Nitrous oxide: Uses in children

A
  • Analgesia/ anxiolysis may expedite the delivery of procedures that are not particularly uncomfortable, but require that the patient not move
  • Allow pt to tolerate unpleasant procedures by reducing/ relieving anxiety, discomfort, pain
  • Increase reaction time and reduces pressure induced pain
    • Does not affect pulpal sensitivity
  • The decision to use N2O analgesia/ anxiolysis must take into consideration:
    • Alternative behavioral guidance modalities
    • The patient’s dental needs
    • The effect on the quality of dental care
    • The patient’s emotional development
    • The patient’s physical considerations
  • Causes most children to feel tingling/warm sensation
    • Appear w/ their hands open, legs limp, trance like expression
  • In some children → they don’t like the feeling of losing control
  • Claustrophobic children → may find nasal hood confining/ unpleasant
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42
Q

Nitrous oxide: Bioenvironmental concerns

A
  • N2O has been associated with bioenvironmental concerns bc of its contribution to the greenhouse effect
  • Nitrous oxide contributes about 5% to greenhouse effect
  • Only 0.35-2% of total nitrous released into the atmosphere is a result of medical applications of N2O gas
  • Natural sources of nitrous
    • Emitted by bacteria in soils and oceans
    • Produced by burning fossil fuels and forests
    • Produced by agricultural practices of soil cultivation and nitrogen fertilization
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43
Q

Nitrous oxide: Objectives

A
  1. Reduce or eliminate anxiety
  2. Reduce untoward movement and reaction to dental treatment
  3. Enhance communication and patient cooperation
  4. Raise the pain reaction threshold
  5. Increase tolerance for longer appointments
  6. Aid in treatment of the mentally/physically disable or medically compromised patient.
  7. Reduce gagging.
  8. Potentiate the effect of sedatives
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44
Q

Nitrous oxide: Disadvantages

A
  1. Lack of potency.
  2. Dependent largely on psychological reassurance.
  3. Interference of the nasal hood with injection to anterior maxillary region
  4. Patient must be able to breathe through the nose
  5. Nitrous oxide pollution
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45
Q

N2O: Indications

A
  1. A fearful, anxious, or difficult patient.
  2. SHCN
  3. A patient whose gag reflex interferes with dental care.
  4. A patient for whom profound local anesthesia cannot be obtained.
  5. A cooperative child undergoing a lengthy dental procedure.
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46
Q

What should be assessed in patient’s med hx before usage of N2O?

A
  1. Allergies and previous allergic or adverse drug reactions.
  2. Current medications including dose, time, route, and site of administration.
  3. Diseases, disorders, or physical abnormalities and pregnancy status.
  4. Previous hospitalization to include the date and purpose.
  5. Recent illnesses (e.g., cold or congestion) that may compromise the airway.
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47
Q

N2O: Contraindications

A
  1. Some chronic obstructive pulmonary diseases
  2. Current upper respiratory tract infections
  3. Recent middle ear disturbance/surgery
  4. Severe emotional disturbances or drug-related dependencies.
  5. First trimester of pregnancy
  6. Treatment with bleomycin sulfate
  7. Methylenetetrahydrofolate reductase deficiency
  8. Cobalamin (vitamin B12) deficiency
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48
Q

When should you consult MD to use N2O?

A
  • Pt has significant underlying med condition
    • Severe obstructive pulmonary disease
    • Congestive heart failure
    • Sickle cell disease
    • Acute otitis media
    • Recent tympanic membrane graft
    • Acute severe head injury
  • Pt is pregnant → contact their pre-natal med provider
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49
Q

N2O: Technique of administration

A
  • Only administered by trained/ licensed individuals who can perform appropriate reversal response [must have training to manage an emergency if pt falls into moderate sedation]
    • Or under direct supervision of [differs per state law]
  • Choose the right size nasal hood
  • Flow rate → 5-6 L/min generally acceptable to most pts
    • Can adjust after observation of reservoir bag
      • The bag should pulsate gently with each breath and should not be either over- or underinflated
  • Introduction of 100% oxygen for 1-2 minutes followed by titration of N2O in 10% intervals is recommended
  • Nitrous concentration should not exceed 50%
    • Studies have demonstrated that gas concentrations dispensed by the flow meter vary significantly from the end-expired alveolar gas concentrations; it is the latter that is responsible for the clinical effects.
  • For sedated effect:
    • Prevent pt from talking and mouth breathing
    • The scavenging vacuum should not be so strong as to prevent adequate ventilation of the lungs with N2O
  • 30-40% N2O to achieve ideal sedation
    • Decrease N2O concentration for easier procedures, and increase for harder
  • Always monitor pts respiratory rate and level of consciousness
  • Effects of nitrous dependent on psychological reassurance
    • Important to continue behavior guidance during tx
  • After tx completed → administer 100% oxygen until pt has returned to pretreatment status
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50
Q

N2O: Monitoring

A
  • Pt’s response to commands → serves as a guide to their level of consciousness
  • Continual clinical observation of the patient’s responsiveness, color, and respiratory rate and rhythm must be performed
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51
Q

N2O: Adverse effects

A
  • Acute/chronic adverse effects is rare
  • Most common adverse effects: nausea and vomiting [0.5-1.2% of pts]
    • Higher incidence noted w:
      • Longer admin of nitrous
      • Fluctuations in nitrous oxide levels
      • Lack of titration
      • Increased concentrations of nitrous oxide
      • A heavy meal prior to administration of nitrous oxide
    • Fasting isn’t necessary, but can recommend lighter meal before admin
  • Other less common adverse effects
    • Silent regurgitation and silent aspiration are rare but occur
      • Concern lies whether pharyngeal- laryngeal reflexes remain intact
      • Avoid this problem by not allowing pt to go into unconscious state
    • Diffusion hypoxia
      • Can occur as a result of rapid release of nitrous oxide from the bloodstream into the alveoli, thereby diluting the concentration of oxygen
      • Can lead to headache, disorientation, and nausea
      • Can be avoided by administering 100% O2 when the nitrous has been terminated
  • Negative outcomes associated with N2O >50% and as an anesthetic during major surgery
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52
Q

N2O: Documentation

A
  • Get informed consent prior to administration of nitrous
  • Pts record should include indication for nitrous, the dosage, duration of procedure and post tx oxygenation procedure
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53
Q

N2O: Facilities, personnel, equipment

A
  • Inhalation equipment must have the capacity for delivering 100%, and never less than 30%
  • Inhalation equipment must have a fail-safe system that is checked and calibrated regularly according to the practitioner’s state laws and regulations
  • Must have an appropriate scavenging system to minimize room air contamination and occupational risk
  • Because moderate sedation may occur, practitioners should have the appropriate training and emergency equipment to manage this
  • An emergency cart should be readily available
    • Should have equipment to resuscitate a non-breathing, unconscious pt and provide continuous support until ER personnel arrive
    • A positive pressure oxygen delivery system capable of administering greater than 90% oxygen at a 10 L/min flow for at least 60 minutes (650 L, “E” cylinder) must be available
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54
Q

N2O: Occupational safety

A
  • Long-term exposure to N2O used as a general anesthetic has been linked to bone marrow suppression and reproductive system disturbances
    • Appropriate scavenging is effective in reducing the reproductive system effects
  • To avoid health hazards → use effective scavenging systems
  • Clinicians should try to minimize the patient’s talking and mouth breathing during N2O administration to prevent expired gas from contaminating the operatory
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55
Q

What has long-term N2O exposure as a general anesthetic been linked to?

A

Bone marrow suppression + reproductive system disturbances

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56
Q

What medicaments should be used as first line interventions for pain management?

A

APAP (acetaminophen)/NSAIDs

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57
Q

Pain: Definition

A
  • Pain: (International Association of the Study of Pain) An unpleasant sensory + emotional experience associated w/ actual or potential tissue damage or described in terms of such damage.
    • Acute
    • Chronic
  • Subjective, emotional, cultural component
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58
Q

Gate Theory of Pain

A
  • Melzack + Wall – 1965
    • The substantial gelatinosa in dorsal horn acts as a “gate” which modulates transmission of sensory information from primary afferent neurons to transmission cells in spinal cord
    • Small-fiber activity “opens” gate while large-fiber activity “closes” gate
    • Descending pathways modify gate – activation of large fiber sensory neurons can modify pain perception
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59
Q

What are infants + young children more sensitive to pain?

A
  • Ascending pathways intact allowing full transmission of pain
  • Descending modulating pathways poorly developed + unable to attenuate pain
  • Do not have cognitive skills to modulate pain
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60
Q

What are effects of improperly managed pain?

A
  • Significant stress rxn w/ ⇡in catecholamines, corticosteroids, suppression of insulin, significantly more complications post-operatively
  • Long-term neurologic change
    • Changes in brain achitecture
    • Changes in dorsal horn (plasticity) → hyperalgesia later in life
    • Plasticity acts as implicit memory of pain
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61
Q

What patients are at higher risk of experiencing pain?

A
  • H/o uncontrolled procedural pain or h/o frequent medical intervention
  • Infants + very young children
  • H/o prematurity
  • Past h/o physical, sexual, or psychological abuse
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62
Q

Wong-Baker FACES Pain Scale

A

Self-reported pain scale – Children >3yo

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63
Q

Visual Analogue Scale

A

Self-reported pain intensity scale – Children >6yo

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64
Q

FLACC

A
  • FLACC = Faces, Legs, Arms, Crying Consolability Scale
    • Observational pain scale for children + adults
    • Requires trained observer
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65
Q

Sucrose + Non-Pharmacologic procedural pain management

A
  • Effective for minor pain in young infants
  • Sucrose has same pathway as opioids; efficacy wanes at 6mo
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66
Q

Ice precooling injection site vs. benzocaine

A

Ice precooling has been shown to be more effective than benzocaine

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67
Q

Topical anesthetic effect – What is the depth of the effect?

A
  • Pharmacologic + psychological
    • Effective on surface up to 2-3mm
    • Subjects who are told they will receive topical for comfort anticipate less pain
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68
Q

How may topical anesthetic be most effective?

A
  • Dry mucosa
  • Longer duration is more effective (5min > 2min > 30 sec)
  • More effective in buccal fold than on palatal tissue OR for deep tissue injections
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69
Q

What type of anesthetic is topical benzocaine (up to 20%)?

A

Ester

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70
Q

What is the reason for the FDA warning w/ benzocaine?

A
  • Children <2yo (i.e. teething gels) due to methemoglobinemia
    • Ferrous iron in the hemoglobin molecule is oxidized to the ferric state to become methemoglobin
    • Cannot carry O2
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71
Q

What other types of topical anesthetic may be used?

A

20% benzocaine, 5% lidocaine, 4% tetracaine

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72
Q

Compound topical anesthetics

A
  • Not regulated by FDA
  • FDA issued warning about use of compounded topical anesthetics + risk of methemoglobinemia w/ benzocaine and/or prilocaine
  • Various formulations (usually higher doses) of tetracaine, lidocaine, benzocaine, prilocaine, and/or phenylephrine
  • Systemic absorption of topical anesthetics must be considered when calculating the total amount of anesthetic dosage administered
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73
Q

LA MOA

A

Reversibly bind to Na+ channels of nerve cells preventing depolarization + impulse propagation

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74
Q

2% lidocaine (xylocaine) 1:100k epi: Duration, Max dosages, mg/1.7mL cartridge

A
  • Duration (in minutes)
    • Max infiltration
      • Pulp = 60
      • Soft tissue = 170
    • Mand block
      • Pulp = 85
      • Soft tissue = 190
  • Max dosage = 4.4mg/kg – Total = 300mg
  • mg/1.7mL cartridge = 34mg
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75
Q

3% mepivacaine (carbocaine) plain: Duration, Max dosages, mg/1.7mL cartridge

A
  • Duration (in minutes)
    • Max infiltration
      • Pulp = 25
      • Soft tissue = 90
    • Mand block
      • Pulp = 40
      • Soft tissue = 165
  • Max dosage = 4.4mg/kg – Total = 300mg
  • mg/1.7mL cartridge = 51mg
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76
Q

4% articaine (septocaine) 1:100k epi: Duration, Max dosages, mg/1.7mL cartridge

A
  • Duration (in minutes)
    • Max infiltration
      • Pulp = 60
      • Soft tissue = 190
    • Mand block
      • Pulp = 90
      • Soft tissue = 230
  • Max dosage = 7.0mg/kg – Total = 500mg
  • mg/1.7mL cartridge = 68mg
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77
Q

4% prilocaine pain: Duration, Max dosages, mg/1.7mL cartridge

A
  • Duration (in minutes)
    • Max infiltration
      • Pulp = 20
      • Soft tissue = 105
    • Mand block
      • Pulp = 55
      • Soft tissue = 190
  • Max dosage = 6.0mg/kg – Total = 400mg
  • mg/1.7mL cartridge = 68mg
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78
Q

0.5% bupivacaine (marcaine) 1:200k epi: Duration, Max dosages, mg/1.7mL cartridge

A
  • Duration (in minutes)
    • Max infiltration
      • Pulp = 40
      • Soft tissue = 340
    • Mand block
      • Pulp = 240
      • Soft tissue = 440
  • Max dosage = 1.3mg/kg – Total = 90mg
  • mg/1.7mL cartridge = 8.5mg
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79
Q

Epi in LA

A
  • ⇓bleeding in area of injection
  • ⇓toxicity by slowing rate of resorption of anesthetic into CV system
  • Recommended, esp when tx includes 2+ quadrants
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80
Q

What is the preservative in LA that patients may have an allergic rxn to? What do we give for LA to patients w/ this allergy?

A

Bisulphite preservative is used in anesthetics w/ epi.

Use anesthetic w/o epi in patients w/ this allergy.

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81
Q

Contraindication] for epi in LA?

A

Hypothyroidism

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82
Q

When should you send a med consult for use of LA?

A
  • Diabetes
  • Significant CV disease
  • Thyroid dysfunction
  • Patients on tricyclic antidepressants, monoamine oxidase inhibitors, phenothiazines
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83
Q

Where is articaine (septocaine) metabolized?

A

Liver + plasma

Has the shortest half-life

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84
Q

FDA approval for use of articaine (septocaine) in children at what age?

A

≥4yo

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85
Q

Which amide LA is associated w/ paresthesia?

A

Articaine (septocaine) + prilocaine

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86
Q

What amide LA is associated w/ a disproportionate number of pediatric LA systemic toxicity reports?

A

Mepivacaine (carbocaine)

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87
Q

Indications for mepivavaine (carbocaine)?

A
  • Significant CV disease
  • Hyperthyroidism
  • Bisulphite sensitivity
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88
Q

Prilocaine

A
  • Can induce formation of methemoglobinemia
  • W/o vasoconstrictor has a higher pH than lidocaine, but no difference in injection pain
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89
Q

Contraindications for prilocaine use

A
  • Patients w/ various anemias
  • Symptoms of hypoxia
  • Taking acetaminophen
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90
Q

Why is bupivacaine (marcaine) not typically used in pediatric dental patients?

A

Due to the long duration of soft tissue anesthesia + ⇑risk of post-operative soft tissue trauma

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91
Q

Indication for bupivacaine (marcaine)?

A

Anticipated post-op pain OR for adolescent patient w/ a “hot” tooth

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92
Q

Infiltration vs. block (in relation to the nerve)

A

Infiltration = terminal branches of nerve

Block = main nerve trunk

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93
Q

IANB

A
  • Innervates primary + permanent mandibular teeth
  • Foramen is 7mm above mandibular plane in adults
    • More inferior + posterior in young children
  • Bilateral blocks are not associated w/ ⇑post-anesthetic soft tissue trauma
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94
Q

PSA nerve block

A
  • Advocated by some for 2nd primary + 1st permanent molars in primary + early mixed dentition due to thick zygomatic bone that overlies apices
  • Highest risk of hematoma
  • Efficacy over infiltration has been questioned
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95
Q

Greater palatine block

A
  • Anesthetizes palatal tissue from tuberosity to canine region
  • Indicated for EXT, long procedures where palatal gingiva is manipulated, or when post-op analgesia is needed
  • Level w/ distal cusp of permanent 1st molars + between tooth and midline of palate
    • If no 1st permanent molar present, injection should be made 10mm distal to primary molar
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96
Q

If no 1st permanent molar is present, where should you drop LA for greater palatine block?

A

If no 1st permanent molar present, injection should be made 10mm distal to primary molar

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97
Q

Nasopalatine injection

A
  • Innervates palatal tissue of maxillary incisors
  • Most painful injection
98
Q

PDL injections in animal model studies

A
  • May be an adjunct to other forms of LA or used alone for single tooth anesthesia
  • ⇑pressure injections associated w/ permanent tooth bud damage in animal models can lead to hypomineralization
  • Injury to succedaneous tooth is not associated w/ computer controlled LA delivery
99
Q

Intrapulpal injection

A
  • Direct deposition into the pulp when other forms of anesthesia are inadequate
    • Effective only when injected under pressure
  • Short duration of action = 15-20min
100
Q

What are hallmarks of inflammatory pain?

A
  • Swelling
  • Hyperthermia
  • Activation of biochemical cascades
101
Q

How is pain processed?

A

Sensory signals travel along afferent trigeminal nerve fibers** and relay information to the **brainstem and higher structures involved w/ pain perception.

102
Q

What is peripheral sensitization?

A

Local increase in nerve membrane activity/excitability → enhanced neuronal response

Primary hyperalgesia: Exaggerated response

103
Q

Central sensitization

A

Enhanced functional status of pain circuits + pain processing at the level of the CNS

  • Characteristics
    • Secondary hyperalgesia: ⇑pain intensity to noxious stimuli outside the area of tissue damage
    • Allodynia: Pain perception following innocuous stimuli like light touch
104
Q

Pain modulation

A
  • Occurs through CNS excitatory + inhibitory processes.
  • Ascending facilitating and descending inhibitory processes enhance or suppress pain experience respectively.
    • Both pharmacologic + nonpharmacologic methods target these processes to alter pain processing.
105
Q

How is pain assessed?

A
  • Self-report
  • Behavioral (vocalization, facial expression, body movement)
  • Biological (heart rate, transcutaneous oxygen, sweating, stress response)
106
Q

What questions should you ask regarding pain?

A

Onset, provoking factors, palliative factors, quality or character, region or location, severity or intensity, timing or duration, and impact on daily activities.

107
Q

At what age can patients likely quantify pain based on a series of faces? (Reference Manual)

A

4-12yo

108
Q

At what age can patients mark pain using a VAS or numeric scale? (Reference Manual)

A

>7yo

109
Q

Validated instruments for pain assessment (Ref Manual)

A
  • Faces Pain Scale (Revised)
  • VAS
  • Numeric rating scale
  • Faces, Legs, Activity, Cry and Consolability score (FLACC)
  • Revised Faces, Legs, Activity, Cry and Consolability (r-FLACC)
  • McGill Pain Questionnaire
110
Q

Diagnostic categories of pain

A
  • Somatic
  • Visceral
  • Neuropathic
111
Q

What type of pain do we typically encounter in dentistry? (Our patients, not referring to the emotional pain of the providers lol)

A
  • Typically inflammatory
    • Somatic
      • Periodontal, alveolar, mucosal
    • Visceral
      • Pulpal
112
Q

Acute vs. Chronic Pain

A
  • Acute pain that fails to respond to treatment may become chronic over time.
  • Chronic pain: Dysfunctional and persists beyond the time for typical tissue healing (i.e. TMD).
113
Q

Profound anesthesia prior to starting tx decreases __

A

⇓central sensitization

114
Q

Pain reduction during LA injection

A
  • Distraction techniques (i.e. jiggling the patient’s cheek) take advantage of the Aß-fiber signal dominance and can significantly reduce the intensity of pain-related C-fiber signaling.
  • Buffering or decreasing acidity of LA with sodium bicarbonate can decrease injection site pain and postop discomfort by increasing pH of the anesthetic.
  • Decreasing LA delivery rate.
115
Q

GA w/o LA may result in what?

A
  • Pain is not experienced during GA, but central sensitization occurs when peripheral nerves are stimulated.
  • Operating w/o LA may result in priming CNS neurons and increased future pain sensitivity.
  • Central sensitization is minimized w/ pre-emptive analgesia or anesthesia.
    • Regional block or infiltration is commonly done prior to surgical procedures to decrease postop pain.
  • Pharmacologic + cardiac considerations along with avoiding the numb sensation and potential for self-inflicted oral trauma are reasons providers may choose not to provide LA during GA.
116
Q

At what age are distraction/imagery techniques most effective?

A
  • >8yo
  • Cognitive (counting, non-procedural talk)
  • Behavioral (videos, games)
  • Significantly effective when measuring pulse rates, respiratory rates, and self-reported pain.
  • Lowers pain perception in younger children.
117
Q

NSAIDs: Properties

A

Anti-inflammatory, analgesic, antipyretic antiplatelet properties.

118
Q

NSAIDs: MOA

A
  • Inhibit prostaglandin synthesis
    • COX (cyclooxygenase)
119
Q

What is included in NSAIDs?

A
  • Salicylic acid: Aspirin
  • Acetic acids: Ketolorac (IV or IN)
    • Useful in moderate-severe acute pain in patients unable to take PO
  • Proprionic acids: Ibuprofen, naproxen
  • COX-2 selective: Celecoxib
120
Q

NSAIDs: Adverse effects

A
  • Inhibition of bone growth and healing
  • Gastritis w/ pain + bleeding
  • Decreased renal blood flow
  • Inhibition of platelet function
  • Increased incidence of CV events
  • Specific concern: Potential to exacerbate asthma due to a shift in leukotrienes.

Due to shared pathways, NSAIDs and steroidal anti-inflammatory medications should not be routinely co-administered.

121
Q

Why should NSAIDs + steroidal anti-inflammatory meds not be routinely co-administered?

A

Due to shared pathways, NSAIDs and steroidal anti-inflammatory medications should not be routinely co-administered.

122
Q

What is APAP (acetaminophen) efficacious for?

A

Mild to moderate pain, antipyretic

Centrally-acting

No effects on gastric mucosal lining or platelets

123
Q

APAP (acetaminophen): MOA

A

Blockade of prostaglandin + substance P production

124
Q

Why does rectal administration of APAP (acetaminophen) have higher bioavailability + faster onset than PO?

A

It partially bypasses hepatic metabolism

125
Q

What is multimodal therapy (w/ NSAIDs)?

A

Alternating/staggering NSAIDs

126
Q

Common uses for opioid analgesics

A
  • Pain w/ cancer
  • Sickle cell disease
  • Osteogenesis imperfecta
  • Epidermolysis bullosa
  • Neuromuscular disease
127
Q

Major concerns of opioid analgesics

A

Efficacy, safety, misuse, accidental deaths

128
Q

Opioid Analgesics: MOA

A
  • Interact w/ differentially w/ μ, 𝛋, and 𝛅 receptors in the CNS.
    • Opioid antagonists act on receptors located in the brain, spinal cord, and digestive tract.
  • Pathways of opioid receptor signaling are multiple and include G-protein receptor coupling, cAMP inhibition, and calcium channel inhibition.
129
Q

What does activating opioid receptors cause?

A
  • Respiratory depression
  • Pupil constriction (miosis)
  • Euphoria
  • Sedation
  • Physical dependence
  • Endocrine disruption
  • Suppression of opiate withdrawal
  • Pruritis (itching) may also occur due to histamine release that accompanies some opioid analgesics
130
Q

Naloxone

A

μ opioid receptor competitive antagonist usually administered parenterally to counter opioid overdose

131
Q

Opioids w/ active metabolites: Which opioids and by what enzyme?

A

Codeine, tramadol, hydrocodone are broken down in the liver into active metabolites by highly variable cytochrome CYP2D6.

132
Q

Why have the FDA + European Medicines Agency have issued warnings + contraindications statements on codeine and tramadol?

A
  • Ineffective in some children due to poor drug metabolism.
  • Hyper-metabolizers break these prodrugs into their active forms too quickly, potentially resulting in overdose, respiratory depression and death.
  • Hydrocodone also relies on cytochrome p450 metabolism and has similar adverse effects
  • Evidence is not convincing that these medications provide appropriate analgesia, especially with concerns of safety.
133
Q

2017 – FDA issued a warning for codeine + tramadol

A
  • Not safe for patients <12yo.
  • 12-17yo age group, should not be used in high-risk patients (e.g. obesity, OSA, lung tissue disease)
  • Should not be used when breastfeeding due to active metabolites present in breastmilk
134
Q

What opioids do not have active metabolites?

A

Naturally occurring morphine + synthetics oxycodone and fentanyl do not have CYP2D6 considerations since they do not contain CYP2D6 metabolites

135
Q

Morphine

A
  • Rapid relief of severe pain for 2-3 hours
  • Associated w/ histamine release + respiratory depression
136
Q

Fentanyl

A
  • 100x more potent than morphine
  • Ultra-short acting
  • Used for invasive procedures + sedatives

Adverse reaction: Chest wall rigidity

137
Q

Adverse reaction to fentanyl

A

Adverse reaction: Chest wall rigidity

138
Q

Codeine

A
  • Rapidly acting codeine has a longer half-life than morphine and is more potent.
  • Single agent vs. combined with aspirin, ibuprofen, acetaminophen
  • Considered off label in children 12yo and younger
139
Q

Opioid Concerns + CDC and Prevention Recommendations

A
  • Limit opioids for moderate to severe pain
    • Restrict prescription to 3 days
    • Provide concurrent pharmacologic and non-pharmacologic therapy
  • Advises against overlap benzodiazepines + opioid prescriptions because of increased risk for potential respiratory depression.
140
Q

Caution with commercial opioids combined w/ acetaminophen

A

Commercial opioids often combined with acetaminophen, the potential for hepatic failure from toxic levels of acetaminophen also must be considered

141
Q

Phentolamine mesylate

A
  • Reversible, non-selective alpha adrenergic antagonist which results in vasodilation and redistribution of LA away from injection site
  • Reduces duration of soft tissue anesthesia by approximately half
  • FDA approval for children >6yo
  • Should not be used to reverse LA toxicity
142
Q

FDA approved use for phentolamine – at what age?

A

>6yo

143
Q

LA Overdose: Causes

A
  • Inadvertent intravascular injection at a rapid rate
  • Excess dosage
144
Q

Signs of LA overdose

A
  • Biphasic clinical manifestation
    • Initial excitatory reaction (inhibitory neurons are depressed)
      • Circumoral tingling, dizziness, tinnitus, increased heart rate, and BP
    • Later depressive reaction
      • Drowsiness, loss of consciousness, seizure, cardiovascular collapse

Other: Needle breakage, prolonged anesthesia or paresthesia, facial nerve paralysis, trismus, hematoma

145
Q

Ibuprofen

A
  • NSAID
  • First line for dental pain if not contraindicated
  • Associations between development of asthma + maternal and infant use of acetaminophen + ibuprofen, but further research is needed
  • Efficacy equivalent to morphine w/ fewer side effects
146
Q

Aspirin in children

A
  • Not recommended for children
  • Some teething gels contain aspirin-like product called choline salicylate
147
Q

Naproxen OTC use by children

A

Not recommended for OTC use in <12yo

148
Q

Naproxen: MOA

A

Reversibly inhibits COX-1 and 2 enzymes resulting in decreased formation of prostaglandin precursors

149
Q

Opioids MOA

A

Bind to opioid receptors in CNS, causing inhibition of ascending pain pathways, altering perception of and response to pain; cause generalized CNS depression

150
Q

Metabolism of codeine, hydrocodone, and oxycodone

A

Catalyzed in the liver by cytochrome P450 enzymes CYP3A4 + CYP2D6

151
Q

Drug-drug interactions that occur with opioids

A
  • Drug-drug interactions can occur with opioids and CYP3A4 inhibitors leading to an increase in drug concentration and potential toxicity
    • CYP3A4 inhibitors:
      • Macrolide antibiotics
      • Proton pump inhibitors
      • Selective serotonin reuptake inhibitors
      • Natural supplements like St. John’s wart + Echinacea
152
Q

Opioids: Adverse effects

A
  • Nausea/vomiting
  • Constipation
  • Sedation/cognitive function
  • Respiratory depression
153
Q

Codeine: FDA Blackbox warning

A
  • Ultra-rapid metabolizers have polymorphism of liver cytochrome enzyme CYP2D6 + convert codeine to high levels of morphine rapidly.
  • FDA blackbox warning due to death following tonsillectomy
  • Contraindicated in children at risk for sleep apnea
154
Q

Poor metabolizers may have inadequate pain control with codeine because? What is codeine’s metabolite?

A

Codeine is a prodrug, meaning that its analgesic effect is due to its metabolite, morphine

155
Q

DEA Schedule for Codeine?

A

FDA Schedule IV allows it to be called into a pharmacy and does not require a written prescription

156
Q

Hydrocodone

A
  • Both parent drug + metabolite have analgesic effects, though parent drug is a weak analgesics
  • CYP2D6 converts hydrocodone into hydromorphone, a more potent opioid
157
Q

DEA Schedule for Hydrocodone?

A

DEA Schedule II requires a written prescription

158
Q

DEA Schedule for oxycodone

A

DEA Schedule II requires a written prescription

159
Q

Systemic analgesics dosing

A
  • “Around the clock” is preferred to PRN for first 36-48hrs
  • Alternating ibuprofen + acetaminophen every 3hrs is safe for short term use with compliant caregivers
160
Q

What type of injuries occur in more than half of child abuse cases?

A

Craniofacial, head, face and neck injuries

161
Q

What is the most common site for inflicted oral injuries?

A

Lips (54%), followed by the oral mucosa, teeth, gingiva, and tongue

162
Q

An intercanine distance measuring more than __ is suspicious for an adult human bite

A

3.0 cm

163
Q

More than __ of children who are victims of human trafficking still see a health care professional

A

One quarter

164
Q

Child trafficking victims have __ risk for dental problems because they “often suffer from inadequate nutrition leading to retarded growth and poorly formed teeth, as well as dental caries, infections and tooth loss.”

A

Twice the risk

165
Q

Orientation to dental environment

A
  • The non-clinical office staff plays an important role in behavior guidance.
    • first point of contact
  • The tone of the communication should be welcoming.
  • From a behavioral standpoint, many factors are important when appointment times are determined.
  • Appointment-related concerns include patient age, presence of a special health care need, the need for sedation, distance the parent/patient travels, length of appointment, additional staffing requirements, parent’s work schedule, and time of day.
  • Appointment duration should not be prolonged beyond a patient’s tolerance level solely for the practitioner’s convenience.
  • A child- friendly reception area (e.g., age-appropriate toys and games) can provide a distraction for and comfort young patients.
    • These first impressions may influence future behaviors.
166
Q

What are the 4 essential ingredients of communication?

A
  • the sender,
  • the message, including the facial expression and body language of the sender,
  • the context or setting in which the message is sent, and
  • the receiver
167
Q

What to do if the parent refuses treatment after discussing risks/benefits and alternative of proposed treatment and behavior guidance techniques?

A
  • If the parent refuses treatment after discussions of the risks/benefits and alternatives of the proposed treatment and behavior guidance techniques, an informed refusal form should be signed by the parent and retained in the patient’s record.
    • If the dentist believes the informed refusal violates proper standard of care, he should recommend the patient seek another opinion and/or dismiss the patient from the practice.
168
Q

Frankl behavioral rating scale

A
      • Definitely negative. Refusal of tx, forceful crying, fearfulness, or any other overt evidence of extreme negativism.
    • Negative. Reluctance to accept tx, uncooperative, some evidence of negative attitude but not pronounced (sullen, withdrawn).
    • Positive. Acceptance of tx, cautious behavior at times, willingness to comply with the dentist, at times with reservation, but patient follows the dentist’s directions cooperatively.
  1. ++ Definitively positive. Good rapport with the dentist, interest in the dental procedures, laughter and enjoyment.
169
Q

Goals of behavior guidance

A
  1. establish communication,
  2. alleviate the child’s dental fear and anxiety,
  3. promote patient’s and parents’ awareness of the need for good oral health and the process by which it is achieved,
  4. promote the child’s positive attitude toward oral health care,
  5. build a trusting relationship between dentist/staff and child/parent, and
  6. provide quality oral health care in a comfortable, minimally- restrictive, safe, and effective manner.
170
Q

Communication and communicative guidance

A
  • asking questions and active/reflective listening can help establish rapport and trust.
  • The dentist may establish teacher/student roles in order to develop an educated patient and deliver quality dental treatment safely.
  • Once a procedure begins, bi-directional communication should be maintained, and the dentist should consider the child as an active participant in his well-being and care.
    • With this two-way interchange of information, the dentist also can provide one-way guidance of behavior through directives.
  • Use of self-disclosing assertiveness techniques (e.g., “I need you to open your mouth so I can check your teeth”, “I need you to sit still so we can take an X-ray”) tells the child exactly what is required to be cooperative.
  • The dentist can ask the child ‘yes’ or ‘no’ questions where the child can answer with a ‘thumbs up’ or ‘thumbs down’ response.
171
Q

Positive pre-visit imagery

A
  • Description: Patients preview positive photographs or images of dentistry and dental treatment before the dental appointment
  • Objectives:
    • provide children and parents with visual information on what to expect during the dental visit; and
    • provide children with context to be able to ask providers relevant questions before dental procedures commence.
  • Indications: Use with any patient.
  • Contraindication: None.
172
Q

Direct observation

A
  • Description: Patients are shown a video or are permitted to directly observe a young cooperative patient undergoing dental treatment
  • Objectives:
    • familiarize the patient with the dental setting and specific steps involved in a dental procedure; and
    • provide an opportunity for the patient and parent to ask questions about the dental procedure in a safe environment
  • Indications: Use with any patient.
  • Contraindications: None
173
Q

Tell-show-do

A
  • Description: verbal explanations of procedures in phrases appropriate to the developmental level of the patient (tell); demonstrations for the patient of the visual, auditory, olfactory, and tactile aspects of the procedure in a carefully defined, nonthreatening setting (show); and then, without deviating from the explanation and demonstration, completion of the procedure (do).
  • The tell-show-do technique operates with communication skills (verbal and nonverbal) and positive reinforcement.
  • Objectives:
    • teach the patient important aspects of the dental visit and familiarize the patient with the dental setting and armamentarium; and
    • shape the patient’s response to procedures through desensitization and well-described expectations.
  • Indications: Use with any patient.
  • Contraindications: None.
174
Q

Ask-tell-ask

A
  • Description: This technique involves inquiring about the patient’s visit and feelings toward or about any planned procedures (ask); explaining the procedures through dem- onstrations and non-threatening language appropriate to the cognitive level of the patient (tell); and again inquiring if the patient understands and how she feels about the impending treatment (ask)
  • Objectives:
    • assess anxiety that may lead to noncompliant behavior during treatment;
    • teach the patient about the procedures and their imple- mentation; and
    • confirm the patient is comfortable with the treatment before proceeding.
175
Q

Voice control

A
  • Description: deliberate alteration of voice volume, tone, or pace to influence and direct the patient’s behavior.
  • An explanation before its use may prevent misunderstanding.
  • Objectives: The objectives of voice control are to:
    • gain the patient’s attention and compliance;
    • avert negative or avoidance behavior; and
    • establish appropriate adult-child roles.
  • Indications: Use with any patient.
  • Contraindications: Patients who are hearing impaired.
176
Q

Nonverbal communication

A
  • Description: reinforcement and guidance of behavior through appropriate contact, posture, facial expression, and body language.
  • Objectives:
    • enhance the effectiveness of other communicative
      guidance technique; and
    • gain or maintain the patient’s attention and compliance.
  • Indications: Use with any patient.
  • Contraindications: None.
177
Q

Positive reinforcement and descriptive praise

A
  • Description: Positive reinforcement rewards desired behaviors thereby strengthening the likelihood of recurrence of those behaviors.
    • Social reinforcers include positive voice modulation, facial expression, verbal praise, and appropriate physical demonstrations of affection by all members of the dental team.
    • Descriptive praise emphasizes specific cooperative behaviors (e.g., “Thank you for sitting still”, “You are doing a great job keeping your hands in your lap”) rather than a generalized praise (e.g., “Good job”).
    • Nonsocial reinforcers include tokens and toys.
  • Objective:
    • to reinforce desired behavior
  • Indications: Use with any patient.
  • Contraindications: None.
178
Q

Distraction

A
  • Description: diverting the patient’s attention from what may be perceived as an unpleasant procedure
    • achieved by imagination (e.g., stories), clinic design, and audio (e.g., music) and/or visual (e.g., television, virtual reality eye- glasses) effects.
  • Giving the patient a short break during a stressful procedure can be an effective use of distraction before considering more advanced behavior guidance techniques
  • Objectives:
    • decrease the perception of unpleasantness
    • avert negative or avoidance behavior.
  • Indications: Use with any patient.
  • Contraindications: None.
179
Q

Memory restructuring

A
  • Description: behavioral approach in which memories associated with a negative or difficult event (e.g., first dental visit, local anesthesia, restorative pro- cedure, extraction) are restructured into positive memories using information suggested after the event has taken place.
  • This approach was utilized with children who received local anesthesia at an initial restorative dental visit and showed a change in local anesthesia-related fears and behaviors at subsequent treatment visits.
  • Restructuring involves four components:
    • visual reminders;
    • positive reinforcement through verbalization;
    • concrete examples to encode sensory details; and
    • sense of accomplishment.
  • A visual reminder could be a photograph of the child smiling at the initial visit (i.e., prior to the difficult experience).
  • Positive reinforcement through verbalization could be asking if the child had told her parent what a good job she had done at the last appointment.
  • The child is asked to role-play and to tell the dentist what she had told the parent.
  • Concrete examples to encoding sensory details include praising the child for specific positive behavior such as keeping her hands on her lap or opening her mouth wide when asked.
  • The child then is asked to demonstrate these behaviors, which leads to a sense of accomplishment.
  • Objectives: The objectives of memory restructuring are to:
    • restructure difficult or negative past dental experiences;
    • improve patient behaviors at subsequent dental visits.
  • Indications: Use with patients who had a negative or difficult dental visits.
  • Contraindications: None.
180
Q

Desensitization to dental setting and procedures

A
  • Description: psychological technique- diminishes emotional responsiveness to a negative, aversive, or positive stimulus after progressive exposure to it.
  • Patients are exposed gradually through a series of sessions to components of the dental appointment that cause them anxiety.
  • Patients may review information regarding the dental office and environment at home with a preparation book or video or by viewing the practice website. Parents may model actions (e.g., opening mouth and touching cheek) and practice with the child at home using a dental mirror.
  • Successful approximations would continue with an office tour during non-clinical hours and another visit in the dental operatory to explore the environment.
  • Objectives:
    • proceed with dental care after habituation and successful
      progression of exposure to the environment;
    • identify his fears;
    • develop relaxation techniques for those fears; and
    • be gradually exposed, with developed techniques, to
      situations that evoke his fears and diminish the emotional
      Responses.
  • Indications: Use with patients who have experienced fear-invoking stimuli, anxiety, and/or neurodevelopmental disorders (e.g., autism spectrum disorder
  • Contraindications: None.
181
Q

Enhancing control

A
  • Description: allow the patient to assume an active role in the dental experience.
    • The dentist provides the patient a signal (e.g., raising a hand) to use if he becomes uncomfortable or needs to briefly interrupt care.
    • The patient should practice this gesture before treatment is initiated to emphasize it is a limited movement away from the operatory field.
    • When the patient employs the signal during dental procedures, the dentist should quickly respond with a pause in treatment and acknowledge the patient’s concern.
    • Enhancing control has been shown to be effective in reducing intraoperative pain.
  • Objectives: to allow a patient to have some measure of control during treatment in order to contain emotions and deter disruptive behaviors.
  • Indications: Use with patients who can communicate.
  • Contraindications: None, but if used prematurely, fear may increase due to an implied concern about the impending procedure.
182
Q

Communication techniques for parents (and age-appropriate patients)

A
  • Because parents are the legal guardians of minors, successful bi-directional communication between the dentist/staff and the parent is essential to assure effective guidance of the child’s behavior.
  • Communication techniques such as ask-tell-ask, teach back, and motivational interviewing can reflect the dentist/staff’s caring for and engaging in a patient/ parent centered-approach.
  • These techniques are presented in Appendix 3.
183
Q

Parental presence/absence

A
  • Description: A wide diversity exists in practitioner philosophy and parents’ attitude regarding parental presence/absence during pediatric dental treatment.
    • Parental involvement, especially in their children’s health care, has changed dramatically in recent years
    • Practitioners should become accustomed to this added involvement of parents and welcome the questions and concerns for their children. Practitioners must consider parents’ desires and wishes and be open to a paradigm shift in their own thinking.
  • Objectives: The objectives of parental presence/absence for parents are to:
    • participate in examinations and treatment;
    • offer physical and psychological support; and
    • observe the reality of their child’s treatment.
  • The objectives:
    • gain the patient’s attention and improve compliance;
    • avert negative or avoidance behaviors;
    • establish appropriate dentist-child roles;
    • enhance effective communication among the dentist,
    • child, and parent;
    • minimize anxiety and achieve a positive dental experience;
    • facilitate rapid informed consent for changes in treatment or behavior guidance.
  • Indications: Use with any patient.
  • Contraindications: Parents who are unwilling or unable to extend effective support.
184
Q

Sensory-adapted dental environments (SADE)

A
  • Description: includes adaptions of the clinical setting (e.g., dimmed lighting, moving projections such as fish or bubbles on the ceiling, soothing background music, application of wrap/blanket around the child to provide deep pressure input) to produce a calming effect.
  • Objectives: enhance relaxation and avert negative or avoidance behaviors
  • Indications: Use with patients having autism spectrum disorder, sensory processing difficulties, other disabilities, or dental anxiety
  • Contraindications: None.
185
Q

Animal-assisted therapy (AAT)

A
  • Description: It is a goal-oriented intervention which utilizes a trained animal in a healthcare setting to improve interactions or decrease a patient’s anxiety, pain, or distress.
    • Unlike animal-assisted activities (e.g., a pet entertains patients in the waiting area), AAT appointments are scheduled for specific time and duration to include an animal that has undergone temperament testing, rigorous training, and certification.
    • The animal, which is available for companionship during the dental visit, can help break communication barriers and enable the patient to establish a safe and comforting relationship, thereby reducing treatment- related stress.
  • For each visit, the goals and results of the intervention should be documented.
  • Objectives:
    • enhance interactions between the patient and dental team;
    • calm or comfort an anxious or fearful patient;
    • provide a distraction from a potentially stressful situation;
    • decrease perceived pain
      The health and safety of the animal and its handler need
      to be maintained.
  • Indications: Use AAT as an adjunctive technique to decrease
    a patient’s anxiety, pain, or emotional distress.
  • Contraindications: The contraindications for the parent:
    • allergy or other medical condition (e.g., asthma, compromised immune system) aggravated by exposure to the animal; and
    • lack of interest in or fear of the therapy animal.
    • The contraindications for the parent:
      • a situation that presents a significant risk to one’s health or safety
186
Q

Picture exchange communication system (PECS)

A
  • Description: communication technique developed for individuals with limited to no verbal communication abilities, specifically those with autism.
  • The individual shares a picture card with a recognizable symbol to express a request or thought. PECS has a one-to-one correspondence with objects, people, and concepts, thereby reducing the degree of ambiguity in communication.
  • The patient is able to initiate communication, and no special training is
    required by the recipient.
  • Objectives:
    • to allow individuals with limited to no verbal communication abilities to express requests or thoughts using symbolic imagery.
    • A prepared picture board may be present for the dental appointment so the dentist can communicate the steps required for completion (e.g., pictures of a dental mirror, handpiece).
  • The patient may have symbols (e.g., a stop sign) to show they need a brief interruption in the procedure.
  • Indications:
    • Use as an adjunctive approach to assist individuals with limited to no verbal communication abilities improve exchange of ideas.
187
Q

Nitrous oxide/oxygen inhalation

A
  • Description: safe and effective technique to reduce anxiety and enhance effective communication.
  • Rapid onset of action, the effects easily are titrated and reversible, and recovery is rapid and complete.
  • It mediates a variable degree of analgesia, amnesia, and gag reflex reduction.
  • If nitrous oxide/oxygen inhalation is used greater than 50% or in combination with other sedating medications (e.g., benzodiazepines, opioids), the likelihood for moderate or deep sedation increases.
    • clinician must be prepared to institute the guidelines for moderate or deep sedation.
  • Objectives:
    • reduce or eliminate anxiety;
    • reduce untoward movement and reaction to dental treatment;
    • enhance communication and patient cooperation;
    • raise the pain reaction threshold;
    • increase tolerance for longer appointments;
    • aid in treatment of the mentally/physically disabled or medically compromised patients;
    • reduce gagging; and
    • potentiate the effect of sedatives.
  • Indications:
    • a fearful, anxious, or obstreperous (noisy, unruly) patient;
    • certain patients with SHCN;
    • a patient whose gag reflex interferes with dental care;
    • a patient for whom profound local anesthesia cannot be obtained; and
    • a cooperative child undergoing a lengthy dental procedure.
  • Contraindications:
    • some chronic obstructive pulmonary diseases;
    • current upper respiratory tract infections;
    • recent middle ear disturbance/surgery;
    • severe emotional disturbances or drug-related dependencies;
    • first trimester of pregnancy;
    • treatment with bleomycin sulfate;
    • methylenetetrahydrofolate reductase (MTHR) deficiency
    • cobalamin (vitamin B-12) deficiency
188
Q

The following need to be considered when deciding on sedation:

A
  • alternative behavioral guidance modalities
  • dental needs of the patient
  • the effect on the quality of dental care
  • the patient’s emotional development
  • the patient’s medical and physical considerations.
189
Q

Goals of sedation:

A
  • patient safety
  • minimize physical discomfort and pain
  • manage anxiety
  • minimize psychological trauma
  • maximize the potential for amnesia
  • manage behavior and/or movement so as to allow the safe completion of the procedure
  • return the patient to a state in which safe discharge from medical supervision, as determined by recognized criteria, is possible.
190
Q

Indications of sedation

A
  • fearful/anxious patients for whom basic behavior guidance techniques have not been successful
  • patients who cannot cooperate due to a lack of psychological or emotional maturity and/or mental, physical, or medical conditions
  • patients for whom the use of sedation may protect the developing psyche and/or reduce medical risk
191
Q

Contraindications of sedation

A
  • cooperative patient with minimal dental needs
  • predisposing medical and/or physical conditions which would make sedation inadvisable.
192
Q

Documentation of sedation

A
  • informed consent that is obtained from the parent and documented prior to the use of sedation
  • pre- and post-operative instructions and information provided to the parent
  • health evaluation
  • a time-based record that includes the name, route, site, time, dosage, and effect on patient of administered drugs
  • the patient’s level of consciousness, responsiveness, heart rate, blood pressure, respiratory rate, and oxygen saturation prior to treatment, at the time of treatment, and postoperatively until predetermined discharge criteria have been attained
  • adverse events (if any) and their treatment
  • time and condition of the patient at discharge.
193
Q

Protective stabilization

A

physical limitation of a patient’s movement by a person or restrictive equipment, materials or devices for a finite period of time in order to safely provide examination, diagnosis, and/or treatment

194
Q

Papoose considerations

A
  • Management customized to individual needs of child and desires of parent - may include sedation, GA, protective stabilization, or referral to another dentist
  • consider patient’s oral health needs, emotional and cognitive development levels, medical and physical conditions, and parental preferences
  • alternative approaches (ex. treatment options or deferral, sedation, GA) and potential impact on quality of care and the patient’s well-being in discussion
  • over 50% use and acceptance of protective stabilization devices by board certified pediatric dentists
  • Practitioner gender, practice setting, region, and perception of parental acceptance - factors for use and acceptance
195
Q

Patient selection for papoose

A
  • prevent or minimize psychological stress and decrease risk of physical injury to patient, the parent, and staff
  • at least one state (Colorado) requires training after dental school to use protective stabilization
196
Q

Papoose consent

A
  • informed consent
  • benefits and risks of protective stabilization and alternative treatment options (interim therapeutic restoration [ITR], SDF, treatment deferral) and alternative behavior guidance techniques (ex. sedation, general anesthesia) discussed and assist parent in determining most appropriate approach
  • should occur on a day separate from treatment
  • obtained and documented in patient’s record prior to using
  • behavior change during treatment - further consent obtained and documented
  • when appropriate - explanation to patient about restraint and allow patient to respond
  • minor - not have statutory right to give or refuse consent for treatment but child’s wishes and feelings (assent) should be considered
  • adolescents or adults with mild intellectual disabilities - patient assent for protective stabilization should be considered
  • 50% of states have adopted the patient-oriented standard
    • Practitioner liable if parent not received all essential information to accept or reject proposed treatment
197
Q

Papoose – parental presence

A
  • may help child and parent during a difficult experience
  • 92% mothers in one study believed they should have been with their child when placed on board to increase the child’s security and/ or comfort
  • 90% recognized that immobilization protected the child from harm
  • if parents are denied access, they must be informed of the reason with documentation of the explanation in the patient’s chart
  • parent has the right to terminate use of restraint at any time if believe child may be experiencing physical or psychological trauma due to immobilization
  • complete treatment to a safe point before ending appointment
198
Q

Papoose techniques

A
  • only when less restrictive interventions are not effective
  • not to be used as discipline, convenience, or retaliation
  • should not induce pain
  • if history of combative behavior that could harm patient and staff
  • when immobilization is indicated, the least restrictive alternative or technique should be used
  • need accurate, comprehensive, and up-to-date medical history
  • conditions (ex asthma) - may compromise respiratory function or neuromuscular or bone/skeletal disorders (may need additional positioning aids due to rigid extremities)
  • should begin in conjunction with distraction techniques
  • gradually increase or decrease levels of restriction
  • Full-body protective stabilization - sequential manner
  • head hold activated last
199
Q

Ideal characteristics of papoose equipment

A
  • easily used
  • appropriately sized for patient
  • soft and contoured to minimize potential injury
  • specifically designed for patient stabilization (not improvised equipment)
  • able to be disinfected
  • stabilization patients extremities: Posey straps, hook and loop straps, seat belts or an extra assistant.
  • Full body: Papoose Board and Pedi-Wrap
  • Stabilization for the head - forearm-body support, a head positioner, or an extra assistant
  • mouth prop in a compliant child is not considered protective stabilization
200
Q

Monitoring of patient in papoose

A
  • awareness/assessment of the patient’s physical and psychological well-being
  • tightness of the stabilization
  • severe emotional stress - terminated as soon as possible to prevent possible physical or psychological trauma
  • removal of restraints - sequentially with short pauses between stages to assess the patient’s level of cooperation
  • intra-operatively (unplanned intervention), debriefing is beneficial for parent/patient and discuss management for future appointments
201
Q

Patients with SHCN – papoose

A
  • distraction, shaping, modeling, sensory integration, desensitization, and reinforcement – are regarded as alternatives
  • Non-pharmacological behavior guidance - effective in autism spectrum disorders
  • Studies - deep pressure from an immobilization device (Papoose board) provided comfort, reduced effects of stressful stimuli, and were observed to be non-harmful to special needs patients
  • Be cautious when patient receiving multiple medications
  • potential adverse CNS or cardiac events may increase when protective stabilization is instituted on patients receiving psychotropic or other medications
202
Q

Papoose indications

A
  • requires immediate diagnosis and/or urgent limited treatment and cannot cooperate due to developmental levels (emotional or cognitive), lack of maturity, or medical/physical conditions
  • urgent care and uncontrolled movements risk the safety of the patient, staff, dentist, or parent without the use of protective stabilization
  • previously cooperative patient who quickly becomes uncooperative and cooperation, safety and stabilization cannot be regained by basic behavior guidance techniques
  • uncooperative patient who requires limited (quadrant) treatment and sedation or general anesthesia may not be an option because the patient does not meet sedation criteria or because of a long operating room wait time, financial considerations, and/or parental preferences after other options have been discussed
  • sedated patient who requires limited stabilization
  • SHCN with uncontrolled movements that would be harmful or significantly interfere with the quality of care
203
Q

Papoose contraindications

A
  • cooperative non-sedated patient
  • uncooperative patient when there is not a clear need to provide treatment at that particular visit
  • patient who cannot be immobilized safely due to associated medical, psychological, or physical conditions
  • patient with a history of physical or psychological trauma, including physical or sexual abuse or other trauma that would place the individual at greater psychological risk during restraint
  • patient with non-emergent treatment needs in order to accomplish full mouth or multiple quadrant dental treatment
  • practitioner’s convenience
  • dental team without knowledge and skills in patient selection and restraining techniques to prevent or minimize psychological stress and/or decrease risk of physical injury
204
Q

Papoose risks

A
  • consider patient’s emotional and cognitive developmental levels and potential physical and psychological effect
  • minor bruises and scratches (more serious injuries have been reported)
  • fewer injuries with passive stabilization compared to active stabilization, and with the use of planned passive stabilization compared to emergent situations
  • Overheating
  • never be unattended - can roll off
  • may not allow for complete extension of the neck - may compromise airway patency, especially in young children or sedated patients (can use neck roll)
  • significant release of adrenal catecholamines - excessive release may sensitize the heart and cause rhythm disturbances
  • physical and psychological health of the patient should override other factors (practitioner convenience, financial compensation)
205
Q

Papoose documentation

A
  • indication for stabilization
  • type of stabilization
  • informed consent
  • reason for parental exclusion during protective stabilization (when applicable)
  • the duration of application
  • behavior evaluation/rating during stabilization
  • any untoward outcomes (skin markings)
  • management for future appointments
206
Q

Classic behavioral theories

A
  • Psychoanalytic
    • Freud
    • Erickson
  • Behaviorism
    • Pavlov
    • Skinner
    • Social learning
  • Cognitive development theory
207
Q

Psychoanalytic: Freud

A
  • Children progress through predictable psychosexual stages of early childhood development
  • Child’s behavior is oriented towards certain parts of that body
208
Q

Psychoanalytic: Erickson

A
  • Psychosocial development proceeds by critical steps or psychosocial crises that shape personality
  • Basic trust → autonomy → initiative → industry → identity → intimacy → generativity → integrity
209
Q

Behaviorism: Pavlov

A

Classical conditioning – one stimulus is associated w/ another through experience

210
Q

Behaviorism: Skinner

A

Operant – consequence of behavior is in itself stimulus that can affect future behavior

211
Q

Operant conditioning

A
212
Q

Behaviorism: Social learning

A

Learning can occur through observing others in addition to direct experience

213
Q

Piaget’s stages of cognitive development: Sensorimotor

A
  • Age: 0-2yo
  • Experience is through movement + senses
  • Cognitive changes: Categorize things, enhance memory
    • Object permanence: If you cannot see it, then it is not there
    • Symbolic play
  • Emotional changes: Fear of strangers (7-12mo) + separation anxiety (6-18mo)
214
Q

Piaget’s stages of cognitive development: Preoperational

A
  • Age: 2-7yo
  • Children use language literally + are egocentric
  • Cognitive changes:
    • Preconceptual (2-4yo): Mental imagery, concentration
    • Intuitive thoughts (4-7yo): Classification, reading + writing, longer attention span
  • Emotional changes:
    • Self control (3-6yo): Conscious
    • Aggression: Inability to demonstrate self control, instrumental aggression to accomplish a goal, hostile aggression to harm another
  • Social: Play, gender identity, toxic stress
215
Q

Piaget’s stages of cognitive development: Concrete operational

A
  • Age: 7-11yo
  • Children can think logically but not abstractly
  • Cognitive changes: Literacy, mental representations of actions (think about what they can do or what they have done)
  • Emotional changes: Accepting societal norms, delayed gratification, body image, peer relationships
  • Social: Positive attitude, self-confidence, peer influences, meaningful friendships
216
Q

Piaget’s stages of cognitive development: Formal operational

A
  • Age: 11+yo
  • Children can think abstractly + are increasingly concerned about the opinion of others
  • Cognitive changes: Abstract thinking, analysis of information, “rebel, complainer, accuser,” idealism leading to disillusionment, introspective + analytic, egocentric, opinionated, argumentative
  • Emotional changes: Attractive vs. unattractive, loved vs. unloved, masculine vs. feminine, sexuality, love
  • Social: Ability to establish + maintain loving relationships, bullying, alcohol, substance abuse, sexual promiscuity, gender
217
Q

Common characteristics of 2yo

A
  • Gross motor skills; just developing self-help skills
  • Very attached to parent
  • Plays alone; rarely shares
  • Limited vocab
218
Q

Common characteristics of 3yo

A
  • Less egocentric
  • Likes to please
  • Active imagination
  • Closely attached to parent
219
Q

Common characteristics of 4yo

A
  • Tries to impose power
  • Develops small social groups
  • Expansive period – reaches out from parent
  • Many independent self-help skills
220
Q

Common characteristics of 5yo

A
  • Deliberate
  • Takes pride in possessions
  • Relinquishes comfort objects
  • Plays cooperatively with peers
221
Q

Erickson’s stages of psychosocial development is based on what?

A

Freud’s psychosexual stages

222
Q

Erickson’s stages of psychosocial development – Eight stages of man

A
  • Basic trust - 0-18mo
  • Autonomy - 18mo-3yo
  • Initiative - 3-6yo
  • Industry - 7-11yo
  • Personal identity - 12-17yo
  • Intimacy, Generativity, Ego integrity - all adult stages
223
Q

Erickson’s eight stages of man: Basic trust

A

Basic Trust: 0-18mo

  • Bonding b/w parent + child
  • “Maternal deprivation” connection to “failure to thrive”
  • “Separation anxiety” – reflection of success in this stage
  • Failure to develop = mistrust
224
Q

Erickson’s eight stages of man: Autonomy

A

Autonomy: 18mo-3yo

  • “Terrible twos”
  • Development of individual identity
  • Allowing choices is a good thing
  • “Consistently enforced limits on behavior at this time allow the child to further develop trust in a predictable environment”
  • Failure to develop = Shame
225
Q

Erickson’s eight stages of man: Initiative

A

Initiative: 3-6yo

  • Increase autonomy, planning + pursuit
  • Extreme curiosity + questioning
  • Aggressive talking
  • Modeling behavior – feeling of accomplishment
  • Failure = Guilt
226
Q

Erickson’s eight stages of man: Industry

A

Industry: 7-11yo

  • Academic + social skills
  • Mastery of skills, competition, cooperation
  • Decrease of parents as role models
  • Increase peers as influences
  • Failure = Inferiority
227
Q

Erickson’s eight stages of man: Personal identity

A

Personal identity: 12-17yo

  • Feelings of belonging
  • Romantic relationships
  • Responsibilities
  • Separation from family + peer group
  • Failure = Role of confusion
228
Q

Erickson’s eight stages of man: Intimacy, Generativity, Ego Integrity

A

All adult stages

229
Q

Most motor skills are before __ of age

A

Most motor skills are before 25mo of age

230
Q

Variables influencing dental behavior

A
  • Parental anxiety: related to child dental anxiety
    • Mother more often than fathers
    • Greatest in the children <4yo
  • Stress: Normal + necessary for survival
    • Toxic stress: stress that continues over a prolonged period + has lifelong effects
      • Child abuse, chronic exposure to drugs/violence in the home, parental depression, mental illness, economic
      • Affects long term outcomes
    • Dandelion vs. orchid child: Some are more sensitive to the envt, some are resilient no matter the situation
  • Medical experiences: Positive visits are correlated w/ positive dental behaviors
    • “Adultified” so many visits that are little adults
    • Pain during previous health care visits
    • Parental attitudes
    • Awareness of dental problem – when a child knows that they have teeth issues + behave negatively
  • General behavior problems
    • Dental fears are correlated w/ general fears
    • Some children have behavior problems only in the dental envt bc of poor experiences
  • Dental fear: 9-20% report dental fear
  • Temperament: Impulsivity + negative emotionality is associated w/ behavior problems
    • Easy, difficult, slow to warm up
231
Q

Parenting styles: Baumrind typology

A
  • Authoritative
  • Authoritarian
  • Permissive/indulgent
  • Neglectful/uninvolved
232
Q

Authoritative parenting style

A
  • High affection, high rules
  • Clear standards, assertive
  • Disciplinary methods are supportive
  • Decisions made by adult authority
  • This style is associated w/ more desirable child behavior
233
Q

Authoritarian parenting style

A
  • High rules, low affection
  • Obedient + status oriented
  • Well-ordered, structured envts w/ clear rules
  • Decisions made by adult authority
234
Q

Neglectful/Uninvolved parenting style

A
  • Low demand, low response
  • May include rejecting-neglecting + neglectful
  • Low parental involvement + low expectations for behavior
235
Q

Permissive/Indulgent parenting style

A
  • Low rules, high affection
  • Decisions made by child
  • Nontraditional lenient
  • Does not require mature behavior
  • Allows considerable self-regulation + avoidance of confrontation
  • Democratic vs. nondirective
236
Q

Behavior shaping

A

State the goal, explain necessity, divide the explanation for the procedure, give explanations at the child’s level of understanding, use successive approximation, reinforce appropriate behavior, disregard minor inappropriate behavior

237
Q

Operant conditioning

A

Response to past behaviors influence future behaviors

238
Q

Positive reinforcement

A

Pleasant stimulus w/ reward

239
Q

Omission or time out

A

Pleasant stimulus withdrawn

240
Q

Negative reinforcement (escape)

A

Unpleasant stimulus withdrawn

241
Q

Punishment

A

Unpleasant stimulus introduced